Relation between adiposity and vascular events, malignancy and mortality in patients with stable cerebrovascular disease

Background: Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m − 2 ) and cardiovascular events is often described. Wh...

Celý popis

Uloženo v:
Podrobná bibliografie
Vydáno v:International Journal of Obesity Ročník 41; číslo 12; s. 1775 - 1781
Hlavní autoři: Jaspers, N E M, Dorresteijn, J A N, van der Graaf, Y, Westerink, J, Kappelle, L J, Nathoe, H M, Algra, A, Visseren, F L J
Médium: Journal Article
Jazyk:angličtina
Vydáno: London Nature Publishing Group UK 01.12.2017
Nature Publishing Group
Témata:
692/308/174
692/499
692/699/2743/393
692/699/75/593/1370
692/700/139/2818
692/700/2817
692/700/459
Abdomen
Adipose tissue
Adiposity - physiology
Adult
Aged
Body Mass Index
Body size
Cardiovascular diseases
Cause of Death - trends
Cerebrovascular disease
Cerebrovascular diseases
Cerebrovascular Disorders - etiology
Cerebrovascular Disorders - mortality
Cerebrovascular Disorders - physiopathology
Chi-square test
Complications and side effects
Confidence intervals
Epidemiology
Female
Hazard assessment
Health Promotion and Disease Prevention
Health risk assessment
Humans
Hypertension
Hypertension - etiology
Hypertension - mortality
Hypertension - physiopathology
Internal Medicine
Male
Malignancy
Medicine
Medicine & Public Health
Metabolic Diseases
Middle Aged
Mortality
Neoplasms - mortality
Neoplasms - physiopathology
Nonlinear systems
Nonlinearity
Obesity
Obesity, Abdominal - complications
Obesity, Abdominal - mortality
Obesity, Abdominal - physiopathology
original-article
Patient outcomes
Patients
Prognosis
Proportional Hazards Models
Prospective Studies
Public Health
Reference Values
Risk analysis
Risk Factors
Statistical analysis
Statistical models
Statistical tests
Ultrasound
Waist Circumference
Young Adult
Netherlands
ISSN:0307-0565, 1476-5497, 1476-5497
On-line přístup:Získat plný text
Tagy: Přidat tag
Žádné tagy, Buďte první, kdo vytvoří štítek k tomuto záznamu!
Abstract Background: Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m − 2 ) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD. Methods: During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the χ 2 -statistic of the nonlinear term was significant ( P -value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations. Results: The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9–29.3) kg m 2 for vascular mortality and 28.1 (95% CI, 19.0–38.2)) kg m − 2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7–134.8) cm for females and 94.8 (95% CI, 80.3–100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00–1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05–1.42). Conclusions: In patients with CeVD, a BMI around 27–28 kg m − 2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
AbstractList Background:Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m−2) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD.Methods:During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the χ2-statistic of the nonlinear term was significant (P-value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations.Results:The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9–29.3) kg m2 for vascular mortality and 28.1 (95% CI, 19.0–38.2)) kg m−2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7–134.8) cm for females and 94.8 (95% CI, 80.3–100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00–1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05–1.42).Conclusions:In patients with CeVD, a BMI around 27–28 kg m−2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Background: Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m − 2 ) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD. Methods: During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the χ 2 -statistic of the nonlinear term was significant ( P -value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations. Results: The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9–29.3) kg m 2 for vascular mortality and 28.1 (95% CI, 19.0–38.2)) kg m − 2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7–134.8) cm for females and 94.8 (95% CI, 80.3–100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00–1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05–1.42). Conclusions: In patients with CeVD, a BMI around 27–28 kg m − 2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Background: Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m.sup.-.sup.2) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD. Methods: During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the [chi].sup.2-statistic of the nonlinear term was significant (P-value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations. Results: The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m.sup.2 for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m.sup.-.sup.2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42). Conclusions: In patients with CeVD, a BMI around 27-28 kg m.sup.-.sup.2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Background: Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m[sup.-][sup.2]) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD. Methods: During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the [chi][sup.2]-statistic of the nonlinear term was significant (P-value [less than] 0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations. Results: The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m[sup.2] for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m[sup.-][sup.2] for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42). Conclusions: In patients with CeVD, a BMI around 27-28 kg m[sup.-][sup.2] relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal. International Journal of Obesity (2017) 41, 1775-1781; doi: 10.1038/ijo.2017.184; published online 12 September 2017
Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m.sup.-.sup.2) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD. During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the [chi].sup.2-statistic of the nonlinear term was significant (P-value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations. The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m.sup.2 for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m.sup.-.sup.2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42). In patients with CeVD, a BMI around 27-28 kg m.sup.-.sup.2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Background:Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m- 2 ) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD.Methods:During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the χ2 -statistic of the nonlinear term was significant (P-value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations.Results:The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m2 for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m- 2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42).Conclusions:In patients with CeVD, a BMI around 27-28 kg m- 2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m ) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD. During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the χ -statistic of the nonlinear term was significant (P-value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations. The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42). In patients with CeVD, a BMI around 27-28 kg m relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m-2) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD.BACKGROUNDAbdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m-2) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD.During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the χ2-statistic of the nonlinear term was significant (P-value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations.METHODSDuring a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the χ2-statistic of the nonlinear term was significant (P-value<0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations.The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m2 for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m-2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42).RESULTSThe relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m2 for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m-2 for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42).In patients with CeVD, a BMI around 27-28 kg m-2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.CONCLUSIONSIn patients with CeVD, a BMI around 27-28 kg m-2 relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable cerebrovascular disease (CeVD). A U-shaped relation between body mass index (BMI, kg m[sup.-][sup.2]) and cardiovascular events is often described. Whether this U-shape persists for abdominal adiposity, and consequently which reference values should guide clinical practice, is unclear. We described the relation between multiple adiposity measurements and risk of vascular events, vascular mortality, malignancy and all-cause mortality in patients with clinically stable CeVD. During a median follow-up time of 6.8 years, 1767 patients were prospectively followed. Relations were assessed using multivariable adjusted Cox proportional hazards models. Adiposity was assessed with BMI, waist circumference (stratified by gender) and the contribution of visceral fat to total abdominal fat (VAT%) measured using ultrasound. Relations were nonlinear if the [chi][sup.2]-statistic of the nonlinear term was significant (P-value [less than] 0.05). Nadirs were reported for nonlinear and hazard ratios (HRs) for linear relations. The relations between BMI and outcomes were nonlinear with nadirs ranging between 27.1 (95% confidence interval (CI) 21.9-29.3) kg m[sup.2] for vascular mortality and 28.1 (95% CI, 19.0-38.2)) kg m[sup.-][sup.2] for malignancy. The relation between waist circumference and all-cause mortality was nonlinear with a nadir of 84.0 (95% CI, 18.7-134.8) cm for females and 94.8 (95% CI, 80.3-100.1) cm for males. No nonlinearity was detected for VAT%. A 1-s.d. (9.8%) increase in VAT% was related to both vascular (HR, 1.23, 95% CI 1.00-1.51) and all-cause mortality (HR, 1.22, 95% CI 1.05-1.42). In patients with CeVD, a BMI around 27-28 kg m[sup.-][sup.2] relates to the lowest risk of vascular events, vascular mortality, malignancy and all-cause mortality. However, increasing abdominal adiposity confers a higher risk of all-cause mortality. Thus, whereas traditional BMI cutoffs may be re-evaluated in this population, striving for low abdominal obesity should remain a goal.
