Permissive Summer Temperatures of the 2010 European West Nile Fever Upsurge

In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation. Pearson and l...

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Vydáno v:PloS one Ročník 8; číslo 2; s. e56398
Hlavní autoři: Paz, Shlomit, Malkinson, Dan, Green, Manfred S., Tsioni, Gil, Papa, Anna, Danis, Kostas, Sirbu, Anca, Ceianu, Cornelia, Katalin, Krisztalovics, Ferenczi, Emőke, Zeller, Herve, Semenza, Jan C.
Médium: Journal Article
Jazyk:angličtina
Vydáno: United States Public Library of Science 19.02.2013
Public Library of Science (PLoS)
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ISSN:1932-6203, 1932-6203
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Abstract In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation. Pearson and lag correlations, binary and multinomial logistic regressions were used to assess the relationship between the climatic parameters and these outbreaks. For human morbidity, significant (<0.05) positive correlations were observed between a number of WNF cases and temperature, with a geographic latitude gradient: northern ("colder") countries displayed strong correlations with a lag of up to four weeks, in contrast to southern ("warmer") countries, where the response was immediate. The correlations with RH were weaker, while the association with precipitation was not consistent. Horse morbidity started three weeks later than in humans where integrated surveillance was conducted, and no significant associations with temperature or RH were found for lags of 0 to 4 weeks. Significant temperature deviations during summer months might be considered environmental precursors of WNF outbreaks in humans, particularly at more northern latitudes. These insights can guide vector abatement strategies by health practitioners in areas at risk for persistent transmission cycles.
AbstractList In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation. Pearson and lag correlations, binary and multinomial logistic regressions were used to assess the relationship between the climatic parameters and these outbreaks. For human morbidity, significant (<0.05) positive correlations were observed between a number of WNF cases and temperature, with a geographic latitude gradient: northern ("colder") countries displayed strong correlations with a lag of up to four weeks, in contrast to southern ("warmer") countries, where the response was immediate. The correlations with RH were weaker, while the association with precipitation was not consistent. Horse morbidity started three weeks later than in humans where integrated surveillance was conducted, and no significant associations with temperature or RH were found for lags of 0 to 4 weeks. Significant temperature deviations during summer months might be considered environmental precursors of WNF outbreaks in humans, particularly at more northern latitudes. These insights can guide vector abatement strategies by health practitioners in areas at risk for persistent transmission cycles.
In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation. Pearson and lag correlations, binary and multinomial logistic regressions were used to assess the relationship between the climatic parameters and these outbreaks. For human morbidity, significant (<0.05) positive correlations were observed between a number of WNF cases and temperature, with a geographic latitude gradient: northern ("colder") countries displayed strong correlations with a lag of up to four weeks, in contrast to southern ("warmer") countries, where the response was immediate. The correlations with RH were weaker, while the association with precipitation was not consistent. Horse morbidity started three weeks later than in humans where integrated surveillance was conducted, and no significant associations with temperature or RH were found for lags of 0 to 4 weeks. Significant temperature deviations during summer months might be considered environmental precursors of WNF outbreaks in humans, particularly at more northern latitudes. These insights can guide vector abatement strategies by health practitioners in areas at risk for persistent transmission cycles.
Background In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation. Methods Pearson and lag correlations, binary and multinomial logistic regressions were used to assess the relationship between the climatic parameters and these outbreaks. Results For human morbidity, significant (<0.05) positive correlations were observed between a number of WNF cases and temperature, with a geographic latitude gradient: northern ("colder") countries displayed strong correlations with a lag of up to four weeks, in contrast to southern ("warmer") countries, where the response was immediate. The correlations with RH were weaker, while the association with precipitation was not consistent. Horse morbidity started three weeks later than in humans where integrated surveillance was conducted, and no significant associations with temperature or RH were found for lags of 0 to 4 weeks. Conclusions Significant temperature deviations during summer months might be considered environmental precursors of WNF outbreaks in humans, particularly at more northern latitudes. These insights can guide vector abatement strategies by health practitioners in areas at risk for persistent transmission cycles.
Background In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation. Methods Pearson and lag correlations, binary and multinomial logistic regressions were used to assess the relationship between the climatic parameters and these outbreaks. Results For human morbidity, significant (<0.05) positive correlations were observed between a number of WNF cases and temperature, with a geographic latitude gradient: northern (“colder”) countries displayed strong correlations with a lag of up to four weeks, in contrast to southern (“warmer”) countries, where the response was immediate. The correlations with RH were weaker, while the association with precipitation was not consistent. Horse morbidity started three weeks later than in humans where integrated surveillance was conducted, and no significant associations with temperature or RH were found for lags of 0 to 4 weeks. Conclusions Significant temperature deviations during summer months might be considered environmental precursors of WNF outbreaks in humans, particularly at more northern latitudes. These insights can guide vector abatement strategies by health practitioners in areas at risk for persistent transmission cycles.
