The endothelial barrier and cancer metastasis: Does the protective facet of platelet function matter?

[Display omitted] Overwhelming evidence suggests that platelets have a detrimental role in promoting cancer spread via platelet–cancer cell interactions linked to thrombotic mechanisms. On the other hand, a beneficial role of platelets in the preservation of the endothelial barrier in inflammatory c...

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Published in:Biochemical pharmacology Vol. 176; p. 113886
Main Authors: Smeda, Marta, Przyborowski, Kamil, Stojak, Marta, Chlopicki, Stefan
Format: Journal Article
Language:English
Published: England Elsevier Inc 01.06.2020
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ISSN:0006-2952, 1873-2968, 1873-2968
Online Access:Get full text
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Summary:[Display omitted] Overwhelming evidence suggests that platelets have a detrimental role in promoting cancer spread via platelet–cancer cell interactions linked to thrombotic mechanisms. On the other hand, a beneficial role of platelets in the preservation of the endothelial barrier in inflammatory conditions has been recently described, a phenomenon that could also operate in cancer–related inflammation. It is tempting to speculate that some antiplatelet strategies to combat cancer metastasis may impair the endogenous platelet–dependent mechanisms preserving endothelial barrier function. If the protective function of platelets is impaired, it may lead to increased endothelial permeability and more efficient cancer cell intravasation in the primary tumor and cancer cell extravasation at metastatic sites. In this commentary, we discuss current evidence that could support this hypothesis.
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ISSN:0006-2952
1873-2968
1873-2968
DOI:10.1016/j.bcp.2020.113886