Audience Academic
Author Algra, A
van der Graaf, Y
Visseren, F L J
Nathoe, H M
Jaspers, N E M
Kappelle, L J
Dorresteijn, J A N
Westerink, J
Author_xml – sequence: 1
  givenname: N E M
  surname: Jaspers
  fullname: Jaspers, N E M
  organization: Department of Vascular Medicine, University Medical Center Utrecht
– sequence: 2
  givenname: J A N
  surname: Dorresteijn
  fullname: Dorresteijn, J A N
  organization: Department of Vascular Medicine, University Medical Center Utrecht
– sequence: 3
  givenname: Y
  surname: van der Graaf
  fullname: van der Graaf, Y
  organization: Julius Center for Health Sciences and Primary Care, University Medical Center Utrecht
– sequence: 4
  givenname: J
  surname: Westerink
  fullname: Westerink, J
  organization: Department of Vascular Medicine, University Medical Center Utrecht
– sequence: 5
  givenname: L J
  surname: Kappelle
  fullname: Kappelle, L J
  organization: Department of Neurology and Neurosurgery, Brain Center Rudolf Magnus, University Medical Center Utrecht
– sequence: 6
  givenname: H M
  surname: Nathoe
  fullname: Nathoe, H M
  organization: Department of Cardiology, University Medical Center Utrecht
– sequence: 7
  givenname: A
  surname: Algra
  fullname: Algra, A
  organization: Julius Center for Health Sciences and Primary Care, University Medical Center Utrecht, Department of Neurology and Neurosurgery, Brain Center Rudolf Magnus, University Medical Center Utrecht
– sequence: 8
  givenname: F L J
  surname: Visseren
  fullname: Visseren, F L J
  email: f.l.j.visseren@umcutrecht.nl
  organization: Department of Vascular Medicine, University Medical Center Utrecht
BackLink https://www.ncbi.nlm.nih.gov/pubmed/28775374$$D View this record in MEDLINE/PubMed
BookMark eNqNkktr3DAUhUVJaSZpd10XQ6F0EU_1sCx5GUJfECiUdm1k-XpGgyxNJTmPf1-5k4ZJSIeiheDqO-dKuucEHTnvAKHXBC8JZvKD2fglxUQsiayeoQWpRF3yqhFHaIEZFiXmNT9GJzFuMMacY_oCHVMpBGeiWqCb72BVMt4VHaRrAFeo3mx9NOm2UK4vrlTUk1WhgCtwKZ4Vo7Jm5ZTTu_PRh5QrmTau2GanmSquTVoXManOQqEhQBf8vVFvIqgIL9HzQdkIr-72U_Tz08cfF1_Ky2-fv16cX5aaS5LKDupaUU00lbrRg-QwCF7XAwOleKMwUKY50UPPOgnQMS4Z50Muc0I7xgZ2it7vfLfB_5ogpnY0UYO1yoGfYksaWteSMUkz-vYRuvFTcPl2La0J57SpyUGKNIJSUclmj1opC61xg09B6bl1e15nQOYxNQcpTkRFGRUsU8snqLx6GI3OaRhMrj-w_S_Bfod3e4I1KJvW0dtpDkZ86HwQ3Hd8c_dLUzdC326DGVW4bf8mLwNnO0AHH2OA4R4huJ2D3eZgt3Ow2xzsjNNHuDbpT3Dz04z9l6jciWL2disIe6N6iv8NAMAHGg
CitedBy_id crossref_primary_10_1016_j_bone_2018_12_009
crossref_primary_10_1089_met_2020_0064
crossref_primary_10_1016_j_semcancer_2023_03_008
crossref_primary_10_1186_s12889_023_16950_8
crossref_primary_10_1016_j_orcp_2022_11_003
crossref_primary_10_1016_j_orcp_2023_09_004
crossref_primary_10_1186_s41043_025_00980_6
crossref_primary_10_1016_j_hnm_2025_200313
crossref_primary_10_1111_obr_13088
Cites_doi 10.1093/eurheartj/ehs340
10.1093/eurheartj/ehv347
10.1016/j.jclinepi.2007.08.012
10.1111/j.1467-789X.2007.00411.x
10.1016/S0140-6736(08)60269-X
10.1001/jama.2016.4666
10.1172/JCI117936
10.1136/bmj.d1732
10.1111/j.1467-789X.2011.00914.x
10.1016/0959-8049(95)00355-M
10.1161/01.HYP.0000154361.47683.d3
10.1016/j.atherosclerosis.2010.04.029
10.1007/s10552-015-0654-9
10.1016/j.jacc.2013.11.004
10.1111/obr.12272
10.1097/EDE.0000000000000493
10.1007/s00125-012-2639-5
10.1161/STROKEAHA.115.009128
10.1111/obr.12377
10.1097/HJH.0b013e328357f18a
10.1001/jamaneurol.2014.1017
10.1016/j.amjcard.2015.02.024
10.1016/j.atherosclerosis.2015.05.026
10.1161/HYPERTENSIONAHA.109.130005
10.1038/oby.2010.248
10.1002/oby.21239
10.1161/01.HYP.25.4.560
10.1161/CIRCHEARTFAILURE.113.001041
10.1093/ajcn/77.4.857
10.1158/1055-9965.EPI-09-0372
10.1023/A:1007621514757
10.1038/ijo.2011.115
ContentType Journal Article
Copyright Macmillan Publishers Limited, part of Springer Nature. 2017
COPYRIGHT 2017 Nature Publishing Group
Copyright Nature Publishing Group Dec 2017
Macmillan Publishers Limited, part of Springer Nature. 2017.
Copyright_xml – notice: Macmillan Publishers Limited, part of Springer Nature. 2017
– notice: COPYRIGHT 2017 Nature Publishing Group
– notice: Copyright Nature Publishing Group Dec 2017
– notice: Macmillan Publishers Limited, part of Springer Nature. 2017.
CorporateAuthor on behalf of the SMART Study Group
CorporateAuthor_xml – name: on behalf of the SMART Study Group
DBID AAYXX
CITATION
CGR
CUY
CVF
ECM
EIF
NPM
3V.
7T2
7TK
7TS
7X2
7X7
7XB
88E
88G
8AO
8C1
8FE
8FH
8FI
8FJ
8FK
ABUWG
AEUYN
AFKRA
ATCPS
AZQEC
BBNVY
BENPR
BHPHI
C1K
CCPQU
DWQXO
FYUFA
GHDGH
GNUQQ
HCIFZ
K9.
