The role of mesenchymal stem cells in hematopoietic stem cell transplantation: prevention and treatment of graft-versus-host disease

Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both pro...

Full description

Saved in:
Bibliographic Details
Published in:Stem cell research & therapy Vol. 10; no. 1; pp. 182 - 13
Main Authors: Zhao, Lu, Chen, Shanquan, Yang, Panxin, Cao, Hongcui, Li, Lanjuan
Format: Journal Article
Language:English
Published: London BioMed Central 21.06.2019
BioMed Central Ltd
BMC
Subjects:
ISSN:1757-6512, 1757-6512
Online Access:Get full text
Tags: Add Tag
No Tags, Be the first to tag this record!
Abstract Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention. Methods We systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest. Results Ultimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death. Conclusions MSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings.
AbstractList The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention. We systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest. Ultimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death. MSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings.
Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention. Methods We systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest. Results Ultimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death. Conclusions MSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings. Keywords: Mesenchymal stem cells, Stem cell transplantation, Graft-versus-host disease, Meta-analysis
Abstract Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention. Methods We systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest. Results Ultimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death. Conclusions MSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings.
Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention. Methods We systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest. Results Ultimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death. Conclusions MSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings.
The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention. We systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest. Ultimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death. MSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings.
The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention.BACKGROUNDThe use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host disease (aGVHD and cGVHD, respectively), in which immune cells from the donor attack healthy recipient tissues. GVHD presents both prophylactic and therapeutic challenges, and overall survival is poor. Mesenchymal stem cells (MSCs) show considerable promise in the treatment of GVHD because of their potential immunomodulatory activity. Multiple studies have been performed to explore the possible benefit of MSCs in GVHD, but the results of these studies are sometimes conflicting. Therefore, we performed a systematic review and meta-analysis to estimate the effect of MSC infusion on GVHD treatment and prevention.We systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest.METHODSWe systematically searched the MEDLINE (PubMed), Cochrane Library, EMBASE, ClinicalTrials.gov, and SinoMed CBM databases to identify studies published before February 2018 involving patients with hematologic malignancies undergoing HSCT and receiving MSC-based or conventional therapy. We included studies if they reported on the outcomes of interest.Ultimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death.RESULTSUltimately, 10 studies were selected from among 413 candidates. According to our meta-analyses, compared with conventional treatment, MSC therapy demonstrated substantial improvements in terms of complete response (CR) and overall survival for cGVHD. However, MSC therapy did not show substantial improvements in terms of engraftment, the incidence of aGVHD, relapse, death, death due to relapse, or death due to infection. Subgroup analyses showed that MSCs derived from the umbilical cord (U-MSCs) and MSC infusion after HSCT substantially improved engraftment and cGVHD incidence, whereas MSCs derived from bone marrow (B-MSCs) and MSC infusion before HSCT shows no improvement. In addition, B-MSCs and MSC infusion before HSCT tend to prolong engraftment time, as well as increase the rates of relapse and death.MSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings.CONCLUSIONSMSC infusion can reduce cGVHD but not aGVHD incidence and showed a positive effect in patients who already had aGVHD. For GVHD prevention, the use of U-MSCs and MSC infusion after HSCT were optimal for reducing cGVHD incidence and promoting engraftment, and might help decrease the incidence rate of relapse and death. However, B-MSCs and MSC infusion before HSCT may be harmful to patients and thus require serious consideration. A lack of robust evidence, owing to the small number of studies and small sample sizes, indicates a need for further high-quality clinical trials including large numbers of patients to validate our findings.
