miR-126 Regulates Distinct Self-Renewal Outcomes in Normal and Malignant Hematopoietic Stem Cells

To investigate miRNA function in human acute myeloid leukemia (AML) stem cells (LSC), we generated a prognostic LSC-associated miRNA signature derived from functionally validated subpopulations of AML samples. For one signature miRNA, miR-126, high bioactivity aggregated all in vivo patient sample L...

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Vydané v:Cancer cell Ročník 29; číslo 2; s. 214
Hlavní autori: Lechman, Eric R, Gentner, Bernhard, Ng, Stanley W K, Schoof, Erwin M, van Galen, Peter, Kennedy, James A, Nucera, Silvia, Ciceri, Fabio, Kaufmann, Kerstin B, Takayama, Naoya, Dobson, Stephanie M, Trotman-Grant, Aaron, Krivdova, Gabriela, Elzinga, Janneke, Mitchell, Amanda, Nilsson, Björn, Hermans, Karin G, Eppert, Kolja, Marke, Rene, Isserlin, Ruth, Voisin, Veronique, Bader, Gary D, Zandstra, Peter W, Golub, Todd R, Ebert, Benjamin L, Lu, Jun, Minden, Mark, Wang, Jean C Y, Naldini, Luigi, Dick, John E
Médium: Journal Article
Jazyk:English
Vydavateľské údaje: United States 08.02.2016
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ISSN:1878-3686, 1878-3686
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Abstract To investigate miRNA function in human acute myeloid leukemia (AML) stem cells (LSC), we generated a prognostic LSC-associated miRNA signature derived from functionally validated subpopulations of AML samples. For one signature miRNA, miR-126, high bioactivity aggregated all in vivo patient sample LSC activity into a single sorted population, tightly coupling miR-126 expression to LSC function. Through functional studies, miR-126 was found to restrain cell cycle progression, prevent differentiation, and increase self-renewal of primary LSC in vivo. Compared with prior results showing miR-126 regulation of normal hematopoietic stem cell (HSC) cycling, these functional stem effects are opposite between LSC and HSC. Combined transcriptome and proteome analysis demonstrates that miR-126 targets the PI3K/AKT/MTOR signaling pathway, preserving LSC quiescence and promoting chemotherapy resistance.
AbstractList To investigate miRNA function in human acute myeloid leukemia (AML) stem cells (LSC), we generated a prognostic LSC-associated miRNA signature derived from functionally validated subpopulations of AML samples. For one signature miRNA, miR-126, high bioactivity aggregated all in vivo patient sample LSC activity into a single sorted population, tightly coupling miR-126 expression to LSC function. Through functional studies, miR-126 was found to restrain cell cycle progression, prevent differentiation, and increase self-renewal of primary LSC in vivo. Compared with prior results showing miR-126 regulation of normal hematopoietic stem cell (HSC) cycling, these functional stem effects are opposite between LSC and HSC. Combined transcriptome and proteome analysis demonstrates that miR-126 targets the PI3K/AKT/MTOR signaling pathway, preserving LSC quiescence and promoting chemotherapy resistance.To investigate miRNA function in human acute myeloid leukemia (AML) stem cells (LSC), we generated a prognostic LSC-associated miRNA signature derived from functionally validated subpopulations of AML samples. For one signature miRNA, miR-126, high bioactivity aggregated all in vivo patient sample LSC activity into a single sorted population, tightly coupling miR-126 expression to LSC function. Through functional studies, miR-126 was found to restrain cell cycle progression, prevent differentiation, and increase self-renewal of primary LSC in vivo. Compared with prior results showing miR-126 regulation of normal hematopoietic stem cell (HSC) cycling, these functional stem effects are opposite between LSC and HSC. Combined transcriptome and proteome analysis demonstrates that miR-126 targets the PI3K/AKT/MTOR signaling pathway, preserving LSC quiescence and promoting chemotherapy resistance.
To investigate miRNA function in human acute myeloid leukemia (AML) stem cells (LSC), we generated a prognostic LSC-associated miRNA signature derived from functionally validated subpopulations of AML samples. For one signature miRNA, miR-126, high bioactivity aggregated all in vivo patient sample LSC activity into a single sorted population, tightly coupling miR-126 expression to LSC function. Through functional studies, miR-126 was found to restrain cell cycle progression, prevent differentiation, and increase self-renewal of primary LSC in vivo. Compared with prior results showing miR-126 regulation of normal hematopoietic stem cell (HSC) cycling, these functional stem effects are opposite between LSC and HSC. Combined transcriptome and proteome analysis demonstrates that miR-126 targets the PI3K/AKT/MTOR signaling pathway, preserving LSC quiescence and promoting chemotherapy resistance.
