The volatile anesthetic isoflurane differentially inhibits voltage-gated sodium channel currents between pyramidal and parvalbumin neurons in the prefrontal cortex

How volatile anesthetics work remains poorly understood. Modulations of synaptic neurotransmission are the direct cellular mechanisms of volatile anesthetics in the central nervous system. Volatile anesthetics such as isoflurane may reduce neuronal interaction by differentially inhibiting neurotrans...

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Vydané v:	Frontiers in neural circuits Ročník 17; s. 1185095
Hlavní autori:	Qiu, Jingxuan, Yang, Yaoxin, Liu, Jin, Zhao, Wenling, Li, Qian, Zhu, Tao, Liang, Peng, Zhou, Cheng
Médium:	Journal Article
Jazyk:	English
Vydavateľské údaje:	Switzerland Frontiers Research Foundation 16.06.2023 Frontiers Media S.A
Predmet:	Anesthetics Anesthetics, Inhalation - pharmacology Animals Brain Brain slice preparation Central nervous system Cerebrospinal fluid Channel gating cortex Electrophysiological recording Exocytosis Glutamatergic transmission Isoflurane Isoflurane - pharmacology Laboratories Mice Neurons Neuroscience Neurotransmission Parvalbumin parvalbumin neurons Parvalbumins Prefrontal Cortex Pyramidal cells Pyramidal Cells - physiology pyramidal neurons Simulation Sodium channels (voltage-gated) Software Unconsciousness voltage-gated sodium channel (Nav) Voltage-Gated Sodium Channels γ-Aminobutyric acid United States > US China cortex pyramidal neurons voltage-gated sodium channel (Nav) isoflurane parvalbumin neurons
ISSN:	1662-5110, 1662-5110
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