Variable NK cell receptors exemplified by human KIR3DL1/S1

Variegated expression of variable NK cell receptors for polymorphic MHC class I broadens the range of an individual's NK cell response and the capacity for populations and species to survive disease epidemics and population bottlenecks. On evolutionary time scales, this component of immunity is...

Full description

Saved in:
Bibliographic Details
Published in:The Journal of immunology (1950) Vol. 187; no. 1; p. 11
Main Authors: Parham, Peter, Norman, Paul J, Abi-Rached, Laurent, Guethlein, Lisbeth A
Format: Journal Article
Language:English
Published: England 01.07.2011
Subjects:
ISSN:1550-6606, 1550-6606
Online Access:Get more information
Tags: Add Tag
No Tags, Be the first to tag this record!
Abstract Variegated expression of variable NK cell receptors for polymorphic MHC class I broadens the range of an individual's NK cell response and the capacity for populations and species to survive disease epidemics and population bottlenecks. On evolutionary time scales, this component of immunity is exceptionally dynamic, unstable, and short-lived, being dependent on coevolution of ligands and receptors subject to varying, competing selection pressures. Consequently these systems of variable NK cell receptors are largely species specific and have recruited different classes of glycoprotein, even within the primate order of mammals. Such disparity helps to explain substantial differences in NK cell biology between humans and animal models, for which the population genetics is largely ignored. KIR3DL1/S1, which recognizes the Bw4 epitope of HLA-A and -B and is the most extensively studied of the variable NK cell receptors, exemplifies how variation in all possible parameters of function is recruited to diversify the human NK cell response.
AbstractList Variegated expression of variable NK cell receptors for polymorphic MHC class I broadens the range of an individual's NK cell response and the capacity for populations and species to survive disease epidemics and population bottlenecks. On evolutionary time scales, this component of immunity is exceptionally dynamic, unstable, and short-lived, being dependent on coevolution of ligands and receptors subject to varying, competing selection pressures. Consequently these systems of variable NK cell receptors are largely species specific and have recruited different classes of glycoprotein, even within the primate order of mammals. Such disparity helps to explain substantial differences in NK cell biology between humans and animal models, for which the population genetics is largely ignored. KIR3DL1/S1, which recognizes the Bw4 epitope of HLA-A and -B and is the most extensively studied of the variable NK cell receptors, exemplifies how variation in all possible parameters of function is recruited to diversify the human NK cell response.Variegated expression of variable NK cell receptors for polymorphic MHC class I broadens the range of an individual's NK cell response and the capacity for populations and species to survive disease epidemics and population bottlenecks. On evolutionary time scales, this component of immunity is exceptionally dynamic, unstable, and short-lived, being dependent on coevolution of ligands and receptors subject to varying, competing selection pressures. Consequently these systems of variable NK cell receptors are largely species specific and have recruited different classes of glycoprotein, even within the primate order of mammals. Such disparity helps to explain substantial differences in NK cell biology between humans and animal models, for which the population genetics is largely ignored. KIR3DL1/S1, which recognizes the Bw4 epitope of HLA-A and -B and is the most extensively studied of the variable NK cell receptors, exemplifies how variation in all possible parameters of function is recruited to diversify the human NK cell response.
Variegated expression of variable NK cell receptors for polymorphic MHC class I broadens the range of an individual's NK cell response and the capacity for populations and species to survive disease epidemics and population bottlenecks. On evolutionary time scales, this component of immunity is exceptionally dynamic, unstable, and short-lived, being dependent on coevolution of ligands and receptors subject to varying, competing selection pressures. Consequently these systems of variable NK cell receptors are largely species specific and have recruited different classes of glycoprotein, even within the primate order of mammals. Such disparity helps to explain substantial differences in NK cell biology between humans and animal models, for which the population genetics is largely ignored. KIR3DL1/S1, which recognizes the Bw4 epitope of HLA-A and -B and is the most extensively studied of the variable NK cell receptors, exemplifies how variation in all possible parameters of function is recruited to diversify the human NK cell response.
