Neuroinflammation in glaucoma: A new opportunity

Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which may also be a key in other common neurodegenerations such as Alzheimer's disease, will lead to targeted therapeutics for a disease that af...

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Veröffentlicht in:Experimental eye research Jg. 157; S. 20 - 27
Hauptverfasser: Williams, Pete A., Marsh-Armstrong, Nick, Howell, Gareth R., Bosco, Alejandra, Danias, John, Simon, John, Di Polo, Adriana, Kuehn, Markus H., Przedborski, Serge, Raff, Martin, Trounce, Ian
Format: Journal Article
Sprache:Englisch
Veröffentlicht: England Elsevier Ltd 01.04.2017
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ISSN:0014-4835, 1096-0007, 1096-0007
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Abstract Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which may also be a key in other common neurodegenerations such as Alzheimer's disease, will lead to targeted therapeutics for a disease that affects as many as 80 million people worldwide. Here, we define neuroinflammation as any immune-relevant response by a variety of cell types including astrocytes, microglia, and peripherally derived cells occurring in the optic nerve head and/or retina. In this review article, we first discuss clinical evidence for neuroinflammation in glaucoma and define neuroinflammation in glaucoma. We then review the inflammatory pathways that have been associated with glaucoma. Finally, we set out key research directions that we believe will greatly advance our understanding of the role of neuroinflammation in glaucoma. This review arose from a discussion of neuroinflammation in glaucoma at the 2015 meeting of The Lasker/IRRF Initiative for Innovation in Vision Science. This manuscript sets out to summarize one of these sessions; “Inflammation and Glaucomatous Neurodegeneration”, as well as to review the current state of the literature surrounding neuroinflammation in glaucoma. •We describe the clinical relevance of inflammation in glaucoma.•We define the sites of neuroinflammation in glaucoma.•We discuss the roles of different cells and pathways in glaucoma.•We discuss suggested 5 year goals as set about by the participants of the 2015 Lasker/IRRF Initiative session “Inflammation and Glaucomatous Neurodegeneration”.
AbstractList Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which may also be a key in other common neurodegenerations such as Alzheimer’s disease, will lead to targeted therapeutics for a disease that affects as many as 80 million people worldwide. Here, we define neuroinflammation as any immune-relevant response by a variety of cell types including astrocytes, microglia, and peripherally derived cells occurring in the optic nerve head and/or retina. In this review article, we first discuss clinical evidence for neuroinflammation in glaucoma and define neuroinflammation in glaucoma. We then review the inflammatory pathways that have been associated with glaucoma. Finally, we set out key research directions that we believe will greatly advance our understanding of the role of neuroinflammation in glaucoma. This review arose from a discussion of neuroinflammation in glaucoma at the 2015 meeting of the The Lasker/IRRF Initiative for Innovation in Vision Science. This manuscript sets out to summarize one of these sessions; “Inflammation and Glaucomatous Neurodegeneration”, as well as to review the current state of the literature surrounding neuroinflammation in glaucoma.
Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which may also be a key in other common neurodegenerations such as Alzheimer's disease, will lead to targeted therapeutics for a disease that affects as many as 80 million people worldwide. Here, we define neuroinflammation as any immune-relevant response by a variety of cell types including astrocytes, microglia, and peripherally derived cells occurring in the optic nerve head and/or retina. In this review article, we first discuss clinical evidence for neuroinflammation in glaucoma and define neuroinflammation in glaucoma. We then review the inflammatory pathways that have been associated with glaucoma. Finally, we set out key research directions that we believe will greatly advance our understanding of the role of neuroinflammation in glaucoma. This review arose from a discussion of neuroinflammation in glaucoma at the 2015 meeting of The Lasker/IRRF Initiative for Innovation in Vision Science. This manuscript sets out to summarize one of these sessions; "Inflammation and Glaucomatous Neurodegeneration", as well as to review the current state of the literature surrounding neuroinflammation in glaucoma.
Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which may also be a key in other common neurodegenerations such as Alzheimer's disease, will lead to targeted therapeutics for a disease that affects as many as 80 million people worldwide. Here, we define neuroinflammation as any immune-relevant response by a variety of cell types including astrocytes, microglia, and peripherally derived cells occurring in the optic nerve head and/or retina. In this review article, we first discuss clinical evidence for neuroinflammation in glaucoma and define neuroinflammation in glaucoma. We then review the inflammatory pathways that have been associated with glaucoma. Finally, we set out key research directions that we believe will greatly advance our understanding of the role of neuroinflammation in glaucoma. This review arose from a discussion of neuroinflammation in glaucoma at the 2015 meeting of The Lasker/IRRF Initiative for Innovation in Vision Science. This manuscript sets out to summarize one of these sessions; "Inflammation and Glaucomatous Neurodegeneration", as well as to review the current state of the literature surrounding neuroinflammation in glaucoma.Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which may also be a key in other common neurodegenerations such as Alzheimer's disease, will lead to targeted therapeutics for a disease that affects as many as 80 million people worldwide. Here, we define neuroinflammation as any immune-relevant response by a variety of cell types including astrocytes, microglia, and peripherally derived cells occurring in the optic nerve head and/or retina. In this review article, we first discuss clinical evidence for neuroinflammation in glaucoma and define neuroinflammation in glaucoma. We then review the inflammatory pathways that have been associated with glaucoma. Finally, we set out key research directions that we believe will greatly advance our understanding of the role of neuroinflammation in glaucoma. This review arose from a discussion of neuroinflammation in glaucoma at the 2015 meeting of The Lasker/IRRF Initiative for Innovation in Vision Science. This manuscript sets out to summarize one of these sessions; "Inflammation and Glaucomatous Neurodegeneration", as well as to review the current state of the literature surrounding neuroinflammation in glaucoma.
Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which may also be a key in other common neurodegenerations such as Alzheimer's disease, will lead to targeted therapeutics for a disease that affects as many as 80 million people worldwide. Here, we define neuroinflammation as any immune-relevant response by a variety of cell types including astrocytes, microglia, and peripherally derived cells occurring in the optic nerve head and/or retina. In this review article, we first discuss clinical evidence for neuroinflammation in glaucoma and define neuroinflammation in glaucoma. We then review the inflammatory pathways that have been associated with glaucoma. Finally, we set out key research directions that we believe will greatly advance our understanding of the role of neuroinflammation in glaucoma. This review arose from a discussion of neuroinflammation in glaucoma at the 2015 meeting of The Lasker/IRRF Initiative for Innovation in Vision Science. This manuscript sets out to summarize one of these sessions; “Inflammation and Glaucomatous Neurodegeneration”, as well as to review the current state of the literature surrounding neuroinflammation in glaucoma. •We describe the clinical relevance of inflammation in glaucoma.•We define the sites of neuroinflammation in glaucoma.•We discuss the roles of different cells and pathways in glaucoma.•We discuss suggested 5 year goals as set about by the participants of the 2015 Lasker/IRRF Initiative session “Inflammation and Glaucomatous Neurodegeneration”.
Author Simon, John
Williams, Pete A.
Marsh-Armstrong, Nick
Di Polo, Adriana
Trounce, Ian
Danias, John
Howell, Gareth R.
Kuehn, Markus H.
Przedborski, Serge
Raff, Martin
Bosco, Alejandra
AuthorAffiliation 4 Graduate Program of Genetics, Sackler School of Graduate Biomedical Sciences, Tufts University, Boston, MA, USA
2 The Solomon H. Snyder Department of Neuroscience, The Johns Hopkins University School of Medicine, Baltimore, MD, USA
3 Hugo W. Moser Research Institute, Kennedy Krieger Institute, Baltimore, MD, USA
1 The Jackson Laboratory, Bar Harbor, ME, USA
AuthorAffiliation_xml – name: 4 Graduate Program of Genetics, Sackler School of Graduate Biomedical Sciences, Tufts University, Boston, MA, USA
– name: 2 The Solomon H. Snyder Department of Neuroscience, The Johns Hopkins University School of Medicine, Baltimore, MD, USA
– name: 1 The Jackson Laboratory, Bar Harbor, ME, USA
– name: 3 Hugo W. Moser Research Institute, Kennedy Krieger Institute, Baltimore, MD, USA
Author_xml – sequence: 1
  givenname: Pete A.