LK8
M0K
M0S
M1P
M2M
M7P
PHGZM
PHGZT
PJZUB
PKEHL
PPXIY
PQEST
PQGLB
PQQKQ
PQUKI
PRINS
PSYQQ
Q9U
7X8
DOI 10.1038/ijo.2017.184
DatabaseName CrossRef
Medline
MEDLINE
MEDLINE (Ovid)
MEDLINE
MEDLINE
PubMed
ProQuest Central (Corporate)
Health and Safety Science Abstracts (Full archive)
Neurosciences Abstracts
Physical Education Index
Agricultural Science Collection
Health & Medical Collection (ProQuest)
ProQuest Central (purchase pre-March 2016)
Medical Database (Alumni Edition)
Psychology Database (Alumni)
ProQuest Pharma Collection
Public Health Database (ProQuest)
ProQuest SciTech Collection
ProQuest Natural Science Collection
ProQuest Hospital Collection
Hospital Premium Collection (Alumni Edition)
ProQuest Central (Alumni) (purchase pre-March 2016)
ProQuest Central (Alumni)
ProQuest One Sustainability
ProQuest Central UK/Ireland
Agricultural & Environmental Science Collection
ProQuest Central Essentials
Biological Science Collection
ProQuest Central
Natural Science Collection
Environmental Sciences and Pollution Management
ProQuest One Community College
ProQuest Central
Proquest Health Research Premium Collection
Health Research Premium Collection (Alumni)
ProQuest Central Student
SciTech Collection (ProQuest)
ProQuest Health & Medical Complete (Alumni)
Biological Sciences
Agricultural Science Database
ProQuest Health & Medical Collection
Medical Database
Psychology Database (ProQuest)
Biological Science Database (ProQuest)
ProQuest Central Premium
ProQuest One Academic
ProQuest Health & Medical Research Collection
ProQuest One Academic Middle East (New)
ProQuest One Health & Nursing
ProQuest One Academic Eastern Edition (DO NOT USE)
ProQuest One Applied & Life Sciences
ProQuest One Academic (retired)
ProQuest One Academic UKI Edition
ProQuest Central China
ProQuest One Psychology
ProQuest Central Basic
MEDLINE - Academic
DatabaseTitle CrossRef
MEDLINE
Medline Complete
MEDLINE with Full Text
PubMed
MEDLINE (Ovid)
Agricultural Science Database
ProQuest One Psychology
ProQuest Central Student
ProQuest One Academic Middle East (New)
ProQuest Central Essentials
ProQuest Health & Medical Complete (Alumni)
ProQuest Central (Alumni Edition)
SciTech Premium Collection
ProQuest One Community College
ProQuest One Health & Nursing
ProQuest Natural Science Collection
ProQuest Pharma Collection
ProQuest Central China
Physical Education Index
Environmental Sciences and Pollution Management
ProQuest Central
ProQuest One Applied & Life Sciences
ProQuest One Sustainability
ProQuest Health & Medical Research Collection
Health Research Premium Collection
Health and Medicine Complete (Alumni Edition)
Natural Science Collection
ProQuest Central Korea
Health & Medical Research Collection
Agricultural & Environmental Science Collection
Biological Science Collection
Health & Safety Science Abstracts
ProQuest Central (New)
ProQuest Medical Library (Alumni)
ProQuest Public Health
ProQuest Biological Science Collection
ProQuest Central Basic
ProQuest One Academic Eastern Edition
Agricultural Science Collection
ProQuest Hospital Collection
Health Research Premium Collection (Alumni)
ProQuest Psychology Journals (Alumni)
Biological Science Database
ProQuest SciTech Collection
Neurosciences Abstracts
ProQuest Hospital Collection (Alumni)
ProQuest Health & Medical Complete
ProQuest Medical Library
ProQuest Psychology Journals
ProQuest One Academic UKI Edition
ProQuest One Academic
ProQuest One Academic (New)
ProQuest Central (Alumni)
MEDLINE - Academic
DatabaseTitleList Agricultural Science Database





Agricultural Science Database

MEDLINE
MEDLINE - Academic
Database_xml – sequence: 1
  dbid: NPM
  name: PubMed
  url: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 2
  dbid: BENPR
  name: ProQuest Central
  url: https://www.proquest.com/central
  sourceTypes: Aggregation Database
DeliveryMethod fulltext_linktorsrc
Discipline Medicine
Public Health
Diet & Clinical Nutrition
Recreation & Sports
EISSN 1476-5497
EndPage 1781
ExternalDocumentID A689284769
A517423273
28775374
10_1038_ijo_2017_184
Genre Journal Article
GeographicLocations Netherlands
GeographicLocations_xml – name: Netherlands
GroupedDBID .55
.GJ
29J
36B
39C
5RE
7X2
7X7
8R4
8R5
A8Z
ABDBF
ABOCM
ABUWG
ACUHS
ADBBV
AFFNX
AI.
ALMA_UNASSIGNED_HOLDINGS
ATCPS
AZQEC
B0M
BAWUL
BENPR
BHPHI
BPHCQ
DIK
DWQXO
EAD
EAP
EBC
EBD
EBS
EMB
EMK
EMOBN
EPL
ESX
F5P
FYUFA
GNUQQ
HCIFZ
IAO
IHR
ITC
J5H
M0K
M1P
M2M
MVM
NAO
OK1
Q2X
RNT
RNTTT
SV3
TUS
VH1
WH7
X7M
ZGI
ZXP
~8M
AAYXX
CITATION
---
-Q-
..I
.L3
.XZ
0R~
1CY
2FS
2WC
4.4
406
53G
5GY
70F
88E
8AO
8C1
8FE
8FH
8FI
8FJ
AACDK
AAHBH
AANZL
AASML
AATNV
AAWTL
AAYZH
ABAKF
ABAWZ
ABBRH
ABCQX
ABDBE
ABFSG
ABIVO
ABJNI
ABLJU
ABRTQ
ABZZP
ACAOD
ACGFS
ACKTT
ACPRK
ACRQY
ACSTC
ACZOJ
ADHUB
ADXHL
AEFQL
AEJRE
AEMSY
AENEX
AEUYN
AEVLU
AEXYK
AEZWR
AFBBN
AFDZB
AFHIU
AFKRA
AFRAH
AFSHS
AGAYW
AGHAI
AGQEE
AHMBA
AHSBF
AHWEU
AIGIU
AILAN
AIXLP
AJRNO
ALFFA
ALIPV
AMYLF
APEBS
ATHPR
AXYYD
AYFIA
BBNVY
BKKNO
BVXVI
CCPQU
CGR
CS3
CUY
CVF
DNIVK
DPUIP
DU5
E3Z
EBLON
ECM
EE.
EIF
EIOEI
EJD
FDQFY
FERAY
FIGPU
FIZPM
FSGXE
HMCUK
HZ~
IHW
INH
INR
IPY
IWAJR
JSO
JZLTJ
KQ8
L7B
M7P
NPM
NQJWS
O9-
OVD
P2P
P6G
PHGZM
PHGZT
PJZUB
PPXIY
PQGLB
PQQKQ
PROAC
PSQYO
PSYQQ
RNS
ROL
SNX
SNYQT
SOHCF
SOJ
SRMVM
SWTZT
TAOOD
TBHMF
TDRGL
TEORI
TR2
TSG
UKHRP
~KM
3V.
7T2
7TK
7TS
7XB
8FK
C1K
K9.
LK8
PKEHL
PQEST
PQUKI
PRINS
Q9U
7X8
PUEGO
ID FETCH-LOGICAL-c581t-be66a2c1c28c9cf85ef7566f3eaa59a0e23c51cfd3b8eeb358355f0e2512b33f3
IEDL.DBID M7P
ISICitedReferencesCount 9
ISICitedReferencesURI http://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=Summon&SrcAuth=ProQuest&DestLinkType=CitingArticles&DestApp=WOS_CPL&KeyUT=000417032800008&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
ISSN 0307-0565
1476-5497
IngestDate Fri Sep 05 13:15:51 EDT 2025
Mon Oct 06 18:23:58 EDT 2025
Mon Oct 06 17:31:39 EDT 2025
Sat Nov 29 13:04:13 EST 2025
Sat Nov 29 13:29:00 EST 2025
Sat Nov 29 09:53:16 EST 2025
Sat Nov 29 10:16:55 EST 2025
Thu May 22 21:08:44 EDT 2025
Thu May 22 21:21:59 EDT 2025
Mon Jul 21 05:48:17 EDT 2025
Sat Nov 29 04:30:58 EST 2025
Tue Nov 18 21:59:12 EST 2025
Fri Feb 21 02:39:41 EST 2025
IsPeerReviewed true
IsScholarly true
Issue 12
Language English
LinkModel DirectLink
MergedId FETCHMERGED-LOGICAL-c581t-be66a2c1c28c9cf85ef7566f3eaa59a0e23c51cfd3b8eeb358355f0e2512b33f3
Notes ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
content type line 14
content type line 23
PMID 28775374
PQID 1972274892
PQPubID 38864
PageCount 7
ParticipantIDs proquest_miscellaneous_1926683382
proquest_journals_2615529612
proquest_journals_1972274892
gale_infotracmisc_A689284769
gale_infotracmisc_A517423273
gale_infotracacademiconefile_A689284769
gale_infotracacademiconefile_A517423273
gale_healthsolutions_A689284769
gale_healthsolutions_A517423273
pubmed_primary_28775374
crossref_primary_10_1038_ijo_2017_184
crossref_citationtrail_10_1038_ijo_2017_184
springer_journals_10_1038_ijo_2017_184
PublicationCentury 2000
PublicationDate 2017-12-01
PublicationDateYYYYMMDD 2017-12-01
PublicationDate_xml – month: 12