ArticleNumber 182
Audience Academic
Author Zhao, Lu
Chen, Shanquan
Li, Lanjuan
Cao, Hongcui
Yang, Panxin
Author_xml – sequence: 1
  givenname: Lu
  surname: Zhao
  fullname: Zhao, Lu
  organization: State Key Laboratory for Diagnosis and Treatment of Infectious Diseases, the First Affiliated Hospital, College of Medicine, Zhejiang University, Collaborative Innovation Center for Diagnosis and Treatment of Infectious Diseases
– sequence: 2
  givenname: Shanquan
  surname: Chen
  fullname: Chen, Shanquan
  organization: The School of Clinical Medicine, University of Cambridge
– sequence: 3
  givenname: Panxin
  surname: Yang
  fullname: Yang, Panxin
  organization: State Key Laboratory for Diagnosis and Treatment of Infectious Diseases, the First Affiliated Hospital, College of Medicine, Zhejiang University, Collaborative Innovation Center for Diagnosis and Treatment of Infectious Diseases
– sequence: 4
  givenname: Hongcui
  orcidid: 0000-0002-6604-6867
  surname: Cao
  fullname: Cao, Hongcui
  email: hccao@zju.edu.cn
  organization: State Key Laboratory for Diagnosis and Treatment of Infectious Diseases, the First Affiliated Hospital, College of Medicine, Zhejiang University, Collaborative Innovation Center for Diagnosis and Treatment of Infectious Diseases
– sequence: 5
  givenname: Lanjuan
  surname: Li
  fullname: Li, Lanjuan
  organization: State Key Laboratory for Diagnosis and Treatment of Infectious Diseases, the First Affiliated Hospital, College of Medicine, Zhejiang University, Collaborative Innovation Center for Diagnosis and Treatment of Infectious Diseases
BackLink https://www.ncbi.nlm.nih.gov/pubmed/31227011$$D View this record in MEDLINE/PubMed
BookMark eNp9kl2L1DAUhousuOu6P8AbKQiiF12T9CONF8Ky-DGwIOjch9P0dJqhTcYkHdx7f7ipXdcZkW0vesh53jfNyfs0OTHWYJI8p-SS0rp662nOap4RKjI6F-JRckZ5ybOqpOzkoD5NLrzfkvjkOSFV8SQ5zSljnFB6lvxc95g6O2Bqu3REj0b1tyMMqQ84pgqHwafapD2OEOzOagxa_e2lwYHxuwFMgKCteZfuHO7RzHUKpo19hDDGhdl-46AL2R6dn3zWWx_SVnsEj8-Sxx0MHi_uvufJ-uOH9fXn7ObLp9X11U2mOKlC1paccUbyqlNQ00LlDQHF86ZuOTYMKBfAKIquKLuGko51TUEEoIJGoWir_DxZLbatha3cOT2Cu5UWtPy9YN1GgovnG1DmJWEdw1zVWBY1Ys1pkVPRsgKEEKqIXu8Xr93UjNiqeEQHw5HpccfoXm7sXlZlXQs6G7y-M3D2-4Q-yFH7eaZg0E5eMlaUVbzYakZfLugG4q9p09noqGZcXpWCcFoWOYnU5X-o-LY4ahWz0-m4fiR4cySITMAfYQOT93L17esx--qA7RGG0Hs7TPNF-2PwxeFc7gfyJ3ERoAugnPXeYXePUCLnYMsl2DIGW87BliJq-D8apZfAxSPq4UElW5Q-7mI26OTWTs7EkD0g-gX2MQx5
CitedBy_id crossref_primary_10_1186_s13287_021_02181_4
crossref_primary_10_3389_fcell_2020_587866
crossref_primary_10_1016_j_ebiom_2019_08_073
crossref_primary_10_1080_14728222_2021_1992383
crossref_primary_10_3390_biology12060792
crossref_primary_10_1007_s13346_021_00934_5
crossref_primary_10_1016_j_prp_2024_155354
crossref_primary_10_1186_s40164_023_00426_x
crossref_primary_10_1093_stcltm_szac009
crossref_primary_10_1155_2023_9672658
crossref_primary_10_1016_j_omtm_2025_101527
crossref_primary_10_1183_16000617_0042_2021
crossref_primary_10_1016_j_tice_2022_101998
crossref_primary_10_3390_ijms22158121
crossref_primary_10_1007_s12015_021_10164_4
crossref_primary_10_3389_fimmu_2021_659621
crossref_primary_10_1097_SHK_0000000000001644
crossref_primary_10_3390_ijms241411530
crossref_primary_10_1186_s13287_021_02170_7
crossref_primary_10_1089_cell_2021_0043
crossref_primary_10_1186_s12935_021_02300_4
crossref_primary_10_3389_fcell_2021_663316
crossref_primary_10_3390_ijms221910474
crossref_primary_10_1080_14728222_2024_2405091
crossref_primary_10_1007_s12015_020_10086_7
crossref_primary_10_1186_s13287_021_02159_2
crossref_primary_10_3389_fphar_2022_1027961
crossref_primary_10_1016_j_transci_2024_104051
crossref_primary_10_3389_fimmu_2021_728190
crossref_primary_10_3390_biomedicines10061391
crossref_primary_10_3390_genes12122008
crossref_primary_10_1016_j_ejphar_2024_176996
crossref_primary_10_1186_s13287_025_04303_8
crossref_primary_10_3390_ijms26020847
crossref_primary_10_1089_scd_2021_0352
crossref_primary_10_1155_2022_1052166
crossref_primary_10_1080_08830185_2023_2252007
crossref_primary_10_1186_s13287_022_03112_7
crossref_primary_10_1016_j_aanat_2023_152131
crossref_primary_10_1186_s11658_023_00485_2
crossref_primary_10_2217_pme_2021_0111
crossref_primary_10_3390_nu15163558
crossref_primary_10_1042_BSR20211986
crossref_primary_10_1007_s13337_021_00743_x
crossref_primary_10_3390_ijms24098126
crossref_primary_10_3389_fneur_2021_621392
crossref_primary_10_1038_s41598_020_60524_2
crossref_primary_10_1111_nep_70054
crossref_primary_10_3389_fimmu_2022_1045168
crossref_primary_10_1080_08923973_2024_2384913
crossref_primary_10_3389_fcell_2021_570179
crossref_primary_10_1186_s13287_022_02838_8
crossref_primary_10_56875_2589_0646_1111
crossref_primary_10_1002_sctm_20_0345
crossref_primary_10_3390_ijms22179231
crossref_primary_10_3389_fimmu_2020_583564
crossref_primary_10_3389_fcell_2021_749822
crossref_primary_10_3390_vaccines8010062
crossref_primary_10_1080_17474086_2020_1738214
crossref_primary_10_1016_j_intimp_2024_112689
crossref_primary_10_1186_s13287_021_02304_x
crossref_primary_10_1186_s13287_025_04321_6
