Extracellular Matrix Orchestration of Tissue Remodeling in the Chronically Inflamed Mouse Colon

Chronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing cancer. Extensive tissue remodeling is a hallmark of such illnesses, and is both a consequence and a mediator of disease progression. Despite...

Celý popis

Uložené v:
Podrobná bibliografia
Vydané v:Cellular and molecular gastroenterology and hepatology Ročník 17; číslo 4; s. 639 - 656
Hlavní autori: Moutin, Elisa B., Bons, Joanna, Giavara, Giada, Lourenco, Filipe, Pan, Deng, Burton, Jordan B., Shah, Samah, Colombé, Mathilde, Gascard, Philippe, Tlsty, Thea, Schilling, Birgit, Winton, Douglas J.
Médium: Journal Article
Jazyk:English
Vydavateľské údaje: United States Elsevier Inc 2024
Predmet:
ISSN:2352-345X, 2352-345X
On-line prístup:Získať plný text
Tagy: Pridať tag
Žiadne tagy, Buďte prvý, kto otaguje tento záznam!
Abstract Chronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing cancer. Extensive tissue remodeling is a hallmark of such illnesses, and is both a consequence and a mediator of disease progression. Despite previous characterization of epithelial and stromal remodeling during inflammatory bowel disease, a complete understanding of its impact on disease progression is lacking. A comprehensive proteomic pipeline using data-independent acquisition was applied to decellularized colon samples from the Muc2 knockout (Muc2KO) mouse model of colitis for an in-depth characterization of extracellular matrix remodeling. Unique proteomic profiles of the matrisomal landscape were extracted from prepathologic and overt colitis. Integration of proteomics and transcriptomics data sets extracted from the same murine model produced network maps describing the orchestrating role of matrisomal proteins in tissue remodeling during the progression of colitis. The in-depth proteomic workflow used here allowed the addition of 34 proteins to the known colon matrisomal signature. Protein signatures of prepathologic and pathologic colitic states were extracted, differentiating the 2 states by expression of small leucine-rich proteoglycans. We outlined the role of this class and other matrisomal proteins in tissue remodeling during colitis, as well as the potential for coordinated regulation of cell types by matrisomal ligands. Our work highlights a central role for matrisomal proteins in tissue remodeling during colitis and defines orchestrating nodes that can be exploited in the selection of therapeutic targets. [Display omitted]
AbstractList Chronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing cancer. Extensive tissue remodeling is a hallmark of such illnesses, and is both a consequence and a mediator of disease progression. Despite previous characterization of epithelial and stromal remodeling during inflammatory bowel disease, a complete understanding of its impact on disease progression is lacking.BACKGROUND & AIMSChronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing cancer. Extensive tissue remodeling is a hallmark of such illnesses, and is both a consequence and a mediator of disease progression. Despite previous characterization of epithelial and stromal remodeling during inflammatory bowel disease, a complete understanding of its impact on disease progression is lacking.A comprehensive proteomic pipeline using data-independent acquisition was applied to decellularized colon samples from the Muc2 knockout (Muc2KO) mouse model of colitis for an in-depth characterization of extracellular matrix remodeling. Unique proteomic profiles of the matrisomal landscape were extracted from prepathologic and overt colitis. Integration of proteomics and transcriptomics data sets extracted from the same murine model produced network maps describing the orchestrating role of matrisomal proteins in tissue remodeling during the progression of colitis.METHODSA comprehensive proteomic pipeline using data-independent acquisition was applied to decellularized colon samples from the Muc2 knockout (Muc2KO) mouse model of colitis for an in-depth characterization of extracellular matrix remodeling. Unique proteomic profiles of the matrisomal landscape were extracted from prepathologic and overt colitis. Integration of proteomics and transcriptomics data sets extracted from the same murine model produced network maps describing the orchestrating role of matrisomal proteins in tissue remodeling during the progression of colitis.The in-depth proteomic workflow used here allowed the addition of 34 proteins to the known colon matrisomal signature. Protein signatures of prepathologic and pathologic colitic states were extracted, differentiating the 2 states by expression of small leucine-rich proteoglycans. We outlined the role of this class and other matrisomal proteins in tissue remodeling during colitis, as well as the potential for coordinated regulation of cell types by matrisomal ligands.RESULTSThe in-depth proteomic workflow used here allowed the addition of 34 proteins to the known colon matrisomal signature. Protein signatures of prepathologic and pathologic colitic states were extracted, differentiating the 2 states by expression of small leucine-rich proteoglycans. We outlined the role of this class and other matrisomal proteins in tissue remodeling during colitis, as well as the potential for coordinated regulation of cell types by matrisomal ligands.Our work highlights a central role for matrisomal proteins in tissue remodeling during colitis and defines orchestrating nodes that can be exploited in the selection of therapeutic targets.CONCLUSIONSOur work highlights a central role for matrisomal proteins in tissue remodeling during colitis and defines orchestrating nodes that can be exploited in the selection of therapeutic targets.
Chronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing cancer. Extensive tissue remodeling is a hallmark of such illnesses, and is both a consequence and a mediator of disease progression. Despite previous characterization of epithelial and stromal remodeling during inflammatory bowel disease, a complete understanding of its impact on disease progression is lacking. A comprehensive proteomic pipeline using data-independent acquisition was applied to decellularized colon samples from the Muc2 knockout (Muc2KO) mouse model of colitis for an in-depth characterization of extracellular matrix remodeling. Unique proteomic profiles of the matrisomal landscape were extracted from prepathologic and overt colitis. Integration of proteomics and transcriptomics data sets extracted from the same murine model produced network maps describing the orchestrating role of matrisomal proteins in tissue remodeling during the progression of colitis. The in-depth proteomic workflow used here allowed the addition of 34 proteins to the known colon matrisomal signature. Protein signatures of prepathologic and pathologic colitic states were extracted, differentiating the 2 states by expression of small leucine-rich proteoglycans. We outlined the role of this class and other matrisomal proteins in tissue remodeling during colitis, as well as the potential for coordinated regulation of cell types by matrisomal ligands. Our work highlights a central role for matrisomal proteins in tissue remodeling during colitis and defines orchestrating nodes that can be exploited in the selection of therapeutic targets. [Display omitted]
Background & AimsChronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing cancer. Extensive tissue remodeling is a hallmark of such illnesses, and is both a consequence and a mediator of disease progression. Despite previous characterization of epithelial and stromal remodeling during inflammatory bowel disease, a complete understanding of its impact on disease progression still is lacking. MethodsA comprehensive proteomic pipeline using data-independent acquisition was applied to decellularized colon samples from the Muc2 knockout ( Muc2KO) mouse model of colitis for an in-depth characterization of extracellular matrix remodeling. Unique proteomic profiles of the matrisomal landscape were extracted from prepathologic and overt colitis. Integration of proteomics and transcriptomics data sets extracted from the same murine model produced network maps describing the orchestrating role of matrisomal proteins in tissue remodeling during the progression of colitis. ResultsThe in-depth proteomic workflow used here allowed the addition of 34 proteins to the known colon matrisomal signature. Protein signatures of prepathologic and pathologic colitic states were extracted, differentiating the 2 states by expression of small leucine-rich proteoglycans. We outlined the role of this class and other matrisomal proteins in tissue remodeling during colitis, as well as the potential for coordinated regulation of cell types by matrisomal ligands. ConclusionsOur work highlights a central role for matrisomal proteins in tissue remodeling during colitis and defines orchestrating nodes that can be exploited in the selection of therapeutic targets.
Chronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing cancer. Extensive tissue remodeling is a hallmark of such illnesses, and is both a consequence and a mediator of disease progression. Despite previous characterization of epithelial and stromal remodeling during inflammatory bowel disease, a complete understanding of its impact on disease progression is lacking. A comprehensive proteomic pipeline using data-independent acquisition was applied to decellularized colon samples from the Muc2 knockout (Muc2 ) mouse model of colitis for an in-depth characterization of extracellular matrix remodeling. Unique proteomic profiles of the matrisomal landscape were extracted from prepathologic and overt colitis. Integration of proteomics and transcriptomics data sets extracted from the same murine model produced network maps describing the orchestrating role of matrisomal proteins in tissue remodeling during the progression of colitis. The in-depth proteomic workflow used here allowed the addition of 34 proteins to the known colon matrisomal signature. Protein signatures of prepathologic and pathologic colitic states were extracted, differentiating the 2 states by expression of small leucine-rich proteoglycans. We outlined the role of this class and other matrisomal proteins in tissue remodeling during colitis, as well as the potential for coordinated regulation of cell types by matrisomal ligands. Our work highlights a central role for matrisomal proteins in tissue remodeling during colitis and defines orchestrating nodes that can be exploited in the selection of therapeutic targets.
Author Pan, Deng
Winton, Douglas J.
Lourenco, Filipe
Bons, Joanna
Gascard, Philippe
Shah, Samah
Colombé, Mathilde
Schilling, Birgit
Tlsty, Thea
Moutin, Elisa B.
Burton, Jordan B.
Giavara, Giada
Author_xml – sequence: 1
  givenname: Elisa B.
  orcidid: 0000-0002-9029-1520
  surname: Moutin
  fullname: Moutin, Elisa B.
  organization: Cancer Research UK Cambridge Institute, Li Ka Shing Centre, Cambridge, United Kingdom
– sequence: 2
  givenname: Joanna
  surname: Bons
  fullname: Bons, Joanna
  organization: Buck Institute for Research on Aging, Novato, California
– sequence: 3
  givenname: Giada
  surname: Giavara
  fullname: Giavara, Giada
  organization: Cancer Research UK Cambridge Institute, Li Ka Shing Centre, Cambridge, United Kingdom
– sequence: 4
  givenname: Filipe
  surname: Lourenco
  fullname: Lourenco, Filipe
  organization: Cancer Research UK Cambridge Institute, Li Ka Shing Centre, Cambridge, United Kingdom
– sequence: 5
  givenname: Deng
  surname: Pan
  fullname: Pan, Deng
  organization: Department of Pathology, University of California, San Francisco, California
– sequence: 6
  givenname: Jordan B.
  surname: Burton
  fullname: Burton, Jordan B.
  organization: Buck Institute for Research on Aging, Novato, California
– sequence: 7
  givenname: Samah
  surname: Shah
  fullname: Shah, Samah
  organization: Buck Institute for Research on Aging, Novato, California
– sequence: 8
  givenname: Mathilde
  surname: Colombé
  fullname: Colombé, Mathilde
  organization: Cancer Research UK Cambridge Institute, Li Ka Shing Centre, Cambridge, United Kingdom
– sequence: 9
  givenname: Philippe
  surname: Gascard
  fullname: Gascard, Philippe
  organization: Department of Pathology, University of California, San Francisco, California
– sequence: 10
  givenname: Thea
  surname: Tlsty
  fullname: Tlsty, Thea
  organization: Department of Pathology, University of California, San Francisco, California
– sequence: 11
  givenname: Birgit
  surname: Schilling
  fullname: Schilling, Birgit
  organization: Buck Institute for Research on Aging, Novato, California
– sequence: 12
  givenname: Douglas J.
  orcidid: 0000-0001-6067-7927
  surname: Winton
  fullname: Winton, Douglas J.
  email: doug.winton@cruk.cam.ac.uk
  organization: Cancer Research UK Cambridge Institute, Li Ka Shing Centre, Cambridge, United Kingdom
BackLink https://www.ncbi.nlm.nih.gov/pubmed/38199279$$D View this record in MEDLINE/PubMed
BookMark eNqFkV9rFTEQxYNUbK39BILk0Ze7zb_dbRAFuVQttBRqBd9CNjvpzZpNarIrvd_erLcVEUqfMjDnTM785iXaCzEAQq8pqSihzfFQDWa82VSMMFERWhHCn6EDxmu24qL-vvdPvY-Och4IIVS0TUvqF2ifn1ApWSsPkDq9m5I24P3sdcIXekruDl8ms4FcGpOLAUeLr13OM-ArGGMP3oUb7AKeNoDXmxSDM9r7LT4L1usRenwR51xa0cfwCj232mc4un8P0bdPp9frL6vzy89n64_nKyMaOq1430igpOuYtBxII2x7wngnbdsKRqnmTPe2q7lgTBhWF0mtRc2lsa3RvDf8EL3dzb1N8edcsqvR5WUtHaCkUUxSLoSsmSzSN_fSuStp1W1yo05b9QClCPhOYFLMOYH9K6FELfTVoP7QVwt9Ragq9Ivr_c4FZc1fDpLKxkEw0LsEZlJ9dE_4P_znNwX0gvYHbCEPcU6hEFRUZaaI-rrcdzkvE-W0hNMy4N3jA578_jctX7aJ
Cites_doi 10.1007/s00418-008-0435-6
10.1002/pmic.201100463
10.1039/C8MO00239H
10.1002/advs.201901198
10.1016/j.cell.2019.06.029
10.1136/gutjnl-2019-319514
10.1046/j.1432-0436.2002.700908.x
10.1016/j.matbio.2017.11.014
10.1038/cdd.2016.117
10.1111/1467-9868.00346
10.1021/acs.jproteome.7b00170
10.1089/ten.tea.2009.0602
10.15252/embr.201439246
10.1074/mcp.RA117.000314
10.1016/j.stem.2018.07.002
10.1021/acs.jproteome.2c00526
10.1016/j.ceb.2004.07.010
10.1093/bioinformatics/btu393
10.1111/apt.14526
10.1074/mcp.O111.016717
10.1074/mcp.M111.014647
10.1053/j.gastro.2011.01.057
10.1126/science.1069094
10.1093/nar/gkn698
10.1093/nar/gkac1009
10.1002/pmic.202200021
10.1053/j.gastro.2020.04.074
10.1038/s41467-019-10769-x
10.1016/S0945-053X(98)90121-4
10.1016/j.cell.2018.08.067
10.1152/ajpgi.00398.2003
10.1016/S0016-5085(99)70339-2
10.1038/s41467-017-00249-5
10.1136/gutjnl-2012-303252
10.1126/science.827022
10.3390/cancers14235903
10.1016/j.matbio.2020.11.001
10.1074/jbc.R800020200
10.1016/j.matbio.2015.02.003
10.1016/S0140-6736(12)60150-0
10.1053/j.gastro.2006.04.020
10.1016/0022-5193(82)90388-5
10.1136/gutjnl-2012-303207
10.1002/term.2061
10.1002/ibd.21027
10.3389/fmed.2021.610189
ContentType Journal Article
Copyright 2024 The Authors
The Authors
Copyright © 2024 The Authors. Published by Elsevier Inc. All rights reserved.
Copyright_xml – notice: 2024 The Authors
– notice: The Authors
– notice: Copyright © 2024 The Authors. Published by Elsevier Inc. All rights reserved.
DBID 6I.
