Vagus nerve stimulation inhibits activation of coagulation and fibrinolysis during endotoxemia in rats

Background: Sepsis and endotoxemia are associated with concurrent activation of inflammation and the hemostatic mechanism, which both contribute to organ dysfunction and death. Electrical vagus nerve stimulation (VNS) has been found to inhibit tumor necrosis factor (TNF)‐α release during endotoxemia...

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Veröffentlicht in:	Journal of thrombosis and haemostasis Jg. 4; H. 9; S. 1997 - 2002
Hauptverfasser:	VAN WESTERLOO, D. J., GIEBELEN, I. A. J., MEIJERS, J. C. M., DAALHUISEN, J., DE VOS, A. F., LEVI, M., VAN DER POLL, T.
Format:	Journal Article
Sprache:	Englisch
Veröffentlicht:	Oxford, UK Blackwell Publishing Ltd 01.09.2006
Schlagworte:	Animals Blood Coagulation coagulation cytokines Electric Stimulation Electric Stimulation Therapy Endotoxemia - pathology Endotoxemia - therapy endotoxin Fibrinolysis Inflammation - prevention & control Interleukin-10 - analysis Interleukin-6 - analysis Kinetics lipopolysaccharide Lipopolysaccharides - administration & dosage Male Rats Rats, Inbred Lew Tumor Necrosis Factor-alpha - analysis Vagus Nerve
ISSN:	1538-7933, 1538-7836, 1538-7836
Online-Zugang:	Volltext
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