crossref_primary_10_3390_ijms24032987
crossref_primary_10_1016_j_bbcan_2024_189189
crossref_primary_10_1073_pnas_2404210121
crossref_primary_10_3390_ijms26125837
crossref_primary_10_1016_j_jcyt_2021_07_009
crossref_primary_10_3389_fimmu_2023_1241068
crossref_primary_10_3389_fimmu_2021_626755
crossref_primary_10_1182_blood_2019000951
crossref_primary_10_1016_j_arcmed_2023_102853
crossref_primary_10_3389_fimmu_2020_00792
crossref_primary_10_1111_apm_70012
crossref_primary_10_7717_peerj_15388
crossref_primary_10_3389_fcell_2022_951764
crossref_primary_10_1016_j_trim_2024_102130
crossref_primary_10_3389_fcell_2022_873603
crossref_primary_10_3390_ijms22179531
crossref_primary_10_1016_j_intimp_2021_107669
crossref_primary_10_1186_s13287_024_03824_y
crossref_primary_10_1089_scd_2020_0191
crossref_primary_10_1186_s13287_024_03949_0
crossref_primary_10_1016_j_jtct_2022_10_030
crossref_primary_10_3389_fbioe_2020_575057
crossref_primary_10_4252_wjSC_v13_i2_177
crossref_primary_10_1016_j_intimp_2025_115018
crossref_primary_10_1186_s12951_025_03297_y
crossref_primary_10_1007_s00784_023_05366_8
crossref_primary_10_3389_fimmu_2021_609544
crossref_primary_10_1177_20406207221134409
crossref_primary_10_3390_cancers16162892
crossref_primary_10_3390_ijms21030708
crossref_primary_10_1186_s12967_025_06623_y
crossref_primary_10_1016_j_jds_2024_11_029
crossref_primary_10_1007_s10238_021_00701_3
crossref_primary_10_3389_fcell_2024_1380785
crossref_primary_10_3389_fphar_2022_1013740
crossref_primary_10_4274_tjh_galenos_2022_2022_0084
crossref_primary_10_1002_JLB_6MA0921_752RR
crossref_primary_10_2147_ITT_S457366
crossref_primary_10_1016_j_bulcan_2022_11_001
crossref_primary_10_1186_s13063_023_07305_0
crossref_primary_10_3390_cells10040961
crossref_primary_10_3389_fcell_2020_599276
crossref_primary_10_1016_j_jcyt_2020_08_012
Cites_doi 10.1016/j.bbmt.2014.09.030
10.1016/j.exphem.2005.01.012
10.1080/1744666X.2016.1208086
10.1182/blood.V76.8.1464.1464
10.1016/j.jcyt.2012.10.002
10.1134/S0006297914120104
10.3727/096368912X655217
10.1182/blood-2005-07-2657
10.1016/j.bbmt.2009.12.426
10.1073/pnas.0603824103
10.1038/sj.leu.2405090
10.1182/blood-2005-07-2775
10.1089/scd.2010.0447
10.1111/j.1365-2141.2005.05409.x
10.1016/j.bbmt.2009.12.057
10.1038/leu.2015.89
10.1016/S2352-3026(15)00224-0
10.1002/eji.200636416
10.1016/S0301-472X(01)00769-X
10.1038/nrclinonc.2015.234
10.1097/00062752-200011000-00007
10.1155/2012/968213
10.1007/s12185-012-1218-3
10.1016/S0140-6736(09)60237-3
10.1097/TP.0b013e31825940a4
10.2174/1389450116666150722140633
10.1186/1756-8722-5-19
10.1182/blood.V116.21.1256.1256
10.1038/emm.2013.2
10.1634/stemcells.2008-0456
10.1016/S0140-6736(04)16104-7
10.1136/bmj.315.7109.629
10.1016/j.blre.2005.11.002
10.1200/JCO.2015.65.3642
10.1056/NEJMoa1203517
10.3727/096368912X663533
10.1007/s11892-016-0734-6
10.1186/1756-8722-7-14
10.1097/01.tp.0000244572.24780.54
10.1634/stemcells.2007-0381
10.1200/JCO.2010.33.7212
10.1016/j.bbmt.2012.04.005
10.1146/annurev.immunol.25.022106.141606
10.1182/blood-2004-11-4557
ContentType Journal Article
Copyright The Author(s). 2019
COPYRIGHT 2019 BioMed Central Ltd.
Copyright_xml – notice: The Author(s). 2019
– notice: COPYRIGHT 2019 BioMed Central Ltd.
DBID C6C
AAYXX
CITATION
NPM
ISR
7X8
5PM
DOA
DOI 10.1186/s13287-019-1287-9
DatabaseName Springer Nature OA Free Journals
CrossRef
PubMed
Gale In Context: Science
MEDLINE - Academic
PubMed Central (Full Participant titles)
DOAJ Directory of Open Access Journals
DatabaseTitle CrossRef
PubMed
MEDLINE - Academic
DatabaseTitleList





PubMed
MEDLINE - Academic
Database_xml – sequence: 1
  dbid: DOA
  name: DOAJ Directory of Open Access Journals
  url: https://www.doaj.org/
  sourceTypes: Open Website
– sequence: 2
  dbid: NPM
  name: PubMed
  url: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 3
  dbid: 7X8
  name: MEDLINE - Academic
  url: https://search.proquest.com/medline
  sourceTypes: Aggregation Database
DeliveryMethod fulltext_linktorsrc
Discipline Biology
EISSN 1757-6512
EndPage 13
ExternalDocumentID oai_doaj_org_article_3502f2e3c8e548ee8714319d24a999c4
PMC6588914
A590715430
31227011
10_1186_s13287_019_1287_9
Genre Research Support, Non-U.S. Gov't
Journal Article
Review
GrantInformation_xml – fundername: National Key Research and Development Program of China
  grantid: 2016YFA0101001
– fundername: National Natural Science Foundation of China
  grantid: 81620108028
  funderid: http://dx.doi.org/10.13039/501100001809
– fundername: ;
  grantid: 81620108028
– fundername: ;
  grantid: 2016YFA0101001
GroupedDBID ---
0R~
53G
5VS
7X7
88E
8FE
8FH
8FI
8FJ
AAFWJ
AAJSJ
AASML
ABDBF
ABUWG
ACGFS
ACIHN
ACJQM
ACPRK
ACUHS
ADBBV
ADUKV
AEAQA
AENEX
AFKRA
AFPKN
AHBYD
AHMBA
AHYZX
ALMA_UNASSIGNED_HOLDINGS
AMKLP
AMTXH
AOIAM
AOIJS
BAPOH
BAWUL
BBNVY
BCNDV
BENPR
BFQNJ
BHPHI
BMC
BPHCQ
BVXVI
C6C
CCPQU
DIK
E3Z
EBD
EBLON
EBS
EJD
EMOBN
ESX
F5P
FYUFA
GROUPED_DOAJ
GX1
HCIFZ
HMCUK
HYE
IAO
IEA
IHR
IHW
INH
INR
ISR
ITC
KQ8
LK8
M1P
M7P
M~E
O5R
O5S
OK1
P2P
PGMZT
PHGZM
PHGZT
PIMPY
PJZUB
PPXIY
PQGLB
PQQKQ
PROAC
PSQYO
PUEGO
RBZ
ROL
RPM
RSV
SBL
SOJ
SV3
TUS
UKHRP
AAYXX
AFFHD
CITATION
-56
-5G
-BR
3V.
