Cardiolipin Synthesis in Brown and Beige Fat Mitochondria Is Essential for Systemic Energy Homeostasis

Activation of energy expenditure in thermogenic fat is a promising strategy to improve metabolic health, yet the dynamic processes that evoke this response are poorly understood. Here we show that synthesis of the mitochondrial phospholipid cardiolipin is indispensable for stimulating and sustaining...

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Vydáno v:Cell metabolism Ročník 28; číslo 1; s. 159
Hlavní autoři: Sustarsic, Elahu G, Ma, Tao, Lynes, Matthew D, Larsen, Michael, Karavaeva, Iuliia, Havelund, Jesper F, Nielsen, Carsten H, Jedrychowski, Mark P, Moreno-Torres, Marta, Lundh, Morten, Plucinska, Kaja, Jespersen, Naja Z, Grevengoed, Trisha J, Kramar, Barbara, Peics, Julia, Hansen, Jakob B, Shamsi, Farnaz, Forss, Isabel, Neess, Ditte, Keipert, Susanne, Wang, Jianing, Stohlmann, Katharina, Brandslund, Ivan, Christensen, Cramer, Jørgensen, Marit E, Linneberg, Allan, Pedersen, Oluf, Kiebish, Michael A, Qvortrup, Klaus, Han, Xianlin, Pedersen, Bente Klarlund, Jastroch, Martin, Mandrup, Susanne, Kjær, Andreas, Gygi, Steven P, Hansen, Torben, Gillum, Matthew P, Grarup, Niels, Emanuelli, Brice, Nielsen, Søren, Scheele, Camilla, Tseng, Yu-Hua, Færgeman, Nils J, Gerhart-Hines, Zachary
Médium: Journal Article
Jazyk:angličtina
Vydáno: United States 03.07.2018
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ISSN:1932-7420, 1932-7420
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Abstract Activation of energy expenditure in thermogenic fat is a promising strategy to improve metabolic health, yet the dynamic processes that evoke this response are poorly understood. Here we show that synthesis of the mitochondrial phospholipid cardiolipin is indispensable for stimulating and sustaining thermogenic fat function. Cardiolipin biosynthesis is robustly induced in brown and beige adipose upon cold exposure. Mimicking this response through overexpression of cardiolipin synthase (Crls1) enhances energy consumption in mouse and human adipocytes. Crls1 deficiency in thermogenic adipocytes diminishes inducible mitochondrial uncoupling and elicits a nuclear transcriptional response through endoplasmic reticulum stress-mediated retrograde communication. Cardiolipin depletion in brown and beige fat abolishes adipose thermogenesis and glucose uptake, which renders animals insulin resistant. We further identify a rare human CRLS1 variant associated with insulin resistance and show that adipose CRLS1 levels positively correlate with insulin sensitivity. Thus, adipose cardiolipin has a powerful impact on organismal energy homeostasis through thermogenic fat bioenergetics.
AbstractList Activation of energy expenditure in thermogenic fat is a promising strategy to improve metabolic health, yet the dynamic processes that evoke this response are poorly understood. Here we show that synthesis of the mitochondrial phospholipid cardiolipin is indispensable for stimulating and sustaining thermogenic fat function. Cardiolipin biosynthesis is robustly induced in brown and beige adipose upon cold exposure. Mimicking this response through overexpression of cardiolipin synthase (Crls1) enhances energy consumption in mouse and human adipocytes. Crls1 deficiency in thermogenic adipocytes diminishes inducible mitochondrial uncoupling and elicits a nuclear transcriptional response through endoplasmic reticulum stress-mediated retrograde communication. Cardiolipin depletion in brown and beige fat abolishes adipose thermogenesis and glucose uptake, which renders animals insulin resistant. We further identify a rare human CRLS1 variant associated with insulin resistance and show that adipose CRLS1 levels positively correlate with insulin sensitivity. Thus, adipose cardiolipin has a powerful impact on organismal energy homeostasis through thermogenic fat bioenergetics.Activation of energy expenditure in thermogenic fat is a promising strategy to improve metabolic health, yet the dynamic processes that evoke this response are poorly understood. Here we show that synthesis of the mitochondrial phospholipid cardiolipin is indispensable for stimulating and sustaining thermogenic fat function. Cardiolipin biosynthesis is robustly induced in brown and beige adipose upon cold exposure. Mimicking this response through overexpression of cardiolipin synthase (Crls1) enhances energy consumption in mouse and human adipocytes. Crls1 deficiency in thermogenic adipocytes diminishes inducible mitochondrial uncoupling and elicits a nuclear transcriptional response through endoplasmic reticulum stress-mediated retrograde communication. Cardiolipin depletion in brown and beige fat abolishes adipose thermogenesis and glucose uptake, which renders animals insulin resistant. We further identify a rare human CRLS1 variant associated with insulin resistance and show that adipose CRLS1 levels positively correlate with insulin sensitivity. Thus, adipose cardiolipin has a powerful impact on organismal energy homeostasis through thermogenic fat bioenergetics.
