No bidirectional relationship between depression and periodontitis: A genetic correlation and Mendelian randomization study

Observational and research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and examined directionality of causation. The study used summary statistics from published genome wide association studies, with sample sizes ranging from 45,5...

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Published in:Frontiers in immunology Vol. 13; p. 918404
Main Authors: Nolde, Michael, Holtfreter, Birte, Kocher, Thomas, Alayash, Zoheir, Reckelkamm, Stefan Lars, Ehmke, Benjamin, Baurecht, Hansjörg, Baumeister, Sebastian-Edgar
Format: Journal Article
Language:English
Published: Switzerland Frontiers Media S.A 22.07.2022
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ISSN:1664-3224, 1664-3224
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Abstract Observational and research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and examined directionality of causation. The study used summary statistics from published genome wide association studies, with sample sizes ranging from 45,563 to 797,563 individuals of European ancestry. We performed linkage disequilibrium score regression (LDSC) to estimate global correlation and used Heritability Estimation from Summary Statistics (ρ-HESS) to further examine local genetic correlation. Latent Heritable Confounder Mendelian randomization (LHC-MR), Causal Analysis using Summary Effect estimates (CAUSE), and conventional MR approaches assessed bidirectional causation. LDSC observed only weak genetic correlation (r = 0.06, P-Value = 0.619) between depression and periodontitis. Analysis of local genetic correlation using ρ-HESS did not reveal loci of significant local genetic covariance. LHC-MR, CAUSE and conventional MR models provided no support for bidirectional causation between depression and periodontitis, with odds ratios ranging from 1.00 to 1.06 in either direction. Results do not support shared heritability or a causal connection between depression and periodontitis.
AbstractList Observational and in-vivo research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and examined directionality of causation.BackgroundObservational and in-vivo research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and examined directionality of causation.The study used summary statistics from published genome wide association studies, with sample sizes ranging from 45,563 to 797,563 individuals of European ancestry. We performed linkage disequilibrium score regression (LDSC) to estimate global correlation and used Heritability Estimation from Summary Statistics (ρ-HESS) to further examine local genetic correlation. Latent Heritable Confounder Mendelian randomization (LHC-MR), Causal Analysis using Summary Effect estimates (CAUSE), and conventional MR approaches assessed bidirectional causation.MethodsThe study used summary statistics from published genome wide association studies, with sample sizes ranging from 45,563 to 797,563 individuals of European ancestry. We performed linkage disequilibrium score regression (LDSC) to estimate global correlation and used Heritability Estimation from Summary Statistics (ρ-HESS) to further examine local genetic correlation. Latent Heritable Confounder Mendelian randomization (LHC-MR), Causal Analysis using Summary Effect estimates (CAUSE), and conventional MR approaches assessed bidirectional causation.LDSC observed only weak genetic correlation (rg = 0.06, P-Value = 0.619) between depression and periodontitis. Analysis of local genetic correlation using ρ-HESS did not reveal loci of significant local genetic covariance. LHC-MR, CAUSE and conventional MR models provided no support for bidirectional causation between depression and periodontitis, with odds ratios ranging from 1.00 to 1.06 in either direction.ResultsLDSC observed only weak genetic correlation (rg = 0.06, P-Value = 0.619) between depression and periodontitis. Analysis of local genetic correlation using ρ-HESS did not reveal loci of significant local genetic covariance. LHC-MR, CAUSE and conventional MR models provided no support for bidirectional causation between depression and periodontitis, with odds ratios ranging from 1.00 to 1.06 in either direction.Results do not support shared heritability or a causal connection between depression and periodontitis.ConclusionsResults do not support shared heritability or a causal connection between depression and periodontitis.