AAFTH
AAYXX
CITATION
CGR
CUY
CVF
ECM
EIF
NPM
7X8
DOI 10.1016/j.jcmgh.2024.01.003
DatabaseName ScienceDirect Open Access Titles
Elsevier:ScienceDirect:Open Access
CrossRef
Medline
MEDLINE
MEDLINE (Ovid)
MEDLINE
MEDLINE
PubMed
MEDLINE - Academic
DatabaseTitle CrossRef
MEDLINE
Medline Complete
MEDLINE with Full Text
PubMed
MEDLINE (Ovid)
MEDLINE - Academic
DatabaseTitleList MEDLINE - Academic



MEDLINE
Database_xml – sequence: 1
  dbid: NPM
  name: PubMed
  url: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 2
  dbid: 7X8
  name: MEDLINE - Academic
  url: https://search.proquest.com/medline
  sourceTypes: Aggregation Database
DeliveryMethod fulltext_linktorsrc
Discipline Medicine
EISSN 2352-345X
EndPage 656
ExternalDocumentID 38199279
10_1016_j_jcmgh_2024_01_003
S2352345X24000031
1_s2_0_S2352345X24000031
Genre Research Support, Non-U.S. Gov't
Journal Article
Research Support, N.I.H., Extramural
GrantInformation_xml – fundername: CRUK Cambridge Institute
  grantid: A24456
  funderid: https://doi.org/10.13039/501100022011
– fundername: National Cancer Institute
  grantid: A29074; A27145; 5R35CA197694; 5R50CA211543
  funderid: https://doi.org/10.13039/100000054
– fundername: Wellcome Trust
  grantid: 103805
  funderid: https://doi.org/10.13039/100010269
– fundername: NCI NIH HHS
  grantid: R50 CA211543
– fundername: Wellcome Trust
– fundername: NCI NIH HHS
  grantid: R35 CA197694
– fundername: Cancer Research UK
  grantid: 27145
– fundername: NIH HHS
  grantid: S10 OD028654
GroupedDBID .1-
.FO
0R~
1P~
4.4
457
53G
AAEDT
AAEDW
AAIKJ
AALRI
AAXUO
AAYWO
ABMAC
ACGFS
ACVFH
ADBBV
ADCNI
ADEZE
ADRAZ
ADVLN
AEUPX
AEVXI
AEXQZ
AFJKZ
AFPUW
AFRHN
AFTJW
AGHFR
AIGII
AITUG
AJUYK
AKBMS
AKRWK
AKYEP
ALMA_UNASSIGNED_HOLDINGS
AMRAJ
AOIJS
APXCP
BCNDV
EBS
EJD
FDB
GROUPED_DOAJ
HYE
KQ8
M41
M48
O9-
OC.
OK1
ON0
ROL
RPM
SSZ
Z5R
AACTN
0SF
6I.
AAFTH
NCXOZ
AAYXX
CITATION
CGR
CUY
CVF
ECM
EIF
NPM
7X8
ID FETCH-LOGICAL-c461t-3d69e10bb29f3e064f7823b9f774211a32adfb534224c253e05a4539cf7ca3dc3
ISICitedReferencesCount 3
ISICitedReferencesURI http://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=Summon&SrcAuth=ProQuest&DestLinkType=CitingArticles&DestApp=WOS_CPL&KeyUT=001202089000001&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
ISSN 2352-345X
IngestDate Wed Oct 01 14:40:58 EDT 2025
Mon Jul 21 05:52:40 EDT 2025
Wed Nov 12 18:36:48 EST 2025
Sat Mar 30 16:20:42 EDT 2024
Wed Apr 02 07:43:54 EDT 2025
Tue Aug 26 16:32:12 EDT 2025
IsDoiOpenAccess true
IsOpenAccess true
IsPeerReviewed true
IsScholarly true
Issue 4
Keywords DIA Proteomics
Proteoglycans
Grem1
SDS
DTT
MS
iRT
It
ACN
ECM
UC
Serpin H1
SLRP
TEAB
Cell-Matrix Interactions
TGF-β
Lamc1
Inflammatory Bowel Disease
FA
DIA
triethylammonium bicarbonate
Integrin
mass spectrometry
sodium dodecyl sulfate
acetonitrile
ulcerative colitis
formic acid
dithiothreitol
Laminin subunit gamma 1
data-independent acquisition
indexed retention time
transforming growth factor-β
Gremlin 1
small leucine-rich proteoglycan
extracellular matrix
Serpin family H member 1
Language English
License This is an open access article under the CC BY license.
Copyright © 2024 The Authors. Published by Elsevier Inc. All rights reserved.
LinkModel OpenURL
MergedId FETCHMERGED-LOGICAL-c461t-3d69e10bb29f3e064f7823b9f774211a32adfb534224c253e05a4539cf7ca3dc3
Notes ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
content type line 23
ORCID 0000-0001-6067-7927
0000-0002-9029-1520
OpenAccessLink http://dx.doi.org/10.1016/j.jcmgh.2024.01.003
PMID 38199279
PQID 2913449529
PQPubID 23479
PageCount 18
ParticipantIDs proquest_miscellaneous_2913449529
pubmed_primary_38199279
crossref_primary_10_1016_j_jcmgh_2024_01_003
elsevier_sciencedirect_doi_10_1016_j_jcmgh_2024_01_003
elsevier_clinicalkeyesjournals_1_s2_0_S2352345X24000031
elsevier_clinicalkey_doi_10_1016_j_jcmgh_2024_01_003
PublicationCentury 2000
PublicationDate 2024
2024-00-00
20240101
PublicationDateYYYYMMDD 2024-01-01
PublicationDate_xml – year: 2024
  text: 2024
PublicationDecade 2020
PublicationPlace United States
PublicationPlace_xml – name: United States
PublicationTitle Cellular and molecular gastroenterology and hepatology
PublicationTitleAlternate Cell Mol Gastroenterol Hepatol
PublicationYear 2024
Publisher Elsevier Inc
Publisher_xml – name: Elsevier Inc
References Kinchen, Chen, Parikh (bib39) 2018; 175
Coussens, Werb (bib1) 2018; 23
Schaefer, Iozzo (bib31) 2008; 283
Burger (bib51) 2018; 17
Mak, Ng (bib38) 2020; 21
Love, Huber, Anders (bib55) 2014
Shimshoni, Adir, Afi (bib35) 2020; 96
Van der Sluis, De Koning, De Bruijn (bib26) 2006; 131
Isserlin R, Voisin V, Ailles L, et al. Cell-cell interaction database [DATASET]. Zenodo, 2020.
