Salt intake and blood pressure response to percutaneous renal denervation in resistant hypertension

The effect of lowering sympathetic nerve activity by renal denervation (RDN) is highly variable. With the exception of office systolic blood pressure (BP), predictors of the BP‐lowering effect have not been identified. Because dietary sodium intake influences sympathetic drive, and, conversely, symp...

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Vydané v:The journal of clinical hypertension (Greenwich, Conn.) Ročník 19; číslo 11; s. 1125 - 1133
Hlavní autori: Beus, Esther, Jager, Rosa L., Beeftink, Martine M., Sanders, Margreet F., Spiering, Wilko, Vonken, Evert‐Jan, Voskuil, Michiel, Bots, Michiel L., Blankestijn, Peter J., Elgersma, O, IJsselmuiden, AJJ, Valk, PHM, Smak Gregoor, P, Roodenburg, S, Meuwissen, M, Dewilde, W, Hunze, I, Hollander, J, Vincent, HH, Rensing, B, Bos, WJ, Weverwijk, I, Tonino, PAL, Brueren, BRG, Konings, CJAM, Gompel, H Hendrix‐van, Heeg, JE, Lambert, J, Smit, JJ, lvan, A, Berends, A, Jager, B, Laverman, G, Vries, PAM, Balen, A, Stoel, M, Steggerda, R, Niamut, L, Bossen, W, Biermann, J, Knot, I, Peels, JOJ, Swart, JB, Kimman, G, Bax, W, Meij, Y, Reekers, J, Wardeh, AJ, Groeneveld, JHM, Dille, C, Hemmelder, MH, Folkeringa, R, Sietzema, M, Wassenaar, C, Parlevliet, K, Aengevaeren, W, Tjon, M, Hovens, M, Berg, A, Monajemi, H, Kleij, FGH, chramm, A, Wiersum, A, Kroon, B, Haan, M, Das, M, Jongen, H, Herben, E, Rennenberg, R, Blankestijn, PJ, Bots, ML, Dijker, B, Frederix, GWJ, Jager, RL, Maarseveen, E, Sanders, MF, Spiering, W, Velikopolskaia, IS, Vendrig, L, Verloop, WL, Vink, EE, Vonken, EPA, Wit, GA, Vincent, J, Roes, CB, Deinum, J, Rabelink, AJ, Lenders, J, Eijkemans, R
Médium: Journal Article
Jazyk:English
Vydavateľské údaje: United States John Wiley and Sons Inc 01.11.2017
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ISSN:1524-6175, 1751-7176, 1751-7176
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Abstract The effect of lowering sympathetic nerve activity by renal denervation (RDN) is highly variable. With the exception of office systolic blood pressure (BP), predictors of the BP‐lowering effect have not been identified. Because dietary sodium intake influences sympathetic drive, and, conversely, sympathetic activity influences salt sensitivity in hypertension, we investigated 24‐hour urinary sodium excretion in participants of the SYMPATHY trial. SYMPATHY investigated RDN in patients with resistant hypertension. Both 24‐hour ambulatory and office BP measurements were end points. No relationship was found for baseline sodium excretion and change in BP 6 months after RDN in multivariable‐adjusted regression analysis. Change in the salt intake–measured BP relationships at 6 months vs baseline was used as a measure for salt sensitivity. BP was 8 mm Hg lower with similar salt intake after RDN, suggesting a decrease in salt sensitivity. However, the change was similar in the control group, and thus not attributable to RDN.
AbstractList The effect of lowering sympathetic nerve activity by renal denervation (RDN) is highly variable. With the exception of office systolic blood pressure (BP), predictors of the BP‐lowering effect have not been identified. Because dietary sodium intake influences sympathetic drive, and, conversely, sympathetic activity influences salt sensitivity in hypertension, we investigated 24‐hour urinary sodium excretion in participants of the SYMPATHY trial. SYMPATHY investigated RDN in patients with resistant hypertension. Both 24‐hour ambulatory and office BP measurements were end points. No relationship was found for baseline sodium excretion and change in BP 6 months after RDN in multivariable‐adjusted regression analysis. Change in the salt intake–measured BP relationships at 6 months vs baseline was used as a measure for salt sensitivity. BP was 8 mm Hg lower with similar salt intake after RDN, suggesting a decrease in salt sensitivity. However, the change was similar in the control group, and thus not attributable to RDN.
