Repressing expression of difficult‐to‐express recombinant proteins during the selection process increases productivity of CHO stable pools

NRC publication: Yes

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Published in:Biotechnology and bioengineering Vol. 120; no. 10; pp. 2840 - 2852
Main Authors: Maltais, Jean‐Sébastien, Lord Dufour, Simon, Morasse, Audrey, Stuible, Matthew, Loignon, Martin, Durocher, Yves
Format: Journal Article
Language:English
Published: United States Wiley 01.10.2023
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ISSN:1097-0290, 0006-3592, 1097-0290
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Abstract NRC publication: Yes
More than half of licensed therapeutic recombinant proteins (r‐proteins) are manufac-tured using constitutively‐expressing, stably‐transfected Chinese hamster ovary (CHO)clones. While constitutive CHO expression systems have proven their efficacy for themanufacturing of monoclonal antibodies, many next‐generation therapeutics such ascytokines and bispecific antibodies as well as biological targets such as ectodomains oftransmembrane receptors remain intrinsically challenging to produce. Herein, weexploited a cumate‐inducible CHO platform allowing reduced expression of variousclasses of r‐proteins during selection of stable pools. Following stable pool generation,fed‐batch productions showed that pools generated without cumate (OFF‐pools) weresignificantly more productive than pools selected in the presence of cumate (ON‐pools)for 8 out of the 10 r‐proteins tested, including cytokines, G‐protein coupled receptors(GPCRs), the HVEM membrane receptor ectodomain, the multifunctional protein HighMobility Group protein B1 (HMGB1), as well as monoclonal and bispecific T‐cell engagerantibodies. We showed that OFF‐pools contain a significantly larger proportion of cellsproducing high levels of r‐proteins and that these cells tend to proliferate faster whenexpression is turned off, suggesting that r‐protein overexpression imposes a metabolicburden on the cells. Cell viability was lower and pool recovery was delayed duringselection of ON‐pools (mimicking constitutive expression), suggesting that high producerswere likely lost or overgrown by faster‐growing, low‐producing cells. We also observed acorrelation between the expression levels of the GPCRs with Binding immunoglobulinProtein, an endoplasmic reticulum (ER) stress marker. Taken together, these data suggestthat using an inducible system to minimize r‐protein expression during stable CHO poolselection reduces cellular stresses, including ER stress and metabolic burden, leading topools with greater frequency of high‐expressing cells, resulting in improved volumetricproductivity.
AbstractList More than half of licensed therapeutic recombinant proteins (r‐proteins) are manufactured using constitutively‐expressing, stably‐transfected Chinese hamster ovary (CHO) clones. While constitutive CHO expression systems have proven their efficacy for the manufacturing of monoclonal antibodies, many next‐generation therapeutics such as cytokines and bispecific antibodies as well as biological targets such as ectodomains of transmembrane receptors remain intrinsically challenging to produce. Herein, we exploited a cumate‐inducible CHO platform allowing reduced expression of various classes of r‐proteins during selection of stable pools. Following stable pool generation, fed‐batch productions showed that pools generated without cumate (OFF‐pools) were significantly more productive than pools selected in the presence of cumate (ON‐pools) for 8 out of the 10 r‐proteins tested, including cytokines, G‐protein coupled receptors (GPCRs), the HVEM membrane receptor ectodomain, the multifunctional protein High Mobility Group protein B1 (HMGB1), as well as monoclonal and bispecific T‐cell engager antibodies. We showed that OFF‐pools contain a significantly larger proportion of cells producing high levels of r‐proteins and that these cells tend to proliferate faster when expression is turned off, suggesting that r‐protein overexpression imposes a metabolic burden on the cells. Cell viability was lower and pool recovery was delayed during selection of ON‐pools (mimicking constitutive expression), suggesting that high producers were likely lost or overgrown by faster‐growing, low‐producing cells. We also observed a correlation between the expression levels of the GPCRs with Binding immunoglobulin Protein, an endoplasmic reticulum (ER) stress marker. Taken together, these data suggest that using an inducible system to minimize r‐protein expression during stable CHO pool selection reduces cellular stresses, including ER stress and metabolic burden, leading to pools with greater frequency of high‐expressing cells, resulting in improved volumetric productivity.
