Body size and risk of MS in two cohorts of US women
To examine whether obesity during childhood, adolescence, or adulthood is associated with an increased risk of multiple sclerosis (MS). Women in the Nurses' Health Study (n = 121,700) and Nurses' Health Study II (n = 116,671) provided information on weight at age 18 and weight and height a...
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| Vydáno v: | Neurology Ročník 73; číslo 19; s. 1543 |
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| Hlavní autoři: | , , |
| Médium: | Journal Article |
| Jazyk: | angličtina |
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United States
10.11.2009
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| ISSN: | 1526-632X, 1526-632X |
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| Abstract | To examine whether obesity during childhood, adolescence, or adulthood is associated with an increased risk of multiple sclerosis (MS).
Women in the Nurses' Health Study (n = 121,700) and Nurses' Health Study II (n = 116,671) provided information on weight at age 18 and weight and height at baseline, from which body mass index was derived. Women also selected silhouettes representing their body size at ages 5, 10, and 20. Over the total 40 years of follow-up in both cohorts combined, we confirmed 593 cases of MS. Cox proportional hazards models, adjusting for age, latitude of residence, ethnicity, and cigarette smoking, were used to estimate the rate ratios and 95% confidence intervals (CI).
Obesity at age 18 (body mass index > or =30 kg/m(2)) was associated with a greater than twofold increased risk of MS (multivariate relative risk(pooled) = 2.25, 95% CI: 1.50-3.37, p trend <0.001). After adjusting for body size at age 20, having a large body size at ages 5 or 10 was not associated with risk of MS, whereas a large body size at age 20 was associated with a 96% increased risk of MS (95% CI: 1.33-2.89, p trend = 0.009). No significant association was found between adult body mass and MS risk.
Obese adolescents have an increased risk of developing multiple sclerosis (MS). Although the mechanisms of this association remain uncertain, this result suggests that prevention of adolescent obesity may contribute to reduced MS risk. |
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| AbstractList | To examine whether obesity during childhood, adolescence, or adulthood is associated with an increased risk of multiple sclerosis (MS).OBJECTIVETo examine whether obesity during childhood, adolescence, or adulthood is associated with an increased risk of multiple sclerosis (MS).Women in the Nurses' Health Study (n = 121,700) and Nurses' Health Study II (n = 116,671) provided information on weight at age 18 and weight and height at baseline, from which body mass index was derived. Women also selected silhouettes representing their body size at ages 5, 10, and 20. Over the total 40 years of follow-up in both cohorts combined, we confirmed 593 cases of MS. Cox proportional hazards models, adjusting for age, latitude of residence, ethnicity, and cigarette smoking, were used to estimate the rate ratios and 95% confidence intervals (CI).METHODSWomen in the Nurses' Health Study (n = 121,700) and Nurses' Health Study II (n = 116,671) provided information on weight at age 18 and weight and height at baseline, from which body mass index was derived. Women also selected silhouettes representing their body size at ages 5, 10, and 20. Over the total 40 years of follow-up in both cohorts combined, we confirmed 593 cases of MS. Cox proportional hazards models, adjusting for age, latitude of residence, ethnicity, and cigarette smoking, were used to estimate the rate ratios and 95% confidence intervals (CI).Obesity at age 18 (body mass index > or =30 kg/m(2)) was associated with a greater than twofold increased risk of MS (multivariate relative risk(pooled) = 2.25, 95% CI: 1.50-3.37, p trend <0.001). After adjusting for body size at age 20, having a large body size at ages 5 or 10 was not associated with risk of MS, whereas a large body size at age 20 was associated with a 96% increased risk of MS (95% CI: 1.33-2.89, p trend = 0.009). No significant association was found between adult body mass and MS risk.RESULTSObesity at age 18 (body mass index > or =30 kg/m(2)) was associated with a greater than twofold increased risk of MS (multivariate relative risk(pooled) = 2.25, 95% CI: 1.50-3.37, p trend <0.001). After adjusting for body size at age 20, having a large body size at ages 5 or 10 was not associated with risk of MS, whereas a large body size at age 20 was associated with a 96% increased risk of MS (95% CI: 1.33-2.89, p trend = 0.009). No significant association was found between adult body mass and MS risk.Obese adolescents have an increased risk of developing multiple sclerosis (MS). Although the mechanisms of this association remain uncertain, this result suggests that prevention of adolescent obesity may contribute to reduced MS risk.CONCLUSIONSObese adolescents have an increased risk of developing multiple sclerosis (MS). Although the mechanisms of this association remain uncertain, this result suggests that prevention