cGAS-STING, inflammasomes and pyroptosis: an overview of crosstalk mechanism of activation and regulation

Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Main...

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Published in:Cell communication and signaling Vol. 22; no. 1; pp. 22 - 21
Main Authors: Liu, Jingwen, Zhou, Jing, Luan, Yuling, Li, Xiaoying, Meng, Xiangrui, Liao, Wenhao, Tang, Jianyuan, Wang, Zheilei
Format: Journal Article
Language:English
Published: London BioMed Central 09.01.2024
BioMed Central Ltd
Springer Nature B.V
BMC
Subjects:
Analysis
Autoimmune diseases
Bacterial infections
Biomedical and Life Sciences
Caspase 1
Cell Biology
Cell membranes
Cellular stress response
cGAS-STING
Chronic infection
Coronaviruses
Crosstalk network
Cytokines
Cytokines and Growth Factors
Dendritic cells
Development and progression
Diseases
Health aspects
Herpes viruses
Homeostasis
Immune response
Immunity, Innate
Infection
Infections
Inflammasome
Inflammasomes
Inflammation
Inflammatory diseases
Innate immunity
Interferon gamma
Kinases
Life Sciences
Membrane permeability
Mitochondrial DNA
Mitochondrial permeability transition pore
Nucleotidyltransferases
Pathogens
Permeability
Phosphorylation
Protein-Ligand Interactions
Proteins
Pyroptosis
Radiation
Receptors
Regulatory proteins
Review
Sensors
Signal transduction
Vaccines
Zika virus
Maryland
China
Inflammasome
Inflammation
Pyroptosis
Crosstalk network
cGAS-STING
Diseases
ISSN:1478-811X, 1478-811X
Online Access:Get full text
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Abstract Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Main body of the abstract Cellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis. Short conclusion This review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention.
AbstractList Abstract Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Main body of the abstract Cellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis. Short conclusion This review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention.
Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Main body of the abstract Cellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis. Short conclusion This review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention. Keywords: cGAS-STING, Inflammasome, Pyroptosis, Inflammation, Crosstalk network, Diseases
Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Cellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis. This review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention.
Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Main body of the abstract Cellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis. Short conclusion This review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention.
Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Cellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis.BACKGROUNDIntracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear. Cellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis.This review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention.SHORT CONCLUSIONThis review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention.
Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear.
BackgroundIntracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic inflammation, cancer progression and organ degeneration, but the mechanism and regulation of the crosstalk network remain unclear.Main body of the abstractCellular stress disrupts mitochondrial homeostasis, facilitates the opening of mitochondrial permeability transition pore and the leakage of mitochondrial DNA to cell membrane, triggers inflammatory responses by activating cGAS-STING signaling, and subsequently induces inflammasomes activation and the onset of pyroptosis. Meanwhile, the inflammasome-associated protein caspase-1, Gasdermin D, the CARD domain of ASC and the potassium channel are involved in regulating cGAS-STING pathway. Importantly, this crosstalk network has a cascade amplification effect that exacerbates the immuno-inflammatory response, worsening the pathological process of inflammatory and autoimmune diseases. Given the importance of this crosstalk network of cGAS-STING, inflammasomes and pyroptosis in the regulation of innate immunity, it is emerging as a new avenue to explore the mechanisms of multiple disease pathogenesis. Therefore, efforts to define strategies to selectively modulate cGAS-STING, inflammasomes and pyroptosis in different disease settings have been or are ongoing. In this review, we will describe how this mechanistic understanding is driving possible therapeutics targeting this crosstalk network, focusing on the interacting or regulatory proteins, pathways, and a regulatory mitochondrial hub between cGAS-STING, inflammasomes, and pyroptosis.Short conclusionThis review aims to provide insight into the critical roles and regulatory mechanisms of the crosstalk network of cGAS-STING, inflammasomes and pyroptosis, and to highlight some promising directions for future research and intervention.
ArticleNumber 22
Audience Academic
Author Liu, Jingwen
Luan, Yuling
Li, Xiaoying
Zhou, Jing
Meng, Xiangrui
Liao, Wenhao
Tang, Jianyuan
Wang, Zheilei
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– sequence: 2
  givenname: Jing
  surname: Zhou
  fullname: Zhou, Jing
  organization: The Second Hospital of Ningbo
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  givenname: Yuling
  surname: Luan
  fullname: Luan, Yuling
  organization: Putuo Hospital, Shanghai University of Traditional Chinese Medicine
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  givenname: Xiaoying
  surname: Li
  fullname: Li, Xiaoying
  organization: Yueyang Hospital of Integrated Traditional Chinese and Western Medicine, Shanghai University of Traditional Chinese Medicine
– sequence: 5
  givenname: Xiangrui
  surname: Meng
  fullname: Meng, Xiangrui
  organization: Hospital of Chengdu University of Traditional Chinese Medicine
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  organization: Hospital of Chengdu University of Traditional Chinese Medicine
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  givenname: Jianyuan
  surname: Tang
  fullname: Tang, Jianyuan
  email: tangjy@cdutcm.edu.cn
  organization: Hospital of Chengdu University of Traditional Chinese Medicine, TCM Regulating Metabolic Diseases Key Laboratory of Sichuan Province, Hospital of Chengdu University of Traditional Chinese Medicine
– sequence: 8
  givenname: Zheilei
  surname: Wang
  fullname: Wang, Zheilei
  email: wangzl1993@outlook.com