ACRMQ
ADINQ
ALIPV
C24
NPM
7X8
5PM
ID FETCH-LOGICAL-c706t-d57272036fca814c3b0ac73b8d7eb2a179a21e9f45fb10f2fb409aecabce9d63
IEDL.DBID RSV
ISICitedReferencesCount 116
ISICitedReferencesURI http://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=Summon&SrcAuth=ProQuest&DestLinkType=CitingArticles&DestApp=WOS_CPL&KeyUT=000472532600001&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
ISSN 1757-6512
IngestDate Tue Oct 14 19:08:24 EDT 2025
Tue Nov 04 01:57:23 EST 2025
Wed Oct 01 11:55:42 EDT 2025
Tue Nov 11 10:07:24 EST 2025
Tue Nov 04 18:02:20 EST 2025
Thu Nov 13 15:32:10 EST 2025
Thu May 22 20:58:48 EDT 2025
Thu Jan 02 22:59:10 EST 2025
Sat Nov 29 01:46:14 EST 2025
Tue Nov 18 21:13:26 EST 2025
Sat Sep 06 07:28:30 EDT 2025
IsDoiOpenAccess true
IsOpenAccess true
IsPeerReviewed true
IsScholarly true
Issue 1
Keywords Graft-versus-host disease
Stem cell transplantation
Mesenchymal stem cells
Meta-analysis
Language English
License Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
LinkModel DirectLink
MergedId FETCHMERGED-LOGICAL-c706t-d57272036fca814c3b0ac73b8d7eb2a179a21e9f45fb10f2fb409aecabce9d63
Notes ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
ObjectType-Review-3
content type line 23
ORCID 0000-0002-6604-6867
OpenAccessLink https://link.springer.com/10.1186/s13287-019-1287-9
PMID 31227011
PQID 2245612864
PQPubID 23479
PageCount 13
ParticipantIDs doaj_primary_oai_doaj_org_article_3502f2e3c8e548ee8714319d24a999c4
pubmedcentral_primary_oai_pubmedcentral_nih_gov_6588914
proquest_miscellaneous_2245612864
gale_infotracmisc_A590715430
gale_infotracacademiconefile_A590715430
gale_incontextgauss_ISR_A590715430
gale_healthsolutions_A590715430
pubmed_primary_31227011
crossref_primary_10_1186_s13287_019_1287_9
crossref_citationtrail_10_1186_s13287_019_1287_9
springer_journals_10_1186_s13287_019_1287_9
PublicationCentury 2000
PublicationDate 2019-06-21
PublicationDateYYYYMMDD 2019-06-21
PublicationDate_xml – month: 06
  year: 2019
  text: 2019-06-21
  day: 21
PublicationDecade 2010
PublicationPlace London
PublicationPlace_xml – name: London
– name: England
PublicationTitle Stem cell research & therapy
PublicationTitleAbbrev Stem Cell Res Ther
PublicationTitleAlternate Stem Cell Res Ther
PublicationYear 2019
Publisher BioMed Central
BioMed Central Ltd
BMC
Publisher_xml – name: BioMed Central
– name: BioMed Central Ltd
– name: BMC
References M Secco (1287_CR47) 2008; 26
A Fierabracci (1287_CR12) 2016; 17
PJ Martin (1287_CR5) 2012; 18
A Uccelli (1287_CR11) 2006; 36
S Hashmi (1287_CR2) 2016; 3
L Gao (1287_CR9) 2016; 34
F Locatelli (1287_CR49) 2017; 13
JY Cahn (1287_CR10) 2005; 106
IN Shipounova (1287_CR34) 2014; 79
A Bartholomew (1287_CR14) 2002; 30
A Corcione (1287_CR16) 2006; 107
K Liu (1287_CR25) 2011; 20
K Le Blanc (1287_CR22) 2004; 363
EJ Kim (1287_CR43) 2013; 45
PJ Martin (1287_CR8) 1990; 76
1287_CR30
1287_CR39
S Wang (1287_CR21) 2012; 5
JR Wingard (1287_CR52) 2011; 29
1287_CR35
J Xiang (1287_CR33) 2017; 21
SM Devine (1287_CR20) 2000; 7
PJ Martin (1287_CR32) 2010; 16
F Dazzi (1287_CR19) 2006; 20
H Ning (1287_CR24) 2008; 22
C Anasetti (1287_CR44) 2012; 367
1287_CR29
1287_CR28
M Remberger (1287_CR38) 2012; 96
N Petinaty (1287_CR4) 2010; 116
1287_CR27
1287_CR26
K Zhao (1287_CR37) 2015; 21
MT Van Lint (1287_CR7) 1998; 92
ZY Zhang (1287_CR45) 2009; 27
R Ramasamy (1287_CR17) 2007; 83
Z Sun (1287_CR41) 2014; 7
YY Dan (1287_CR13) 2006; 103
G Kogler (1287_CR42) 2005; 33
LL Lu (1287_CR46) 2006; 91
GM Spaggiari (1287_CR18) 2006; 107
LA Welniak (1287_CR3) 2007; 25
CK Chan (1287_CR48) 2012; 94
K Wu (1287_CR40) 2013; 22
BL Te (1287_CR51) 2015; 29
CG Kanakry (1287_CR1) 2016; 13
