Reprogramming of the circadian clock by nutritional challenge

Circadian rhythms and cellular metabolism are intimately linked. Here, we reveal that a high-fat diet (HFD) generates a profound reorganization of specific metabolic pathways, leading to widespread remodeling of the liver clock. Strikingly, in addition to disrupting the normal circadian cycle, HFD c...

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Veröffentlicht in:Cell Jg. 155; H. 7; S. 1464
Hauptverfasser: Eckel-Mahan, Kristin L, Patel, Vishal R, de Mateo, Sara, Orozco-Solis, Ricardo, Ceglia, Nicholas J, Sahar, Saurabh, Dilag-Penilla, Sherry A, Dyar, Kenneth A, Baldi, Pierre, Sassone-Corsi, Paolo
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Sprache:Englisch
Veröffentlicht: United States 19.12.2013
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ISSN:1097-4172, 1097-4172
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Abstract Circadian rhythms and cellular metabolism are intimately linked. Here, we reveal that a high-fat diet (HFD) generates a profound reorganization of specific metabolic pathways, leading to widespread remodeling of the liver clock. Strikingly, in addition to disrupting the normal circadian cycle, HFD causes an unexpectedly large-scale genesis of de novo oscillating transcripts, resulting in reorganization of the coordinated oscillations between coherent transcripts and metabolites. The mechanisms underlying this reprogramming involve both the impairment of CLOCK:BMAL1 chromatin recruitment and a pronounced cyclic activation of surrogate pathways through the transcriptional regulator PPARγ. Finally, we demonstrate that it is specifically the nutritional challenge, and not the development of obesity, that causes the reprogramming of the clock and that the effects of the diet on the clock are reversible.
AbstractList Circadian rhythms and cellular metabolism are intimately linked. Here, we reveal that a high-fat diet (HFD) generates a profound reorganization of specific metabolic pathways, leading to widespread remodeling of the liver clock. Strikingly, in addition to disrupting the normal circadian cycle, HFD causes an unexpectedly large-scale genesis of de novo oscillating transcripts, resulting in reorganization of the coordinated oscillations between coherent transcripts and metabolites. The mechanisms underlying this reprogramming involve both the impairment of CLOCK:BMAL1 chromatin recruitment and a pronounced cyclic activation of surrogate pathways through the transcriptional regulator PPARγ. Finally, we demonstrate that it is specifically the nutritional challenge, and not the development of obesity, that causes the reprogramming of the clock and that the effects of the diet on the clock are reversible.Circadian rhythms and cellular metabolism are intimately linked. Here, we reveal that a high-fat diet (HFD) generates a profound reorganization of specific metabolic pathways, leading to widespread remodeling of the liver clock. Strikingly, in addition to disrupting the normal circadian cycle, HFD causes an unexpectedly large-scale genesis of de novo oscillating transcripts, resulting in reorganization of the coordinated oscillations between coherent transcripts and metabolites. The mechanisms underlying this reprogramming involve both the impairment of CLOCK:BMAL1 chromatin recruitment and a pronounced cyclic activation of surrogate pathways through the transcriptional regulator PPARγ. Finally, we demonstrate that it is specifically the nutritional challenge, and not the development of obesity, that causes the reprogramming of the clock and that the effects of the diet on the clock are reversible.
Circadian rhythms and cellular metabolism are intimately linked. Here, we reveal that a high-fat diet (HFD) generates a profound reorganization of specific metabolic pathways, leading to widespread remodeling of the liver clock. Strikingly, in addition to disrupting the normal circadian cycle, HFD causes an unexpectedly large-scale genesis of de novo oscillating transcripts, resulting in reorganization of the coordinated oscillations between coherent transcripts and metabolites. The mechanisms underlying this reprogramming involve both the impairment of CLOCK:BMAL1 chromatin recruitment and a pronounced cyclic activation of surrogate pathways through the transcriptional regulator PPARγ. Finally, we demonstrate that it is specifically the nutritional challenge, and not the development of obesity, that causes the reprogramming of the clock and that the effects of the diet on the clock are reversible.
Author Sassone-Corsi, Paolo
Sahar, Saurabh
Patel, Vishal R
Dyar, Kenneth A
Eckel-Mahan, Kristin L
Baldi, Pierre
Orozco-Solis, Ricardo
Ceglia, Nicholas J
de Mateo, Sara
Dilag-Penilla, Sherry A
Author_xml – sequence: 1
  givenname: Kristin L
  surname: Eckel-Mahan
  fullname: Eckel-Mahan, Kristin L
  organization: Center for Epigenetics and Metabolism, Department of Biological Chemistry, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 2
  givenname: Vishal R
  surname: Patel
  fullname: Patel, Vishal R
  organization: Institute for Genomics and Bioinformatics, School of Information and Computer Sciences, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 3
  givenname: Sara
  surname: de Mateo
  fullname: de Mateo, Sara
  organization: Center for Epigenetics and Metabolism, Department of Biological Chemistry, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 4
  givenname: Ricardo
  surname: Orozco-Solis
  fullname: Orozco-Solis, Ricardo
  organization: Center for Epigenetics and Metabolism, Department of Biological Chemistry, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 5
  givenname: Nicholas J
  surname: Ceglia
  fullname: Ceglia, Nicholas J
  organization: Institute for Genomics and Bioinformatics, School of Information and Computer Sciences, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 6
  givenname: Saurabh
  surname: Sahar
  fullname: Sahar, Saurabh
  organization: Center for Epigenetics and Metabolism, Department of Biological Chemistry, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 7
  givenname: Sherry A
  surname: Dilag-Penilla
  fullname: Dilag-Penilla, Sherry A
  organization: Center for Epigenetics and Metabolism, Department of Biological Chemistry, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 8
  givenname: Kenneth A
  surname: Dyar
  fullname: Dyar, Kenneth A
  organization: Venetian Institute of Molecular Medicine, Padova 35129, Italy
– sequence: 9
  givenname: Pierre
  surname: Baldi
  fullname: Baldi, Pierre
  organization: Institute for Genomics and Bioinformatics, School of Information and Computer Sciences, University of California, Irvine, Irvine, CA 92697, USA
– sequence: 10
  givenname: Paolo
  surname: Sassone-Corsi
  fullname: Sassone-Corsi, Paolo
  email: psc@uci.edu
  organization: Center for Epigenetics and Metabolism, Department of Biological Chemistry, University of California, Irvine, Irvine, CA 92697, USA. Electronic address: psc@uci.edu
BackLink https://www.ncbi.nlm.nih.gov/pubmed/24360271$$D View this record in MEDLINE/PubMed
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References 24590185 - Nat Rev Endocrinol. 2014 Apr;10(4):191-2
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Snippet Circadian rhythms and cellular metabolism are intimately linked. Here, we reveal that a high-fat diet (HFD) generates a profound reorganization of specific...
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SubjectTerms Animals
ARNTL Transcription Factors - metabolism
Circadian Clocks
Circadian Rhythm
CLOCK Proteins - metabolism
Diet, High-Fat
Gene Expression Regulation
Male
Metabolic Networks and Pathways
Mice
Mice, Inbred C57BL
Obesity - metabolism
PPAR gamma - metabolism
Transcriptome
Title Reprogramming of the circadian clock by nutritional challenge
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