  organization: Hospital of Chengdu University of Traditional Chinese Medicine, TCM Regulating Metabolic Diseases Key Laboratory of Sichuan Province, Hospital of Chengdu University of Traditional Chinese Medicine
BackLink https://www.ncbi.nlm.nih.gov/pubmed/38195584$$D View this record in MEDLINE/PubMed
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Cites_doi 10.1038/s41586-019-1605-5
10.1038/ncomms14392
10.1186/s12974-020-01830-4
10.1016/j.tins.2020.10.008
10.1038/ni.1932
10.15252/embj.201694696
10.1126/science.aau1208
10.15252/embj.201488029
10.1172/JCI121842
10.3390/ijms20194924
10.1002/anie.202107265
10.1111/acel.13186
10.1136/jitc-2022-006481
10.1073/pnas.1308331110
10.1152/ajprenal.00534.2015
10.1038/s41577-021-00588-x
10.1002/anie.202210988
10.1016/j.immuni.2019.02.009
10.1002/ctm2.228
10.1074/jbc.M116.736991
10.1038/s41586-023-06373-1
10.1038/s41419-022-05047-6
10.1172/JCI136329
10.1016/j.immuni.2014.01.010
10.1038/s41418-022-01033-9
10.1016/j.phrs.2019.104349
10.3389/fcell.2021.717610
10.1016/j.molcel.2019.03.021
10.1016/j.ijbiomac.2020.12.127
10.1111/bph.13825
10.1038/ncomms10555
10.1016/j.jhep.2012.03.035
10.1111/jnc.15678
10.3389/fimmu.2022.1006395
10.7150/thno.37574
10.1016/j.jhep.2020.07.041
10.1038/s41586-022-04802-1
10.1016/j.chom.2016.03.001
10.1016/j.jaci.2021.10.027
10.3389/fcimb.2016.00182
10.1186/s13045-020-00916-z
10.1084/jem.20121960
10.1038/s41401-020-00525-z
10.1002/advs.202102534
10.1016/j.immuni.2022.06.007
10.1016/j.immuni.2021.05.004
10.1681/ASN.2006060639
10.1038/ni.3118
10.1021/acs.jmedchem.9b01039
10.1084/jem.20151613
10.1038/s41580-020-0244-x
10.1038/s41416-022-01775-w
10.3389/fimmu.2022.973089
10.3390/cancers12010079
10.1126/scisignal.aat9900
10.1007/s10753-021-01529-5
10.3389/fimmu.2022.905109
10.1038/s41586-022-04421-w
10.1155/2021/1552127
10.1074/jbc.M110.109736
10.1371/journal.pone.0029004
10.1161/CIRCULATIONAHA.113.002208
10.1111/acel.13594
10.1113/jphysiol.2010.190926
10.1038/nature18268
10.1128/mBio.00944-17
10.1016/j.biomaterials.2011.05.009
10.1002/jmv.28472
10.1371/journal.ppat.1005809
10.1146/annurev-virology-092917-043244
10.1016/j.redox.2022.102305
10.1016/j.immuni.2020.05.013
10.1038/cmi.2016.25
10.1038/s41423-021-00801-w
10.3389/fimmu.2020.575818
10.1002/eji.201847589
10.3389/fimmu.2017.00873
10.55563/clinexprheumatol/eakvlv
10.1016/j.immuni.2022.07.011
10.1126/sciimmunol.abi9007
10.1093/nar/gky777
10.1038/s41591-019-0675-0
10.1126/science.1162667
10.1016/j.intimp.2023.110104
10.1002/eji.201545839
10.1038/s41577-021-00524-z
10.1172/JCI83658
10.1186/s12974-022-02511-0
10.1038/s41556-022-00894-z
10.1073/pnas.1717190115
10.1016/S0140-6736(13)60647-9
10.1073/pnas.2011226118
10.1016/j.jaci.2017.05.015
10.1515/hmbci-2014-0022
10.1002/ctm2.269
10.1038/s41380-018-0286-z
10.1371/journal.ppat.1008335
10.1016/j.cyto.2018.04.032
10.1089/ars.2017.7096
10.4049/jimmunol.1700725
10.1126/science.aau0810
10.1038/s41423-021-00670-3
10.1016/j.addr.2021.114020
10.1073/pnas.1607769113
10.1016/j.molmed.2014.11.008
10.3390/diagnostics10030152
10.2147/DDDT.S251623
10.3390/ijms22031048
10.1038/s41419-022-04664-5
10.1038/ni.1960
10.1186/s12985-017-0891-5
10.1038/s41419-019-1777-9
10.4049/jimmunol.1700698
10.1093/hmg/ddi229
10.1093/jmcb/mjz020
10.1016/j.immuni.2018.03.017
10.1038/ni.2509
10.1016/j.immuni.2011.02.006
10.4049/jimmunol.1002468
10.4049/jimmunol.0900557
10.1111/liv.14895
10.1007/s12975-020-00840-x
10.1186/s13073-021-00863-5
10.1038/s12276-019-0333-0
10.1146/annurev-pathmechdis-032521-102529
10.1021/acs.nanolett.2c04883
10.1016/j.cell.2013.09.049
10.1038/nrneph.2017.2
10.3389/fcimb.2017.00169
10.1523/JNEUROSCI.4452-12.2013
10.1074/jbc.M116.769695
10.1126/science.aau1330
10.1038/s41420-022-01046-w
10.1172/JCI149258
10.1038/ni.2239
10.1038/nature14156
10.3389/fimmu.2022.1010911
10.1126/science.aab3145
10.1152/physrev.00015.2003
10.1681/ASN.2010020143
10.1038/ni.3558
10.1074/jbc.RA117.000152
10.1038/s12276-022-00729-9
10.3389/fimmu.2020.624597
10.1016/j.autrev.2021.102867
10.1093/nar/gkv008
10.1016/j.chom.2015.05.003
10.1080/15548627.2017.1293766
10.1038/s41392-021-00752-8
10.1038/ncomms15030
10.4049/jimmunol.1701492
10.1016/j.cell.2021.06.028
10.1016/j.immuni.2018.07.006
10.1002/jmv.29013
10.1016/j.trecan.2022.07.002
10.3390/antiox10030485
10.3389/fonc.2020.598107
10.4049/jimmunol.1402764
10.1002/hep.26592
10.1016/j.ebiom.2022.104299
10.3389/fimmu.2021.682736
10.1016/j.cytogfr.2021.06.002
10.4049/jimmunol.1701177
10.3389/fimmu.2023.1130423
10.1053/j.gastro.2018.09.010
10.1016/j.ceb.2019.02.002
10.1111/j.1600-065X.2011.01053.x
10.15252/emmm.202013076
10.3389/fphar.2022.907209
10.1172/jci.insight.159430
10.1038/s41571-020-00459-9
10.1158/2159-8290.CD-19-0761
10.1371/journal.ppat.1006383
10.1172/JCI148852
10.15252/emmm.201911002
10.1111/cei.12457
10.3389/fimmu.2019.00474
10.1038/s42003-022-03868-x
10.1002/hep.31752
10.1016/j.taap.2021.115495
10.1038/cddis.2016.460
10.1128/mSystems.00336-21
10.1038/nature18629
10.1523/JNEUROSCI.0774-19.2019
10.1182/blood-2016-07-730556
10.1016/j.redox.2019.101215
10.1016/S0140-6736(18)30312-X
10.1371/journal.ppat.1008079
10.1083/jcb.201503023
10.1371/journal.ppat.1005019
10.1073/pnas.2123227119
10.1038/s41467-020-18916-5
10.1016/j.cell.2015.12.057
10.18632/oncotarget.17057
10.1126/science.1230751
10.3389/fimmu.2019.02536
10.1161/HYPERTENSIONAHA.122.20004
10.1111/obr.12520
10.1016/j.immuni.2019.06.025
10.1016/j.biopha.2022.113680
10.1038/s41467-018-02936-3
10.1186/s12974-023-02737-6
10.1016/j.ijrobp.2023.06.221
10.1186/s12943-020-01247-w
10.1016/j.redox.2023.102691
10.14336/AD.2019.0116
10.1073/pnas.1818281116
10.1371/journal.ppat.1008294
10.3389/fimmu.2019.02274
10.1016/j.mcn.2005.07.017
10.1016/j.jhep.2020.04.001
10.1016/j.celrep.2019.09.050
10.1155/2022/1392896
10.3389/fimmu.2013.00440
10.1038/s41419-020-02985-x
10.4049/jimmunol.1901508
10.1038/s41586-018-0705-y
10.1016/j.immuni.2017.03.020
10.1242/jcs.091181
10.4049/jimmunol.1502538
10.1186/1471-2148-12-140
10.1038/nri3921
10.1084/jem.20171749
10.1016/j.celrep.2017.02.057
10.1038/s43018-022-00468-w
10.1681/ASN.2015040376
10.1002/anie.202303010
10.1016/j.immuni.2015.08.013
10.1038/nature18590
10.1016/j.immuni.2016.06.015
10.1016/j.cmet.2022.02.016
10.15252/embj.201899347
10.1186/s13045-019-0721-x
10.1016/j.cell.2017.09.039
10.1084/jem.20182192
10.1128/mBio.01553-16
10.1016/j.immuni.2017.02.011
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Issue 1
Keywords Inflammasome
Inflammation
Pyroptosis
Crosstalk network
cGAS-STING
Diseases
Language English
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References YG Liu (1466_CR236) 2017; 8
T Taguchi (1466_CR17) 2019; 59
A Li (1466_CR204) 2019; 12
W Zhong (1466_CR81) 2020; 19
J Kwon (1466_CR9) 2020; 10
E Sefik (1466_CR208) 2022; 606
MS Dick (1466_CR26) 2017; 8
I Jorgensen (1466_CR29) 2016; 213