Activation of energy expenditure in thermogenic fat is a promising strategy to improve metabolic health, yet the dynamic processes that evoke this response are poorly understood. Here we show that synthesis of the mitochondrial phospholipid cardiolipin is indispensable for stimulating and sustaining thermogenic fat function. Cardiolipin biosynthesis is robustly induced in brown and beige adipose upon cold exposure. Mimicking this response through overexpression of cardiolipin synthase (Crls1) enhances energy consumption in mouse and human adipocytes. Crls1 deficiency in thermogenic adipocytes diminishes inducible mitochondrial uncoupling and elicits a nuclear transcriptional response through endoplasmic reticulum stress-mediated retrograde communication. Cardiolipin depletion in brown and beige fat abolishes adipose thermogenesis and glucose uptake, which renders animals insulin resistant. We further identify a rare human CRLS1 variant associated with insulin resistance and show that adipose CRLS1 levels positively correlate with insulin sensitivity. Thus, adipose cardiolipin has a powerful impact on organismal energy homeostasis through thermogenic fat bioenergetics.
Author Lundh, Morten
Stohlmann, Katharina
Grarup, Niels
Lynes, Matthew D
Havelund, Jesper F
Jespersen, Naja Z
Neess, Ditte
Wang, Jianing
Jørgensen, Marit E
Pedersen, Oluf
Jedrychowski, Mark P
Moreno-Torres, Marta
Linneberg, Allan
Peics, Julia
Pedersen, Bente Klarlund
Gerhart-Hines, Zachary
Jastroch, Martin
Kjær, Andreas
Emanuelli, Brice
Ma, Tao
Plucinska, Kaja
Larsen, Michael
Gillum, Matthew P
Kramar, Barbara
Mandrup, Susanne
Shamsi, Farnaz
Nielsen, Carsten H
Tseng, Yu-Hua
Færgeman, Nils J
Brandslund, Ivan
Sustarsic, Elahu G
Gygi, Steven P
Forss, Isabel
Hansen, Torben
Hansen, Jakob B
Keipert, Susanne
Kiebish, Michael A
Karavaeva, Iuliia
Grevengoed, Trisha J
Qvortrup, Klaus
Nielsen, Søren
Han, Xianlin
Christensen, Cramer
Scheele, Camilla
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  surname: Sustarsic
  fullname: Sustarsic, Elahu G
  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark; Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark
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  givenname: Tao
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  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark; Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark
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  surname: Lynes
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  organization: Section on Integrative Physiology and Metabolism, Joslin Diabetes Center, Harvard Medical School, Boston, MA 02215, USA
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  givenname: Michael
  surname: Larsen
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  organization: Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark
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  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark; Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark
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  surname: Nielsen
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  organization: Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark; Department of Clinical Physiology, Nuclear Medicine & PET and Cluster for Molecular Imaging, Rigshospitalet, Copenhagen 2200, Denmark
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  surname: Lundh
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  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark
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  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark
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  givenname: Naja Z
  surname: Jespersen
  fullname: Jespersen, Naja Z
  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark; Centre of Inflammation and Metabolism and Centre for Physical Activity Research, Rigshospitalet, University Hospital of Copenhagen, Copenhagen 2200, Denmark
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  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark
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  givenname: Barbara
  surname: Kramar
  fullname: Kramar, Barbara
  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark
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  givenname: Julia