JL Ferrara (1287_CR6) 2009; 373
B Puissant (1287_CR15) 2005; 129
M Egger (1287_CR31) 1997; 315
KH Wu (1287_CR36) 2013; 22
J Galipeau (1287_CR23) 2013; 15
TP Griffin (1287_CR50) 2016; 16
References_xml – volume: 21
  start-page: 97
  issue: 1
  year: 2015
  ident: 1287_CR37
  publication-title: Biol Blood Marrow Transplant
  doi: 10.1016/j.bbmt.2014.09.030
– volume: 33
  start-page: 573
  issue: 5
  year: 2005
  ident: 1287_CR42
  publication-title: Exp Hematol
  doi: 10.1016/j.exphem.2005.01.012
– volume: 13
  start-page: 43
  issue: 1
  year: 2017
  ident: 1287_CR49
  publication-title: Expert Rev Clin Immunol
  doi: 10.1080/1744666X.2016.1208086
– volume: 76
  start-page: 1464
  issue: 8
  year: 1990
  ident: 1287_CR8
  publication-title: Blood.
  doi: 10.1182/blood.V76.8.1464.1464
– volume: 15
  start-page: 2
  year: 2013
  ident: 1287_CR23
  publication-title: Cytotherapy.
  doi: 10.1016/j.jcyt.2012.10.002
– volume: 79
  start-page: 1363
  issue: 12
  year: 2014
  ident: 1287_CR34
  publication-title: Biochemistry (Mosc)
  doi: 10.1134/S0006297914120104
– volume: 22
  start-page: 723
  issue: 4
  year: 2013
  ident: 1287_CR40
  publication-title: Cell Transplant
  doi: 10.3727/096368912X655217
– volume: 107
  start-page: 367
  issue: 1
  year: 2006
  ident: 1287_CR16
  publication-title: Blood.
  doi: 10.1182/blood-2005-07-2657
– ident: 1287_CR39
  doi: 10.1016/j.bbmt.2009.12.426
– ident: 1287_CR28
– volume: 103
  start-page: 9912
  issue: 26
  year: 2006
  ident: 1287_CR13
  publication-title: Proc Natl Acad Sci U S A
  doi: 10.1073/pnas.0603824103
– volume: 22
  start-page: 593
  issue: 3
  year: 2008
  ident: 1287_CR24
  publication-title: Leukemia.
  doi: 10.1038/sj.leu.2405090
– ident: 1287_CR30
– volume: 107
  start-page: 1484
  issue: 4
  year: 2006
  ident: 1287_CR18
  publication-title: Blood.
  doi: 10.1182/blood-2005-07-2775
– volume: 20
  start-page: 1679
  issue: 10
  year: 2011
  ident: 1287_CR25
  publication-title: Stem Cells Dev
  doi: 10.1089/scd.2010.0447
– volume: 129
  start-page: 118
  issue: 1
  year: 2005
  ident: 1287_CR15
  publication-title: Br J Haematol
  doi: 10.1111/j.1365-2141.2005.05409.x
– volume: 16
  start-page: S169
  issue: 2, Supplement 2
  year: 2010
  ident: 1287_CR32
  publication-title: Biol Blood Marrow Transplant
  doi: 10.1016/j.bbmt.2009.12.057
– volume: 29
  start-page: 1839
  issue: 9
  year: 2015
  ident: 1287_CR51
  publication-title: Leukemia.
  doi: 10.1038/leu.2015.89
– volume: 3
  start-page: e45
  issue: 1
  year: 2016
  ident: 1287_CR2
  publication-title: Lancet Haematol
  doi: 10.1016/S2352-3026(15)00224-0
– volume: 36
  start-page: 2566
  issue: 10
  year: 2006
  ident: 1287_CR11
  publication-title: Eur J Immunol
  doi: 10.1002/eji.200636416
– volume: 30
  start-page: 42
  issue: 1
  year: 2002
  ident: 1287_CR14
  publication-title: Exp Hematol
  doi: 10.1016/S0301-472X(01)00769-X
– volume: 13
  start-page: 132
  issue: 2
  year: 2016
  ident: 1287_CR1
  publication-title: Nat Rev Clin Oncol
  doi: 10.1038/nrclinonc.2015.234
– volume: 21
  start-page: 4679
  issue: 29
  year: 2017
  ident: 1287_CR33
  publication-title: Chin J Tissue Eng Res
– volume: 7
  start-page: 358
  issue: 6
  year: 2000
  ident: 1287_CR20
  publication-title: Curr Opin Hematol
  doi: 10.1097/00062752-200011000-00007
– ident: 1287_CR35
  doi: 10.1155/2012/968213
– volume: 96
  start-page: 822
  issue: 6
  year: 2012
  ident: 1287_CR38
  publication-title: Int J Hematol
  doi: 10.1007/s12185-012-1218-3
– ident: 1287_CR27
– volume: 373
  start-page: 1550
  issue: 9674
  year: 2009
  ident: 1287_CR6
  publication-title: Lancet.