RA Aglietti (1466_CR58) 2016; 113
1466_CR214
H Konno (1466_CR125) 2013; 155
R Karki (1466_CR23) 2020; 204
M Brault (1466_CR94) 2018; 200
A Decout (1466_CR18) 2021; 21
L Corrales (1466_CR32) 2016; 196
K Ellwanger (1466_CR72) 2018; 293
Q Chen (1466_CR207) 2016; 533
B Cannon (1466_CR105) 2004; 84
G Pisano (1466_CR52) 2017; 14
N Miura (1466_CR232) 2015; 43
1466_CR205
S Paik (1466_CR21) 2021; 18
M Jiang (1466_CR8) 2020; 13
Z Liu (1466_CR112) 2022; 52
Y Wu (1466_CR24) 2015; 179
TP Fleming (1466_CR132) 2018; 391
EE Gray (1466_CR35) 2016; 45
M Bühler (1466_CR45) 2023; 20
X Xu (1466_CR233) 2023; 135
H Jiang (1466_CR36) 2021; 8
J Petrasek (1466_CR97) 2013; 110
Y Yu (1466_CR150) 2019; 129
S Van Herck (1466_CR13) 2021; 179
W Wang (1466_CR86) 2020; 16
SM Man (1466_CR89) 2016; 46
L Bai (1466_CR224) 2022; 2022
R Zhang (1466_CR75) 2023; 95
P Xu (1466_CR180) 2019; 10
Y Ran (1466_CR179) 2021; 2021
S Yan (1466_CR34) 2018; 200
B Han (1466_CR177) 2021; 13
N Lv (1466_CR141) 2022; 21
L Ning (1466_CR142) 2020; 10
G Lawrence (1466_CR102) 2022; 55
M Yan (1466_CR139) 2022; 8
T Abe (1466_CR73) 2017; 46
RA Ratsimandresy (1466_CR37) 2013; 4
J Cui (1466_CR71) 2012; 13
P Gurung (1466_CR99) 2015; 21
MI Gröschel (1466_CR230) 2017; 18
D Zhang (1466_CR137) 2022; 24
KE Wiens (1466_CR114) 2016; 12
RL Brunette (1466_CR38) 2012; 209
AA Kogan (1466_CR201) 2022; 119
J Hou (1466_CR203) 2021; 18
S Yuan (1466_CR220) 2017; 27
Y Li (1466_CR155) 2022; 19
W Gong (1466_CR174) 2016; 310
MJ Barrera (1466_CR198) 2021; 20
AP West (1466_CR100) 2015; 520
Y Peng (1466_CR5) 2020; 17
D Wang (1466_CR184) 2017; 13
SM Afify (1466_CR129) 2022; 127
Y Cui (1466_CR96) 2016; 6
J Wu (1466_CR157) 2019; 10
K Hansen (1466_CR54) 2014; 33
1466_CR84
MM Costa Franco (1466_CR93) 2018; 200
T Montanari (1466_CR107) 2017; 18
J Liu (1466_CR183) 2022; 154
GJ Seo (1466_CR119) 2018; 9
JD Domizio (1466_CR212) 2022; 603
Q Cheng (1466_CR27) 2021; 42
B Schoenebeck (1466_CR190) 2005; 30
C Zhang (1466_CR226) 2022; 13
NH Lameire (1466_CR165) 2013; 382
D Jia (1466_CR164) 2021; 60
UA Nuber (1466_CR186) 2005; 14
R Panchanathan (1466_CR43) 2010; 185
SJ Webster (1466_CR80) 2017; 13
BR Kim (1466_CR115) 2020; 16
L Wang (1466_CR189) 2017; 8
N Kopitar-Jerala (1466_CR118) 2017; 8
T Wu (1466_CR55) 2021; 418
L Sborgi (1466_CR61) 2016; 35
MN Lee (1466_CR47) 2013; 14
1466_CR76
N Samson (1466_CR10) 2022; 3
H Yong (1466_CR154) 2021; 41
D Furman (1466_CR134) 2019; 25
B Bai (1466_CR30) 2020; 11
1466_CR193
J Gautheron (1466_CR156) 2020; 73
JF Almine (1466_CR46) 2017; 8
R Xiao (1466_CR213) 2022; 13
Y Wang (1466_CR11) 2020; 19
H Zhang (1466_CR83) 2022; 5
M Rosina (1466_CR109) 2022; 34
XY Wu (1466_CR160) 2022; 2022
KP Hopfner (1466_CR101) 2020; 21
G Yang (1466_CR222) 2008; 322
H Maekawa (1466_CR168) 2019; 29
J Luo (1466_CR15) 2019; 10
L Zhang (1466_CR68) 2014; 40
1466_CR64
H Zhang (1466_CR2) 2020; 63
1466_CR185
X Song (1466_CR91) 2019; 39
X Luo (1466_CR151) 2018; 155
A Vilaysane (1466_CR173) 2010; 21
N Zhao (1466_CR209) 2021; 61
OC Kwon (1466_CR192) 2021; 13
MA Ansari (1466_CR51) 2015; 11
S Mani (1466_CR221) 2013; 127
L Chunfa (1466_CR49) 2017; 7
Y Yang (1466_CR70) 2018; 48
YT Wu (1466_CR217) 2023; 118
S Gaul (1466_CR159) 2021; 74
1466_CR56
1466_CR53
Z Wang (1466_CR147) 2021; 9
J Ding (1466_CR59) 2016; 535
1466_CR175
B Lin (1466_CR199) 2022; 149
HY Li (1466_CR161) 2011; 32
A Molyvdas (1466_CR22) 2018; 110
KD Mayer-Barber (1466_CR117) 2017; 14
S Inokuchi (1466_CR200) 2022; 40
H Qiao (1466_CR124) 2023; 95
Q Li (1466_CR135) 2020; 12
G Szabo (1466_CR149) 2012; 57
A Iracheta-Vellve (1466_CR153) 2016; 291
C Baatarjav (1466_CR33) 2022; 29
1466_CR42
W Eldahshan (1466_CR176) 2019; 147
V Kumar (1466_CR39) 2020; 11
RM de Carvalho (1466_CR148) 2022; 17
Y Zhang (1466_CR215) 2023; 23
1466_CR169
Y Wu (1466_CR216) 2022; 13
G Guarda (1466_CR116) 2011; 34
L Unterholzner (1466_CR40) 2010; 11
M Motwani (1466_CR16) 2019; 116
S Kuenzel (1466_CR77) 2010; 184
NQ Dat (1466_CR162) 2021; 73
Q Chen (1466_CR6) 2016; 17
J Han (1466_CR140) 2022; 79
D Denk (1466_CR128) 2022; 8
X Liu (1466_CR122) 2018; 46
MF Gulen (1466_CR138) 2023; 620
T Su (1466_CR228) 2019; 9
J Zi (1466_CR121) 2020; 10
H Wobma (1466_CR195) 2022; 13
SA Schattgen (1466_CR41) 2011; 243
R Wassermann (1466_CR113) 2015; 17
Y Wang (1466_CR25) 2019; 11
SM Vora (1466_CR210) 2021; 21
D Xu (1466_CR146) 2021; 12
Y Aachoui (1466_CR31) 2013; 339
Z Ma (1466_CR227) 2018; 5
S Aits (1466_CR85) 2013; 126
G Long (1466_CR143) 2022; 13
1466_CR145
BD Paul (1466_CR19) 2021; 44
KM Storek (1466_CR44) 2015; 194
P Wang (1466_CR74) 2015; 350
T Kimura (1466_CR126) 2015; 210
W Chen (1466_CR50) 2019; 10
D Chen (1466_CR95) 2018; 115
A Wree (1466_CR158) 2014; 59
P Xu (1466_CR178) 2021; 12
P Shen (1466_CR225) 2022; 45
J Wu (1466_CR98) 2019; 216
Y Messaoud-Nacer (1466_CR144) 2022; 13
C Zheng (1466_CR66) 2021; 169
1466_CR14
JA Cridland (1466_CR88) 2012; 12
R Ding (1466_CR182) 2022; 19
Z Zhang (1466_CR188) 2021; 26
NP Manes (1466_CR4) 2021; 6
Y Wang (1466_CR57) 2017; 46
GN Barber (1466_CR238) 2015; 15
H Xian (1466_CR104) 2022; 55
JM Ramanjulu (1466_CR12) 2018; 564
C Ma (1466_CR231) 2021; 6
CL Evavold (1466_CR163) 2021; 184
SL Ming (1466_CR229) 2020; 11
X Li (1466_CR67) 2019; 50
Z Chen (1466_CR131) 2020; 11
H Xian (1466_CR152) 2021; 54
KV Swanson (1466_CR127) 2017; 214
FR Greten (1466_CR130) 2019; 51
W Zhang (1466_CR197) 2022; 54
M Karmakar (1466_CR20) 2016; 7
A Fenzl (1466_CR106) 2014; 19
I Banerjee (1466_CR62) 2018; 49
G Meng (1466_CR219) 2018; 175
L Cao (1466_CR48) 2019; 15
LM Zhang (1466_CR194) 2022; 162
Y Xiao (1466_CR87) 2023; 62
Z Zhong (1466_CR103) 2016; 164
YY Ling (1466_CR235) 2022; 61
E Eren (1466_CR63) 2017; 292
D Cohen (1466_CR3) 2019; 574
N Tsuji (1466_CR167) 2016; 27
Y Tian (1466_CR218) 2020; 14
X Liu (1466_CR60) 2016; 535
G Wang (1466_CR110) 2019; 74
Y Du (1466_CR136) 2023; 14
X Zhang (1466_CR7) 2020; 53
M Correa-Costa (1466_CR172) 2011; 6
C Wang (1466_CR120) 2018; 48
AA Basiorka (1466_CR196) 2016; 128
LS Chawla (1466_CR166) 2017; 13
T Inoue (1466_CR170) 2016; 126
MM Gaidt (1466_CR79) 2017; 171
1466_CR111
HY Peng (1466_CR191) 2013; 33
1466_CR237
AJ Chui (1466_CR65) 2019; 364
N Potere (1466_CR211) 2022; 85
1466_CR234
S Xu (1466_CR181) 2021; 11
NL Denning (1466_CR123) 2019; 10
SM Man (1466_CR92) 2015; 16
J Ahn (1466_CR1) 2019; 51
F Liu (1466_CR90) 2017; 199
F Lang (1466_CR187) 2010; 588
L Gu (1466_CR202) 2022; 13
A Neerincx (1466_CR78) 2010; 285
EA Miao (1466_CR28) 2010; 11
N Li (1466_CR82) 2019; 24
I Kojima (1466_CR171) 2007; 18
H Guo (1466_CR69) 2016; 19
J Dupaul-Chicoine (1466_CR206) 2015; 43
1466_CR108
H Renz (1466_CR133) 2017; 140
1466_CR223
References_xml – volume: 574
  start-page: 691
  issue: 7780
  year: 2019
  ident: 1466_CR3
  publication-title: Nature.