Author Naldini, Luigi
Ciceri, Fabio
Ng, Stanley W K
Schoof, Erwin M
Nilsson, Björn
Golub, Todd R
Mitchell, Amanda
Takayama, Naoya
Gentner, Bernhard
Hermans, Karin G
Nucera, Silvia
Lu, Jun
Krivdova, Gabriela
Dick, John E
Marke, Rene
van Galen, Peter
Wang, Jean C Y
Trotman-Grant, Aaron
Bader, Gary D
Kennedy, James A
Lechman, Eric R
Kaufmann, Kerstin B
Ebert, Benjamin L
Zandstra, Peter W
Elzinga, Janneke
Minden, Mark
Dobson, Stephanie M
Isserlin, Ruth
Eppert, Kolja
Voisin, Veronique
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  givenname: Eric R
  surname: Lechman
  fullname: Lechman, Eric R
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 2
  givenname: Bernhard
  surname: Gentner
  fullname: Gentner, Bernhard
  organization: San Raffaele Telethon Institute for Gene Therapy, San Raffaele Hospital, Milan 20132, Italy; Vita Salute San Raffaele University, San Raffaele Scientific Institute, San Raffaele Hospital, Milan 20132, Italy; Hematology and Bone Marrow Transplantation Unit, San Raffaele Hospital, Milan 20132, Italy
– sequence: 3
  givenname: Stanley W K
  surname: Ng
  fullname: Ng, Stanley W K
  organization: Department of Chemical Engineering and Applied Chemistry, Institute of Biomaterials and Biomedical Engineering, University of Toronto, Toronto, ON M5G 2M9, Canada; The Donnelly Centre, University of Toronto, Toronto, ON M5S 3E1, Canada
– sequence: 4
  givenname: Erwin M
  surname: Schoof
  fullname: Schoof, Erwin M
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 5
  givenname: Peter
  surname: van Galen
  fullname: van Galen, Peter
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 6
  givenname: James A
  surname: Kennedy
  fullname: Kennedy, James A
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Medicine, University of Toronto, Toronto, ON M5G 2M9, Canada
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  givenname: Fabio
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  organization: Vita Salute San Raffaele University, San Raffaele Scientific Institute, San Raffaele Hospital, Milan 20132, Italy; Hematology and Bone Marrow Transplantation Unit, San Raffaele Hospital, Milan 20132, Italy
– sequence: 9
  givenname: Kerstin B
  surname: Kaufmann
  fullname: Kaufmann, Kerstin B
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 10
  givenname: Naoya
  surname: Takayama
  fullname: Takayama, Naoya
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 11
  givenname: Stephanie M
  surname: Dobson
  fullname: Dobson, Stephanie M
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 12
  givenname: Aaron
  surname: Trotman-Grant
  fullname: Trotman-Grant, Aaron
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
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  givenname: Gabriela
  surname: Krivdova
  fullname: Krivdova, Gabriela
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 14
  givenname: Janneke
  surname: Elzinga
  fullname: Elzinga, Janneke
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 15
  givenname: Amanda
  surname: Mitchell
  fullname: Mitchell, Amanda
  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
– sequence: 16
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  givenname: Karin G
  surname: Hermans
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  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada
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  organization: Princess Margaret Cancer Centre, University Health Network, University of Toronto, Toronto, ON M5G 1L7, Canada; Department of Molecular Genetics, University of Toronto, Toronto, ON M5G 1L7, Canada; Princess Margaret Cancer Research Tower, Room 8-301, 101 College Street, Toronto M5G 1L7, Canada. Electronic address: jdick@uhnresearch.ca
BackLink https://www.ncbi.nlm.nih.gov/pubmed/26832662$$D View this record in MEDLINE/PubMed
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ContentType Journal Article
Copyright Copyright © 2016 The Authors. Published by Elsevier Inc. All rights reserved.
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Snippet To investigate miRNA function in human acute myeloid leukemia (AML) stem cells (LSC), we generated a prognostic LSC-associated miRNA signature derived from...
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SubjectTerms Animals
Antineoplastic Agents - pharmacology
Cell Line, Tumor
Gene Knockdown Techniques
Hematopoietic Stem Cells - pathology
Heterografts
Humans
Leukemia, Myeloid, Acute - genetics
Leukemia, Myeloid, Acute - pathology
Mice
Mice, SCID
MicroRNAs - genetics
MicroRNAs - physiology
Phosphatidylinositol 3-Kinases - metabolism
Prognosis
Proto-Oncogene Proteins c-akt - metabolism
TOR Serine-Threonine Kinases - metabolism
Title miR-126 Regulates Distinct Self-Renewal Outcomes in Normal and Malignant Hematopoietic Stem Cells
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