Basal ganglia, thalamus and neocortical atrophy predicting slowed cognitive processing in multiple sclerosis

Information-processing speed (IPS) slowing is a primary cognitive deficit in multiple sclerosis (MS). Basal ganglia, thalamus and neocortex are thought to have a key role for efficient information-processing, yet the specific relative contribution of these structures for MS-related IPS impairment is...

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Bibliographic Details
Published in:	Journal of neurology Vol. 259; no. 1; pp. 139 - 146
Main Authors:	Batista, Sonia, Zivadinov, Robert, Hoogs, Marietta, Bergsland, Niels, Heininen-Brown, Mari, Dwyer, Michael G., Weinstock-Guttman, Bianca, Benedict, Ralph H. B.
Format:	Journal Article
Language:	English
Published:	Berlin/Heidelberg Springer-Verlag 01.01.2012 Springer Springer Nature B.V
Subjects:	Adult Atrophy Basal ganglia Basal Ganglia - pathology Biological and medical sciences Brain Cognition Disorders - etiology Cognition Disorders - psychology Cognitive ability Computer programs Cortex Data Interpretation, Statistical Female Globus pallidus Humans Image Processing, Computer-Assisted Information processing Magnetic Resonance Imaging Male Measuring techniques Medical sciences Medicine Medicine & Public Health Middle Aged Multiple sclerosis Multiple Sclerosis - complications Multiple Sclerosis - pathology Multiple Sclerosis - psychology Multiple sclerosis and variants. Guillain barré syndrome and other inflammatory polyneuropathies. Leukoencephalitis Neocortex - pathology Neurology Neuropsychological Tests Neuropsychology Neuroradiology Neurosciences Nucleus accumbens Original Communication Predictive Value of Tests Psychology Putamen Sensory integration software Substantia grisea Thalamus Thalamus - pathology Volumetric analysis Thalamus Multiple sclerosis Basal ganglia Magnetic resonance imaging Neocortex Cognitive dysfunction Nervous system diseases Cognitive disorder Central nervous system Basal ganglion Nuclear magnetic resonance imaging Encephalon Inflammatory disease Atrophy Central nervous system disease
ISSN:	0340-5354, 1432-1459, 1432-1459
Online Access:	Get full text
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