More than half of licensed therapeutic recombinant proteins (r-proteins) are manufactured using constitutively-expressing, stably-transfected Chinese hamster ovary (CHO) clones. While constitutive CHO expression systems have proven their efficacy for the manufacturing of monoclonal antibodies, many next-generation therapeutics such as cytokines and bispecific antibodies as well as biological targets such as ectodomains of transmembrane receptors remain intrinsically challenging to produce. Herein, we exploited a cumate-inducible CHO platform allowing reduced expression of various classes of r-proteins during selection of stable pools. Following stable pool generation, fed-batch productions showed that pools generated without cumate (OFF-pools) were significantly more productive than pools selected in the presence of cumate (ON-pools) for 8 out of the 10 r-proteins tested, including cytokines, G-protein coupled receptors (GPCRs), the HVEM membrane receptor ectodomain, the multifunctional protein High Mobility Group protein B1 (HMGB1), as well as monoclonal and bispecific T-cell engager antibodies. We showed that OFF-pools contain a significantly larger proportion of cells producing high levels of r-proteins and that these cells tend to proliferate faster when expression is turned off, suggesting that r-protein overexpression imposes a metabolic burden on the cells. Cell viability was lower and pool recovery was delayed during selection of ON-pools (mimicking constitutive expression), suggesting that high producers were likely lost or overgrown by faster-growing, low-producing cells. We also observed a correlation between the expression levels of the GPCRs with Binding immunoglobulin Protein, an endoplasmic reticulum (ER) stress marker. Taken together, these data suggest that using an inducible system to minimize r-protein expression during stable CHO pool selection reduces cellular stresses, including ER stress and metabolic burden, leading to pools with greater frequency of high-expressing cells, resulting in improved volumetric productivity.
NRC publication: Yes
More than half of licensed therapeutic recombinant proteins (r-proteins) are manufactured using constitutively-expressing, stably-transfected Chinese hamster ovary (CHO) clones. While constitutive CHO expression systems have proven their efficacy for the manufacturing of monoclonal antibodies, many next-generation therapeutics such as cytokines and bispecific antibodies as well as biological targets such as ectodomains of transmembrane receptors remain intrinsically challenging to produce. Herein, we exploited a cumate-inducible CHO platform allowing reduced expression of various classes of r-proteins during selection of stable pools. Following stable pool generation, fed-batch productions showed that pools generated without cumate (OFF-pools) were significantly more productive than pools selected in the presence of cumate (ON-pools) for 8 out of the 10 r-proteins tested, including cytokines, G-protein coupled receptors (GPCRs), the HVEM membrane receptor ectodomain, the multifunctional protein High Mobility Group protein B1 (HMGB1), as well as monoclonal and bispecific T-cell engager antibodies. We showed that OFF-pools contain a significantly larger proportion of cells producing high levels of r-proteins and that these cells tend to proliferate faster when expression is turned off, suggesting that r-protein overexpression imposes a metabolic burden on the cells. Cell viability was lower and pool recovery was delayed during selection of ON-pools (mimicking constitutive expression), suggesting that high producers were likely lost or overgrown by faster-growing, low-producing cells. We also observed a correlation between the expression levels of the GPCRs with Binding immunoglobulin Protein, an endoplasmic reticulum (ER) stress marker. Taken together, these data suggest that using an inducible system to minimize r-protein expression during stable CHO pool selection reduces cellular stresses, including ER stress and metabolic burden, leading to pools with greater frequency of high-expressing cells, resulting in improved volumetric productivity.More than half of licensed therapeutic recombinant proteins (r-proteins) are manufactured using constitutively-expressing, stably-transfected Chinese hamster ovary (CHO) clones. While constitutive CHO expression systems have proven their efficacy for the manufacturing of monoclonal antibodies, many next-generation therapeutics such as cytokines and bispecific antibodies as well as biological targets such as ectodomains of transmembrane receptors remain intrinsically challenging to produce. Herein, we exploited a cumate-inducible CHO platform allowing reduced expression of various classes of r-proteins during selection of stable pools. Following stable pool generation, fed-batch productions showed that pools generated without cumate (OFF-pools) were significantly more productive than pools selected in the presence of cumate (ON-pools) for 8 out of the 10 r-proteins tested, including cytokines, G-protein coupled receptors (GPCRs), the HVEM membrane receptor ectodomain, the multifunctional protein High Mobility Group protein B1 (HMGB1), as well as monoclonal and bispecific T-cell engager antibodies. We showed that OFF-pools contain a significantly larger proportion of cells producing high levels of r-proteins and that these cells tend to proliferate faster when expression is turned off, suggesting that r-protein overexpression imposes a metabolic burden on the cells. Cell viability was lower and pool recovery was delayed during selection of ON-pools (mimicking constitutive expression), suggesting that high producers were likely lost or overgrown by faster-growing, low-producing cells. We also observed a correlation between the expression levels of the GPCRs with Binding immunoglobulin Protein, an endoplasmic reticulum (ER) stress marker. Taken together, these data suggest that using an inducible system to minimize r-protein expression during stable CHO pool selection reduces cellular stresses, including ER stress and metabolic burden, leading to pools with greater frequency of high-expressing cells, resulting in improved volumetric productivity.