  doi: 10.1038/s41586-019-1605-5
– volume: 8
  start-page: 14392
  year: 2017
  ident: 1466_CR46
  publication-title: Nat Commun.
  doi: 10.1038/ncomms14392
– volume: 17
  start-page: 165
  issue: 1
  year: 2020
  ident: 1466_CR5
  publication-title: J Neuroinflammation.
  doi: 10.1186/s12974-020-01830-4
– volume: 44
  start-page: 83
  issue: 2
  year: 2021
  ident: 1466_CR19
  publication-title: Trends Neurosci.
  doi: 10.1016/j.tins.2020.10.008
– volume: 11
  start-page: 997
  issue: 11
  year: 2010
  ident: 1466_CR40
  publication-title: Nat Immunol.
  doi: 10.1038/ni.1932
– volume: 35
  start-page: 1766
  issue: 16
  year: 2016
  ident: 1466_CR61
  publication-title: EMBO J.
  doi: 10.15252/embj.201694696
– volume: 364
  start-page: 82
  issue: 6435
  year: 2019
  ident: 1466_CR65
  publication-title: Science (New York, NY).
  doi: 10.1126/science.aau1208
– volume: 33
  start-page: 1654
  issue: 15
  year: 2014
  ident: 1466_CR54
  publication-title: EMBO J.
  doi: 10.15252/embj.201488029
– volume: 129
  start-page: 546
  issue: 2
  year: 2019
  ident: 1466_CR150
  publication-title: J Clin Invest.
  doi: 10.1172/JCI121842
– ident: 1466_CR108
  doi: 10.3390/ijms20194924
– volume: 60
  start-page: 21449
  issue: 39
  year: 2021
  ident: 1466_CR164
  publication-title: Angewandte Chemie (International ed in English).
  doi: 10.1002/anie.202107265
– volume: 19
  start-page: e13186
  issue: 8
  year: 2020
  ident: 1466_CR81
  publication-title: Aging Cell.
  doi: 10.1111/acel.13186
– ident: 1466_CR205
  doi: 10.1136/jitc-2022-006481
– volume: 110
  start-page: 16544
  issue: 41
  year: 2013
  ident: 1466_CR97
  publication-title: Proc Natl Acad Sci U S A.
  doi: 10.1073/pnas.1308331110
– volume: 310
  start-page: F1081
  issue: 10
  year: 2016
  ident: 1466_CR174
  publication-title: Am J Physiol Renal Physiol.
  doi: 10.1152/ajprenal.00534.2015
– volume: 21
  start-page: 694
  issue: 11
  year: 2021
  ident: 1466_CR210
  publication-title: Nat Rev Immunol.
  doi: 10.1038/s41577-021-00588-x
– volume: 61
  start-page: e202210988
  issue: 43
  year: 2022
  ident: 1466_CR235
  publication-title: Angewandte Chemie (International ed in English).
  doi: 10.1002/anie.202210988
– volume: 50
  start-page: 591
  issue: 3
  year: 2019
  ident: 1466_CR67
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2019.02.009
– volume: 10
  start-page: e228
  issue: 7
  year: 2020
  ident: 1466_CR142
  publication-title: Clin Transl Med.
  doi: 10.1002/ctm2.228
– volume: 291
  start-page: 26794
  issue: 52
  year: 2016
  ident: 1466_CR153
  publication-title: J Biol Chem.
  doi: 10.1074/jbc.M116.736991
– volume: 620
  start-page: 374
  issue: 7973
  year: 2023
  ident: 1466_CR138
  publication-title: Nature.
  doi: 10.1038/s41586-023-06373-1
– volume: 13
  start-page: 583
  issue: 7
  year: 2022
  ident: 1466_CR202
  publication-title: Cell Death Dis.
  doi: 10.1038/s41419-022-05047-6
– ident: 1466_CR175
  doi: 10.1172/JCI136329
– volume: 40
  start-page: 329
  issue: 3
  year: 2014
  ident: 1466_CR68
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2014.01.010
– volume: 29
  start-page: 2487
  issue: 12
  year: 2022
  ident: 1466_CR33
  publication-title: Cell Death Differ.
  doi: 10.1038/s41418-022-01033-9
– volume: 147
  start-page: 104349
  year: 2019
  ident: 1466_CR176
  publication-title: Pharmacol Res.
  doi: 10.1016/j.phrs.2019.104349
– volume: 9
  start-page: 717610
  year: 2021
  ident: 1466_CR147
  publication-title: Front Cell Dev Biol.
  doi: 10.3389/fcell.2021.717610
– volume: 74
  start-page: 844
  issue: 4
  year: 2019
  ident: 1466_CR110
  publication-title: Mol Cell.
  doi: 10.1016/j.molcel.2019.03.021
– volume: 169
  start-page: 407
  year: 2021
  ident: 1466_CR66
  publication-title: Int J Biol Macromol.
  doi: 10.1016/j.ijbiomac.2020.12.127
– volume: 175
  start-page: 1146
  issue: 8
  year: 2018
  ident: 1466_CR219
  publication-title: Br J Pharmacol.
  doi: 10.1111/bph.13825
– volume: 7
  start-page: 10555
  year: 2016
  ident: 1466_CR20
  publication-title: Nat Commun.
  doi: 10.1038/ncomms10555
– volume: 57
  start-page: 642
  issue: 3
  year: 2012
  ident: 1466_CR149
  publication-title: J Hepatol.
  doi: 10.1016/j.jhep.2012.03.035
– volume: 162
  start-page: 444
  issue: 5
  year: 2022
  ident: 1466_CR194
  publication-title: J Neurochem.
  doi: 10.1111/jnc.15678
– volume: 13
  start-page: 1006395
  year: 2022
  ident: 1466_CR213
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2022.1006395
– volume: 9
  start-page: 7759
  issue: 25
  year: 2019
  ident: 1466_CR228
  publication-title: Theranostics.
  doi: 10.7150/thno.37574
– volume: 74
  start-page: 156
  issue: 1
  year: 2021
  ident: 1466_CR159
  publication-title: J Hepatol.
  doi: 10.1016/j.jhep.2020.07.041
– volume: 606
  start-page: 585
  issue: 7914
  year: 2022
  ident: 1466_CR208
  publication-title: Nature.
  doi: 10.1038/s41586-022-04802-1
– volume: 19
  start-page: 515
  issue: 4
  year: 2016
  ident: 1466_CR69
  publication-title: Cell Host Microbe.
  doi: 10.1016/j.chom.2016.03.001
– volume: 149
  start-page: 819
  issue: 3
  year: 2022
  ident: 1466_CR199
  publication-title: J Allergy Clin Immunol.
  doi: 10.1016/j.jaci.2021.10.027
– volume: 6
  start-page: 182
  year: 2016
  ident: 1466_CR96
  publication-title: Front Cell Infect Microbiol.
  doi: 10.3389/fcimb.2016.00182
– volume: 13
  start-page: 81
  issue: 1
  year: 2020
  ident: 1466_CR8
  publication-title: J Hematol Oncol.
  doi: 10.1186/s13045-020-00916-z
– volume: 209
  start-page: 1969
  issue: 11
  year: 2012
  ident: 1466_CR38
  publication-title: J Exp Med.
  doi: 10.1084/jem.20121960
– volume: 42
  start-page: 954
  issue: 6
  year: 2021
  ident: 1466_CR27
  publication-title: Acta Pharmacol Sin.
  doi: 10.1038/s41401-020-00525-z
– volume: 8
  start-page: e2102534
  issue: 22
  year: 2021
  ident: 1466_CR36
  publication-title: Adv Sci (Weinh).
  doi: 10.1002/advs.202102534
– volume: 55
  start-page: 1370
  issue: 8
  year: 2022
  ident: 1466_CR104
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2022.06.007
– volume: 54
  start-page: 1463
  issue: 7
  year: 2021
  ident: 1466_CR152
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2021.05.004
– volume: 18
  start-page: 1218
  issue: 4
  year: 2007
  ident: 1466_CR171
  publication-title: J Am Soc Nephrol : JASN.
  doi: 10.1681/ASN.2006060639
– volume: 16
  start-page: 467
  issue: 5
  year: 2015
  ident: 1466_CR92
  publication-title: Nat Immunol.