Nakayama, Batchelder, Lee, Tarantal (bib7) 2010; 16
Ullah, Busch, Rabien (bib9) 2020; 7
Collins, Hunter, Liu (bib22) 2017; 8
Bruderer, Bernhardt, Gandhi (bib23) 2017; 16
Charni, Aloni-Grinstein, Molchadsky (bib43) 2017; 24
Hocking, Shinomura, McQuillan (bib32) 1998; 17
Velcich, Yang, Heyer (bib25) 2002; 295
Rana, Zreiqat, Benkirane-Jessel (bib8) 2017; 11
Marangio, Biccari, D’Angelo (bib15) 2022; 14
Gu, Gu, Eils (bib54) 2014; 30
Kamburov, Weirling, Lehrach, Herwig (bib52) 2009; 37
Fukushima, Fiocchi (bib44) 2004; 286
Rath, Haller (bib46) 2012; 18
Escher, Reiter, MacLean (bib49) 2012; 12
(bib56) 2016; 174
Mitsialis, Wall, Liu (bib41) 2020; 159
Czarnewski, Parigi, Sorini (bib29) 2019; 10
Beltran, Nos, Dasi (bib45) 2010; 16
Baugh, Perry, Hollander (bib12) 1999; 117
Khaloian, Rath, Hammoudi (bib47) 2020; 69
Koelink, Overbeek, Braber (bib18) 2014; 63
Kirov, Sasson, Zhang (bib11) 2019; 15
Rifkin, Rifkin, Zilberberg (bib37) 2018; 71–72
Ordás, Eckmann, Talamini (bib16) 2012; 380
Bons, Pan, Shah (bib24) 2023; 23
Solé, Unzeta (bib30) 2011
Moll, Divo, Langbein (bib28) 2008; 129
Mott, Werb (bib5) 2004; 16
Johansson, Gustafsson, Holmen-Larsson (bib36) 2014; 63
Smillie, Biton, Ordovas-Montanes (bib40) 2019; 178
Storey (bib50) 2002; 64
Naba, Clauser, Hoersh (bib53) 2012; 11
Ullman, Itzkowitz (bib20) 2011; 140
Muncie, Weaver (bib6) 2019; 130
Mortensen, Lindholm, Langholm (bib17) 2019; 10
Bissell, Hall, Parry (bib10) 1982; 99
Gordon, Agrawal, Willis (bib19) 2018; 47
Ingber (bib13) 2002; 70
McCabe, Saviola, Hansen (bib3) 2023; 22
Gillet, Navarro, Tate (bib21) 2012; 11
Iozzo, Schaefer (bib33) 2015; 42
Sakakura, Nishizuka, Dawe (bib4) 1976; 194
Troup, Njue, Kliewer (bib42) 2003; 9
Tomic, Morrissey, Kozar (bib48) 2018; 23
Pompili, Latella, Gaudio, Sferra, Vetuschi (bib2) 2021; 8
Pickup, Mouw, Weaver (bib14) 2014; 15
Shao (bib27) 2023; 51
Burger (10.1016/j.jcmgh.2024.01.003_bib51) 2018; 17
Charni (10.1016/j.jcmgh.2024.01.003_bib43) 2017; 24
Coussens (10.1016/j.jcmgh.2024.01.003_bib1) 2018; 23
McCabe (10.1016/j.jcmgh.2024.01.003_bib3) 2023; 22
Khaloian (10.1016/j.jcmgh.2024.01.003_bib47) 2020; 69
Smillie (10.1016/j.jcmgh.2024.01.003_bib40) 2019; 178
(10.1016/j.jcmgh.2024.01.003_bib56) 2016; 174
Mortensen (10.1016/j.jcmgh.2024.01.003_bib17) 2019; 10
Ullah (10.1016/j.jcmgh.2024.01.003_bib9) 2020; 7
Rana (10.1016/j.jcmgh.2024.01.003_bib8) 2017; 11
Mott (10.1016/j.jcmgh.2024.01.003_bib5) 2004; 16
Mak (10.1016/j.jcmgh.2024.01.003_bib38) 2020; 21
Collins (10.1016/j.jcmgh.2024.01.003_bib22) 2017; 8
Sakakura (10.1016/j.jcmgh.2024.01.003_bib4) 1976; 194
Pompili (10.1016/j.jcmgh.2024.01.003_bib2) 2021; 8
10.1016/j.jcmgh.2024.01.003_bib34
Love (10.1016/j.jcmgh.2024.01.003_bib55) 2014
Bons (10.1016/j.jcmgh.2024.01.003_bib24) 2023; 23
Tomic (10.1016/j.jcmgh.2024.01.003_bib48) 2018; 23
Shao (10.1016/j.jcmgh.2024.01.003_bib27) 2023; 51
Van der Sluis (10.1016/j.jcmgh.2024.01.003_bib26) 2006; 131
Johansson (10.1016/j.jcmgh.2024.01.003_bib36) 2014; 63
Nakayama (10.1016/j.jcmgh.2024.01.003_bib7) 2010; 16
Gordon (10.1016/j.jcmgh.2024.01.003_bib19) 2018; 47
Naba (10.1016/j.jcmgh.2024.01.003_bib53) 2012; 11
Ordás (10.1016/j.jcmgh.2024.01.003_bib16) 2012; 380
Hocking (10.1016/j.jcmgh.2024.01.003_bib32) 1998; 17
Velcich (10.1016/j.jcmgh.2024.01.003_bib25) 2002; 295
Gu (10.1016/j.jcmgh.2024.01.003_bib54) 2014; 30
Schaefer (10.1016/j.jcmgh.2024.01.003_bib31) 2008; 283
Baugh (10.1016/j.jcmgh.2024.01.003_bib12) 1999; 117
Troup (10.1016/j.jcmgh.2024.01.003_bib42) 2003; 9
Escher (10.1016/j.jcmgh.2024.01.003_bib49) 2012; 12
Rifkin (10.1016/j.jcmgh.2024.01.003_bib37) 2018; 71–72
Kamburov (10.1016/j.jcmgh.2024.01.003_bib52) 2009; 37
Mitsialis (10.1016/j.jcmgh.2024.01.003_bib41) 2020; 159
Marangio (10.1016/j.jcmgh.2024.01.003_bib15) 2022; 14
Ingber (10.1016/j.jcmgh.2024.01.003_bib13) 2002; 70
Czarnewski (10.1016/j.jcmgh.2024.01.003_bib29) 2019; 10
Muncie (10.1016/j.jcmgh.2024.01.003_bib6) 2019; 130
Rath (10.1016/j.jcmgh.2024.01.003_bib46) 2012; 18
Solé (10.1016/j.jcmgh.2024.01.003_bib30) 2011
Koelink (10.1016/j.jcmgh.2024.01.003_bib18) 2014; 63
Ullman (10.1016/j.jcmgh.2024.01.003_bib20) 2011; 140
Moll (10.1016/j.jcmgh.2024.01.003_bib28) 2008; 129
Shimshoni (10.1016/j.jcmgh.2024.01.003_bib35) 2020; 96
Kirov (10.1016/j.jcmgh.2024.01.003_bib11) 2019; 15
Beltran (10.1016/j.jcmgh.2024.01.003_bib45) 2010; 16
Storey (10.1016/j.jcmgh.2024.01.003_bib50) 2002; 64
Bissell (10.1016/j.jcmgh.2024.01.003_bib10) 1982; 99
Pickup (10.1016/j.jcmgh.2024.01.003_bib14) 2014; 15
Bruderer (10.1016/j.jcmgh.2024.01.003_bib23) 2017; 16
Kinchen (10.1016/j.jcmgh.2024.01.003_bib39) 2018; 175
Gillet (10.1016/j.jcmgh.2024.01.003_bib21) 2012; 11
Iozzo (10.1016/j.jcmgh.2024.01.003_bib33) 2015; 42
Fukushima (10.1016/j.jcmgh.2024.01.003_bib44) 2004; 286
38331388 - Cell Mol Gastroenterol Hepatol. 2024;17(4):660-661. doi: 10.1016/j.jcmgh.2024.01.019.
References_xml – volume: 11
  start-page: 1
  year: 2012
  end-page: 17
  ident: bib21
  article-title: Targeted data extraction of the MS/MS spectra generated by data-independent acquisition: a new concept for consistent and accurate proteome analysis
  publication-title: Mol Cell Proteomics
– volume: 174
  start-page: 245
  year: 2016
  end-page: 246
  ident: bib56
  article-title: Elegant Graphics for Data Analysis
  publication-title: Journal of the Royal Statistical Society Series A: Statistics in Society ggplot2
– volume: 70
  start-page: 547
  year: 2002
  end-page: 560
  ident: bib13
  article-title: Cancer as a disease of epithelial-mesenchymal interactions and extracellular matrix regulation
  publication-title: Differentiation
– volume: 17
  start-page: 1
  year: 1998
  end-page: 19
  ident: bib32
  article-title: Leucine-rich repeat glycoproteins of the extracellular matrix
  publication-title: Matrix Biol
– volume: 15
  start-page: 1243
  year: 2014
  end-page: 1253
  ident: bib14
  article-title: The extracellular matrix modulates the hallmarks of cancer
  publication-title: EMBO Rep
– volume: 14
  start-page: 5903
  year: 2022
  ident: bib15
  article-title: The study of the extracellular matrix in chronic inflammation: a way to prevent cancer initiation?
  publication-title: Cancers
– volume: 21
  start-page: 332
  year: 2020
  end-page: 335
  ident: bib38
  article-title: Epidemiology of fibrostenosing inflammatory bowel disease
  publication-title: J Dig Dis
– volume: 24
  start-page: 8
  year: 2017
  end-page: 14
  ident: bib43
  article-title: P53 on the crossroad between regeneration and cancer
  publication-title: Cell Death Differ
– volume: 17
  start-page: 12
  year: 2018
  end-page: 22
  ident: bib51
  article-title: Gentle introduction to the statistical foundations of false discovery rate in quantitative proteomics
  publication-title: J Proteome Res
– volume: 16
  start-page: 2296
  year: 2017
  end-page: 2309
  ident: bib23
  article-title: Optimization of experimental parameters in data-independent mass spectrometry significantly increases depth and reproducibility of results
  publication-title: Mol Cell Proteomics
– reference: Isserlin R, Voisin V, Ailles L, et al. Cell-cell interaction database [DATASET]. Zenodo, 2020.
