TREM1/3 Deficiency Impairs Tissue Repair After Acute Kidney Injury and Mitochondrial Metabolic Flexibility in Tubular Epithelial Cells

Long-term sequelae of acute kidney injury (AKI) are associated with incomplete recovery of renal function and the development of chronic kidney disease (CKD), which can be mediated by aberrant innate immune activation, mitochondrial pathology, and accumulation of senescent tubular epithelial cells (...

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Vydané v:	Frontiers in immunology Ročník 10; s. 1469
Hlavní autori:	Tammaro, Alessandra, Scantlebery, Angelique M. L., Rampanelli, Elena, Borrelli, Cristiana, Claessen, Nike, Butter, Loes M., Soriani, Alessandra, Colonna, Marco, Leemans, Jaklien C., Dessing, Mark C., Florquin, Sandrine
Médium:	Journal Article
Jazyk:	English
Vydavateľské údaje:	Switzerland Frontiers Media S.A 09.07.2019
Predmet:	epithelial innate immunity Immunology ischemia/reperfusion injury maladaptive repair mitochondrial metabolism renal repair tubular cell senescence tubular cell senescence maladaptive repair renal repair epithelial innate immunity ischemia/reperfusion injury mitochondrial metabolism
ISSN:	1664-3224, 1664-3224
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