  doi: 10.1038/ni.3118
– volume: 63
  start-page: 3785
  issue: 8
  year: 2020
  ident: 1466_CR2
  publication-title: J Med Chem.
  doi: 10.1021/acs.jmedchem.9b01039
– volume: 213
  start-page: 2113
  issue: 10
  year: 2016
  ident: 1466_CR29
  publication-title: J Exp Med.
  doi: 10.1084/jem.20151613
– volume: 21
  start-page: 501
  issue: 9
  year: 2020
  ident: 1466_CR101
  publication-title: Nat Rev Mol Cell Biol.
  doi: 10.1038/s41580-020-0244-x
– volume: 127
  start-page: 193
  issue: 2
  year: 2022
  ident: 1466_CR129
  publication-title: Br J Cancer.
  doi: 10.1038/s41416-022-01775-w
– volume: 13
  start-page: 973089
  year: 2022
  ident: 1466_CR143
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2022.973089
– ident: 1466_CR237
  doi: 10.3390/cancers12010079
– ident: 1466_CR185
  doi: 10.1126/scisignal.aat9900
– volume: 45
  start-page: 74
  issue: 1
  year: 2022
  ident: 1466_CR225
  publication-title: Inflammation.
  doi: 10.1007/s10753-021-01529-5
– volume: 13
  start-page: 905109
  year: 2022
  ident: 1466_CR195
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2022.905109
– volume: 603
  start-page: 145
  issue: 7899
  year: 2022
  ident: 1466_CR212
  publication-title: Nature.
  doi: 10.1038/s41586-022-04421-w
– volume: 2021
  start-page: 1552127
  year: 2021
  ident: 1466_CR179
  publication-title: Oxidative Med Cell Longev.
  doi: 10.1155/2021/1552127
– volume: 285
  start-page: 26223
  issue: 34
  year: 2010
  ident: 1466_CR78
  publication-title: J Biol Chem.
  doi: 10.1074/jbc.M110.109736
– volume: 6
  start-page: e29004
  issue: 12
  year: 2011
  ident: 1466_CR172
  publication-title: PLoS One.
  doi: 10.1371/journal.pone.0029004
– volume: 127
  start-page: 2523
  issue: 25
  year: 2013
  ident: 1466_CR221
  publication-title: Circulation.
  doi: 10.1161/CIRCULATIONAHA.113.002208
– volume: 21
  start-page: e13594
  issue: 4
  year: 2022
  ident: 1466_CR141
  publication-title: Aging Cell.
  doi: 10.1111/acel.13594
– volume: 588
  start-page: 3349
  issue: Pt 18
  year: 2010
  ident: 1466_CR187
  publication-title: J Physiol.
  doi: 10.1113/jphysiol.2010.190926
– volume: 533
  start-page: 493
  issue: 7604
  year: 2016
  ident: 1466_CR207
  publication-title: Nature.
  doi: 10.1038/nature18268
– ident: 1466_CR42
  doi: 10.1128/mBio.00944-17
– volume: 32
  start-page: 5994
  issue: 26
  year: 2011
  ident: 1466_CR161
  publication-title: Biomaterials.
  doi: 10.1016/j.biomaterials.2011.05.009
– volume: 95
  start-page: e28472
  issue: 2
  year: 2023
  ident: 1466_CR124
  publication-title: J Med Virol.
  doi: 10.1002/jmv.28472
– volume: 12
  start-page: e1005809
  issue: 8
  year: 2016
  ident: 1466_CR114
  publication-title: PLoS Pathog.
  doi: 10.1371/journal.ppat.1005809
– volume: 5
  start-page: 341
  issue: 1
  year: 2018
  ident: 1466_CR227
  publication-title: Annu Rev Virol.
  doi: 10.1146/annurev-virology-092917-043244
– volume: 52
  start-page: 102305
  year: 2022
  ident: 1466_CR112
  publication-title: Redox Biol.
  doi: 10.1016/j.redox.2022.102305
– volume: 53
  start-page: 43
  issue: 1
  year: 2020
  ident: 1466_CR7
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2020.05.013
– volume: 14
  start-page: 22
  issue: 1
  year: 2017
  ident: 1466_CR117
  publication-title: Cell Mol Immunol.
  doi: 10.1038/cmi.2016.25
– volume: 19
  start-page: 92
  issue: 1
  year: 2022
  ident: 1466_CR155
  publication-title: Cell Mol Immunol.
  doi: 10.1038/s41423-021-00801-w
– volume: 11
  start-page: 575818
  year: 2020
  ident: 1466_CR229
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2020.575818
– volume: 48
  start-page: 1817
  issue: 11
  year: 2018
  ident: 1466_CR70
  publication-title: Eur J Immunol.
  doi: 10.1002/eji.201847589
– volume: 8
  start-page: 873
  year: 2017
  ident: 1466_CR118
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2017.00873
– volume: 40
  start-page: 522
  issue: 3
  year: 2022
  ident: 1466_CR200
  publication-title: Clin Exp Rheumatol.
  doi: 10.55563/clinexprheumatol/eakvlv
– volume: 55
  start-page: 1331
  issue: 8
  year: 2022
  ident: 1466_CR102
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2022.07.011
– ident: 1466_CR214
  doi: 10.1126/sciimmunol.abi9007
– volume: 46
  start-page: 9601
  issue: 18
  year: 2018
  ident: 1466_CR122
  publication-title: Nucleic Acids Res.
  doi: 10.1093/nar/gky777
– volume: 25
  start-page: 1822
  issue: 12
  year: 2019
  ident: 1466_CR134
  publication-title: Nat Med.
  doi: 10.1038/s41591-019-0675-0
– volume: 322
  start-page: 587
  issue: 5901
  year: 2008
  ident: 1466_CR222
  publication-title: Science (New York, NY).
  doi: 10.1126/science.1162667
– volume: 118
  start-page: 110104
  year: 2023
  ident: 1466_CR217
  publication-title: Int Immunopharmacol.
  doi: 10.1016/j.intimp.2023.110104
– volume: 46
  start-page: 269
  issue: 2
  year: 2016
  ident: 1466_CR89
  publication-title: Eur J Immunol.
  doi: 10.1002/eji.201545839
– volume: 21
  start-page: 548
  issue: 9
  year: 2021
  ident: 1466_CR18
  publication-title: Nat Rev Immunol.
  doi: 10.1038/s41577-021-00524-z
– volume: 126
  start-page: 1939
  issue: 5
  year: 2016
  ident: 1466_CR170
  publication-title: J Clin Invest.
  doi: 10.1172/JCI83658
– volume: 19
  start-page: 137
  issue: 1
  year: 2022
  ident: 1466_CR182
  publication-title: J Neuroinflammation.
  doi: 10.1186/s12974-022-02511-0
– volume: 24
  start-page: 766
  issue: 5
  year: 2022
  ident: 1466_CR137
  publication-title: Nat Cell Biol.
  doi: 10.1038/s41556-022-00894-z
– volume: 115
  start-page: 3930
  issue: 15
  year: 2018
  ident: 1466_CR95
  publication-title: Proc Natl Acad Sci U S A.
  doi: 10.1073/pnas.1717190115
– volume: 382
  start-page: 170
  issue: 9887
  year: 2013
  ident: 1466_CR165
  publication-title: Lancet (London, England).
  doi: 10.1016/S0140-6736(13)60647-9
– ident: 1466_CR193
  doi: 10.1073/pnas.2011226118
– volume: 140
  start-page: 24
  issue: 1
  year: 2017
  ident: 1466_CR133
  publication-title: J Allergy Clin Immunol.
  doi: 10.1016/j.jaci.2017.05.015
– volume: 19
  start-page: 25
  issue: 1
  year: 2014
  ident: 1466_CR106
  publication-title: Horm Mol Biol Clin Investig.
  doi: 10.1515/hmbci-2014-0022
– volume: 11
  start-page: e269
  issue: 1
  year: 2021
  ident: 1466_CR181
  publication-title: Clin Transl Med.
  doi: 10.1002/ctm2.269
– volume: 26
  start-page: 586
  issue: 2
  year: 2021
  ident: 1466_CR188
  publication-title: Mol Psychiatry.
  doi: 10.1038/s41380-018-0286-z
– volume: 16
  start-page: e1008335
  issue: 3
  year: 2020
  ident: 1466_CR86
  publication-title: PLoS Pathog.
  doi: 10.1371/journal.ppat.1008335
– volume: 110
  start-page: 389
  year: 2018
  ident: 1466_CR22
  publication-title: Cytokine.
