Does Endoplasmic Reticulum (ER) Stress Contribute to T-cell Exhaustion in B-ALL?

Glucose deprivation in T lymphocytes can trigger compensatory metabolic pathways, potentially contributing to T-cell exhaustion. Additionally, it may induce the unfolded protein response (UPR), ultimately resulting in endoplasmic reticulum (ER) stress. To examine the transcriptional profiles of endo...

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Vydané v:	Iranian journal of immunology Ročník 22; číslo 2; s. 100
Hlavní autori:	Kahrizi, Amir, Akbar, Armin, Najafi, Ahmad, Asgarian-Omran, Hossein, Karami, Hossein, Naderisorki, Mohammad, Karimi, Alireza, Tehrani, Mohsen
Médium:	Journal Article
Jazyk:	English
Vydavateľské údaje:	Iran Shiraz Institute for Cancer Research 01.06.2025
Predmet:	CD8 antigen Cell activation Endoplasmic reticulum Gene expression Lymphocytes Lymphocytes T Metabolic pathways Peripheral blood Protein folding
ISSN:	1735-1383, 1735-367X, 1735-367X
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