– volume: 16
  start-page: 2207
  year: 2010
  end-page: 2216
  ident: bib7
  article-title: Decellularized rhesus monkey kidney as a three-dimensional scaffold for renal tissue engineering
  publication-title: Tissue Eng
– volume: 18
  start-page: 1364
  year: 2012
  end-page: 1377
  ident: bib46
  article-title: Mitochondria at the interface between danger signaling and metabolism : role of unfolded protein responses in chronic inflammation
  publication-title: Basic Sci Rev
– volume: 12
  start-page: 1111
  year: 2012
  end-page: 1121
  ident: bib49
  article-title: Using iRT, a normalized retention time for more targeted measurement of peptides
  publication-title: Proteomics
– volume: 23
  year: 2023
  ident: bib24
  article-title: Data-independent acquisition and quantification of extracellular matrix from human lung in chronic inflammation-associated carcinomas
  publication-title: Proteomics
– volume: 10
  start-page: 1
  year: 2019
  end-page: 11
  ident: bib29
  article-title: Conserved transcriptomic profile between mouse and human colitis allows unsupervised patient stratification
  publication-title: Nat Commun
– volume: 71–72
  start-page: 90
  year: 2018
  end-page: 99
  ident: bib37
  article-title: LTBPs in biology and medicine: LTBP diseases
  publication-title: Matrix Biol
– volume: 23
  start-page: 436
  year: 2018
  end-page: 443
  ident: bib48
  article-title: Phospho-regulation of ATOH1 is required for plasticity of secretory progenitors and tissue regeneration
  publication-title: Cell Stem Cell
– volume: 16
  start-page: 76
  year: 2010
  end-page: 86
  ident: bib45
  article-title: Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohn’s disease
  publication-title: Inflamm Bowel Dis
– volume: 37
  start-page: 623
  year: 2009
  end-page: 628
  ident: bib52
  article-title: ConsensusPathDB - a database for integrating human functional interaction networks
  publication-title: Nucleic Acids Res
– volume: 69
  start-page: 1939
  year: 2020
  end-page: 1951
  ident: bib47
  article-title: Mitochondrial impairment drives intestinal stem cell transition into dysfunctional Paneth cells predicting Crohn’s disease recurrence
  publication-title: Gut
– volume: 51
  start-page: D1519
  year: 2023
  end-page: D1530
  ident: bib27
  article-title: MatrisomeDB 2.0: 2023 updates to the ECM-protein knowledge database
  publication-title: Nucleic Acids Res
– volume: 9
  start-page: 207
  year: 2003
  end-page: 214
  ident: bib42
  article-title: Reduced expression of the small leucine-rich proteoglycans, lumican, and decorin is associated with poor outcome in node-negative invasive breast cancer
  publication-title: Clin Cancer Res
– volume: 64
  start-page: 479
  year: 2002
  end-page: 498
  ident: bib50
  article-title: A direct approach to false discovery rates
  publication-title: J R Stat Soc B Stat Methodol
– volume: 10
  start-page: 977
  year: 2019
  end-page: 993
  ident: bib17
  article-title: The intestinal tissue homeostasis–the role of extracellular matrix remodeling in inflammatory bowel disease
  publication-title: Gastroenterol Hepatol
– volume: 131
  start-page: 117
  year: 2006
  end-page: 129
  ident: bib26
  article-title: Muc2-deficient mice spontaneously develop colitis, indicating that MUC2 is critical for colonic protection
  publication-title: Gastroenterology
– volume: 8
  start-page: 1
  year: 2021
  end-page: 19
  ident: bib2
  article-title: The charming world of the extracellular matrix: a dynamic and protective network of the intestinal wall
  publication-title: Front Med
– start-page: 103543
  year: 2011
  end-page: 103557
  ident: bib30
  article-title: Vascular cell lines expressing SSAO/VAP-1: a new experimental tool to study its involvement in vascular diseases
  publication-title: Biol Cell
– volume: 42
  start-page: 11
  year: 2015
  end-page: 55
  ident: bib33
  article-title: Proteoglycan form and function: a comprehensive nomenclature of proteoglycans
  publication-title: Matrix Biol
– volume: 117
  start-page: 814
  year: 1999
  end-page: 822
  ident: bib12
  article-title: Matrix metalloproteinase levels are elevated in inflammatory bowel disease
  publication-title: Gastroenterology
– volume: 8
  start-page: 1
  year: 2017
  end-page: 11
  ident: bib22
  article-title: Multi-laboratory assessment of reproducibility, qualitative and quantitative performance of SWATH-mass spectrometry
  publication-title: Nat Commun
– volume: 140
  start-page: 1807
  year: 2011
  end-page: 1816
  ident: bib20
  article-title: Intestinal inflammation and cancer
  publication-title: Gastroenterology
– volume: 99
  start-page: 31
  year: 1982
  end-page: 68
  ident: bib10
  article-title: How does the extracellular matrix direct gene expression?
  publication-title: J Theor Biol
– volume: 11
  year: 2012
  ident: bib53
  article-title: The matrisome: in silico definition and in vivo characterization by proteomics of normal and tumor extracellular matrices
  publication-title: Mol Cell Proteomics
– volume: 63
  start-page: 578
  year: 2014
  end-page: 587
  ident: bib18
  article-title: Collagen degradation and neutrophilic infiltration: a vicious circle in inflammatory bowel disease
  publication-title: Gut
– volume: 178
  start-page: 714
  year: 2019
  end-page: 730
  ident: bib40
  article-title: Intra- and inter-cellular rewiring of the human colon during ulcerative colitis
  publication-title: Cell
– volume: 194
  start-page: 1439
  year: 1976
  end-page: 1441
  ident: bib4
  article-title: Mesenchyme-dependent morphogenesis and epithelium-specific cytodifferentiation in mouse mammary gland
  publication-title: Science
– volume: 380
  start-page: 1606
  year: 2012
  end-page: 1619
  ident: bib16
  article-title: Ulcerative colitis
  publication-title: Lancet
– volume: 96
  start-page: 47
  year: 2020
  end-page: 68
  ident: bib35
  article-title: Distinct extracellular–matrix remodeling events precede symptoms of inflammation
  publication-title: Matrix Biol
– volume: 283
  start-page: 21305
  year: 2008
  end-page: 21309
  ident: bib31
  article-title: Biological functions of the small leucine-rich proteoglycans: from genetics to signal transduction
  publication-title: J Biol Chem
– volume: 47
  start-page: 922
  year: 2018
  end-page: 939
  ident: bib19
  article-title: Fibrosis in ulcerative colitis is directly linked to severity and chronicity of mucosal inflammation
  publication-title: Aliment Pharmacol Ther
– volume: 30
  start-page: 2811
  year: 2014
  end-page: 2812
  ident: bib54
  article-title: Circlize implements and enhances circular visualization in R
  publication-title: Bioinformatics
– volume: 129
  start-page: 705
  year: 2008
  end-page: 733
  ident: bib28
