Deubiquitinases at the interplay between hematopoietic stem cell aging and myelodysplastic transformation

Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting p...

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Published in:FEBS letters Vol. 598; no. 22; pp. 2807 - 2808
Main Authors: Citterio, Elisabetta, Ronchi, Antonella Ellena
Format: Journal Article
Language:English
Published: England John Wiley and Sons Inc 01.11.2024
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ISSN:0014-5793, 1873-3468, 1873-3468
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Abstract Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting protein ubiquitination ensure proper proteostasis, a key process in HSC maintenance and fitness. Hematopoietic stem cell (HSC) function declines during aging, leading to a higher risk of hematopoietic malignancies. By reverting protein ubiquitination, deubiquitinating enzymes (DUBs) ensure protein homeostasis, a key process in HSC maintenance. Here, we discuss how alterations in DUBs function exacerbate HSC aging, predisposing them to myelodysplastic syndrome and leukaemia. The potential of DUBs as therapeutic targets is highlighted.
AbstractList Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting protein ubiquitination ensure proper proteostasis, a key process in HSC maintenance and fitness. Hematopoietic stem cell (HSC) function declines during aging, leading to a higher risk of hematopoietic malignancies. By reverting protein ubiquitination, deubiquitinating enzymes (DUBs) ensure protein homeostasis, a key process in HSC maintenance. Here, we discuss how alterations in DUBs function exacerbate HSC aging, predisposing them to myelodysplastic syndrome and leukaemia. The potential of DUBs as therapeutic targets is highlighted.
Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting protein ubiquitination ensure proper proteostasis, a key process in HSC maintenance and fitness.
Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting protein ubiquitination ensure proper proteostasis, a key process in HSC maintenance and fitness.Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting protein ubiquitination ensure proper proteostasis, a key process in HSC maintenance and fitness.
Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting protein ubiquitination ensure proper proteostasis, a key process in HSC maintenance and fitness. image
Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the increased prevalence of haematological diseases and hematopoietic malignancies in the elderly. Deubiquitinating enzymes (DUBs) by reverting protein ubiquitination ensure proper proteostasis, a key process in HSC maintenance and fitness. Hematopoietic stem cell (HSC) function declines during aging, leading to a higher risk of hematopoietic malignancies. By reverting protein ubiquitination, deubiquitinating enzymes (DUBs) ensure protein homeostasis, a key process in HSC maintenance. Here, we discuss how alterations in DUBs function exacerbate HSC aging, predisposing them to myelodysplastic syndrome and leukaemia. The potential of DUBs as therapeutic targets is highlighted.
Author Ronchi, Antonella Ellena
Citterio, Elisabetta
AuthorAffiliation 1 Department of Biotechnology and Biosciences University of Milano‐Bicocca 20126 Milan Italy
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Issue 22
Keywords Hematopoietic Stem Cells (HSC)
Ubiquitination
Deubiquitinating enzymes (DUBs)
Myelodysplastic Syndromes (MDS)
Aging
Deubiquitinases
Acute Myeloid Leukemia (AML)
Language English
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2024 The Author(s). FEBS Letters published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies.
This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.
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Snippet Hematopoietic stem cells (HSC) maintain blood production throughout life. Nevertheless, HSC functionality deteriorates upon physiological aging leading to the...
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pubmed
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wiley
SourceType Open Access Repository
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StartPage 2807
SubjectTerms Acute Myeloid Leukemia (AML)
Aging
Aging - metabolism
Aging - pathology
Animals
blood
Cellular Senescence
Deubiquitinases
Deubiquitinating enzymes (DUBs)
Deubiquitinating Enzymes - genetics
Deubiquitinating Enzymes - metabolism
elderly
Graphical Review
hematopoietic stem cells
Hematopoietic Stem Cells (HSC)
Hematopoietic Stem Cells - enzymology
Hematopoietic Stem Cells - metabolism
Hematopoietic Stem Cells - pathology
Humans
Myelodysplastic Syndromes (MDS)
Myelodysplastic Syndromes - enzymology
Myelodysplastic Syndromes - genetics
Myelodysplastic Syndromes - metabolism
Myelodysplastic Syndromes - pathology
Ubiquitination
ubiquitinyl hydrolase 1
Title Deubiquitinases at the interplay between hematopoietic stem cell aging and myelodysplastic transformation
URI https://onlinelibrary.wiley.com/doi/abs/10.1002%2F1873-3468.14991
https://www.ncbi.nlm.nih.gov/pubmed/39108012
https://www.proquest.com/docview/3089879881
https://www.proquest.com/docview/3154265388
https://pubmed.ncbi.nlm.nih.gov/PMC11586589
Volume 598
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