Neuropeptide Y directly reduced apoptosis of granulosa cells, and the expression of NPY and its receptors in PCOS subjects

Background Most women with anovulatory infertility show polycystic ovarian syndrome (PCOS), and androgen excess is known as a key factor involved in pathogenicity of PCOS. However, the mechanism of follicular developmental arrest in PCOS is not completely understood. The reproductive function of Neu...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Journal of ovarian research Jg. 16; H. 1; S. 1 - 10
Hauptverfasser:	Urata, Yoko, Salehi, Reza, Wyse, Brandon A., Jahangiri, Sahar, Librach, Clifford L., Tzeng, Chii-Ruey, Osuga, Yutaka, Tsang, Benjamin
Format:	Journal Article
Sprache:	Englisch
Veröffentlicht:	London BioMed Central 31.08.2023 BioMed Central Ltd Springer Nature B.V BMC
Schlagworte:	Analysis Androgens Apoptosis Body fat Cell culture Cell growth Cell proliferation Dihydrotestosterone Enzyme-linked immunosorbent assay Follicular fluid Folliculogenesis Granulosa cells Gynecology Hyperandrogenism Infertility Medicine Medicine & Public Health Metabolism mRNA Neuropeptide Neuropeptide Y Neuropeptides Ovaries Pathogenesis Pathogenicity Phenotypes Polycystic ovarian syndrome Polycystic ovary syndrome Protein folding Proteins Receptor mechanisms Reproductive Medicine RNA Stein-Leventhal syndrome Neuropeptide Y Polycystic ovarian syndrome Hyperandrogenism Neuropeptide Apoptosis
ISSN:	1757-2215, 1757-2215
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!