  article-title: The human keratins: biology and pathology
  publication-title: Histochem Cell Biol
– volume: 7
  start-page: 1
  year: 2020
  end-page: 10
  ident: bib9
  article-title: Adult tissue extracellular matrix determines tissue specification of human iPSC-derived embryonic stage mesodermal precursor cells
  publication-title: Adv Sci
– volume: 175
  start-page: 372
  year: 2018
  end-page: 386.e17
  ident: bib39
  article-title: Structural remodeling of the human colonic mesenchyme in inflammatory bowel disease
  publication-title: Cell
– volume: 130
  start-page: 1
  year: 2019
  end-page: 30
  ident: bib6
  article-title: The physical and biochemical properties of the extracellular matrix regulate cell fate
  publication-title: Curr Top Dev Biol
– volume: 286
  start-page: 804
  year: 2004
  end-page: 813
  ident: bib44
  article-title: Paradoxical decrease of mitochondrial DNA deletions in epithelial cells of active ulcerative colitis patients
  publication-title: Am J Physiol Gastrointest Liver Physiol
– volume: 16
  start-page: 558
  year: 2004
  end-page: 564
  ident: bib5
  article-title: Regulation of matrix biology by matrix metalloproteinases
  publication-title: Curr Opin Cell Biol
– volume: 15
  start-page: 67
  year: 2019
  end-page: 76
  ident: bib11
  article-title: Degradation of the extracellular matrix is part of the pathology of ulcerative colitis
  publication-title: Mol Omics
– volume: 23:
  year: 2018
  ident: bib1
  article-title: Inflammation and cancer
  publication-title: Environmental Health and Preventive Medicine
– volume: 11
  start-page: 942
  year: 2017
  end-page: 965
  ident: bib8
  article-title: Development of decellularized scaffolds for stem cell-driven tissue engineering
  publication-title: J Tissue Eng Regen Med
– volume: 63
  start-page: 281
  year: 2014
  end-page: 291
  ident: bib36
  article-title: Bacteria penetrate the normally impenetrable inner colon mucus layer in both murine colitis models and patients with ulcerative colitis
  publication-title: Gut
– volume: 159
  start-page: 591
  year: 2020
  end-page: 608
  ident: bib41
  article-title: Single-cell analyses of colon and blood reveal distinct immune cell signatures of ulcerative colitis and Crohn’s disease
  publication-title: Gastroenterology
– start-page: 1
  year: 2014
  end-page: 21
  ident: bib55
  article-title: Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2
  publication-title: Genome Biology
– volume: 295
  start-page: 1726
  year: 2002
  end-page: 1729
  ident: bib25
  article-title: Colorectal cancer in mice genetically deficient in the mucin Muc2
  publication-title: Science
– volume: 22
  start-page: 790
  year: 2023
  end-page: 801
  ident: bib3
  article-title: Mass spectrometry-based atlas of extracellular matrix proteins across 25 mouse organs
  publication-title: J Proteome Res
– volume: 129
  start-page: 705
  year: 2008
  ident: 10.1016/j.jcmgh.2024.01.003_bib28
  article-title: The human keratins: biology and pathology
  publication-title: Histochem Cell Biol
  doi: 10.1007/s00418-008-0435-6
– volume: 12
  start-page: 1111
  year: 2012
  ident: 10.1016/j.jcmgh.2024.01.003_bib49
  article-title: Using iRT, a normalized retention time for more targeted measurement of peptides
  publication-title: Proteomics
  doi: 10.1002/pmic.201100463
– volume: 15
  start-page: 67
  year: 2019
  ident: 10.1016/j.jcmgh.2024.01.003_bib11
  article-title: Degradation of the extracellular matrix is part of the pathology of ulcerative colitis
  publication-title: Mol Omics
  doi: 10.1039/C8MO00239H
– volume: 7
  start-page: 1
  year: 2020
  ident: 10.1016/j.jcmgh.2024.01.003_bib9
  article-title: Adult tissue extracellular matrix determines tissue specification of human iPSC-derived embryonic stage mesodermal precursor cells
  publication-title: Adv Sci
  doi: 10.1002/advs.201901198
– volume: 178
  start-page: 714
  year: 2019
  ident: 10.1016/j.jcmgh.2024.01.003_bib40
  article-title: Intra- and inter-cellular rewiring of the human colon during ulcerative colitis
  publication-title: Cell
  doi: 10.1016/j.cell.2019.06.029
– volume: 69
  start-page: 1939
  year: 2020
  ident: 10.1016/j.jcmgh.2024.01.003_bib47
  article-title: Mitochondrial impairment drives intestinal stem cell transition into dysfunctional Paneth cells predicting Crohn’s disease recurrence
  publication-title: Gut
  doi: 10.1136/gutjnl-2019-319514
– volume: 70
  start-page: 547
  year: 2002
  ident: 10.1016/j.jcmgh.2024.01.003_bib13
  article-title: Cancer as a disease of epithelial-mesenchymal interactions and extracellular matrix regulation
  publication-title: Differentiation
  doi: 10.1046/j.1432-0436.2002.700908.x
– volume: 71–72
  start-page: 90
  year: 2018
  ident: 10.1016/j.jcmgh.2024.01.003_bib37
  article-title: LTBPs in biology and medicine: LTBP diseases
  publication-title: Matrix Biol
  doi: 10.1016/j.matbio.2017.11.014
– volume: 24
  start-page: 8
  year: 2017
  ident: 10.1016/j.jcmgh.2024.01.003_bib43
  article-title: P53 on the crossroad between regeneration and cancer
  publication-title: Cell Death Differ
  doi: 10.1038/cdd.2016.117
– volume: 64
  start-page: 479
  year: 2002
  ident: 10.1016/j.jcmgh.2024.01.003_bib50
  article-title: A direct approach to false discovery rates
  publication-title: J R Stat Soc B Stat Methodol
  doi: 10.1111/1467-9868.00346
– volume: 17
  start-page: 12
  year: 2018
  ident: 10.1016/j.jcmgh.2024.01.003_bib51
  article-title: Gentle introduction to the statistical foundations of false discovery rate in quantitative proteomics
  publication-title: J Proteome Res
  doi: 10.1021/acs.jproteome.7b00170
– volume: 16
  start-page: 2207
  year: 2010
  ident: 10.1016/j.jcmgh.2024.01.003_bib7
  article-title: Decellularized rhesus monkey kidney as a three-dimensional scaffold for renal tissue engineering
  publication-title: Tissue Eng
  doi: 10.1089/ten.tea.2009.0602
– volume: 174
  start-page: 245
  issue: 1
  year: 2016
  ident: 10.1016/j.jcmgh.2024.01.003_bib56
  article-title: Elegant Graphics for Data Analysis
  publication-title: Journal of the Royal Statistical Society Series A: Statistics in Society ggplot2
– volume: 15
  start-page: 1243
  year: 2014
  ident: 10.1016/j.jcmgh.2024.01.003_bib14
  article-title: The extracellular matrix modulates the hallmarks of cancer
  publication-title: EMBO Rep
  doi: 10.15252/embr.201439246
– volume: 16
  start-page: 2296
  year: 2017
  ident: 10.1016/j.jcmgh.2024.01.003_bib23
  article-title: Optimization of experimental parameters in data-independent mass spectrometry significantly increases depth and reproducibility of results
  publication-title: Mol Cell Proteomics
  doi: 10.1074/mcp.RA117.000314
– volume: 23
  start-page: 436
  year: 2018