of adolescent obesity may contribute to reduced MS risk. To examine whether obesity during childhood, adolescence, or adulthood is associated with an increased risk of multiple sclerosis (MS). Women in the Nurses' Health Study (n = 121,700) and Nurses' Health Study II (n = 116,671) provided information on weight at age 18 and weight and height at baseline, from which body mass index was derived. Women also selected silhouettes representing their body size at ages 5, 10, and 20. Over the total 40 years of follow-up in both cohorts combined, we confirmed 593 cases of MS. Cox proportional hazards models, adjusting for age, latitude of residence, ethnicity, and cigarette smoking, were used to estimate the rate ratios and 95% confidence intervals (CI). Obesity at age 18 (body mass index > or =30 kg/m(2)) was associated with a greater than twofold increased risk of MS (multivariate relative risk(pooled) = 2.25, 95% CI: 1.50-3.37, p trend <0.001). After adjusting for body size at age 20, having a large body size at ages 5 or 10 was not associated with risk of MS, whereas a large body size at age 20 was associated with a 96% increased risk of MS (95% CI: 1.33-2.89, p trend = 0.009). No significant association was found between adult body mass and MS risk. Obese adolescents have an increased risk of developing multiple sclerosis (MS). Although the mechanisms of this association remain uncertain, this result suggests that prevention of adolescent obesity may contribute to reduced MS risk. |
| Author | Chitnis, Tanuja Ascherio, Alberto Munger, Kassandra L |
| Author_xml | – sequence: 1 givenname: Kassandra L surname: Munger fullname: Munger, Kassandra L organization: Department of Nutrition, Harvard School of Public Health, Boston, MA 02115, USA – sequence: 2 givenname: Tanuja surname: Chitnis fullname: Chitnis, Tanuja – sequence: 3 givenname: Alberto surname: Ascherio fullname: Ascherio, Alberto |
| BackLink | https://www.ncbi.nlm.nih.gov/pubmed/19901245$$D View this record in MEDLINE/PubMed |
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| References_xml | – reference: 7304477 - Am J Clin Nutr. 1981 Nov;34(11):2359-63 – reference: 9839742 - Int J Epidemiol. 1998 Oct;27(5):845-52 – reference: 10563617 - Neurology. 1999 Nov 10;53(8):1711-8 – reference: 16148162 - J Immunol. 2005 Sep 15;175(6):4119-26 – reference: 17444504 - Ann Neurol. 2007 Apr;61(4):288-99 – reference: 12532024 - Science. 2003 Feb 14;299(5609):1033-6 – reference: 7584987 - Nat Med. 1995 Nov;1(11):1155-61 – reference: 15067033 - J Exp Med. 2004 Apr 5;199(7):971-9 – reference: 16388368 - Neurol Sci. 2005 Dec;26(5):334-9 – reference: 17656603 - Eur J Endocrinol. 2007 Aug;157(2):225-32 – reference: 10966885 - Am J Clin Nutr. 2000 Sep;72(3):690-3 – reference: 12519845 - J Clin Endocrinol Metab. 2003 Jan;88(1):157-61 – reference: 8333427 - Am J Epidemiol. 1993 Jul 1;138(1):56-64 – reference: 9398739 - J Clin Endocrinol Metab. 1997 Dec;82(12):4196-200 – reference: 2991340 - J Clin Invest. 1985 Jul;76(1):370-3 – reference: 6983505 - Int J Obes. 1982;6(5):473-9 – reference: 3802833 - Control Clin Trials. 1986 Sep;7(3):177-88 – reference: 18272866 - Neurology. 2008 Mar 25;70(13 Pt 2):1113-8 – reference: 15788534 - Proc Natl Acad Sci U S A. 2005 Apr 5;102(14):5150-5 – reference: 17329717 - Am J Epidemiol. 2007 May 15;165(10):1097-109 – reference: 8966212 - Prog Neurobiol. 1995 Nov-Dec;47(4-5):425-48 – reference: 9236259 - Calcif Tissue Int. 1997 Aug;61(2):129-33 – reference: 1705564 - J Clin Invest. 1991 Mar;87(3):1103-7 – reference: 6823524 - Res Publ Assoc Res Nerv Ment Dis. 1983;60:115-20 – reference: 17179460 - JAMA. 2006 Dec 20;296(23):2832-8 – reference: 17307705 - Immunity. 2007 Feb;26(2):241-55 – reference: 3145124 - Calcif Tissue Int. 1988 Oct;43(4):199-201 – reference: 8755567 - Proc Natl Acad Sci U S A. 1996 Jul 23;93(15):7861-4 – reference: 12887108 - Cell Mol Biol (Noisy-le-grand). 2003 Mar;49(2):277-300 – reference: 11234459 - World Health Organ Tech Rep Ser. 2000;894:i-xii, 1-253 – reference: 11525796 - J Neuroimmunol. 2001 Sep 3;119(1):16-29 – reference: 17492755 - Ann Neurol. 2007 Jun;61(6):504-13 – reference: 7489028 - Int J Obes Relat Metab Disord. 1995 Aug;19(8):570-2 – reference: 18191047 - Metabolism. 2008 Feb;57(2):183-91 |
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| Snippet | To examine whether obesity during childhood, adolescence, or adulthood is associated with an increased risk of multiple sclerosis (MS).
Women in the Nurses'... To examine whether obesity during childhood, adolescence, or adulthood is associated with an increased risk of multiple sclerosis (MS).OBJECTIVETo examine... |
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| SubjectTerms | Adult Body Mass Index Body Size - physiology Cohort Studies Female Follow-Up Studies Humans Longitudinal Studies Male Middle Aged Multiple Sclerosis - epidemiology Multiple Sclerosis - etiology Multiple Sclerosis - physiopathology Obesity - complications Obesity - epidemiology Obesity - physiopathology Risk Factors United States - epidemiology |
| Title | Body size and risk of MS in two cohorts of US women |
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