Author Norman, Paul J
Abi-Rached, Laurent
Parham, Peter
Guethlein, Lisbeth A
Author_xml – sequence: 1
  givenname: Peter
  surname: Parham
  fullname: Parham, Peter
  organization: Department of Structural Biology, Stanford University School of Medicine, Stanford, CA 94305, USA
– sequence: 2
  givenname: Paul J
  surname: Norman
  fullname: Norman, Paul J
– sequence: 3
  givenname: Laurent
  surname: Abi-Rached
  fullname: Abi-Rached, Laurent
– sequence: 4
  givenname: Lisbeth A
  surname: Guethlein
  fullname: Guethlein, Lisbeth A
BackLink https://www.ncbi.nlm.nih.gov/pubmed/21690332$$D View this record in MEDLINE/PubMed
BookMark eNpNj0tLw0AUhQep2IfuXcnsXKW980zGndRXaVDwtQ2TyQ2mZJKYacD-eytWcHXO4uPjnCkZNW2DhJwzmEuQZrGpvB-atp6DAS4EPyITphREWoMe_etjMg1hAwAauDwhY860gT0_IVfvtq9sXiN9XFOHdU17dNht2z5Q_ELf1VVZYUHzHf0YvG3oevUsblK2eGGn5Li0dcCzQ87I293t6_IhSp_uV8vrNHJSJNuoLJgBG2OORiuHDArICyXBGaOSXCel5o5xrbRxzEoRQ8G5iXnuCiGkdpLPyOWvt-vbzwHDNvNV-JlqG2yHkCWxEEwlKt6TFwdyyD0WWddX3va77O8u_wYljVeH
CitedBy_id crossref_primary_10_1016_j_jtct_2024_02_014
crossref_primary_10_1155_2013_283805
crossref_primary_10_1371_journal_pone_0099543
crossref_primary_10_1016_j_clim_2016_11_010
crossref_primary_10_1200_JCO_2015_64_9558
crossref_primary_10_1097_QAD_0000000000001735
crossref_primary_10_3390_v11100959
crossref_primary_10_1016_j_bbmt_2011_10_033
crossref_primary_10_1097_COH_0b013e3283522b56
crossref_primary_10_1053_j_gastro_2018_07_019
crossref_primary_10_3389_fimmu_2017_01496
crossref_primary_10_1038_gene_2013_55
crossref_primary_10_3390_cancers13153767
crossref_primary_10_1038_ni_3513
crossref_primary_10_1007_s00262_016_1806_9
crossref_primary_10_3389_fimmu_2021_798235
crossref_primary_10_1186_1479_5876_11_262
crossref_primary_10_1097_MOT_0000000000000103
crossref_primary_10_1111_imr_12329
crossref_primary_10_1007_s00251_013_0724_7
crossref_primary_10_1016_j_it_2017_12_001
crossref_primary_10_3389_fimmu_2019_00024
crossref_primary_10_1111_tan_15251
crossref_primary_10_1016_j_vetimm_2012_09_022
crossref_primary_10_1016_j_placenta_2011_11_020
crossref_primary_10_1016_j_coi_2017_11_003
crossref_primary_10_1016_j_autrev_2012_10_007
crossref_primary_10_1016_j_humimm_2018_10_003
crossref_primary_10_3389_fimmu_2017_00547
crossref_primary_10_1111_cbdd_13658
crossref_primary_10_1155_2015_427217
crossref_primary_10_1016_j_cell_2024_10_005
crossref_primary_10_1371_journal_pone_0225151
crossref_primary_10_3389_fimmu_2017_00581
crossref_primary_10_4049_jimmunol_1201360
crossref_primary_10_1038_s41598_022_21062_1
crossref_primary_10_1158_1078_0432_CCR_18_3041
crossref_primary_10_1371_journal_pcbi_1009059
crossref_primary_10_1371_journal_pone_0064736
crossref_primary_10_1016_j_humimm_2022_07_006
crossref_primary_10_1038_s41409_018_0218_1
crossref_primary_10_1371_journal_pgen_1003938
crossref_primary_10_3389_fimmu_2014_00184
crossref_primary_10_1200_JCO_2016_70_7059
crossref_primary_10_3389_fimmu_2015_00134
crossref_primary_10_4049_jimmunol_1500243
crossref_primary_10_1016_j_humimm_2017_10_008
crossref_primary_10_1371_journal_pone_0163297
crossref_primary_10_1002_eji_201243007
crossref_primary_10_4049_jimmunol_1801178
