Inflammation as the Potential Basis in Depression

There is growing evidence of the association between inflammation and stress-related disorders including depression. The positive correlation between the increased levels of inflammatory cytokines observed in patients with other diseases and the byproduct of the depressive symptoms may be caused by...

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Bibliographic Details
Published in:International neurourology journal Vol. 23; no. Suppl 2; pp. S63 - 71
Main Authors: Maeng, Sung Ho, Hong, Heeok
Format: Journal Article
Language:English
Published: Korea (South) Korean Continence Society 01.11.2019
대한배뇨장애요실금학회
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ISSN:2093-6931, 2093-4777, 2093-6931
Online Access:Get full text
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Summary:There is growing evidence of the association between inflammation and stress-related disorders including depression. The positive correlation between the increased levels of inflammatory cytokines observed in patients with other diseases and the byproduct of the depressive symptoms may be caused by chronic stress. Increased neuroinflammatory responses are capable of activating microglia and astrocytes, which leads to release pro-inflammatory cytokines. Moreover, elevated levels of inflammatory cytokines such as tumor necrosis factor-alpha, interleukin (IL)-1, and IL-6 are causally related to various aspects of depression such as the behavioral symptomatology. Eventually, these elevated cytokines aggravate and propagate neuroinflammation, impairing brain functions. Thus, activated astrocytes and microglia may be potential mediators in neuroinflammatory processes contributing to the development of depression.
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https://doi.org/10.5213/inj.1938226.113
ISSN:2093-6931
2093-4777
2093-6931
DOI:10.5213/inj.1938226.113