  orcidid: 0000-0001-6194-8397
  surname: Williams
  fullname: Williams, Pete A.
  organization: The Jackson Laboratory, Bar Harbor, ME, USA
– sequence: 2
  givenname: Nick
  surname: Marsh-Armstrong
  fullname: Marsh-Armstrong, Nick
  organization: The Solomon H. Snyder Department of Neuroscience, The Johns Hopkins University School of Medicine, Baltimore, MD, USA
– sequence: 3
  givenname: Gareth R.
  surname: Howell
  fullname: Howell, Gareth R.
  email: gareth.howell@jax.org
  organization: The Jackson Laboratory, Bar Harbor, ME, USA
– sequence: 4
  givenname: Alejandra
  surname: Bosco
  fullname: Bosco, Alejandra
  organization: University of Utah, School of Medicine, Salt Lake City, UT, USA
– sequence: 5
  givenname: John
  surname: Danias
  fullname: Danias, John
  organization: Department of Ophthalmology, SUNY Downstate, NY, USA
– sequence: 6
  givenname: John
  surname: Simon
  fullname: Simon, John
  organization: The Jackson Laboratory, Bar Harbor, ME, USA
– sequence: 7
  givenname: Adriana
  surname: Di Polo
  fullname: Di Polo, Adriana
  organization: Department of Neuroscience and Centre de Recherche du Centre Hospitalier de l'Université de Montréal, Université de Montréal, Montréal, Québec, Canada
– sequence: 8
  givenname: Markus H.
  surname: Kuehn
  fullname: Kuehn, Markus H.
  organization: Department of Ophthalmology and Visual Sciences, Carver College of Medicine, University of Iowa, Iowa City, IA, USA
– sequence: 9
  givenname: Serge
  surname: Przedborski
  fullname: Przedborski, Serge
  organization: Departments of Pathology & Cell Biology, Columbia University Medical Center, NY, USA
– sequence: 10
  givenname: Martin
  surname: Raff
  fullname: Raff, Martin
  organization: MRC Laboratory for Molecular Cell Biology, University College London, London, UK
– sequence: 11
  givenname: Ian
  surname: Trounce
  fullname: Trounce, Ian
  organization: Centre for Eye Research Australia, Royal Victorian Eye and Ear Hospital, University of Melbourne, East Melbourne, Victoria, Australia
BackLink https://www.ncbi.nlm.nih.gov/pubmed/28242160$$D View this record in MEDLINE/PubMed
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Keywords Glaucoma
Monocyte
Neurodegeneration
Lasker
Inflammation
Astrocyte
Microglia
Language English
License Copyright © 2017 Elsevier Ltd. All rights reserved.
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Authors contributed equally
The following participated in the session “Inflammation and Glaucomatous Neurodegeneration”: Discussion leaders: Nick Marsh-Armstrong, Gareth R Howell. Scribe: Pete A Williams. Discussion Participants: Alejandra Bosco, John Danias, Adriana Di Polo, Simon John, Markus H. Kuehn, Serge Przedborski, Martin Raff, Ian Trounce
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PublicationTitle Experimental eye research
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Snippet Mounting evidence suggests neuroinflammation is a key process in glaucoma, yet the precise roles are not known. Understanding these complex processes, which...
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SubjectTerms Animals
Astrocyte
Astrocytes - pathology
Glaucoma
Glaucoma - physiopathology
Humans
Inflammation
Inflammation - physiopathology
Lasker
Microglia
Monocyte
Neurodegeneration
Optic Nerve - immunology
Optic Neuritis - physiopathology
Retinal Ganglion Cells - immunology
Title Neuroinflammation in glaucoma: A new opportunity
URI https://dx.doi.org/10.1016/j.exer.2017.02.014
https://www.ncbi.nlm.nih.gov/pubmed/28242160
https://www.proquest.com/docview/1872884242
https://pubmed.ncbi.nlm.nih.gov/PMC5497582
Volume 157
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