In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation.BACKGROUNDIn the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to identify potential drivers of these outbreaks, such as spring and summer temperatures, relative humidity (RH), and precipitation.Pearson and lag correlations, binary and multinomial logistic regressions were used to assess the relationship between the climatic parameters and these outbreaks.METHODSPearson and lag correlations, binary and multinomial logistic regressions were used to assess the relationship between the climatic parameters and these outbreaks.For human morbidity, significant (<0.05) positive correlations were observed between a number of WNF cases and temperature, with a geographic latitude gradient: northern ("colder") countries displayed strong correlations with a lag of up to four weeks, in contrast to southern ("warmer") countries, where the response was immediate. The correlations with RH were weaker, while the association with precipitation was not consistent. Horse morbidity started three weeks later than in humans where integrated surveillance was conducted, and no significant associations with temperature or RH were found for lags of 0 to 4 weeks.RESULTSFor human morbidity, significant (<0.05) positive correlations were observed between a number of WNF cases and temperature, with a geographic latitude gradient: northern ("colder") countries displayed strong correlations with a lag of up to four weeks, in contrast to southern ("warmer") countries, where the response was immediate. The correlations with RH were weaker, while the association with precipitation was not consistent. Horse morbidity started three weeks later than in humans where integrated surveillance was conducted, and no significant associations with temperature or RH were found for lags of 0 to 4 weeks.Significant temperature deviations during summer months might be considered environmental precursors of WNF outbreaks in humans, particularly at more northern latitudes. These insights can guide vector abatement strategies by health practitioners in areas at risk for persistent transmission cycles.CONCLUSIONSSignificant temperature deviations during summer months might be considered environmental precursors of WNF outbreaks in humans, particularly at more northern latitudes. These insights can guide vector abatement strategies by health practitioners in areas at risk for persistent transmission cycles.
Audience Academic
Author Semenza, Jan C.
Paz, Shlomit
Tsioni, Gil
Malkinson, Dan
Ceianu, Cornelia
Green, Manfred S.
Ferenczi, Emőke
Sirbu, Anca
Papa, Anna
Danis, Kostas
Zeller, Herve
Katalin, Krisztalovics
AuthorAffiliation 8 European Center for Disease Prevention and Control (ECDC), Stockholm, Sweden
4 Hellenic Centre of Disease Prevention & Control, Athens, Greece
1 Department of Geography and Environmental Studies, University of Haifa, Haifa, Israel
3 Department of Microbiology, Medical School, Aristotle University of Thessaloniki, Thessaloniki, Greece
6 Cantacuzino National Institute for Research and Development in Microbiology and Immunology, Bucharest, Romania
7 National Center for Epidemiology, Budapest, Hungary
Louisiana State University, United States of America
2 School of Public Health, University of Haifa, Haifa, Israel
5 National Center for Surveillance and Control of Communicable Diseases, Bucharest, Romania
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BackLink https://www.ncbi.nlm.nih.gov/pubmed/23431374$$D View this record in MEDLINE/PubMed
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ContentType Journal Article
Copyright COPYRIGHT 2013 Public Library of Science
2013 Paz et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.
2013 Paz et al 2013 Paz et al
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– notice: 2013 Paz et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License: https://creativecommons.org/licenses/by/4.0/ (the “License”), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.
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Competing Interests: The authors have declared that no competing interests exist.
Conceived and designed the experiments: SP JCS. Performed the experiments: SP DM MSG GT. Analyzed the data: SP DM MSG GT JCS. Contributed reagents/materials/analysis tools: AP KD AS CC KK EF HZ. Wrote the paper: SP JCS.
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Snippet In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this study was to...
Background In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this...
BACKGROUND: In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this...
Background In the summer of 2010, Europe experienced outbreaks of West Nile Fever (WNF) in humans, which was preceded by hot spells. The objective of this...
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StartPage e56398
SubjectTerms Animal Migration
Animals
Biology
Climate change
Correlation
Culex
Culex pipiens
Culicidae
Disease
Disease Outbreaks
Disease prevention
Disease transmission
Encephalitis
Environmental studies
Epidemics
Epidemiology
Europe - epidemiology
Fever
Horse Diseases - epidemiology
Horse Diseases - virology
Horses
Hot spells
Humans
Humidity
Incidence
Infections
Latitude
Medicine
Meningitis
Morbidity
Mosquitoes
open climate campaign
Outbreaks
Precipitation
Precipitation (Meteorology)
Rainfall
Relative humidity
Seasons
Summer
Summer temperatures
Surveillance
Temperature
Temperature effects
Vector-borne diseases
Vectors (Biology)
West Nile fever
West Nile Fever - epidemiology
West Nile Fever - veterinary
West Nile virus
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Title Permissive Summer Temperatures of the 2010 European West Nile Fever Upsurge
URI https://www.ncbi.nlm.nih.gov/pubmed/23431374
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https://doaj.org/article/fea67b0a385c458c8654f64fb9c7629a
http://dx.doi.org/10.1371/journal.pone.0056398
Volume 8
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