  year: 2017
  text: 2017-12-01
  day: 01
PublicationDecade 2010
PublicationPlace London
PublicationPlace_xml – name: London
– name: England
PublicationSubtitle Official journal of the International Association for the Study of Obesity
PublicationTitle International Journal of Obesity
PublicationTitleAbbrev Int J Obes
PublicationTitleAlternate Int J Obes (Lond)
PublicationYear 2017
Publisher Nature Publishing Group UK
Nature Publishing Group
Publisher_xml – name: Nature Publishing Group UK
– name: Nature Publishing Group
References Sharma, Lavie, Borer, Vallakati, Goel, Lopez-Jimenez (CR3) 2015; 115
(CR18) 2000; 894
Dorresteijn, Visseren, Spiering (CR9) 2012; 13
Afzal, Tybjaerg-Hansen, Jensen, Nordestgaard (CR13) 2016; 315
Chirinos, Khan, Bansal, Dries, Feldman, Ford (CR19) 2014; 7
Gast, den Heijer, Smit, Widya, Lamb, de Roos (CR11) 2015; 241
Timpson, Harbord, Davey Smith, Zacho, Tybjaerg-Hansen, Nordestgaard (CR26) 2009; 54
Hansel, Roussel, Elbez, Marre, Krempf, Ikeda (CR2) 2015; 36
Stolk, Meijer, Mali, Grobbee, van der Graaf (CR16) 2003; 77
CR35
van der Sanden, Coebergh, Schouten, Visser, van Leeuwen (CR17) 1995; 31A
Dehlendorff, Andersen, Olsen (CR22) 2014; 71
Doehner, Schenkel, Anker, Springer, Audebert (CR21) 2013; 34
Renehan, Tyson, Egger, Heller, Zwahlen (CR30) 2008; 371
Sperrin, Candlish, Badrick, Renehan, Buchan (CR36) 2016; 27
Faber, van der Graaf, Westerink, Visseren (CR27) 2010; 212
Lee, Huxley, Wildman, Woodward (CR8) 2008; 61
Kanhai, Kappelle, van der Graaf, Uiterwaal, Visseren (CR14) 2012; 36
Howard, Banach, Cushman, Goff, Howard, Lackland (CR7) 2015; 46
McMinn, Steel, Bowman (CR20) 2011; 342
(CR32) 1995; 854
Hotamisligil, Arner, Caro, Atkinson, Spiegelman (CR23) 1995; 95
Ross, Berentzen, Bradshaw, Janssen, Kahn, Katzmarzyk (CR33) 2008; 9
Cabia, Andrade, Carreira, Casanueva, Crujeiras (CR28) 2016; 17
Camhi, Bray, Bouchard, Greenway, Johnson, Newton (CR29) 2011; 19
Grassi, Seravalle, Cattaneo, Bolla, Lanfranchi, Colombo (CR24) 1995; 25
Engeli, Bohnke, Gorzelniak, Janke, Schling, Bader (CR25) 2005; 45
van Kruijsdijk, van der Wall, Visseren (CR6) 2009; 18
Davies, Gaunt, Lewis, Holly, Donovan, Hamdy (CR31) 2015; 26
Jensen, Ryan, Apovian, Ard, Comuzzie, Donato (CR1) 2014; 63
Kaess, Pedley, Massaro, Murabito, Hoffmann, Fox (CR12) 2012; 55
Bagheri, Speakman, Shabbidar, Kazemi, Djafarian (CR4) 2015; 16
Stokes, Preston (CR5) 2015; 23
Dorresteijn, Spiering, Van Der Graaf, Visseren (CR10) 2012; 30
Nadas, Putz, Kolev, Nagy, Jermendy (CR34) 2008; 14
Simons, Algra, van de Laak, Grobbee, van der Graaf (CR15) 1999; 15
G Howard (BFijo2017184_CR7) 2015; 46
GS Hotamisligil (BFijo2017184_CR23) 1995; 95
KB Gast (BFijo2017184_CR11) 2015; 241
JA Dorresteijn (BFijo2017184_CR10) 2012; 30
BFijo2017184_CR35
Obesity: preventing and managing the global epidemic (BFijo2017184_CR18) 2000; 894
J Nadas (BFijo2017184_CR34) 2008; 14
CM Lee (BFijo2017184_CR8) 2008; 61
JA Chirinos (BFijo2017184_CR19) 2014; 7
DR Faber (BFijo2017184_CR27) 2010; 212
NM Davies (BFijo2017184_CR31) 2015; 26
JA Dorresteijn (BFijo2017184_CR9) 2012; 13
NJ Timpson (BFijo2017184_CR26) 2009; 54
PC Simons (BFijo2017184_CR15) 1999; 15
R Ross (BFijo2017184_CR33) 2008; 9
B Hansel (BFijo2017184_CR2) 2015; 36
S Engeli (BFijo2017184_CR25) 2005; 45
DA Kanhai (BFijo2017184_CR14) 2012; 36
AG Renehan (BFijo2017184_CR30) 2008; 371
MD Jensen (BFijo2017184_CR1) 2014; 63
BM Kaess (BFijo2017184_CR12) 2012; 55
GA van der Sanden (BFijo2017184_CR17) 1995; 31A
M Bagheri (BFijo2017184_CR4) 2015; 16
C Dehlendorff (BFijo2017184_CR22) 2014; 71
A Stokes (BFijo2017184_CR5) 2015; 23
S Afzal (BFijo2017184_CR13) 2016; 315
RP Stolk (BFijo2017184_CR16) 2003; 77
A Sharma (BFijo2017184_CR3) 2015; 115
RC van Kruijsdijk (BFijo2017184_CR6) 2009; 18
G Grassi (BFijo2017184_CR24) 1995; 25
W Doehner (BFijo2017184_CR21) 2013; 34
Physical status: the use and interpretation of anthropometry (BFijo2017184_CR32) 1995; 854
J McMinn (BFijo2017184_CR20) 2011; 342
M Sperrin (BFijo2017184_CR36) 2016; 27
B Cabia (BFijo2017184_CR28) 2016; 17
SM Camhi (BFijo2017184_CR29) 2011; 19
26242715 - Eur Heart J. 2015 Oct 21;36(40):2716-28
21447571 - BMJ. 2011 Mar 29;342:d1732
25953369 - Stroke. 2015 Jun;46(6):1595-600
18280327 - Lancet. 2008 Feb 16;371(9612):569-78
26387087 - Cancer Causes Control. 2015 Nov;26(11):1603-16
24886975 - JAMA Neurol. 2014 Aug;71(8):978-84
11234459 - World Health Organ Tech Rep Ser. 2000;894:i-xii, 1-253
26421898 - Obesity (Silver Spring). 2015 Dec;23(12):2485-90
8541107 - Eur J Cancer. 1995 Oct;31A(11):1822-9
17956544 - Obes Rev. 2008 Jul;9(4):312-25
19470880 - Hypertension. 2009 Jul;54(1):84-90
12663283 - Am J Clin Nutr. 2003 Apr;77(4):857-60
26914773 - Obes Rev. 2016 Apr;17 (4):361-76
20494358 - Atherosclerosis. 2010 Sep;212(1):274-80
10608355 - Eur J Epidemiol. 1999 Oct;15(9):773-81
8594834 - World Health Organ Tech Rep Ser. 1995;854:1-452
27075676 - Epidemiology. 2016 Jul;27(4):525-30
7721398 - Hypertension. 1995 Apr;25(4 Pt 1):560-3
23041751 - J Hypertens. 2012 Dec;30(12):2331-7
20948514 - Obesity (Silver Spring). 2011 Feb;19(2):402-8
23076781 - Eur Heart J. 2013 Jan;34(4):268-77
19755644 - Cancer Epidemiol Biomarkers Prev. 2009 Oct;18(10):2569-78
27163987 - JAMA. 2016 May 10;315(18):1989-96
26100677 - Atherosclerosis. 2015 Aug;241(2):547-54
25753533 - Obes Rev. 2015 May;16(5):416-23
21831233 - Obes Rev. 2012 Jan;13(1):17-26
18359190 - J Clin Epidemiol. 2008 Jul;61(7):646-53
25772740 - Am J Cardiol. 2015 May 15;115(10):1428-34
18160939 - Med Sci Monit. 2008 Jan;14(1):CR15-18
21712810 - Int J Obes (Lond). 2012 May;36(5):695-702
7738205 - J Clin Invest. 1995 May;95(5):2409-15
25059422 - Circ Heart Fail. 2014 Sep;7(5):709-16
24239920 - J Am Coll Cardiol. 2014 Jul 1;63(25 Pt B):2985-3023
15630041 - Hypertension. 2005 Mar;45(3):356-62
22898763 - Diabetologia. 2012 Oct;55(10 ):2622-2630
References_xml – volume: 34
  start-page: 268
  year: 2013
  end-page: 277
  ident: CR21
  article-title: Overweight and obesity are associated with improved survival, functional outcome, and stroke recurrence after acute stroke or transient ischaemic attack: observations from the TEMPiS trial
  publication-title: Eur Heart J
  doi: 10.1093/eurheartj/ehs340
– volume: 36
  start-page: 2716
  year: 2015
  end-page: 2728
  ident: CR2
  article-title: Cardiovascular risk in relation to body mass index and use of evidence-based preventive medications in patients with or at risk of atherothrombosis
  publication-title: Eur Heart J
  doi: 10.1093/eurheartj/ehv347
– volume: 61
  start-page: 646
  year: 2008
  end-page: 653
  ident: CR8
  article-title: Indices of abdominal obesity are better discriminators of cardiovascular risk factors than BMI: a meta-analysis
  publication-title: J Clin Epidemiol
  doi: 10.1016/j.jclinepi.2007.08.012
– volume: 9
  start-page: 312
  year: 2008
  end-page: 325
  ident: CR33
  article-title: Does the relationship between waist circumference, morbidity and mortality depend on measurement protocol for waist circumference?