Author Stuible, Matthew
Durocher, Yves
Lord Dufour, Simon
Loignon, Martin
Maltais, Jean‐Sébastien
Morasse, Audrey
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Cites_doi 10.1016/S0959-8049(94)00401-3
10.1016/j.biotechadv.2021.107831
10.1110/ps.051489405
10.1038/ncb0311-184
10.1002/bit.26280
10.1016/j.biotechadv.2011.08.022
10.1371/journal.pone.0183694
10.1073/pnas.89.12.5547
10.1182/blood-2008-12-189266
10.1007/s11095-010-0172-0
10.1007/s10529-014-1537-y
10.1002/btpr.2495
10.1002/btpr.2772
10.3390/ph6050579
10.1016/j.jbiotec.2017.04.028
10.2174/138920110791111960
10.1016/J.JBIOTEC.2022.03.010
10.1002/bit.25687
10.1007/s10616-006-9041-4
10.1016/j.ymben.2012.12.003
10.3390/pr5020020
10.1016/j.imlet.2017.08.010
10.1002/bit.28387
10.3389/fimmu.2020.615603
10.3390/ANTIB8030043
10.1002/btpr.2572
10.3389/fonc.2017.00078
10.1080/19420862.2021.2005507
10.1002/btpr.2548
10.1002/bit.28127
10.1002/btpr.3186
10.1089/ars.2009.2485
10.3389/FBIOE.2021.658325
10.1016/j.biotechadv.2020.107552
10.1016/j.ejpb.2011.01.005
10.1016/J.COCHE.2018.09.010
10.1002/btpr.2467
10.1002/btpr.9
10.1002/bit.22723
10.1007/s10616-005-3765-4
10.1002/btpr.2839
10.1002/btpr.2493
10.1038/sj.embor.7400779
10.1186/1472-6750-6-43
10.1016/J.JBIOTEC.2019.03.009
10.1002/bit.25116
10.1007/s10529-009-0050-1
10.1124/jpet.108.138263
10.1016/J.BBAMCR.2013.06.028
10.1002/bit.20566
10.1016/j.jbiotec.2017.06.009
10.1002/btpr.2439
10.1002/BIT.27995
10.1016/j.ddtec.2021.02.003
10.1016/j.copbio.2009.10.008
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Keywords ER stress
difficult-to-express protein
inducible expression
cumate
productivity
CHO cells
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References e_1_2_9_31_1
e_1_2_9_52_1
e_1_2_9_50_1
e_1_2_9_10_1
e_1_2_9_35_1
e_1_2_9_56_1
e_1_2_9_12_1
e_1_2_9_33_1
e_1_2_9_54_1
e_1_2_9_14_1
e_1_2_9_39_1
e_1_2_9_16_1
e_1_2_9_37_1
e_1_2_9_58_1
e_1_2_9_18_1
e_1_2_9_41_1
e_1_2_9_20_1
e_1_2_9_22_1
e_1_2_9_45_1
e_1_2_9_24_1
e_1_2_9_43_1
e_1_2_9_8_1
e_1_2_9_6_1
Pedrazzoli P. (e_1_2_9_32_1) 1996; 16
e_1_2_9_4_1
e_1_2_9_2_1
Nims R. W. (e_1_2_9_27_1) 2006; 123
e_1_2_9_26_1
e_1_2_9_49_1
e_1_2_9_47_1
Nissen C. (e_1_2_9_28_1) 1994; 30
e_1_2_9_30_1
e_1_2_9_53_1
e_1_2_9_51_1
e_1_2_9_11_1
e_1_2_9_34_1
e_1_2_9_57_1
e_1_2_9_13_1
e_1_2_9_55_1
Querol S. (e_1_2_9_36_1) 1999; 84
e_1_2_9_15_1
e_1_2_9_38_1
e_1_2_9_17_1
e_1_2_9_59_1
e_1_2_9_19_1
e_1_2_9_42_1
e_1_2_9_40_1
e_1_2_9_21_1
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e_1_2_9_23_1
e_1_2_9_44_1
e_1_2_9_7_1
e_1_2_9_5_1
e_1_2_9_3_1
e_1_2_9_9_1
e_1_2_9_25_1
e_1_2_9_27_2
e_1_2_9_48_1