The effect of lowering sympathetic nerve activity by renal denervation (RDN) is highly variable. With the exception of office systolic blood pressure (BP), predictors of the BP-lowering effect have not been identified. Because dietary sodium intake influences sympathetic drive, and, conversely, sympathetic activity influences salt sensitivity in hypertension, we investigated 24-hour urinary sodium excretion in participants of the SYMPATHY trial. SYMPATHY investigated RDN in patients with resistant hypertension. Both 24-hour ambulatory and office BP measurements were end points. No relationship was found for baseline sodium excretion and change in BP 6 months after RDN in multivariable-adjusted regression analysis. Change in the salt intake-measured BP relationships at 6 months vs baseline was used as a measure for salt sensitivity. BP was 8 mm Hg lower with similar salt intake after RDN, suggesting a decrease in salt sensitivity. However, the change was similar in the control group, and thus not attributable to RDN.The effect of lowering sympathetic nerve activity by renal denervation (RDN) is highly variable. With the exception of office systolic blood pressure (BP), predictors of the BP-lowering effect have not been identified. Because dietary sodium intake influences sympathetic drive, and, conversely, sympathetic activity influences salt sensitivity in hypertension, we investigated 24-hour urinary sodium excretion in participants of the SYMPATHY trial. SYMPATHY investigated RDN in patients with resistant hypertension. Both 24-hour ambulatory and office BP measurements were end points. No relationship was found for baseline sodium excretion and change in BP 6 months after RDN in multivariable-adjusted regression analysis. Change in the salt intake-measured BP relationships at 6 months vs baseline was used as a measure for salt sensitivity. BP was 8 mm Hg lower with similar salt intake after RDN, suggesting a decrease in salt sensitivity. However, the change was similar in the control group, and thus not attributable to RDN.
The effect of lowering sympathetic nerve activity by renal denervation ( RDN ) is highly variable. With the exception of office systolic blood pressure ( BP ), predictors of the BP ‐lowering effect have not been identified. Because dietary sodium intake influences sympathetic drive, and, conversely, sympathetic activity influences salt sensitivity in hypertension, we investigated 24‐hour urinary sodium excretion in participants of the SYMPATHY trial. SYMPATHY investigated RDN in patients with resistant hypertension. Both 24‐hour ambulatory and office BP measurements were end points. No relationship was found for baseline sodium excretion and change in BP 6 months after RDN in multivariable‐adjusted regression analysis. Change in the salt intake–measured BP relationships at 6 months vs baseline was used as a measure for salt sensitivity. BP was 8 mm Hg lower with similar salt intake after RDN , suggesting a decrease in salt sensitivity. However, the change was similar in the control group, and thus not attributable to RDN .
Author Herben, E
Folkeringa, R
Hovens, M
Bots, ML
Dille, C
Maarseveen, E
Biermann, J
Hemmelder, MH
Laverman, G
IJsselmuiden, AJJ
Valk, PHM
Vendrig, L
Rensing, B
Spiering, W
Swart, JB
Wardeh, AJ
Beeftink, Martine M.
Parlevliet, K
Spiering, Wilko
Bossen, W
Hunze, I
Vink, EE
Roes, CB
Smak Gregoor, P
Aengevaeren, W
Kleij, FGH
Gompel, H Hendrix‐van
Dijker, B
Lenders, J
Blankestijn, PJ
Rabelink, AJ
lvan, A
Steggerda, R
Jongen, H
Verloop, WL
Deinum, J
Das, M
Peels, JOJ
Jager, RL
Eijkemans, R
Beus, Esther
Weverwijk, I
Sietzema, M
Bos, WJ
Haan, M
Roodenburg, S
Reekers, J
Wiersum, A
Brueren, BRG
Jager, B
Elgersma, O
Voskuil, Michiel
Blankestijn, Peter J.
Kimman, G
Frederix, GWJ
Meuwissen, M
Sanders, MF
Vonken, EPA
Vonken, Evert‐Jan
Knot, I
Bots, Michiel L.
Groeneveld, JHM
Hollander, J
Velikopolskaia, IS
Dewilde, W
Berg, A
Balen, A
Vries, PAM
Sanders, Margreet F.
Vincent, HH
Bax, W
Tonino, PAL
Wassenaar, C
Lambert, J
Niamut, L
Heeg, JE
Konings, CJAM
Kroon, B
Monajemi, H
Wit, GA
Berends, A
Smit, JJ
chramm, A
Vincent, J
Jager, Rosa L.