  publication-title: Obes Rev
  doi: 10.1111/j.1467-789X.2007.00411.x
– volume: 371
  start-page: 569
  year: 2008
  end-page: 578
  ident: CR30
  article-title: Body-mass index and incidence of cancer: a systematic review and meta-analysis of prospective observational studies
  publication-title: Lancet
  doi: 10.1016/S0140-6736(08)60269-X
– volume: 315
  start-page: 1989
  year: 2016
  end-page: 1996
  ident: CR13
  article-title: Change in body mass index associated with lowest mortality in Denmark, 1976-2013
  publication-title: JAMA
  doi: 10.1001/jama.2016.4666
– volume: 95
  start-page: 2409
  year: 1995
  end-page: 2415
  ident: CR23
  article-title: Increased adipose tissue expression of tumor necrosis factor-alpha in human obesity and insulin resistance
  publication-title: J Clin Invest
  doi: 10.1172/JCI117936
– volume: 342
  start-page: d1732
  year: 2011
  ident: CR20
  article-title: Investigation and management of unintentional weight loss in older adults
  publication-title: Br Med J
  doi: 10.1136/bmj.d1732
– ident: CR35
– volume: 13
  start-page: 17
  year: 2012
  end-page: 26
  ident: CR9
  article-title: Mechanisms linking obesity to hypertension
  publication-title: Obes Rev
  doi: 10.1111/j.1467-789X.2011.00914.x
– volume: 31A
  start-page: 1822
  year: 1995
  end-page: 1829
  ident: CR17
  article-title: Cancer incidence in The Netherlands in 1989 and 1990: first results of the nationwide Netherlands cancer registry. Coordinating Committee for Regional Cancer Registries
  publication-title: Eur J Cancer
  doi: 10.1016/0959-8049(95)00355-M
– volume: 45
  start-page: 356
  year: 2005
  end-page: 362
  ident: CR25
  article-title: Weight loss and the renin-angiotensin-aldosterone system
  publication-title: Hypertension
  doi: 10.1161/01.HYP.0000154361.47683.d3
– volume: 212
  start-page: 274
  year: 2010
  end-page: 280
  ident: CR27
  article-title: Increased visceral adipose tissue mass is associated with increased C-reactive protein in patients with manifest vascular diseases
  publication-title: Atherosclerosis
  doi: 10.1016/j.atherosclerosis.2010.04.029
– volume: 26
  start-page: 1603
  year: 2015
  end-page: 1616
  ident: CR31
  article-title: The effects of height and BMI on prostate cancer incidence and mortality: a Mendelian randomization study in 20,848 cases and 20,214 controls from the PRACTICAL consortium
  publication-title: Cancer Causes Control
  doi: 10.1007/s10552-015-0654-9
– volume: 63
  start-page: 2985
  issue: 25 Pt B
  year: 2014
  end-page: 3023
  ident: CR1
  article-title: 2013 AHA/ACC/TOS guideline for the management of overweight and obesity in adults: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines and The Obesity Society
  publication-title: J Am Coll Cardiol
  doi: 10.1016/j.jacc.2013.11.004
– volume: 16
  start-page: 416
  year: 2015
  end-page: 423
  ident: CR4
  article-title: A dose-response meta-analysis of the impact of body mass index on stroke and all-cause mortality in stroke patients: a paradox within a paradox
  publication-title: Obes Rev
  doi: 10.1111/obr.12272
– volume: 854
  start-page: 1
  year: 1995
  end-page: 452
  ident: CR32
  article-title: Report of a WHO expert committee
  publication-title: World Health Organ Tech Rep Ser
– volume: 27
  start-page: 525
  year: 2016
  end-page: 530
  ident: CR36
  article-title: Collider bias is only a partial explanation for the obesity paradox
  publication-title: Epidemiology
  doi: 10.1097/EDE.0000000000000493
– volume: 55
  start-page: 2622
  year: 2012
  end-page: 2630
  ident: CR12
  article-title: The ratio of visceral to subcutaneous fat, a metric of body fat distribution, is a unique correlate of cardiometabolic risk
  publication-title: Diabetologia
  doi: 10.1007/s00125-012-2639-5
– volume: 46
  start-page: 1595
  year: 2015
  end-page: 1600
  ident: CR7
  article-title: Is blood pressure control for stroke prevention the correct goal? The lost opportunity of preventing hypertension
  publication-title: Stroke
  doi: 10.1161/STROKEAHA.115.009128
– volume: 17
  start-page: 361
  year: 2016
  end-page: 376
  ident: CR28
  article-title: A role for novel adipose tissue-secreted factors in obesity-related carcinogenesis
  publication-title: Obes Rev
  doi: 10.1111/obr.12377
– volume: 30
  start-page: 2331
  year: 2012
  end-page: 2337
  ident: CR10
  article-title: Relation between adiposity and hypertension persists after onset of clinically manifest arterial disease
  publication-title: J Hypertens
  doi: 10.1097/HJH.0b013e328357f18a
– volume: 71
  start-page: 978
  year: 2014
  end-page: 984
  ident: CR22
  article-title: Body mass index and death by stroke: no obesity paradox
  publication-title: JAMA Neurol
  doi: 10.1001/jamaneurol.2014.1017
– volume: 115
  start-page: 1428
  year: 2015
  end-page: 1434
  ident: CR3
  article-title: Meta-analysis of the relation of body mass index to all-cause and cardiovascular mortality and hospitalization in patients with chronic heart failure
  publication-title: Am J Cardiol
  doi: 10.1016/j.amjcard.2015.02.024
– volume: 894
  start-page: i
  year: 2000
  end-page: xii
  ident: CR18
  article-title: Report of a WHO consultation
  publication-title: World Health Organ Tech Rep Ser
– volume: 241
  start-page: 547
  year: 2015
  end-page: 554
  ident: CR11
  article-title: Individual contributions of visceral fat and total body fat to subclinical atherosclerosis: the NEO study
  publication-title: Atherosclerosis
  doi: 10.1016/j.atherosclerosis.2015.05.026
– volume: 54
  start-page: 84
  year: 2009
  end-page: 90
  ident: CR26
  article-title: Does greater adiposity increase blood pressure and hypertension risk? Mendelian randomization using the FTO/MC4R genotype
  publication-title: Hypertension