e_1_2_9_29_1
References_xml – volume: 30
  start-page: S12
  issue: 3
  year: 1994
  ident: e_1_2_9_28_1
  article-title: Glycosylation of recombinant human granulocyte colony stimulating factor: Implications for stability and potency
  publication-title: European Journal of Cancer (Oxford, England: 1990)
  doi: 10.1016/S0959-8049(94)00401-3
– ident: e_1_2_9_14_1
  doi: 10.1016/j.biotechadv.2021.107831
– ident: e_1_2_9_38_1
  doi: 10.1110/ps.051489405
– ident: e_1_2_9_47_1
  doi: 10.1038/ncb0311-184
– ident: e_1_2_9_12_1
  doi: 10.1002/bit.26280
– ident: e_1_2_9_59_1
  doi: 10.1016/j.biotechadv.2011.08.022
– ident: e_1_2_9_40_1
  doi: 10.1371/journal.pone.0183694
– ident: e_1_2_9_13_1
  doi: 10.1073/pnas.89.12.5547
– ident: e_1_2_9_3_1
  doi: 10.1182/blood-2008-12-189266
– ident: e_1_2_9_51_1
  doi: 10.1007/s11095-010-0172-0
– ident: e_1_2_9_16_1
  doi: 10.1007/s10529-014-1537-y
– ident: e_1_2_9_37_1
  doi: 10.1002/btpr.2495
– ident: e_1_2_9_48_1
  doi: 10.1002/btpr.2772
– ident: e_1_2_9_21_1
  doi: 10.3390/ph6050579
– volume: 123
  start-page: 153
  year: 2006
  ident: e_1_2_9_27_1
  article-title: Detection of adventitious viruses in biologicals—A rare occurrence
  publication-title: Developments in Biologicals
– ident: e_1_2_9_22_1
  doi: 10.1016/j.jbiotec.2017.04.028
– ident: e_1_2_9_30_1
  doi: 10.2174/138920110791111960
– ident: e_1_2_9_43_1
  doi: 10.1016/J.JBIOTEC.2022.03.010
– ident: e_1_2_9_17_1
  doi: 10.1002/bit.25687
– ident: e_1_2_9_11_1
  doi: 10.1007/s10616-006-9041-4
– ident: e_1_2_9_24_1
  doi: 10.1016/j.ymben.2012.12.003
– ident: e_1_2_9_55_1
  doi: 10.3390/pr5020020
– ident: e_1_2_9_39_1
  doi: 10.1016/j.imlet.2017.08.010
– volume: 84
  start-page: 493
  issue: 6
  year: 1999
  ident: e_1_2_9_36_1
  article-title: Effect of glycosylation of recombinant human granulocytic colony‐stimulating factor on expansion cultures of umbilical cord blood CD34+ cells
  publication-title: Haematologica
– ident: e_1_2_9_20_1
  doi: 10.1002/bit.28387
– ident: e_1_2_9_57_1
  doi: 10.3389/fimmu.2020.615603
– ident: e_1_2_9_53_1
  doi: 10.3390/ANTIB8030043
– ident: e_1_2_9_26_1
  doi: 10.1002/btpr.2572
– ident: e_1_2_9_6_1
  doi: 10.3389/fonc.2017.00078
– ident: e_1_2_9_56_1
  doi: 10.1080/19420862.2021.2005507
– ident: e_1_2_9_54_1
  doi: 10.1002/btpr.2548
– ident: e_1_2_9_19_1
  doi: 10.1002/bit.28127
– ident: e_1_2_9_58_1
  doi: 10.1002/btpr.3186
– ident: e_1_2_9_52_1
  doi: 10.1089/ars.2009.2485
– ident: e_1_2_9_9_1
  doi: 10.3389/FBIOE.2021.658325
– ident: e_1_2_9_27_2
– ident: e_1_2_9_29_1
  doi: 10.1016/j.biotechadv.2020.107552
