Acute Lung Injury Is an Independent Risk Factor for Brain Hypoxia After Severe Traumatic Brain Injury

Abstract BACKGROUND Pulmonary complications are frequently observed after severe traumatic brain injury (TBI), but little is known about the consequences of lung injury on brain tissue oxygenation and metabolism. OBJECTIVE We examined the association between lung function and brain tissue oxygen ten...

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Veröffentlicht in:	Neurosurgery Jg. 67; H. 2; S. 338 - 344
Hauptverfasser:	Oddo, Mauro, Nduom, Edjah, Frangos, Suzanne, MacKenzie, Larami, Chen, Isaac, Maloney-Wilensky, Eileen, Kofke, W. Andrew, Levine, Joshua M., LeRoux, Peter D.
Format:	Journal Article
Sprache:	Englisch
Veröffentlicht:	Hagerstown, MD Oxford University Press 01.08.2010 Lippincott Williams & Wilkins Wolters Kluwer Health, Inc
Schlagworte:	Acute Lung Injury - epidemiology Acute Lung Injury - etiology Adult APACHE Biological and medical sciences Brain Chemistry - physiology Brain Injuries - complications Brain Injuries - epidemiology Brain Injuries - therapy Female Glasgow Coma Scale Humans Hypoxia Hypoxia, Brain - epidemiology Hypoxia, Brain - etiology Intracranial Pressure - physiology Male Medical sciences Middle Aged Monitoring, Physiologic Neurosurgery Oxygen Consumption Respiration, Artificial Risk Factors Surgery (general aspects). Transplantations, organ and tissue grafts. Graft diseases Tomography, X-Ray Computed Traumatic brain injury Acute lung injury Lung function Traumatic brain injury Systemic oxygenation Brain hypoxia Brain tissue oxygen tension Lung disease Oxygen Nervous system diseases Respiratory disease Encephalon Surgery Risk factor Hypoxia Head trauma Oxygenation
ISSN:	0148-396X, 1524-4040, 1524-4040
Online-Zugang:	Volltext
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