  doi: 10.1161/HYPERTENSIONAHA.109.130005
– volume: 19
  start-page: 402
  year: 2011
  end-page: 408
  ident: CR29
  article-title: The relationship of waist circumference and BMI to visceral, subcutaneous, and total body fat: sex and race differences
  publication-title: Obesity
  doi: 10.1038/oby.2010.248
– volume: 23
  start-page: 2485
  year: 2015
  end-page: 2490
  ident: CR5
  article-title: Smoking and reverse causation create an obesity paradox in cardiovascular disease
  publication-title: Obesity
  doi: 10.1002/oby.21239
– volume: 25
  start-page: 560
  issue: 4 Pt 1
  year: 1995
  end-page: 563
  ident: CR24
  article-title: Sympathetic activation in obese normotensive subjects
  publication-title: Hypertension
  doi: 10.1161/01.HYP.25.4.560
– volume: 7
  start-page: 709
  year: 2014
  end-page: 716
  ident: CR19
  article-title: Arterial stiffness, central pressures, and incident hospitalized heart failure in the chronic renal insufficiency cohort study
  publication-title: Circ Heart Failure
  doi: 10.1161/CIRCHEARTFAILURE.113.001041
– volume: 77
  start-page: 857
  year: 2003
  end-page: 860
  ident: CR16
  article-title: Secondary Manifestations of Arterial Disease Study G. Ultrasound measurements of intraabdominal fat estimate the metabolic syndrome better than do measurements of waist circumference
  publication-title: Am J Clin Nutr
  doi: 10.1093/ajcn/77.4.857
– volume: 18
  start-page: 2569
  year: 2009
  end-page: 2578
  ident: CR6
  article-title: Obesity and cancer: the role of dysfunctional adipose tissue
  publication-title: Cancer Epidemiol Biomarkers Prev
  doi: 10.1158/1055-9965.EPI-09-0372
– volume: 15
  start-page: 773
  year: 1999
  end-page: 781
  ident: CR15
  article-title: Second manifestations of ARTerial disease (SMART) study: rationale and design
  publication-title: Eur J Epidemiol
  doi: 10.1023/A:1007621514757
– volume: 14
  start-page: CR15
  year: 2008
  end-page: CR18
  ident: CR34
  article-title: Intraobserver and interobserver variability of measuring waist circumference
  publication-title: Med Sci Monit
– volume: 36
  start-page: 695
  year: 2012
  end-page: 702
  ident: CR14
  article-title: The risk of general and abdominal adiposity in the occurrence of new vascular events and mortality in patients with various manifestations of vascular disease
  publication-title: Int J Obes
  doi: 10.1038/ijo.2011.115
– volume: 241
  start-page: 547
  year: 2015
  ident: BFijo2017184_CR11
  publication-title: Atherosclerosis
  doi: 10.1016/j.atherosclerosis.2015.05.026
– volume: 342
  start-page: d1732
  year: 2011
  ident: BFijo2017184_CR20
  publication-title: Br Med J
  doi: 10.1136/bmj.d1732
– volume: 26
  start-page: 1603
  year: 2015
  ident: BFijo2017184_CR31
  publication-title: Cancer Causes Control
  doi: 10.1007/s10552-015-0654-9
– volume: 16
  start-page: 416
  year: 2015
  ident: BFijo2017184_CR4
  publication-title: Obes Rev
  doi: 10.1111/obr.12272
– volume: 15
  start-page: 773
  year: 1999
  ident: BFijo2017184_CR15
  publication-title: Eur J Epidemiol
  doi: 10.1023/A:1007621514757
– volume: 71
  start-page: 978
  year: 2014
  ident: BFijo2017184_CR22
  publication-title: JAMA Neurol
  doi: 10.1001/jamaneurol.2014.1017
– volume: 212
  start-page: 274
  year: 2010
  ident: BFijo2017184_CR27
  publication-title: Atherosclerosis
  doi: 10.1016/j.atherosclerosis.2010.04.029
– volume: 19
  start-page: 402
  year: 2011
  ident: BFijo2017184_CR29
  publication-title: Obesity
  doi: 10.1038/oby.2010.248
– volume: 371
  start-page: 569
  year: 2008
  ident: BFijo2017184_CR30
  publication-title: Lancet
  doi: 10.1016/S0140-6736(08)60269-X
– volume: 9
  start-page: 312
  year: 2008
  ident: BFijo2017184_CR33
  publication-title: Obes Rev
  doi: 10.1111/j.1467-789X.2007.00411.x
– volume: 36
  start-page: 2716
  year: 2015
  ident: BFijo2017184_CR2
  publication-title: Eur Heart J
  doi: 10.1093/eurheartj/ehv347
– volume: 894
  start-page: i
  year: 2000
  ident: BFijo2017184_CR18
  publication-title: World Health Organ Tech Rep Ser
– volume: 13
  start-page: 17
  year: 2012
  ident: BFijo2017184_CR9
  publication-title: Obes Rev
  doi: 10.1111/j.1467-789X.2011.00914.x
– volume: 25
  start-page: 560
  issue: 4 Pt 1
  year: 1995
  ident: BFijo2017184_CR24
  publication-title: Hypertension
  doi: 10.1161/01.HYP.25.4.560
– volume: 36
  start-page: 695
  year: 2012
  ident: BFijo2017184_CR14
  publication-title: Int J Obes
  doi: 10.1038/ijo.2011.115
– volume: 31A
  start-page: 1822
  year: 1995
  ident: BFijo2017184_CR17
  publication-title: Eur J Cancer
  doi: 10.1016/0959-8049(95)00355-M
– volume: 34
  start-page: 268
  year: 2013
  ident: BFijo2017184_CR21
  publication-title: Eur Heart J
  doi: 10.1093/eurheartj/ehs340
– volume: 854
  start-page: 1
  year: 1995
  ident: BFijo2017184_CR32
  publication-title: World Health Organ Tech Rep Ser
– volume: 95
  start-page: 2409
  year: 1995
  ident: BFijo2017184_CR23
  publication-title: J Clin Invest
  doi: 10.1172/JCI117936
– volume: 14
  start-page: CR15
  year: 2008
  ident: BFijo2017184_CR34
  publication-title: Med Sci Monit
– volume: 18
  start-page: 2569
  year: 2009
  ident: BFijo2017184_CR6
  publication-title: Cancer Epidemiol Biomarkers Prev
  doi: 10.1158/1055-9965.EPI-09-0372
– volume: 77
  start-page: 857
  year: 2003
  ident: BFijo2017184_CR16
  publication-title: Am J Clin Nutr
  doi: 10.1093/ajcn/77.4.857
– volume: 55
  start-page: 2622
  year: 2012
  ident: BFijo2017184_CR12
  publication-title: Diabetologia
  doi: 10.1007/s00125-012-2639-5
– volume: 54
  start-page: 84
  year: 2009
  ident: BFijo2017184_CR26
  publication-title: Hypertension
  doi: 10.1161/HYPERTENSIONAHA.109.130005
– volume: 63
  start-page: 2985
  issue: 25 Pt B