  doi: 10.1016/S0140-6736(09)60237-3
– volume: 94
  start-page: 132
  issue: 2
  year: 2012
  ident: 1287_CR48
  publication-title: Transplantation.
  doi: 10.1097/TP.0b013e31825940a4
– volume: 17
  start-page: 229
  issue: 2
  year: 2016
  ident: 1287_CR12
  publication-title: Curr Drug Targets
  doi: 10.2174/1389450116666150722140633
– volume: 5
  start-page: 19
  year: 2012
  ident: 1287_CR21
  publication-title: J Hematol Oncol
  doi: 10.1186/1756-8722-5-19
– volume: 116
  start-page: 538
  issue: 21
  year: 2010
  ident: 1287_CR4
  publication-title: Blood
  doi: 10.1182/blood.V116.21.1256.1256
– volume: 45
  start-page: e2
  year: 2013
  ident: 1287_CR43
  publication-title: Exp Mol Med
  doi: 10.1038/emm.2013.2
– volume: 27
  start-page: 126
  issue: 1
  year: 2009
  ident: 1287_CR45
  publication-title: Stem Cells
  doi: 10.1634/stemcells.2008-0456
– volume: 363
  start-page: 1439
  year: 2004
  ident: 1287_CR22
  publication-title: Lancet.
  doi: 10.1016/S0140-6736(04)16104-7
– volume: 92
  start-page: 2288
  issue: 7
  year: 1998
  ident: 1287_CR7
  publication-title: Blood.
– ident: 1287_CR26
– volume: 315
  start-page: 629
  issue: 7109
  year: 1997
  ident: 1287_CR31
  publication-title: BMJ.
  doi: 10.1136/bmj.315.7109.629
– volume: 20
  start-page: 161
  issue: 3
  year: 2006
  ident: 1287_CR19
  publication-title: Blood Rev
  doi: 10.1016/j.blre.2005.11.002
– volume: 34
  start-page: 2843
  issue: 24
  year: 2016
  ident: 1287_CR9
  publication-title: J Clin Oncol
  doi: 10.1200/JCO.2015.65.3642
– volume: 367
  start-page: 1487
  issue: 16
  year: 2012
  ident: 1287_CR44
  publication-title: N Engl J Med
  doi: 10.1056/NEJMoa1203517
– volume: 22
  start-page: 2041
  issue: 11
  year: 2013
  ident: 1287_CR36
  publication-title: Cell Transplant
  doi: 10.3727/096368912X663533
– volume: 16
  start-page: 42
  issue: 5
  year: 2016
  ident: 1287_CR50
  publication-title: Curr Diab Rep
  doi: 10.1007/s11892-016-0734-6
– volume: 7
  start-page: 14
  year: 2014
  ident: 1287_CR41
  publication-title: J Hematol Oncol
  doi: 10.1186/1756-8722-7-14
– volume: 83
  start-page: 71
  issue: 1
  year: 2007
  ident: 1287_CR17
  publication-title: Transplantation.
  doi: 10.1097/01.tp.0000244572.24780.54
– volume: 26
  start-page: 146
  issue: 1
  year: 2008
  ident: 1287_CR47
  publication-title: Stem Cells
  doi: 10.1634/stemcells.2007-0381
– volume: 29
  start-page: 2230
  issue: 16
  year: 2011
  ident: 1287_CR52
  publication-title: J Clin Oncol
  doi: 10.1200/JCO.2010.33.7212
– volume: 91
  start-page: 1017
  issue: 8
  year: 2006
  ident: 1287_CR46
  publication-title: Haematologica.
– volume: 18
  start-page: 1150
  issue: 8
  year: 2012
  ident: 1287_CR5
  publication-title: Biol Blood Marrow Transplant.
  doi: 10.1016/j.bbmt.2012.04.005
– ident: 1287_CR29
– volume: 25
  start-page: 139
  year: 2007
  ident: 1287_CR3
  publication-title: Annu Rev Immunol
  doi: 10.1146/annurev.immunol.25.022106.141606
– volume: 106
  start-page: 1495
  issue: 4
  year: 2005
  ident: 1287_CR10
  publication-title: Blood.
  doi: 10.1182/blood-2004-11-4557
SSID ssj0000330064
Score 2.5358183
SecondaryResourceType review_article
Snippet Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic...
The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic graft-versus-host...
Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic...
Abstract Background The use and effectiveness of hematopoietic stem cell transplantation (HSCT) are limited by lethal complications, i.e., acute and chronic...