– ident: e_1_2_9_7_1
  doi: 10.1016/j.ejpb.2011.01.005
– ident: e_1_2_9_44_1
  doi: 10.1016/J.COCHE.2018.09.010
– ident: e_1_2_9_15_1
  doi: 10.1002/btpr.2467
– ident: e_1_2_9_45_1
  doi: 10.1002/btpr.9
– ident: e_1_2_9_5_1
  doi: 10.1002/bit.22723
– ident: e_1_2_9_49_1
  doi: 10.1007/s10616-005-3765-4
– ident: e_1_2_9_31_1
  doi: 10.1002/btpr.2839
– ident: e_1_2_9_42_1
  doi: 10.1002/btpr.2493
– ident: e_1_2_9_46_1
  doi: 10.1038/sj.embor.7400779
– volume: 16
  start-page: 1781
  issue: 4
  year: 1996
  ident: e_1_2_9_32_1
  article-title: Effects of glycosylated and non‐glycosylated G‐CSFs, alone and in combination with other cytokines, on the growth of human progenitor cells
  publication-title: Anticancer Research
– ident: e_1_2_9_25_1
  doi: 10.1186/1472-6750-6-43
– ident: e_1_2_9_33_1
  doi: 10.1016/J.JBIOTEC.2019.03.009
– ident: e_1_2_9_35_1
  doi: 10.1002/bit.25116
– ident: e_1_2_9_4_1
  doi: 10.1007/s10529-009-0050-1
– ident: e_1_2_9_8_1
  doi: 10.1124/jpet.108.138263
– ident: e_1_2_9_41_1
  doi: 10.1016/J.BBAMCR.2013.06.028
– ident: e_1_2_9_18_1
  doi: 10.1002/bit.20566
– ident: e_1_2_9_34_1
  doi: 10.1016/j.jbiotec.2017.06.009
– ident: e_1_2_9_23_1
  doi: 10.1002/btpr.2439
– ident: e_1_2_9_2_1
  doi: 10.1002/BIT.27995
– ident: e_1_2_9_50_1
  doi: 10.1016/j.ddtec.2021.02.003
– ident: e_1_2_9_10_1
  doi: 10.1016/j.copbio.2009.10.008
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More than half of licensed therapeutic recombinant proteins (r‐proteins) are manufactured using constitutively‐expressing, stably‐transfected Chinese hamster...
More than half of licensed therapeutic recombinant proteins (r-proteins) are manufactured using constitutively-expressing, stably-transfected Chinese hamster...
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SubjectTerms Antibodies
Bispecific antibodies
Cell surface receptors
Cell viability
Cellular stress response
CHO cells
cumate
Cytokines
difficult‐to‐express protein
Endoplasmic reticulum
ER stress
High mobility group proteins
HMGB1 protein
inducible expression
Metabolism
Monoclonal antibodies
produc
Productivity
Proteins
Receptors
Title Repressing expression of difficult‐to‐express recombinant proteins during the selection process increases productivity of CHO stable pools
URI https://nrc-publications.canada.ca/eng/view/object/?id=493f0bb0-5867-49bb-be5a-1e1ac9568432
https://www.ncbi.nlm.nih.gov/pubmed/37232536
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https://www.proquest.com/docview/2820015958
Volume 120
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