  year: 2014
  ident: BFijo2017184_CR1
  publication-title: J Am Coll Cardiol
  doi: 10.1016/j.jacc.2013.11.004
– ident: BFijo2017184_CR35
– volume: 61
  start-page: 646
  year: 2008
  ident: BFijo2017184_CR8
  publication-title: J Clin Epidemiol
  doi: 10.1016/j.jclinepi.2007.08.012
– volume: 17
  start-page: 361
  year: 2016
  ident: BFijo2017184_CR28
  publication-title: Obes Rev
  doi: 10.1111/obr.12377
– volume: 27
  start-page: 525
  year: 2016
  ident: BFijo2017184_CR36
  publication-title: Epidemiology
  doi: 10.1097/EDE.0000000000000493
– volume: 46
  start-page: 1595
  year: 2015
  ident: BFijo2017184_CR7
  publication-title: Stroke
  doi: 10.1161/STROKEAHA.115.009128
– volume: 23
  start-page: 2485
  year: 2015
  ident: BFijo2017184_CR5
  publication-title: Obesity
  doi: 10.1002/oby.21239
– volume: 7
  start-page: 709
  year: 2014
  ident: BFijo2017184_CR19
  publication-title: Circ Heart Failure
  doi: 10.1161/CIRCHEARTFAILURE.113.001041
– volume: 30
  start-page: 2331
  year: 2012
  ident: BFijo2017184_CR10
  publication-title: J Hypertens
  doi: 10.1097/HJH.0b013e328357f18a
– volume: 115
  start-page: 1428
  year: 2015
  ident: BFijo2017184_CR3
  publication-title: Am J Cardiol
  doi: 10.1016/j.amjcard.2015.02.024
– volume: 315
  start-page: 1989
  year: 2016
  ident: BFijo2017184_CR13
  publication-title: JAMA
  doi: 10.1001/jama.2016.4666
– volume: 45
  start-page: 356
  year: 2005
  ident: BFijo2017184_CR25
  publication-title: Hypertension
  doi: 10.1161/01.HYP.0000154361.47683.d3
– reference: 25953369 - Stroke. 2015 Jun;46(6):1595-600
– reference: 24886975 - JAMA Neurol. 2014 Aug;71(8):978-84
– reference: 27075676 - Epidemiology. 2016 Jul;27(4):525-30
– reference: 20948514 - Obesity (Silver Spring). 2011 Feb;19(2):402-8
– reference: 23076781 - Eur Heart J. 2013 Jan;34(4):268-77
– reference: 26242715 - Eur Heart J. 2015 Oct 21;36(40):2716-28
– reference: 27163987 - JAMA. 2016 May 10;315(18):1989-96
– reference: 18280327 - Lancet. 2008 Feb 16;371(9612):569-78
– reference: 8541107 - Eur J Cancer. 1995 Oct;31A(11):1822-9
– reference: 26387087 - Cancer Causes Control. 2015 Nov;26(11):1603-16
– reference: 25753533 - Obes Rev. 2015 May;16(5):416-23
– reference: 26421898 - Obesity (Silver Spring). 2015 Dec;23(12):2485-90
– reference: 12663283 - Am J Clin Nutr. 2003 Apr;77(4):857-60
– reference: 19755644 - Cancer Epidemiol Biomarkers Prev. 2009 Oct;18(10):2569-78
– reference: 17956544 - Obes Rev. 2008 Jul;9(4):312-25
– reference: 7738205 - J Clin Invest. 1995 May;95(5):2409-15
– reference: 18160939 - Med Sci Monit. 2008 Jan;14(1):CR15-18
– reference: 8594834 - World Health Organ Tech Rep Ser. 1995;854:1-452
– reference: 18359190 - J Clin Epidemiol. 2008 Jul;61(7):646-53
– reference: 26100677 - Atherosclerosis. 2015 Aug;241(2):547-54
– reference: 15630041 - Hypertension. 2005 Mar;45(3):356-62
– reference: 25059422 - Circ Heart Fail. 2014 Sep;7(5):709-16
– reference: 22898763 - Diabetologia. 2012 Oct;55(10 ):2622-2630
– reference: 23041751 - J Hypertens. 2012 Dec;30(12):2331-7
– reference: 21831233 - Obes Rev. 2012 Jan;13(1):17-26
– reference: 11234459 - World Health Organ Tech Rep Ser. 2000;894:i-xii, 1-253
– reference: 20494358 - Atherosclerosis. 2010 Sep;212(1):274-80
– reference: 21447571 - BMJ. 2011 Mar 29;342:d1732
– reference: 21712810 - Int J Obes (Lond). 2012 May;36(5):695-702
– reference: 26914773 - Obes Rev. 2016 Apr;17 (4):361-76
– reference: 19470880 - Hypertension. 2009 Jul;54(1):84-90
– reference: 25772740 - Am J Cardiol. 2015 May 15;115(10):1428-34
– reference: 10608355 - Eur J Epidemiol. 1999 Oct;15(9):773-81
– reference: 24239920 - J Am Coll Cardiol. 2014 Jul 1;63(25 Pt B):2985-3023
– reference: 7721398 - Hypertension. 1995 Apr;25(4 Pt 1):560-3
SSID ssj0005502
ssj0033214
Score 2.2921183
Snippet Background: Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable...
Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable...
Background: Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable...
Background:Abdominal adiposity is associated with various risk factors including hypertension, and is therefore particularly relevant in patients with stable...
SourceID proquest
gale
pubmed
crossref
springer
SourceType Aggregation Database
Index Database
Enrichment Source
Publisher
StartPage 1775
SubjectTerms 692/308/174
692/499
692/699/2743/393
692/699/75/593/1370
692/700/139/2818
692/700/2817
692/700/459
Abdomen
Adipose tissue
Adiposity - physiology
Adult
Aged
Body Mass Index
Body size
Cardiovascular diseases
Cause of Death - trends
Cerebrovascular disease
Cerebrovascular diseases
Cerebrovascular Disorders - etiology
Cerebrovascular Disorders - mortality
Cerebrovascular Disorders - physiopathology
Chi-square test
Complications and side effects
Confidence intervals
Epidemiology
Female
Hazard assessment
Health Promotion and Disease Prevention
Health risk assessment
Humans
Hypertension
Hypertension - etiology
Hypertension - mortality
Hypertension - physiopathology
Internal Medicine
Male
Malignancy
Medicine
Medicine & Public Health
Metabolic Diseases
Middle Aged
Mortality
Neoplasms - mortality
Neoplasms - physiopathology
Nonlinear systems
Nonlinearity
Obesity
Obesity, Abdominal - complications
Obesity, Abdominal - mortality
Obesity, Abdominal - physiopathology
original-article
Patient outcomes
Patients
Prognosis
Proportional Hazards Models
Prospective Studies
Public Health
Reference Values
Risk analysis
Risk Factors
Statistical analysis
Statistical models
Statistical tests
Ultrasound
Waist Circumference
Young Adult
Title Relation between adiposity and vascular events, malignancy and mortality in patients with stable cerebrovascular disease
URI https://link.springer.com/article/10.1038/ijo.2017.184
https://www.ncbi.nlm.nih.gov/pubmed/28775374