SourceID doaj
pubmedcentral
proquest
gale
pubmed
crossref
springer
SourceType Open Website
Open Access Repository
Aggregation Database
Index Database
Enrichment Source
Publisher
StartPage 182
SubjectTerms Biomedical and Life Sciences
Biomedical Engineering and Bioengineering
Bone marrow transplantation
Care and treatment
Cell Biology
Graft vs. host disease
Graft-versus-host disease
Hematopoietic stem cell transplantation
Life Sciences
Medical research
Mesenchymal stem cells
Meta-analysis
Prevention
Regenerative Medicine/Tissue Engineering
Review
Stem cell transplantation
Stem Cells
SummonAdditionalLinks – databaseName: DOAJ Directory of Open Access Journals
  dbid: DOA
  link: http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwrV3Nb9UwDI_QBBIXxDeFAQEhIYGqtUnbNNwGYoLLhGCH3aI0TbYnbenTax_S7vvDZ6d55XUIuHCrGrdqbCexa_tnQt5obYQGUyKVzDTgoLSw5gpRpbYohWbc4XBoNiEOD-vjY_ltq9UX5oSN8MAj4_Z4mTHHLDe1BePa2hobdueyZYUG28YEJNBMyC1nKuzB4KbDYRvDmHld7fXgdtWYZYno_3AhZwdRwOv_fVfeOpaup0xei5uG4-jgLrkT7Ui6P37_PXLD-vvk1thZ8uIBuQTxU8wcpJ2j51hhZE4vzuEBxG2m-Le-pwtPA2Jrt-wWWMr4a4wOAfL8TI91Sf4DXUakp85T7Vs6pafj609W2g0ppnes-xSLRmiM-jwkRwefjz59SWPDhdSIrBrSthzDspUzus4Lw5sMZMWbuhXggGtYu5rlVrqidE2eOeYa8A61NboxVrYVf0R2fOftE0LBkIKTsQVfqpSFqXjj8tI4Y5kV1vDCJCTbMF-ZCEaOPTHOVHBK6kqN8lIgL4XyUjIh76ZHliMSx9-IP6JEJ0IE0Q43QLVUVC31L9VKyEvUBzVWpE5bgdovJRhmZcGzhLwOFAik4TFT50Sv-159_fF9RvQ2ErkO5mh0LHwATiH21oxyd0YJK93Mhl9tFFPhEKbHedute8UwfA0zr-CbH4-KOk2d54wJ2MUTImYqPOPNfMQvTgPQOFintczhne83yq7iDtf_mfVP_wfrn5HbDJdqVqUs3yU7w2ptn5Ob5uew6Fcvwlq_AoAiV1A
  priority: 102
  providerName: Directory of Open Access Journals
Title The role of mesenchymal stem cells in hematopoietic stem cell transplantation: prevention and treatment of graft-versus-host disease
URI https://link.springer.com/article/10.1186/s13287-019-1287-9
https://www.ncbi.nlm.nih.gov/pubmed/31227011
https://www.proquest.com/docview/2245612864
https://pubmed.ncbi.nlm.nih.gov/PMC6588914
https://doaj.org/article/3502f2e3c8e548ee8714319d24a999c4
Volume 10
WOSCitedRecordID wos000472532600001&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
hasFullText 1
inHoldings 1
isFullTextHit
isPrint
journalDatabaseRights – providerCode: PRVADU
  databaseName: BioMed Central
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: RBZ
  dateStart: 20100101
  isFulltext: true
  titleUrlDefault: https://www.biomedcentral.com/search/
  providerName: BioMedCentral
– providerCode: PRVAON
  databaseName: DOAJ Directory of Open Access Journals
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: DOA
  dateStart: 20100101
  isFulltext: true
  titleUrlDefault: https://www.doaj.org/
  providerName: Directory of Open Access Journals
– providerCode: PRVHPJ
  databaseName: ROAD: Directory of Open Access Scholarly Resources
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: M~E
  dateStart: 20100101
  isFulltext: true
  titleUrlDefault: https://road.issn.org
  providerName: ISSN International Centre
– providerCode: PRVPQU
  databaseName: Biological Science Database
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: M7P
  dateStart: 20150101
  isFulltext: true
  titleUrlDefault: http://search.proquest.com/biologicalscijournals
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: ProQuest Central
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: BENPR
  dateStart: 20150101
  isFulltext: true
  titleUrlDefault: https://www.proquest.com/central
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: ProQuest Health & Medical Collection
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: 7X7
  dateStart: 20150101
  isFulltext: true
  titleUrlDefault: https://search.proquest.com/healthcomplete
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: Publicly Available Content Database
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: PIMPY
  dateStart: 20150101
  isFulltext: true
  titleUrlDefault: http://search.proquest.com/publiccontent