Stoel, M
Meij, Y
Rennenberg, R
Tjon, M
AuthorAffiliation 5 Julius Center for Health Sciences and Primary Care University Medical Center Utrecht Utrecht The Netherlands
1 Department of Nephrology and Hypertension University Medical Center Utrecht Utrecht The Netherlands
2 Department of Cardiology University Medical Center Utrecht Utrecht The Netherlands
4 Department of Radiology University Medical Center Utrecht Utrecht The Netherlands
3 Department of Vascular Medicine University Medical Center Utrecht Utrecht The Netherlands
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– name: 1 Department of Nephrology and Hypertension University Medical Center Utrecht Utrecht The Netherlands
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– name: 5 Julius Center for Health Sciences and Primary Care University Medical Center Utrecht Utrecht The Netherlands
– name: 3 Department of Vascular Medicine University Medical Center Utrecht Utrecht The Netherlands
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BackLink https://www.ncbi.nlm.nih.gov/pubmed/28929577$$D View this record in MEDLINE/PubMed
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CitedBy_id crossref_primary_10_1007_s00467_020_04896_7
crossref_primary_10_1097_HJH_0000000000003655
crossref_primary_10_1097_HJH_0000000000003050
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ContentType Journal Article
Contributor Herben, E
Folkeringa, R
Hovens, M
Verloop, W L
Dille, C
de Vries, Pam
Biermann, J
Hemmelder, M H
Vincent, H H
Laverman, G
IJsselmuiden, Ajj
An Weverwijk, I
Vendrig, L
Smit, J J
Chramm, A
Rensing, B
Spiering, W
Wardeh, A J
Parlevliet, K
Sanders, M F
Bossen, W
Hunze, I
Lvan, A
Smak Gregoor, P
Aengevaeren, W
Dijker, B
Lenders, J
Steggerda, R
Jongen, H
Deinum, J
Das, M
de Swart, J B
Peels, Joj
Eijkemans, R
Sietzema, M
Vink, E E
Roodenburg, S
Reekers, J
Wiersum, A
Brueren, Brg
Bots, M L
Elgersma, O
van der Meij, Y
de Wit, G A
Kimman, G
van den Berg, A
Frederix, Gwj
Meuwissen, M
Konings, Cjam
de Haan, M
van Maarseveen, E
van der Valk, Phm
Vonken, Epa
Blankestijn, P J
Knot, I
Groeneveld, Jhm
Roes, C B
Dewilde, W
de Jager, B
Bax, W
Tonino, Pal
Wassenaar, C
van Balen, A
Lambert, J
Niamut, L
Rabelink, A J
Kroon, B
Monajemi, H
Berends, A
Velikopolskaia, I S
Gompel, H Hendrix-van
Heeg, J E
den Hollander, J
Vincent, J
de Jager, R L
Stoel, M
van der Kleij, Fgh
Bos, W J
Rennenberg, R
Tjon, M
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Issue 11
Keywords renal denervation
sodium intake
hypertension
dietary sodium
salt sensitivity
Language English
License http://onlinelibrary.wiley.com/termsAndConditions#vor
2017 Wiley Periodicals, Inc.
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SYMPATHY is an investigator‐driven trial and received unrestricted grants from the Netherlands Organisation for Health Research and Development (ZonMw, No. 837004006), the Dutch Kidney Foundation (Nierstichting Nederland, No. CPI12.02), and Medtronic Inc.
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Snippet The effect of lowering sympathetic nerve activity by renal denervation (RDN) is highly variable. With the exception of office systolic blood pressure (BP),...
The effect of lowering sympathetic nerve activity by renal denervation ( RDN ) is highly variable. With the exception of office systolic blood pressure ( BP ),...
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SourceType Open Access Repository
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StartPage 1125
SubjectTerms Aged
Antihypertensive Agents - therapeutic use
Blood Pressure - drug effects
Blood Pressure Monitoring, Ambulatory - methods
dietary sodium
Drug Resistance
Female
Humans
hypertension
Hypertension - diagnosis
Hypertension - metabolism
Hypertension - physiopathology
Hypertension - surgery
Kidney - innervation
Kidney - physiopathology
Male
Middle Aged
Original Paper
Renal Denervation
salt sensitivity
Sodium - urine
Sodium Chloride, Dietary - metabolism
sodium intake
Sympathectomy - adverse effects
Sympathectomy - methods
Sympathetic Nervous System - drug effects
Sympathetic Nervous System - physiopathology
Treatment Outcome
Title Salt intake and blood pressure response to percutaneous renal denervation in resistant hypertension
URI https://onlinelibrary.wiley.com/doi/abs/10.1111%2Fjch.13085
https://www.ncbi.nlm.nih.gov/pubmed/28929577
https://www.proquest.com/docview/1941090727
https://pubmed.ncbi.nlm.nih.gov/PMC8030930
Volume 19
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