Does Endoplasmic Reticulum (ER) Stress Contribute to T-cell Exhaustion in B-ALL?

Glucose deprivation in T lymphocytes can trigger compensatory metabolic pathways, potentially contributing to T-cell exhaustion. Additionally, it may induce the unfolded protein response (UPR), ultimately resulting in endoplasmic reticulum (ER) stress. To examine the transcriptional profiles of endo...

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Bibliographic Details

Published in:	Iranian journal of immunology Vol. 22; no. 2; p. 100
Main Authors:	Kahrizi, Amir, Akbar, Armin, Najafi, Ahmad, Asgarian-Omran, Hossein, Karami, Hossein, Naderisorki, Mohammad, Karimi, Alireza, Tehrani, Mohsen
Format:	Journal Article
Language:	English
Published:	Iran Shiraz Institute for Cancer Research 01.06.2025
Subjects:	CD8 antigen Cell activation Endoplasmic reticulum Gene expression Lymphocytes Lymphocytes T Metabolic pathways Peripheral blood Protein folding
ISSN:	1735-1383, 1735-367X, 1735-367X
Online Access:	Get full text
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