  doi: 10.1016/j.cyto.2018.04.032
– volume: 27
  start-page: 634
  issue: 10
  year: 2017
  ident: 1466_CR220
  publication-title: Antioxid Redox Signal.
  doi: 10.1089/ars.2017.7096
– volume: 200
  start-page: 607
  issue: 2
  year: 2018
  ident: 1466_CR93
  publication-title: J Immunol.
  doi: 10.4049/jimmunol.1700725
– ident: 1466_CR14
  doi: 10.1126/science.aau0810
– volume: 18
  start-page: 1141
  issue: 5
  year: 2021
  ident: 1466_CR21
  publication-title: Cell Mol Immunol.
  doi: 10.1038/s41423-021-00670-3
– volume: 179
  start-page: 114020
  year: 2021
  ident: 1466_CR13
  publication-title: Adv Drug Deliv Rev.
  doi: 10.1016/j.addr.2021.114020
– volume: 113
  start-page: 7858
  issue: 28
  year: 2016
  ident: 1466_CR58
  publication-title: Proc Natl Acad Sci U S A.
  doi: 10.1073/pnas.1607769113
– volume: 21
  start-page: 193
  issue: 3
  year: 2015
  ident: 1466_CR99
  publication-title: Trends Mol Med.
  doi: 10.1016/j.molmed.2014.11.008
– ident: 1466_CR169
  doi: 10.3390/diagnostics10030152
– volume: 14
  start-page: 2113
  year: 2020
  ident: 1466_CR218
  publication-title: Drug Des Devel Ther.
  doi: 10.2147/DDDT.S251623
– ident: 1466_CR76
  doi: 10.3390/ijms22031048
– volume: 13
  start-page: 269
  issue: 3
  year: 2022
  ident: 1466_CR144
  publication-title: Cell Death Dis.
  doi: 10.1038/s41419-022-04664-5
– volume: 11
  start-page: 1136
  issue: 12
  year: 2010
  ident: 1466_CR28
  publication-title: Nat Immunol.
  doi: 10.1038/ni.1960
– volume: 14
  start-page: 221
  issue: 1
  year: 2017
  ident: 1466_CR52
  publication-title: Virol J.
  doi: 10.1186/s12985-017-0891-5
– volume: 10
  start-page: 555
  issue: 8
  year: 2019
  ident: 1466_CR180
  publication-title: Cell Death Dis.
  doi: 10.1038/s41419-019-1777-9
– volume: 199
  start-page: 3293
  issue: 9
  year: 2017
  ident: 1466_CR90
  publication-title: J Immunol.
  doi: 10.4049/jimmunol.1700698
– volume: 14
  start-page: 2247
  issue: 15
  year: 2005
  ident: 1466_CR186
  publication-title: Hum Mol Genet.
  doi: 10.1093/hmg/ddi229
– volume: 11
  start-page: 1069
  issue: 12
  year: 2019
  ident: 1466_CR25
  publication-title: J Mol Cell Biol.
  doi: 10.1093/jmcb/mjz020
– volume: 48
  start-page: 675
  issue: 4
  year: 2018
  ident: 1466_CR120
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2018.03.017
– volume: 14
  start-page: 179
  issue: 2
  year: 2013
  ident: 1466_CR47
  publication-title: Nat Immunol.
  doi: 10.1038/ni.2509
– volume: 34
  start-page: 213
  issue: 2
  year: 2011
  ident: 1466_CR116
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2011.02.006
– volume: 185
  start-page: 7385
  issue: 12
  year: 2010
  ident: 1466_CR43
  publication-title: J Immunol (Baltimore, Md : 1950).
  doi: 10.4049/jimmunol.1002468
– volume: 184
  start-page: 1990
  issue: 4
  year: 2010
  ident: 1466_CR77
  publication-title: J Immunol (Baltimore, Md : 1950).
  doi: 10.4049/jimmunol.0900557
– volume: 41
  start-page: 1969
  issue: 8
  year: 2021
  ident: 1466_CR154
  publication-title: Liver Int : Off J Int Assoc Study Liver.
  doi: 10.1111/liv.14895
– volume: 12
  start-page: 643
  issue: 4
  year: 2021
  ident: 1466_CR178
  publication-title: Transl Stroke Res.
  doi: 10.1007/s12975-020-00840-x
– volume: 2022
  start-page: 8123157
  year: 2022
  ident: 1466_CR160
  publication-title: Oxidative Med Cell Longev.
– volume: 13
  start-page: 47
  issue: 1
  year: 2021
  ident: 1466_CR177
  publication-title: Genome Med.
  doi: 10.1186/s13073-021-00863-5
– volume: 51
  start-page: 1
  issue: 12
  year: 2019
  ident: 1466_CR1
  publication-title: Exp Mol Med.
  doi: 10.1038/s12276-019-0333-0
– volume: 17
  start-page: 345
  year: 2022
  ident: 1466_CR148
  publication-title: Annu Rev Pathol.
  doi: 10.1146/annurev-pathmechdis-032521-102529
– volume: 23
  start-page: 2593
  issue: 7
  year: 2023
  ident: 1466_CR215
  publication-title: Nano Lett.
  doi: 10.1021/acs.nanolett.2c04883
– volume: 155
  start-page: 688
  issue: 3
  year: 2013
  ident: 1466_CR125
  publication-title: Cell.
  doi: 10.1016/j.cell.2013.09.049
– volume: 13
  start-page: 241
  issue: 4
  year: 2017
  ident: 1466_CR166
  publication-title: Nat Rev Nephrol.
  doi: 10.1038/nrneph.2017.2
– volume: 7
  start-page: 169
  year: 2017
  ident: 1466_CR49
  publication-title: Front Cell Infect Microbiol.
  doi: 10.3389/fcimb.2017.00169
– volume: 33
  start-page: 5227
  issue: 12
  year: 2013
  ident: 1466_CR191
  publication-title: J Neurosci.
  doi: 10.1523/JNEUROSCI.4452-12.2013
– volume: 292
  start-page: 12691
  issue: 30
  year: 2017
  ident: 1466_CR63
  publication-title: J Biol Chem.
  doi: 10.1074/jbc.M116.769695
– ident: 1466_CR64
  doi: 10.1126/science.aau1330
– volume: 8
  start-page: 258
  issue: 1
  year: 2022
  ident: 1466_CR139
  publication-title: Cell Death Discov.
  doi: 10.1038/s41420-022-01046-w
– ident: 1466_CR234
  doi: 10.1172/JCI149258
– volume: 13
  start-page: 387
  issue: 4
  year: 2012
  ident: 1466_CR71
  publication-title: Nat Immunol.
  doi: 10.1038/ni.2239
– volume: 520
  start-page: 553
  issue: 7548
  year: 2015
  ident: 1466_CR100
  publication-title: Nature.
  doi: 10.1038/nature14156
– volume: 13
  start-page: 1010911
  year: 2022
  ident: 1466_CR216
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2022.1010911
– volume: 350
  start-page: 826
  issue: 6262
  year: 2015
  ident: 1466_CR74
  publication-title: Science (New York, NY).
  doi: 10.1126/science.aab3145
– volume: 84
  start-page: 277
  issue: 1
  year: 2004
  ident: 1466_CR105
  publication-title: Physiol Rev.
  doi: 10.1152/physrev.00015.2003
– volume: 21
  start-page: 1732
  issue: 10
  year: 2010
  ident: 1466_CR173
  publication-title: J Am Soc Nephrol : JASN.
  doi: 10.1681/ASN.2010020143
– volume: 17
  start-page: 1142
  issue: 10
  year: 2016
  ident: 1466_CR6
  publication-title: Nat Immunol.
  doi: 10.1038/ni.3558
– volume: 293
  start-page: 2701
  issue: 8
  year: 2018
  ident: 1466_CR72
  publication-title: J Biol Chem.
  doi: 10.1074/jbc.RA117.000152
– volume: 54
  start-page: 129
  issue: 2
  year: 2022
  ident: 1466_CR197
  publication-title: Exp Mol Med.
  doi: 10.1038/s12276-022-00729-9
– volume: 11
  start-page: 624597
  year: 2020
  ident: 1466_CR39
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2020.624597
– volume: 20
  start-page: 102867
  issue: 8
  year: 2021
  ident: 1466_CR198
  publication-title: Autoimmun Rev.
  doi: 10.1016/j.autrev.2021.102867
– volume: 43
  start-page: 1317
  issue: 3
  year: 2015
  ident: 1466_CR232
  publication-title: Nucleic Acids Res.
  doi: 10.1093/nar/gkv008
– volume: 17
  start-page: 799
  issue: 6
  year: 2015
  ident: 1466_CR113
  publication-title: Cell Host Microbe.