crossref_primary_10_1111_tan_14782
ContentType Journal Article
DBID CGR
CUY
CVF
ECM
EIF
NPM
7X8
DOI 10.4049/jimmunol.0902332
DatabaseName Medline
MEDLINE
MEDLINE (Ovid)
MEDLINE
MEDLINE
PubMed
MEDLINE - Academic
DatabaseTitle MEDLINE
Medline Complete
MEDLINE with Full Text
PubMed
MEDLINE (Ovid)
MEDLINE - Academic
DatabaseTitleList MEDLINE - Academic
MEDLINE
Database_xml – sequence: 1
  dbid: NPM
  name: PubMed
  url: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 2
  dbid: 7X8
  name: MEDLINE - Academic
  url: https://search.proquest.com/medline
  sourceTypes: Aggregation Database
DeliveryMethod no_fulltext_linktorsrc
Discipline Medicine
Biology
EISSN 1550-6606
ExternalDocumentID 21690332
Genre Comparative Study
Review
Journal Article
Research Support, N.I.H., Extramural
GrantInformation_xml – fundername: NIAID NIH HHS
  grantid: R01 AI017892
– fundername: NIAID NIH HHS
  grantid: AI024258
– fundername: NIAID NIH HHS
  grantid: AI017892
– fundername: NIAID NIH HHS
  grantid: R01 AI031168
– fundername: NIAID NIH HHS
  grantid: AI022039
– fundername: NIAID NIH HHS
  grantid: AI031168
GroupedDBID ---
-~X
.55
0R~
18M
1KJ
2WC
34G
39C
3O-
53G
5GY
5RE
5VS
5WD
79B
85S
AARDX
ABCQX
ABDFA
ABEJV
ABGNP
ABJNI
ABOCM
ABPPZ
ABXVV
ACGFO
ACGFS
ACIWK
ACNCT
ACPRK
ADBBV
ADIPN
ADNWM
AENEX
AETEA
AFFNX
AFHIN
AFOSN
AFRAH
AGORE
AHMMS
AHWXS
AIZAD
ALMA_UNASSIGNED_HOLDINGS
ARBBW
BAWUL
BCRHZ
BTFSW
CGR
CUY
CVF
D0L
DIK
DU5
E3Z
EBS
ECM
EIF
EJD
F5P
GX1
H13
IH2
K-O
KQ8
L7B
NPM
OCZFY
OK1
OWPYF
P0W
P2P
PQQKQ
R.V
RHI
ROX
RZQ
SJN
TR2
TWZ
W8F
WH7
WOQ
X7M
XJT
XSW
XTH
YHG
7X8
KOP
ID FETCH-LOGICAL-c438t-fd190a7ebe965ce10d0bd540c9958b68f62c126569c1a4370d22972bcd3346c42
IEDL.DBID 7X8
ISICitedReferencesCount 53
ISICitedReferencesURI http://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=Summon&SrcAuth=ProQuest&DestLinkType=CitingArticles&DestApp=WOS_CPL&KeyUT=000291799300005&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
ISSN 1550-6606
IngestDate Wed Oct 01 13:33:48 EDT 2025
Mon Jul 21 05:43:55 EDT 2025
IsDoiOpenAccess false
IsOpenAccess true
IsPeerReviewed true
IsScholarly true
Issue 1
Language English
LinkModel DirectLink
MergedId FETCHMERGED-LOGICAL-c438t-fd190a7ebe965ce10d0bd540c9958b68f62c126569c1a4370d22972bcd3346c42
Notes ObjectType-Article-2
SourceType-Scholarly Journals-1
ObjectType-Feature-1
ObjectType-Review-3
content type line 23
OpenAccessLink https://www.jimmunol.org/content/jimmunol/187/1/11.full.pdf
PMID 21690332
PQID 873315857
PQPubID 23479
ParticipantIDs proquest_miscellaneous_873315857
pubmed_primary_21690332
PublicationCentury 2000
PublicationDate 2011-Jul-01
20110701
PublicationDateYYYYMMDD 2011-07-01
PublicationDate_xml – month: 07
  year: 2011
  text: 2011-Jul-01
  day: 01
PublicationDecade 2010
PublicationPlace England
PublicationPlace_xml – name: England
PublicationTitle The Journal of immunology (1950)
PublicationTitleAlternate J Immunol
PublicationYear 2011
SSID ssj0006024
Score 2.2757187
SecondaryResourceType review_article
Snippet Variegated expression of variable NK cell receptors for polymorphic MHC class I broadens the range of an individual's NK cell response and the capacity for...