BackgroundObservational and in-vivo research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and examined directionality of causation.MethodsThe study used summary statistics from published genome wide association studies, with sample sizes ranging from 45,563 to 797,563 individuals of European ancestry. We performed linkage disequilibrium score regression (LDSC) to estimate global correlation and used Heritability Estimation from Summary Statistics (ρ-HESS) to further examine local genetic correlation. Latent Heritable Confounder Mendelian randomization (LHC-MR), Causal Analysis using Summary Effect estimates (CAUSE), and conventional MR approaches assessed bidirectional causation.ResultsLDSC observed only weak genetic correlation (rg = 0.06, P-Value = 0.619) between depression and periodontitis. Analysis of local genetic correlation using ρ-HESS did not reveal loci of significant local genetic covariance. LHC-MR, CAUSE and conventional MR models provided no support for bidirectional causation between depression and periodontitis, with odds ratios ranging from 1.00 to 1.06 in either direction.ConclusionsResults do not support shared heritability or a causal connection between depression and periodontitis.
Observational and research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and examined directionality of causation. The study used summary statistics from published genome wide association studies, with sample sizes ranging from 45,563 to 797,563 individuals of European ancestry. We performed linkage disequilibrium score regression (LDSC) to estimate global correlation and used Heritability Estimation from Summary Statistics (ρ-HESS) to further examine local genetic correlation. Latent Heritable Confounder Mendelian randomization (LHC-MR), Causal Analysis using Summary Effect estimates (CAUSE), and conventional MR approaches assessed bidirectional causation. LDSC observed only weak genetic correlation (r = 0.06, P-Value = 0.619) between depression and periodontitis. Analysis of local genetic correlation using ρ-HESS did not reveal loci of significant local genetic covariance. LHC-MR, CAUSE and conventional MR models provided no support for bidirectional causation between depression and periodontitis, with odds ratios ranging from 1.00 to 1.06 in either direction. Results do not support shared heritability or a causal connection between depression and periodontitis.
Author Alayash, Zoheir
Ehmke, Benjamin
Baurecht, Hansjörg
Kocher, Thomas
Baumeister, Sebastian-Edgar
Reckelkamm, Stefan Lars
Nolde, Michael
Holtfreter, Birte
AuthorAffiliation 3 Clinic for Periodontology and Conservative Dentistry, University of Münster , Münster , Germany
1 Institute of Health Services Research in Dentistry, University of Münster , Münster , Germany
4 Department of Epidemiology and Preventive Medicine, University of Regensburg , Regensburg , Germany
2 Department of Restorative Dentistry, Periodontology, Endodontology, and Preventive and Pediatric Dentistry, University Medicine Greifswald , Greifswald , Germany
AuthorAffiliation_xml – name: 2 Department of Restorative Dentistry, Periodontology, Endodontology, and Preventive and Pediatric Dentistry, University Medicine Greifswald , Greifswald , Germany
– name: 1 Institute of Health Services Research in Dentistry, University of Münster , Münster , Germany
– name: 4 Department of Epidemiology and Preventive Medicine, University of Regensburg , Regensburg , Germany
– name: 3 Clinic for Periodontology and Conservative Dentistry, University of Münster , Münster , Germany
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Keywords genetic correlation analysis
depression
genetic correlation
mendelian randomization analysis
periodontitis
Language English
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This article was submitted to Inflammation, a section of the journal Frontiers in Immunology
Reviewed by: Mia Rakic, Complutense University of Madrid, Spain; Chunyu Li, West China Hospital, Sichuan University, China
Edited by: Pedro Paulo Chaves Souza, Universidade Federal de Goiás, Brazil
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Snippet Observational and research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and examined...
Observational and in-vivo research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation and...
BackgroundObservational and in-vivo research suggested a bidirectional relationship between depression and periodontitis. We estimated the genetic correlation...
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SubjectTerms depression
Depression - genetics
genetic correlation
genetic correlation analysis
Genome-Wide Association Study
Humans
Immunology
Linkage Disequilibrium
mendelian randomization analysis
Mendelian Randomization Analysis - methods
periodontitis
Periodontitis - genetics
Title No bidirectional relationship between depression and periodontitis: A genetic correlation and Mendelian randomization study
URI https://www.ncbi.nlm.nih.gov/pubmed/35935963
https://www.proquest.com/docview/2699958371
https://pubmed.ncbi.nlm.nih.gov/PMC9355660
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