  doi: 10.1016/j.chom.2015.05.003
– volume: 13
  start-page: 914
  issue: 5
  year: 2017
  ident: 1466_CR184
  publication-title: Autophagy.
  doi: 10.1080/15548627.2017.1293766
– volume: 6
  start-page: 353
  issue: 1
  year: 2021
  ident: 1466_CR231
  publication-title: Signal Transduct Target Ther.
  doi: 10.1038/s41392-021-00752-8
– volume: 8
  start-page: 15030
  year: 2017
  ident: 1466_CR26
  publication-title: Nat Commun.
  doi: 10.1038/ncomms15030
– volume: 200
  start-page: 2748
  issue: 8
  year: 2018
  ident: 1466_CR94
  publication-title: J Immunol (Baltimore, Md : 1950).
  doi: 10.4049/jimmunol.1701492
– volume: 184
  start-page: 4495
  issue: 17
  year: 2021
  ident: 1466_CR163
  publication-title: Cell.
  doi: 10.1016/j.cell.2021.06.028
– volume: 49
  start-page: 413
  issue: 3
  year: 2018
  ident: 1466_CR62
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2018.07.006
– volume: 95
  start-page: e29013
  issue: 8
  year: 2023
  ident: 1466_CR75
  publication-title: J Med Virol.
  doi: 10.1002/jmv.29013
– volume: 8
  start-page: 901
  issue: 11
  year: 2022
  ident: 1466_CR128
  publication-title: Trends Cancer.
  doi: 10.1016/j.trecan.2022.07.002
– ident: 1466_CR223
  doi: 10.3390/antiox10030485
– volume: 10
  start-page: 598107
  year: 2020
  ident: 1466_CR121
  publication-title: Front Oncol.
  doi: 10.3389/fonc.2020.598107
– volume: 194
  start-page: 3236
  issue: 7
  year: 2015
  ident: 1466_CR44
  publication-title: J Immunol (Baltimore, Md : 1950).
  doi: 10.4049/jimmunol.1402764
– volume: 59
  start-page: 898
  issue: 3
  year: 2014
  ident: 1466_CR158
  publication-title: Hepatology (Baltimore, Md).
  doi: 10.1002/hep.26592
– volume: 85
  start-page: 104299
  year: 2022
  ident: 1466_CR211
  publication-title: EBioMedicine.
  doi: 10.1016/j.ebiom.2022.104299
– volume: 12
  start-page: 682736
  year: 2021
  ident: 1466_CR146
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2021.682736
– volume: 61
  start-page: 2
  year: 2021
  ident: 1466_CR209
  publication-title: Cytokine Growth Factor Rev.
  doi: 10.1016/j.cytogfr.2021.06.002
– volume: 200
  start-page: 1016
  issue: 3
  year: 2018
  ident: 1466_CR34
  publication-title: J Immunol.
  doi: 10.4049/jimmunol.1701177
– volume: 14
  start-page: 1130423
  year: 2023
  ident: 1466_CR136
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2023.1130423
– volume: 155
  start-page: 1971
  issue: 6
  year: 2018
  ident: 1466_CR151
  publication-title: Gastroenterology.
  doi: 10.1053/j.gastro.2018.09.010
– volume: 59
  start-page: 1
  year: 2019
  ident: 1466_CR17
  publication-title: Curr Opin Cell Biol.
  doi: 10.1016/j.ceb.2019.02.002
– volume: 243
  start-page: 109
  issue: 1
  year: 2011
  ident: 1466_CR41
  publication-title: Immunol Rev.
  doi: 10.1111/j.1600-065X.2011.01053.x
– volume: 13
  start-page: e13076
  issue: 4
  year: 2021
  ident: 1466_CR192
  publication-title: EMBO Mol Med.
  doi: 10.15252/emmm.202013076
– volume: 13
  start-page: 907209
  year: 2022
  ident: 1466_CR226
  publication-title: Front Pharmacol.
  doi: 10.3389/fphar.2022.907209
– ident: 1466_CR84
  doi: 10.1172/jci.insight.159430
– volume: 18
  start-page: 261
  issue: 5
  year: 2021
  ident: 1466_CR203
  publication-title: Nat Rev Clin Oncol.
  doi: 10.1038/s41571-020-00459-9
– volume: 10
  start-page: 26
  issue: 1
  year: 2020
  ident: 1466_CR9
  publication-title: Cancer Discov.
  doi: 10.1158/2159-8290.CD-19-0761
– volume: 13
  start-page: e1006383
  issue: 6
  year: 2017
  ident: 1466_CR80
  publication-title: PLoS Pathog.
  doi: 10.1371/journal.ppat.1006383
– ident: 1466_CR111
  doi: 10.1172/JCI148852
– volume: 12
  start-page: e11002
  issue: 4
  year: 2020
  ident: 1466_CR135
  publication-title: EMBO Mol Med.
  doi: 10.15252/emmm.201911002
– volume: 179
  start-page: 277
  issue: 2
  year: 2015
  ident: 1466_CR24
  publication-title: Clin Exp Immunol.
  doi: 10.1111/cei.12457
– volume: 10
  start-page: 474
  year: 2019
  ident: 1466_CR50
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2019.00474
– volume: 5
  start-page: 916
  issue: 1
  year: 2022
  ident: 1466_CR83
  publication-title: Commun Biol.
  doi: 10.1038/s42003-022-03868-x
– volume: 73
  start-page: 2527
  issue: 6
  year: 2021
  ident: 1466_CR162
  publication-title: Hepatology (Baltimore, Md).
  doi: 10.1002/hep.31752
– volume: 418
  start-page: 115495
  year: 2021
  ident: 1466_CR55
  publication-title: Toxicol Appl Pharmacol.
  doi: 10.1016/j.taap.2021.115495
– volume: 8
  start-page: e2579
  issue: 2
  year: 2017
  ident: 1466_CR236
  publication-title: Cell Death Dis.
  doi: 10.1038/cddis.2016.460
– volume: 6
  start-page: e0033621
  issue: 3
  year: 2021
  ident: 1466_CR4
  publication-title: mSystems.
  doi: 10.1128/mSystems.00336-21
– volume: 535
  start-page: 153
  issue: 7610
  year: 2016
  ident: 1466_CR60
  publication-title: Nature.
  doi: 10.1038/nature18629
– volume: 39
  start-page: 6378
  issue: 32
  year: 2019
  ident: 1466_CR91
  publication-title: J Neurosci.
  doi: 10.1523/JNEUROSCI.0774-19.2019
– volume: 128
  start-page: 2960
  issue: 25
  year: 2016
  ident: 1466_CR196
  publication-title: Blood.
  doi: 10.1182/blood-2016-07-730556
– volume: 24
  start-page: 101215
  year: 2019
  ident: 1466_CR82
  publication-title: Redox Biol.
  doi: 10.1016/j.redox.2019.101215
– volume: 391
  start-page: 1842
  issue: 10132
  year: 2018
  ident: 1466_CR132
  publication-title: Lancet.
  doi: 10.1016/S0140-6736(18)30312-X
– volume: 15
  start-page: e1008079
  issue: 10
  year: 2019
  ident: 1466_CR48
  publication-title: PLoS Pathog.
  doi: 10.1371/journal.ppat.1008079
– volume: 210
  start-page: 973
  issue: 6
  year: 2015
  ident: 1466_CR126
  publication-title: J Cell Biol.
  doi: 10.1083/jcb.201503023
– volume: 11
  start-page: e1005019
  issue: 7
  year: 2015
  ident: 1466_CR51
  publication-title: PLoS Pathog.
  doi: 10.1371/journal.ppat.1005019
– volume: 119
  start-page: e2123227119
  issue: 27
  year: 2022
  ident: 1466_CR201
  publication-title: Proc Natl Acad Sci U S A.
  doi: 10.1073/pnas.2123227119
– volume: 11
  start-page: 5077
  issue: 1
  year: 2020
  ident: 1466_CR131
  publication-title: Nat Commun.
  doi: 10.1038/s41467-020-18916-5
– volume: 164
  start-page: 896
  issue: 5
  year: 2016
  ident: 1466_CR103
  publication-title: Cell.
  doi: 10.1016/j.cell.2015.12.057
– volume: 8
  start-page: 42808
  issue: 26
  year: 2017
  ident: 1466_CR189
  publication-title: Oncotarget.
  doi: 10.18632/oncotarget.17057
– volume: 339
  start-page: 975
  issue: 6122
  year: 2013
  ident: 1466_CR31
  publication-title: Science (New York, NY).