  ident: 10.1016/j.jcmgh.2024.01.003_bib48
  article-title: Phospho-regulation of ATOH1 is required for plasticity of secretory progenitors and tissue regeneration
  publication-title: Cell Stem Cell
  doi: 10.1016/j.stem.2018.07.002
– volume: 22
  start-page: 790
  year: 2023
  ident: 10.1016/j.jcmgh.2024.01.003_bib3
  article-title: Mass spectrometry-based atlas of extracellular matrix proteins across 25 mouse organs
  publication-title: J Proteome Res
  doi: 10.1021/acs.jproteome.2c00526
– volume: 16
  start-page: 558
  year: 2004
  ident: 10.1016/j.jcmgh.2024.01.003_bib5
  article-title: Regulation of matrix biology by matrix metalloproteinases
  publication-title: Curr Opin Cell Biol
  doi: 10.1016/j.ceb.2004.07.010
– volume: 30
  start-page: 2811
  year: 2014
  ident: 10.1016/j.jcmgh.2024.01.003_bib54
  article-title: Circlize implements and enhances circular visualization in R
  publication-title: Bioinformatics
  doi: 10.1093/bioinformatics/btu393
– volume: 47
  start-page: 922
  year: 2018
  ident: 10.1016/j.jcmgh.2024.01.003_bib19
  article-title: Fibrosis in ulcerative colitis is directly linked to severity and chronicity of mucosal inflammation
  publication-title: Aliment Pharmacol Ther
  doi: 10.1111/apt.14526
– volume: 11
  start-page: 1
  year: 2012
  ident: 10.1016/j.jcmgh.2024.01.003_bib21
  article-title: Targeted data extraction of the MS/MS spectra generated by data-independent acquisition: a new concept for consistent and accurate proteome analysis
  publication-title: Mol Cell Proteomics
  doi: 10.1074/mcp.O111.016717
– volume: 11
  year: 2012
  ident: 10.1016/j.jcmgh.2024.01.003_bib53
  article-title: The matrisome: in silico definition and in vivo characterization by proteomics of normal and tumor extracellular matrices
  publication-title: Mol Cell Proteomics
  doi: 10.1074/mcp.M111.014647
– volume: 140
  start-page: 1807
  year: 2011
  ident: 10.1016/j.jcmgh.2024.01.003_bib20
  article-title: Intestinal inflammation and cancer
  publication-title: Gastroenterology
  doi: 10.1053/j.gastro.2011.01.057
– start-page: 103543
  year: 2011
  ident: 10.1016/j.jcmgh.2024.01.003_bib30
  article-title: Vascular cell lines expressing SSAO/VAP-1: a new experimental tool to study its involvement in vascular diseases
  publication-title: Biol Cell
– volume: 295
  start-page: 1726
  year: 2002
  ident: 10.1016/j.jcmgh.2024.01.003_bib25
  article-title: Colorectal cancer in mice genetically deficient in the mucin Muc2
  publication-title: Science
  doi: 10.1126/science.1069094
– volume: 37
  start-page: 623
  issue: Suppl 1
  year: 2009
  ident: 10.1016/j.jcmgh.2024.01.003_bib52
  article-title: ConsensusPathDB - a database for integrating human functional interaction networks
  publication-title: Nucleic Acids Res
  doi: 10.1093/nar/gkn698
– volume: 23:
  issue: 860–867
  year: 2018
  ident: 10.1016/j.jcmgh.2024.01.003_bib1
  article-title: Inflammation and cancer
  publication-title: Environmental Health and Preventive Medicine
– volume: 18
  start-page: 1364
  year: 2012
  ident: 10.1016/j.jcmgh.2024.01.003_bib46
  article-title: Mitochondria at the interface between danger signaling and metabolism : role of unfolded protein responses in chronic inflammation
  publication-title: Basic Sci Rev
– volume: 51
  start-page: D1519
  year: 2023
  ident: 10.1016/j.jcmgh.2024.01.003_bib27
  article-title: MatrisomeDB 2.0: 2023 updates to the ECM-protein knowledge database
  publication-title: Nucleic Acids Res
  doi: 10.1093/nar/gkac1009
– volume: 23
  year: 2023
  ident: 10.1016/j.jcmgh.2024.01.003_bib24
  article-title: Data-independent acquisition and quantification of extracellular matrix from human lung in chronic inflammation-associated carcinomas
  publication-title: Proteomics
  doi: 10.1002/pmic.202200021
– volume: 159
  start-page: 591
  year: 2020
  ident: 10.1016/j.jcmgh.2024.01.003_bib41
  article-title: Single-cell analyses of colon and blood reveal distinct immune cell signatures of ulcerative colitis and Crohn’s disease
  publication-title: Gastroenterology
  doi: 10.1053/j.gastro.2020.04.074
– volume: 10
  start-page: 1
  year: 2019
  ident: 10.1016/j.jcmgh.2024.01.003_bib29
  article-title: Conserved transcriptomic profile between mouse and human colitis allows unsupervised patient stratification
  publication-title: Nat Commun
  doi: 10.1038/s41467-019-10769-x
– volume: 17
  start-page: 1
  year: 1998
  ident: 10.1016/j.jcmgh.2024.01.003_bib32
  article-title: Leucine-rich repeat glycoproteins of the extracellular matrix
  publication-title: Matrix Biol
  doi: 10.1016/S0945-053X(98)90121-4
– volume: 175
  start-page: 372
  year: 2018
  ident: 10.1016/j.jcmgh.2024.01.003_bib39
  article-title: Structural remodeling of the human colonic mesenchyme in inflammatory bowel disease
  publication-title: Cell
  doi: 10.1016/j.cell.2018.08.067
– volume: 286
  start-page: 804
  year: 2004
  ident: 10.1016/j.jcmgh.2024.01.003_bib44
  article-title: Paradoxical decrease of mitochondrial DNA deletions in epithelial cells of active ulcerative colitis patients
  publication-title: Am J Physiol Gastrointest Liver Physiol
  doi: 10.1152/ajpgi.00398.2003
– volume: 117
  start-page: 814
  year: 1999
  ident: 10.1016/j.jcmgh.2024.01.003_bib12
  article-title: Matrix metalloproteinase levels are elevated in inflammatory bowel disease
  publication-title: Gastroenterology
  doi: 10.1016/S0016-5085(99)70339-2
– volume: 8
  start-page: 1
  year: 2017
  ident: 10.1016/j.jcmgh.2024.01.003_bib22
  article-title: Multi-laboratory assessment of reproducibility, qualitative and quantitative performance of SWATH-mass spectrometry
  publication-title: Nat Commun
  doi: 10.1038/s41467-017-00249-5
– ident: 10.1016/j.jcmgh.2024.01.003_bib34
– volume: 63
  start-page: 578
  year: 2014
  ident: 10.1016/j.jcmgh.2024.01.003_bib18
  article-title: Collagen degradation and neutrophilic infiltration: a vicious circle in inflammatory bowel disease
  publication-title: Gut
  doi: 10.1136/gutjnl-2012-303252
– volume: 194
  start-page: 1439
  year: 1976
  ident: 10.1016/j.jcmgh.2024.01.003_bib4
  article-title: Mesenchyme-dependent morphogenesis and epithelium-specific cytodifferentiation in mouse mammary gland
  publication-title: Science
  doi: 10.1126/science.827022
– volume: 9
  start-page: 207
  year: 2003
  ident: 10.1016/j.jcmgh.2024.01.003_bib42
  article-title: Reduced expression of the small leucine-rich proteoglycans, lumican, and decorin is associated with poor outcome in node-negative invasive breast cancer
  publication-title: Clin Cancer Res
– volume: 14
  start-page: 5903
  year: 2022
  ident: 10.1016/j.jcmgh.2024.01.003_bib15
  article-title: The study of the extracellular matrix in chronic inflammation: a way to prevent cancer initiation?