https://www.proquest.com/docview/1972274892
https://www.proquest.com/docview/2615529612
https://www.proquest.com/docview/1926683382
Volume 41
WOSCitedRecordID wos000417032800008&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
hasFullText 1
inHoldings 1
isFullTextHit
isPrint
journalDatabaseRights – providerCode: PRVPQU
  databaseName: Agricultural Science Database
  customDbUrl:
  eissn: 1476-5497
  dateEnd: 20171231
  omitProxy: false
  ssIdentifier: ssj0005502
  issn: 0307-0565
  databaseCode: M0K
  dateStart: 19970201
  isFulltext: true
  titleUrlDefault: https://search.proquest.com/agriculturejournals
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: Biological Science Database
  customDbUrl:
  eissn: 1476-5497
  dateEnd: 20171231
  omitProxy: false
  ssIdentifier: ssj0005502
  issn: 0307-0565
  databaseCode: M7P
  dateStart: 19970201
  isFulltext: true
  titleUrlDefault: http://search.proquest.com/biologicalscijournals
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: Health & Medical Collection
  customDbUrl:
  eissn: 1476-5497
  dateEnd: 20171231
  omitProxy: false
  ssIdentifier: ssj0005502
  issn: 0307-0565
  databaseCode: 7X7
  dateStart: 20050101
  isFulltext: true
  titleUrlDefault: https://search.proquest.com/healthcomplete
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: ProQuest Central
  customDbUrl:
  eissn: 1476-5497
  dateEnd: 20171231
  omitProxy: false
  ssIdentifier: ssj0005502
  issn: 0307-0565
  databaseCode: BENPR
  dateStart: 20050101
  isFulltext: true
  titleUrlDefault: https://www.proquest.com/central
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: Psychology Database
  customDbUrl:
  eissn: 1476-5497
  dateEnd: 20171231
  omitProxy: false
  ssIdentifier: ssj0005502
  issn: 0307-0565
  databaseCode: M2M
  dateStart: 20050101
  isFulltext: true
  titleUrlDefault: https://www.proquest.com/psychology
  providerName: ProQuest
– providerCode: PRVPQU
  databaseName: Public Health Database
  customDbUrl:
  eissn: 1476-5497
  dateEnd: 20171231
  omitProxy: false
  ssIdentifier: ssj0005502
  issn: 0307-0565
  databaseCode: 8C1
  dateStart: 19970201
  isFulltext: true
  titleUrlDefault: https://search.proquest.com/publichealth
  providerName: ProQuest
link http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwpV3db9MwED-xDSFe-ChfhVGMBOMBwpK4TuwnNMYmJNSqQiD1LUpcGxV16Wg6BP89d46TtlMHD7xYanxxE9s5_853_h3AC6FTExtbBErIMOjnVgZS6jBQiZZSFBqn2MQlm0iHQzkeq5HfcKt8WGWjE52insw17ZEfUnqsmKhS4nfnPwLKGkXeVZ9CYwf2iCWBu9C90SrEQ4QtlxSnjDwNKyKu-sJHwYdcHk6_0zHAKH0byf7G-nRZS68tU5f8pm45Or39vy9yB255IMqO6plzF66ZsgPdD1OzZAfMs4XO2LAh6-_AjYF3w3egswKbKOwSpVf34FcTWMd88BfLJ1MXFPab5eWENUGvzJFGVW_YGdoA34jwo64_c5YASU9L5vleK0YbxQwhbDEzTJsFmvBt9Czz3qX78PX05Mvxx8Andgi0kNEyKEyS5LGOdCy10lYKY1OElZabPBcqD03MtYi0nfBCGrT2BcJEYfEyopOCc8sfwG45L80jYCq0ylpEVQLtPqVzZbhNFGXh6kvEhkkXXjfDmWnPek7JN2aZ875zmeHgZzT4GQ5-F1620uc128cVcs9oZmT1WdVWSWRHxPuNGDXlV0okOPCICBLVhVdOghQJPpHO_XkIfC-i5Npo6x-Sa23ub0iiqtAbDW2rXr-7mbGZ12RVtpquW6tj8mvHCnFyF5631dQwBe-VZn5BTSAKlJxLlHlYfz9t76LBjgZzij160HxQa_-9pesf__0hn8BNkqwDjvZhd7m4ME_huv65nFaLHuyk49SVEkt5HPVg7_3JcPQZfw3CT1TGg55TIn8AaQ1wfA
linkProvider ProQuest
linkToHtml http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMw1V3db9MwED-NgYAXPspXYTAjse0BwlK7TuwHhCbGtGlrxcOQ9hYS10ZFXTqaDtg_xd_IXeKk7bTB0x54jS-X1L0738U_3w_glTSx5dZlgZYqDLqpU4FSJgx0ZJSSmUETG5RkE3G_r46O9Kcl-F2fhSFYZR0Ty0A9GBv6Rr5J9FicWqXw9yffA2KNot3VmkKjMot9e_YTS7bi3d42_r9rnO98PPywG3hWgcBI1ZkGmY2ilJuO4cpo45S0LsacxgmbplKnoeXCyI5xA5Epi6WmxBxFOryMS2MmhBOo9xpc72IlRFQRPd6bQUpk2PSuEsQAVHdhxCxDetR9KNTm8BsdO-zEbzuqu7Aenl8V5pbFc_u05fK3c_d_m7h7cMcn2myr8oz7sGTzFrS3h3bK1pnvhjpi_ZqMoAU3ex5m0ILWLJlG4ZIIvngAv2rgIPPgNpYOhiXo7Yyl-YDVoF5WNsUq3rBjrHG-UkOTavy4rHRIepgz38-2YPQhnGGKno0sM3Zis0mDDmZ-9-whfL6SmXoEy_k4t0-A6dBp5zBrlFjXapNqK1ykiWWsqzD3jdrwujafxPiu7kQuMkpKdIFQCRpbQsaWoLG1Ya2RPqm6mVwit0qWmFRncZsgmGxRX3PMwWNxqUSEhoYZT6TbsFFKUKDENzKpP--Bv4taji3o-ofknM6VBUkMhWZB0UXD83fXHpL4SF0kM_e4cJjTvj3XWAe04WUzTIoJnJjb8SmpwCxXCaFQ5nHlr83schXHUsQ4o-u1A889-4Kpf_r3l1yFW7uHvYPkYK-__wxu010VuGoFlqeTU_scbpgf02ExeVGGJgZfrtql_wCkC8k_
linkToPdf http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMw1V3db9MwED-NgSZe-ChfhcGMxMYDhKZ2ndgPCE0bFdOg2gNIe8sS10ZFXTqaDti_xl_HXeJk7dTB0x54jS-X1L07_y7--Q7ghTSx5dZlgZYqDHqpU4FSJgx0ZJSSmUETG5bNJuLBQB0e6oMV-F2fhSFaZR0Ty0A9nBj6Rt6h9licSqXwjvO0iIPd_ruT7wF1kKKd1rqdRmUi-_bsJ6Zvxdu9XfyvNznvv_-88yHwHQYCI1V3FmQ2ilJuuoYro41T0roY8Y0TNk2lTkPLhZFd44YiUxbTTol4RTq8jMtkJoQTqPcaXI8FWjGdUt-Zo5fIsKljJagbUF2RERGH9Az8UKjO6BsdQezGb7qqt7A2Xlwh5pbIC3u25VLYv_0_T-IduOUBONuuPOYurNi8Be3dkZ2xLearpI7ZoG5S0IK1T55-0ILWOchG4bJBfHEPftWEQuZJbywdjkoy3BlL8yGryb6sLJZVvGbHmPt8pUIn1fhxmQGR9Chnvs5twegDOUPono0tM3Zqs2nDGmZ-V-0-fLmSmXoAq_kkt4-A6dBp5xBNSsx3tUm1FS7S1H2spxATR214VZtSYny1d2o6Mk5K1oFQCRpeQoaXoOG1YbORPqmqnFwit0FWmVRndJvgmGxTvXPE5rG4VCJCo0MkFOk2vCwlKIDiG5nUnwPB30WlyBZ0_UNyTuf6giSGSLOgaNnw_N21tyQ-ghfJuassHea0n8815gdteN4Mk2IiLeZ2ckoqEP0qIRTKPKx8t5ldruJYihhndKt25rlnL5n6x39_yQ1YQ09OPu4N9p_ATbqp4lytw-psemqfwg3zYzYqps_KKMXg6Ko9-g9rodHJ
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Relation+between+adiposity+and+vascular+events%2C+malignancy+and+mortality+in+patients+with+stable+cerebrovascular+disease&rft.jtitle=International+journal+of+obesity+%282005%29&rft.au=Jaspers%2C+N+E+M&rft.au=Dorresteijn%2C+J+A+N&rft.au=van+der+Graaf%2C+Y&rft.au=Westerink%2C+J&rft.date=2017-12-01&rft.issn=1476-5497&rft.eissn=1476-5497&rft.volume=41&rft.issue=12&rft.spage=1775&rft_id=info:doi/10.1038%2Fijo.2017.184&rft.externalDBID=NO_FULL_TEXT
thumbnail_l http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=0307-0565&client=summon
thumbnail_m http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=0307-0565&client=summon
thumbnail_s http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=0307-0565&client=summon