  doi: 10.1126/science.1230751
– volume: 10
  start-page: 2536
  year: 2019
  ident: 1466_CR123
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2019.02536
– volume: 79
  start-page: 2505
  issue: 11
  year: 2022
  ident: 1466_CR140
  publication-title: Hypertension (Dallas, Tex : 1979).
  doi: 10.1161/HYPERTENSIONAHA.122.20004
– volume: 18
  start-page: 495
  issue: 5
  year: 2017
  ident: 1466_CR107
  publication-title: Obes Rev.
  doi: 10.1111/obr.12520
– volume: 51
  start-page: 27
  issue: 1
  year: 2019
  ident: 1466_CR130
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2019.06.025
– volume: 154
  start-page: 113680
  year: 2022
  ident: 1466_CR183
  publication-title: Biomed Pharmacother = Biomed Pharmacother.
  doi: 10.1016/j.biopha.2022.113680
– volume: 9
  start-page: 613
  issue: 1
  year: 2018
  ident: 1466_CR119
  publication-title: Nat Commun.
  doi: 10.1038/s41467-018-02936-3
– volume: 20
  start-page: 58
  issue: 1
  year: 2023
  ident: 1466_CR45
  publication-title: J Neuroinflammation.
  doi: 10.1186/s12974-023-02737-6
– ident: 1466_CR145
  doi: 10.1016/j.ijrobp.2023.06.221
– volume: 19
  start-page: 136
  issue: 1
  year: 2020
  ident: 1466_CR11
  publication-title: Mol Cancer.
  doi: 10.1186/s12943-020-01247-w
– volume: 62
  start-page: 102691
  year: 2023
  ident: 1466_CR87
  publication-title: Redox Biol.
  doi: 10.1016/j.redox.2023.102691
– volume: 10
  start-page: 1094
  issue: 5
  year: 2019
  ident: 1466_CR157
  publication-title: Aging Dis.
  doi: 10.14336/AD.2019.0116
– volume: 116
  start-page: 7941
  issue: 16
  year: 2019
  ident: 1466_CR16
  publication-title: Proc Natl Acad Sci U S A.
  doi: 10.1073/pnas.1818281116
– volume: 16
  start-page: e1008294
  issue: 3
  year: 2020
  ident: 1466_CR115
  publication-title: PLoS Pathog.
  doi: 10.1371/journal.ppat.1008294
– volume: 10
  start-page: 2274
  year: 2019
  ident: 1466_CR15
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2019.02274
– volume: 30
  start-page: 249
  issue: 2
  year: 2005
  ident: 1466_CR190
  publication-title: Mol Cell Neurosci.
  doi: 10.1016/j.mcn.2005.07.017
– volume: 73
  start-page: 394
  issue: 2
  year: 2020
  ident: 1466_CR156
  publication-title: J Hepatol.
  doi: 10.1016/j.jhep.2020.04.001
– volume: 29
  start-page: 1261
  issue: 5
  year: 2019
  ident: 1466_CR168
  publication-title: Cell Rep.
  doi: 10.1016/j.celrep.2019.09.050
– volume: 2022
  start-page: 1392896
  year: 2022
  ident: 1466_CR224
  publication-title: Oxidative Med Cell Longev.
  doi: 10.1155/2022/1392896
– volume: 4
  start-page: 440
  year: 2013
  ident: 1466_CR37
  publication-title: Front Immunol.
  doi: 10.3389/fimmu.2013.00440
– volume: 11
  start-page: 776
  issue: 9
  year: 2020
  ident: 1466_CR30
  publication-title: Cell Death Dis.
  doi: 10.1038/s41419-020-02985-x
– volume: 204
  start-page: 2514
  issue: 9
  year: 2020
  ident: 1466_CR23
  publication-title: J Immunol (Baltimore, Md : 1950).
  doi: 10.4049/jimmunol.1901508
– volume: 564
  start-page: 439
  issue: 7736
  year: 2018
  ident: 1466_CR12
  publication-title: Nature.
  doi: 10.1038/s41586-018-0705-y
– volume: 46
  start-page: 621
  issue: 4
  year: 2017
  ident: 1466_CR73
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2017.03.020
– volume: 126
  start-page: 1905
  issue: Pt 9
  year: 2013
  ident: 1466_CR85
  publication-title: J Cell Sci.
  doi: 10.1242/jcs.091181
– volume: 196
  start-page: 3191
  issue: 7
  year: 2016
  ident: 1466_CR32
  publication-title: J Immunol (Baltimore, Md : 1950).
  doi: 10.4049/jimmunol.1502538
– volume: 12
  start-page: 140
  year: 2012
  ident: 1466_CR88
  publication-title: BMC Evol Biol.
  doi: 10.1186/1471-2148-12-140
– volume: 15
  start-page: 760
  issue: 12
  year: 2015
  ident: 1466_CR238
  publication-title: Nat Rev Immunol.
  doi: 10.1038/nri3921
– volume: 214
  start-page: 3611
  issue: 12
  year: 2017
  ident: 1466_CR127
  publication-title: J Exp Med.
  doi: 10.1084/jem.20171749
– volume: 18
  start-page: 2752
  issue: 11
  year: 2017
  ident: 1466_CR230
  publication-title: Cell Rep.
  doi: 10.1016/j.celrep.2017.02.057
– volume: 3
  start-page: 1452
  issue: 12
  year: 2022
  ident: 1466_CR10
  publication-title: Nat Cancer.
  doi: 10.1038/s43018-022-00468-w
– volume: 27
  start-page: 2009
  issue: 7
  year: 2016
  ident: 1466_CR167
  publication-title: J Am Soc Nephrol : JASN.
  doi: 10.1681/ASN.2015040376
– volume: 135
  start-page: e202303010
  year: 2023
  ident: 1466_CR233
  publication-title: Angewandte Chemie (International ed in English).
  doi: 10.1002/anie.202303010
– volume: 43
  start-page: 751
  issue: 4
  year: 2015
  ident: 1466_CR206
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2015.08.013
– volume: 535
  start-page: 111
  issue: 7610
  year: 2016
  ident: 1466_CR59
  publication-title: Nature.
  doi: 10.1038/nature18590
– volume: 45
  start-page: 255
  issue: 2
  year: 2016
  ident: 1466_CR35
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2016.06.015
– volume: 34
  start-page: 533
  issue: 4
  year: 2022
  ident: 1466_CR109
  publication-title: Cell Metab.
  doi: 10.1016/j.cmet.2022.02.016
– ident: 1466_CR56
  doi: 10.15252/embj.201899347
– volume: 12
  start-page: 35
  issue: 1
  year: 2019
  ident: 1466_CR204
  publication-title: J Hematol Oncol.
  doi: 10.1186/s13045-019-0721-x
– volume: 171
  start-page: 1110
  issue: 5
  year: 2017
  ident: 1466_CR79
  publication-title: Cell.
  doi: 10.1016/j.cell.2017.09.039
– volume: 216
  start-page: 867
  issue: 4
  year: 2019
  ident: 1466_CR98
  publication-title: J Exp Med.
  doi: 10.1084/jem.20182192
– ident: 1466_CR53
  doi: 10.1128/mBio.01553-16
– volume: 46
  start-page: 393
  issue: 3
  year: 2017
  ident: 1466_CR57
  publication-title: Immunity.
  doi: 10.1016/j.immuni.2017.02.011
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Snippet Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection,...
Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection, chronic...
Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection,...
BackgroundIntracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial infection,...
Abstract Background Intracellular DNA-sensing pathway cGAS-STING, inflammasomes and pyroptosis act as critical natural immune signaling axes for microbial...
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SubjectTerms Analysis
Autoimmune diseases
Bacterial infections
Biomedical and Life Sciences
Caspase 1
Cell Biology
Cell membranes
Cellular stress response
cGAS-STING
Chronic infection
Coronaviruses
Crosstalk network
Cytokines
Cytokines and Growth Factors
Dendritic cells
Development and progression
Diseases
Health aspects
Herpes viruses
Homeostasis
Immune response
Immunity, Innate
Infection
Infections
Inflammasome
Inflammasomes
Inflammation
Inflammatory diseases
Innate immunity
Interferon gamma
Kinases
Life Sciences
Membrane permeability
Mitochondrial DNA
Mitochondrial permeability transition pore
Nucleotidyltransferases
Pathogens
Permeability
Phosphorylation
Protein-Ligand Interactions
Proteins
Pyroptosis
Radiation
Receptors
Regulatory proteins
Review
Sensors
Signal transduction
Vaccines
Zika virus
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Title cGAS-STING, inflammasomes and pyroptosis: an overview of crosstalk mechanism of activation and regulation
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