  providerName: ProQuest
– providerCode: PRVAVX
  databaseName: SpringerLINK
  customDbUrl:
  eissn: 1757-6512
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0000330064
  issn: 1757-6512
  databaseCode: RSV
  dateStart: 20100301
  isFulltext: true
  titleUrlDefault: https://link.springer.com/search?facet-content-type=%22Journal%22
  providerName: Springer Nature
link http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwnV3db9MwELfYBtJe-IYVRjEICQkUkTgfdnjb0Cb2QFV1EypPluPYXaUtqZoWae_84dw5biDjQ4KXqoovUXw5n-98d78j5JVSmiswJYKc6QIclBLWXMKzwCQpVyy2OOyaTfDRSEyn-djXcTebbPdNSNJparesRfauAb9JYJokwvfDn3yL7MBuJ7Bfw-T0c3ewEoKHDvusj2D-9s7eHuSg-n9VyD_tSNezJa-FTN1OdHznv-Zwl9z2hic9aCXlHrlhqvvkVtuK8uoB-QbyQjHVkNaWXmJJkj6_uoQbEOiZ4vF-Q-cVdRCv9aKeY-3jjzG6chjpF6otZKre04WHhqorqqqSdvns-PjZUtlVgPkg6ybAKhPqw0QPydnx0dmHj4Hv0BBoHmaroEzbOG5mtRJRouMihI8bF6Lk4LErWOyKRSa3SWqLKLTMFuBOKqNVoU1eZvEjsl3VldkjFCwv2EpLcL7SPNFZXNgo1VYbZrjRcaIHJNx8Mqk9ejk20biQzosRmWx5K4G3Enkr8wF5092yaKE7_kZ8iHLQESLqtrtQL2fSL2IZpyGzzMRaGHD0jBHYPD7KS5YosLN1MiDPUYpkW8La6Q55kOZgyaVJHA7IS0eByBsVpvbM1Lpp5MnppEf02hPZGuaola-UAE4hWFePcr9HCapB94ZfbMRZ4hDm01WmXjeSYbwbZp7BOz9uxbubehwxxkHtDwjvCX6PN_2Ran7ukMnBnBV5BM98uxF_6VVi82fWP_kn6qdkl-H6CbOARftke7Vcm2fkpv66mjfLIdniU-5-xZDsHB6NxpOhO2YZYlLvGK6NTz6NvwydyvgOK6Zi9Q
linkProvider Springer Nature
linkToHtml http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwnV1Zb9QwELaggOCF-wgUahASUlFEYufkrSCqVpQValeob5bj2NuVWme1ySL1nR_OjOMNpBwSvEXxOIon4zkyM58JeSmlyiW4EmHJVAUBSg17LsmzUCdpLhk3OOwOm8gnk-L4uPzs-7jbdbX7OiXpNLXb1kX2poW4qcAySYTvh4vyMrmSgMFCwPzDoy_Dj5UIInSwsz6D-duZIxvkoPp_Vcg_WaSL1ZIXUqbOEu3e-q813CY3veNJd3pJuUMuaXuXXOuPojy_R76BvFAsNaSNoWfYkqROzs9gAgI9U_y939K5pQ7itVk0c-x9_DFGO4eRfir7Rib7li48NFRjqbQ1HerZ8fGzpTRdiPUgqzbELhPq00T3yXT3w_T9XuhPaAhVHmVdWKd9HjczShZxongVwcflVVHnELFL2OySxbo0SWqqODLMVBBOSq1kpXRZZ_wB2bCN1Y8IBc8LTGkNwVdaJirjlYlTZZRmOteKJyog0fqTCeXRy_EQjVPhopgiEz1vBfBWIG9FGZDtYcqih-74G_E7lIOBEFG33Y1mORN-EwueRswwzVWhIdDTusDD4-OyZokEP1slAdlCKRJ9C-ugO8ROWoInlyY8CsgLR4HIGxZLe2Zy1bZi_-hwRPTKE5kG1qik75QATiFY14hyc0QJqkGNhp-vxVngENbTWd2sWsEw3w0rz-CdH_biPSydx4zloPYDko8Ef8Sb8YidnzhkcnBnizKGZ75ei7_wKrH9M-sf_xP1Frm-N_10IA72Jx-fkBsM91KUhSzeJBvdcqWfkqvqazdvl8-cWvgOnMNe1A
linkToPdf http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwpV1Zb9QwELagHOoLNzRQqEFISKCoiXOat3KsqECrilaob5bj2NuVWme1ySL1nR_OjOMNpBwS4m0VT6L1ZA5PZuYbQp5LqQoJR4mQM1VBgFKDzqVFHuo0KyRLDC67YRPFdFoeH_MDP-e0XVe7r1OSfU8DojTZbndRm17Fy3y3hRiqxJJJhPKHH_wyuZJiHT2G64dfho8sEUTr4HN9NvO3d478kYPt_9U4_-SdLlZOXkifOq80ufnf-7lFbvgDKd3rJeg2uaTtHXKtH1F5fpd8AzmiWIJIG0PPsFVJnZyfwQ0IAE3xs39L55Y66Ndm0cyxJ_LHGu0cdvqp7Buc7Gu68JBRjaXS1nSoc8fHz5bSdCHWiazaELtPqE8f3SNHk_dHbz-EfnJDqIoo78I66_O7uVGyjFOVVBG89KQq6wIieQlGQLJYc5Nmpoojw0wFYabUSlZK8zpP7pMN21i9RSicyMDF1hCUZTxVeVKZOFNGaaYLrZJUBSRavz6hPKo5Dtc4FS66KXPR81YAbwXyVvCAvBxuWfSQHn8jfoMyMRAiGre70Cxnwiu3SLKIGaYTVWoIALUucah8zGuWSjh_qzQgOyhRom9tHWyK2Ms4nPCyNIkC8sxRICKHxZKfmVy1rdg__DwieuGJTAN7VNJ3UACnEMRrRLk9ogSToUbLT9eiLXAJ6-ysblatYJgHh53n8J8f9KI-bD2JGSvAHQSkGCnBiDfjFTs_cYjlcMwteQzPfLVWBeFNZftn1j_8J-odcv3g3UR82p9-fEQ2GapSlIcs3iYb3XKlH5Or6ms3b5dPnIX4DsP3Z7g
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=The+role+of+mesenchymal+stem+cells+in+hematopoietic+stem+cell+transplantation%3A+prevention+and+treatment+of+graft-versus-host+disease&rft.jtitle=Stem+cell+research+%26+therapy&rft.au=Zhao%2C+Lu&rft.au=Chen%2C+Shanquan&rft.au=Yang%2C+Panxin&rft.au=Cao%2C+Hongcui&rft.date=2019-06-21&rft.pub=BioMed+Central+Ltd&rft.issn=1757-6512&rft.eissn=1757-6512&rft.volume=10&rft.issue=1&rft_id=info:doi/10.1186%2Fs13287-019-1287-9&rft.externalDBID=ISR&rft.externalDocID=A590715430
thumbnail_l http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=1757-6512&client=summon
thumbnail_m http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=1757-6512&client=summon
thumbnail_s http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=1757-6512&client=summon