SourceID proquest
pubmed
SourceType Aggregation Database
Index Database
StartPage 11
SubjectTerms Alleles
Animals
Disease Models, Animal
Evolution, Molecular
Histocompatibility Antigens Class I - genetics
Histocompatibility Antigens Class I - immunology
Histocompatibility Antigens Class I - metabolism
HLA-B Antigens - genetics
HLA-B Antigens - immunology
HLA-B Antigens - metabolism
Humans
Killer Cells, Natural - immunology
Killer Cells, Natural - metabolism
Peptide Fragments - immunology
Peptide Fragments - metabolism
Phylogeny
Receptors, KIR3DL1 - genetics
Receptors, KIR3DL1 - immunology
Receptors, KIR3DL1 - metabolism
Receptors, KIR3DS1 - genetics
Receptors, KIR3DS1 - immunology
Receptors, KIR3DS1 - metabolism
Title Variable NK cell receptors exemplified by human KIR3DL1/S1
URI https://www.ncbi.nlm.nih.gov/pubmed/21690332
https://www.proquest.com/docview/873315857
Volume 187
WOSCitedRecordID wos000291799300005&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
hasFullText
inHoldings 1
isFullTextHit
isPrint
link http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwpV3JTsMwEB0BBcSFfSmbfOBqmtiuk3BBCKhAbaOKTb1V8RKpCCWFFkT_nnGSwglx4JJTIlnj8fhl5s08gBNljfJSv0kjKxMqrJFUIcqmPnqTVcYakxZzZjtBHIf9ftSruDnjilY5i4lFoDa5djnyRujEBRHbBuejV-pEo1xxtVLQmIcaRyTjnDro_wwLl16paYsgnEoE6mWVUiAmbjwPXfdF_nLqaIncaY_8hi-Le6a19s8VrsNqBTDJRekRGzBns01YKiUnp5uw3K2K6Vtw9oT_ya5zisRt4hL4BKOfHTn5HWI_raOapwhQiZqSQsmPtG_v-FXHb9z72_DYun64vKGVlALVgocTmhq8-JMAdyySTW19z3jKIFjTUdQMlQxTybTPENtF2k8EDzzDWBQwpQ3nQmrBdmAhyzO7B4QLFSh8L_GkEVzKRKSe6-ZNhWEJxtY6kJl5BuiqbvlJZvP38eDbQHXYLU08GJUjNQbMVetwH_b__vgAVsq8rqPMHkItxWNqj2BRf0yG47fjwgXwGfe6X92ouag
linkProvider ProQuest
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Variable+NK+cell+receptors+exemplified+by+human+KIR3DL1%2FS1&rft.jtitle=The+Journal+of+immunology+%281950%29&rft.au=Parham%2C+Peter&rft.au=Norman%2C+Paul+J&rft.au=Abi-Rached%2C+Laurent&rft.au=Guethlein%2C+Lisbeth+A&rft.date=2011-07-01&rft.eissn=1550-6606&rft.volume=187&rft.issue=1&rft.spage=11&rft_id=info:doi/10.4049%2Fjimmunol.0902332&rft_id=info%3Apmid%2F21690332&rft_id=info%3Apmid%2F21690332&rft.externalDocID=21690332
thumbnail_l http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=1550-6606&client=summon
thumbnail_m http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=1550-6606&client=summon
thumbnail_s http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=1550-6606&client=summon