  publication-title: Cancers
  doi: 10.3390/cancers14235903
– volume: 21
  start-page: 332
  year: 2020
  ident: 10.1016/j.jcmgh.2024.01.003_bib38
  article-title: Epidemiology of fibrostenosing inflammatory bowel disease
  publication-title: J Dig Dis
– volume: 96
  start-page: 47
  year: 2020
  ident: 10.1016/j.jcmgh.2024.01.003_bib35
  article-title: Distinct extracellular–matrix remodeling events precede symptoms of inflammation
  publication-title: Matrix Biol
  doi: 10.1016/j.matbio.2020.11.001
– volume: 283
  start-page: 21305
  year: 2008
  ident: 10.1016/j.jcmgh.2024.01.003_bib31
  article-title: Biological functions of the small leucine-rich proteoglycans: from genetics to signal transduction
  publication-title: J Biol Chem
  doi: 10.1074/jbc.R800020200
– volume: 130
  start-page: 1
  year: 2019
  ident: 10.1016/j.jcmgh.2024.01.003_bib6
  article-title: The physical and biochemical properties of the extracellular matrix regulate cell fate
  publication-title: Curr Top Dev Biol
– volume: 10
  start-page: 977
  year: 2019
  ident: 10.1016/j.jcmgh.2024.01.003_bib17
  article-title: The intestinal tissue homeostasis–the role of extracellular matrix remodeling in inflammatory bowel disease
  publication-title: Gastroenterol Hepatol
– volume: 42
  start-page: 11
  year: 2015
  ident: 10.1016/j.jcmgh.2024.01.003_bib33
  article-title: Proteoglycan form and function: a comprehensive nomenclature of proteoglycans
  publication-title: Matrix Biol
  doi: 10.1016/j.matbio.2015.02.003
– volume: 380
  start-page: 1606
  year: 2012
  ident: 10.1016/j.jcmgh.2024.01.003_bib16
  article-title: Ulcerative colitis
  publication-title: Lancet
  doi: 10.1016/S0140-6736(12)60150-0
– volume: 131
  start-page: 117
  year: 2006
  ident: 10.1016/j.jcmgh.2024.01.003_bib26
  article-title: Muc2-deficient mice spontaneously develop colitis, indicating that MUC2 is critical for colonic protection
  publication-title: Gastroenterology
  doi: 10.1053/j.gastro.2006.04.020
– volume: 99
  start-page: 31
  year: 1982
  ident: 10.1016/j.jcmgh.2024.01.003_bib10
  article-title: How does the extracellular matrix direct gene expression?
  publication-title: J Theor Biol
  doi: 10.1016/0022-5193(82)90388-5
– volume: 63
  start-page: 281
  year: 2014
  ident: 10.1016/j.jcmgh.2024.01.003_bib36
  article-title: Bacteria penetrate the normally impenetrable inner colon mucus layer in both murine colitis models and patients with ulcerative colitis
  publication-title: Gut
  doi: 10.1136/gutjnl-2012-303207
– start-page: 1
  year: 2014
  ident: 10.1016/j.jcmgh.2024.01.003_bib55
  article-title: Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2
  publication-title: Genome Biology
– volume: 11
  start-page: 942
  year: 2017
  ident: 10.1016/j.jcmgh.2024.01.003_bib8
  article-title: Development of decellularized scaffolds for stem cell-driven tissue engineering
  publication-title: J Tissue Eng Regen Med
  doi: 10.1002/term.2061
– volume: 16
  start-page: 76
  year: 2010
  ident: 10.1016/j.jcmgh.2024.01.003_bib45
  article-title: Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohn’s disease
  publication-title: Inflamm Bowel Dis
  doi: 10.1002/ibd.21027
– volume: 8
  start-page: 1
  year: 2021
  ident: 10.1016/j.jcmgh.2024.01.003_bib2
  article-title: The charming world of the extracellular matrix: a dynamic and protective network of the intestinal wall
  publication-title: Front Med
  doi: 10.3389/fmed.2021.610189
– reference: 38331388 - Cell Mol Gastroenterol Hepatol. 2024;17(4):660-661. doi: 10.1016/j.jcmgh.2024.01.019.
SSID ssj0001476705
Score 2.3002367
Snippet Chronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased risk of developing...
Background & AimsChronic inflammatory illnesses are debilitating and recurrent conditions associated with significant comorbidities, including an increased...
SourceID proquest
pubmed
crossref
elsevier
SourceType Aggregation Database
Index Database
Publisher
StartPage 639
SubjectTerms Animals
Cell-Matrix Interactions
Chronic Disease
Colitis - pathology
DIA Proteomics
Disease Progression
Extracellular Matrix - metabolism
Gastroenterology and Hepatology
Inflammatory Bowel Disease
Mice
Proteoglycans
Proteomics
Title Extracellular Matrix Orchestration of Tissue Remodeling in the Chronically Inflamed Mouse Colon
URI https://www.clinicalkey.com/#!/content/1-s2.0-S2352345X24000031
https://www.clinicalkey.es/playcontent/1-s2.0-S2352345X24000031
https://dx.doi.org/10.1016/j.jcmgh.2024.01.003
https://www.ncbi.nlm.nih.gov/pubmed/38199279
https://www.proquest.com/docview/2913449529
Volume 17
WOSCitedRecordID wos001202089000001&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
hasFullText 1
inHoldings 1
isFullTextHit
isPrint
journalDatabaseRights – providerCode: PRVAON
  databaseName: DOAJ Directory of Open Access Journals
  customDbUrl:
  eissn: 2352-345X
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0001476705
  issn: 2352-345X
  databaseCode: DOA
  dateStart: 20150101
  isFulltext: true
  titleUrlDefault: https://www.doaj.org/
  providerName: Directory of Open Access Journals
link http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwtV1Lj9MwELa6C0JcEG_KY2UkbiWrxHbi-girAhLbBYki9WY5ibO06qarpFuVX8rfYWzHaatueUlcosiJE2fmk2c8GX-D0KuM5yTRvAgyzeKACVYEaU5JoHNDXs7iWLmNwqf87Kw_HovPnc4PvxdmOeNl2V-txOV_VTW0gbLN1tm_UHf7UGiAc1A6HEHtcPwjxQ9Wi0qZeLxNMB0aCv5V71NlC2NVrYM4sgIH6dpSOM3GFuOENmy5ajb7DpNHAYABl3Q4v6pNfbtZo0RPbeDfYqLvF77Qbu9cwZvmhuyzWjM8fQO7t9iK4cNDGwaDwWxSq3X957fzsvk7ocqyNRvvJ2qpbFWkHpyuAwmnIBTDxmndcBMd0puRDLd12k11BNzAgLJ47KzSNW1-ruYbmGQbE2_iKJEaG544svId8-AiFdPjaXZxbv5EEWY5W0O6toY-A2Bw8jEKanIcBl_MSMxATLqtmQS3b3ZLKVkTGcqdWw_QDcJhzbax1rfBP8YTbtNr26_0jFg293BnfPu8pn2rIusdje6iO82yBr9xcLyHOrq8j24Nm8SNB0huoRI7VOItVOJ5gR0q8RqVeFJiQCXeQCX2qMQWldii8iH6-m4wOvkQNLU9gowl0SKgeSJ0FKYpEQXV4BcX4KrSVBSwHCFRpChReZHGlIGLmZEYbokVi6nICp4pmmf0ETos56V-gnCkudb9lJtaB4wJuJxEimSUhoUgKS266LWXnbx0FC7S5zZOpRW1NKKWYWSYcruIeflKvzsZ7KkEDP26G7-um66bqaGW-zDSRUnbs3F7nTv7-1e-9OqXYBSMDlWpQfSSmHwaJmIiuuixw0X76SZEIwgXT_95wM_QbTMKF4h8jg4X1ZV-gW5my8Wkro7QAR_3jyzefwKDc-eU
linkProvider Directory of Open Access Journals
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Extracellular+Matrix+Orchestration+of+Tissue+Remodeling+in+the+Chronically+Inflamed+Mouse+Colon&rft.jtitle=Cellular+and+molecular+gastroenterology+and+hepatology&rft.au=Moutin%2C+Elisa+B&rft.au=Bons%2C+Joanna&rft.au=Giavara%2C+Giada&rft.au=Lourenco%2C+Filipe&rft.date=2024&rft.issn=2352-345X&rft.eissn=2352-345X&rft.volume=17&rft.issue=4&rft.spage=639&rft.epage=656&rft_id=info:doi/10.1016%2Fj.jcmgh.2024.01.003&rft.externalDBID=ECK1-s2.0-S2352345X24000031&rft.externalDocID=1_s2_0_S2352345X24000031
thumbnail_m http://cvtisr.summon.serialssolutions.com/2.0.0/image/custom?url=https%3A%2F%2Fcdn.clinicalkey.com%2Fck-thumbnails%2F2352345X%2Fcov200h.gif