Neuropeptide Y directly reduced apoptosis of granulosa cells, and the expression of NPY and its receptors in PCOS subjects

Background Most women with anovulatory infertility show polycystic ovarian syndrome (PCOS), and androgen excess is known as a key factor involved in pathogenicity of PCOS. However, the mechanism of follicular developmental arrest in PCOS is not completely understood. The reproductive function of Neu...

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Bibliographic Details
Published in:	Journal of ovarian research Vol. 16; no. 1; pp. 1 - 10
Main Authors:	Urata, Yoko, Salehi, Reza, Wyse, Brandon A., Jahangiri, Sahar, Librach, Clifford L., Tzeng, Chii-Ruey, Osuga, Yutaka, Tsang, Benjamin
Format:	Journal Article
Language:	English
Published:	London BioMed Central 31.08.2023 BioMed Central Ltd Springer Nature B.V BMC
Subjects:	Analysis Androgens Apoptosis Body fat Cell culture Cell growth Cell proliferation Dihydrotestosterone Enzyme-linked immunosorbent assay Follicular fluid Folliculogenesis Granulosa cells Gynecology Hyperandrogenism Infertility Medicine Medicine & Public Health Metabolism mRNA Neuropeptide Neuropeptide Y Neuropeptides Ovaries Pathogenesis Pathogenicity Phenotypes Polycystic ovarian syndrome Polycystic ovary syndrome Protein folding Proteins Receptor mechanisms Reproductive Medicine RNA Stein-Leventhal syndrome Neuropeptide Y Polycystic ovarian syndrome Hyperandrogenism Neuropeptide Apoptosis
ISSN:	1757-2215, 1757-2215
Online Access:	Get full text
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