  surname: Peics
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  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark; Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark
– sequence: 16
  givenname: Jakob B
  surname: Hansen
  fullname: Hansen, Jakob B
  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark; Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark
– sequence: 17
  givenname: Farnaz
  surname: Shamsi
  fullname: Shamsi, Farnaz
  organization: Section on Integrative Physiology and Metabolism, Joslin Diabetes Center, Harvard Medical School, Boston, MA 02215, USA
– sequence: 18
  givenname: Isabel
  surname: Forss
  fullname: Forss, Isabel
  organization: Department of Biochemistry and Molecular Biology, University of Southern Denmark, Odense 5230, Denmark
– sequence: 19
  givenname: Ditte
  surname: Neess
  fullname: Neess, Ditte
  organization: Department of Biochemistry and Molecular Biology, University of Southern Denmark, Odense 5230, Denmark
– sequence: 20
  givenname: Susanne
  surname: Keipert
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  organization: Helmholtz Diabetes Center and German Diabetes Center (DZD), Helmholtz Zentrum München, Neuherberg 85764, Germany
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  givenname: Jianing
  surname: Wang
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  organization: Steno Diabetes Center, Gentofte 2820, Denmark; National Institute of Public Health, Southern Denmark University, Copenhagen 1353, Denmark
– sequence: 26
  givenname: Allan
  surname: Linneberg
  fullname: Linneberg, Allan
  organization: Research Center for Prevention and Health, Glostrup 2600, Denmark; Center for Clinical Research and Prevention, Bispebjerg and Frederiksberg Hospital, Copenhagen, Denmark; Department of Clinical Medicine, University of Copenhagen, Copenhagen 2200, Denmark
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  givenname: Martin
  surname: Jastroch
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  givenname: Andreas
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  organization: Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark; Department of Clinical Physiology, Nuclear Medicine & PET and Cluster for Molecular Imaging, Rigshospitalet, Copenhagen 2200, Denmark
– sequence: 35
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  organization: Department of Cell Biology, Harvard Medical School, Boston, MA 02115, USA
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  organization: Novo Nordisk Foundation Center for Basic Metabolic Research, University of Copenhagen, Copenhagen 2200, Denmark; Department of Biomedical Sciences, University of Copenhagen, Copenhagen 2200, Denmark. Electronic address: zpg@sund.ku.dk
BackLink https://www.ncbi.nlm.nih.gov/pubmed/29861389$$D View this record in MEDLINE/PubMed
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ContentType Journal Article
Copyright Copyright © 2018 The Author(s). Published by Elsevier Inc. All rights reserved.
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Keywords beige adipose
thermogenesis
brown adipose
mitochondria
lipid metabolism
CHOP-10
phospholipids
CRLS1
cardiolipin
insulin resistance
Language English
License Copyright © 2018 The Author(s). Published by Elsevier Inc. All rights reserved.
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PublicationTitle Cell metabolism
PublicationTitleAlternate Cell Metab
PublicationYear 2018
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Snippet Activation of energy expenditure in thermogenic fat is a promising strategy to improve metabolic health, yet the dynamic processes that evoke this response are...
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StartPage 159
SubjectTerms Adipocytes - metabolism
Adipose Tissue, Beige - metabolism
Adipose Tissue, Brown - metabolism
Animals
Cardiolipins - biosynthesis
Cells, Cultured
Energy Metabolism
Humans
Membrane Proteins - genetics
Membrane Proteins - metabolism
Mice
Mice, Inbred C57BL
Mitochondria - metabolism
Thermogenesis
Transferases (Other Substituted Phosphate Groups) - genetics
Transferases (Other Substituted Phosphate Groups) - metabolism
Title Cardiolipin Synthesis in Brown and Beige Fat Mitochondria Is Essential for Systemic Energy Homeostasis
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