Protocol for elucidating metabolite binding and regulation of TET2 dioxygenase

Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the e...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:STAR protocols Jg. 6; H. 3; S. 104015
Hauptverfasser: Zhang, Shuyuan, Cheng, Zhou-Li, Ye, Dan
Format: Journal Article
Sprache:Englisch
Veröffentlicht: United States Elsevier Inc 19.09.2025
Elsevier
Schlagworte:
ISSN:2666-1667, 2666-1667
Online-Zugang:Volltext
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
Abstract Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation. For complete details on the use and execution of this protocol, please refer to Cheng et al.1 [Display omitted] •Instructions for the purification of highly active, tag-free human TET2CD protein•Procedures for flow cytometry-based detection of TET2 activity in vitro•Guidance on the simultaneous screening of TET2 regulatory metabolites•Steps for STD NMR-based detection of metabolite-TET2 interactions Publisher’s note: Undertaking any experimental protocol requires adherence to local institutional guidelines for laboratory safety and ethics. Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation.
AbstractList Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation. For complete details on the use and execution of this protocol, please refer to Cheng et al. .
Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation. For complete details on the use and execution of this protocol, please refer to Cheng et al.1 •Instructions for the purification of highly active, tag-free human TET2CD protein•Procedures for flow cytometry-based detection of TET2 activity in vitro•Guidance on the simultaneous screening of TET2 regulatory metabolites•Steps for STD NMR-based detection of metabolite-TET2 interactions Publisher’s note: Undertaking any experimental protocol requires adherence to local institutional guidelines for laboratory safety and ethics. Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation.
Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation. For complete details on the use and execution of this protocol, please refer to Cheng et al.1.Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation. For complete details on the use and execution of this protocol, please refer to Cheng et al.1.
Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation. For complete details on the use and execution of this protocol, please refer to Cheng et al.1 [Display omitted] •Instructions for the purification of highly active, tag-free human TET2CD protein•Procedures for flow cytometry-based detection of TET2 activity in vitro•Guidance on the simultaneous screening of TET2 regulatory metabolites•Steps for STD NMR-based detection of metabolite-TET2 interactions Publisher’s note: Undertaking any experimental protocol requires adherence to local institutional guidelines for laboratory safety and ethics. Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for combining biochemical assays and saturation transfer difference (STD) NMR spectroscopy to experimentally validate metabolite binding and assess the effect on TET2 activity. This protocol enables the identification of both TET2 activators and inhibitors, providing a framework for studying the interplay between metabolism and epigenetic regulation.
ArticleNumber 104015
Author Cheng, Zhou-Li
Zhang, Shuyuan
Ye, Dan
Author_xml – sequence: 1
  givenname: Shuyuan
  surname: Zhang
  fullname: Zhang, Shuyuan
  organization: Huashan Hospital, Fudan University, and International Co-laboratory of Medical Epigenetics and Metabolism of Ministry of Science and Technology, and Key Laboratory of Metabolism and Molecular Medicine of Ministry of Education, and Molecular and Cell Biology Lab, Institutes of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai 200032, China
– sequence: 2
  givenname: Zhou-Li
  surname: Cheng
  fullname: Cheng, Zhou-Li
  organization: Huashan Hospital, Fudan University, and International Co-laboratory of Medical Epigenetics and Metabolism of Ministry of Science and Technology, and Key Laboratory of Metabolism and Molecular Medicine of Ministry of Education, and Molecular and Cell Biology Lab, Institutes of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai 200032, China
– sequence: 3
  givenname: Dan
  surname: Ye
  fullname: Ye, Dan
  email: yedan@fudan.edu.cn
  organization: Huashan Hospital, Fudan University, and International Co-laboratory of Medical Epigenetics and Metabolism of Ministry of Science and Technology, and Key Laboratory of Metabolism and Molecular Medicine of Ministry of Education, and Molecular and Cell Biology Lab, Institutes of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai 200032, China
BackLink https://www.ncbi.nlm.nih.gov/pubmed/40773351$$D View this record in MEDLINE/PubMed
BookMark eNp9kUtr3DAUhUVIaR7NH-gieNnNTPWwJAsCoYS0KYS2i-layFfXUw0eaSLZIfn3sZkkJJuuJO79zjlwzwk5jCkiIZ8ZXTLK1NfN8mGX05JTLqdBTZk8IMdcKbVgSunDN_8jclbKhtKJZLxmzUdyVFOthZDsmPz6k9OQIPVVl3KF_QjBuyHEdbXFwbWpDwNWbYh-Hrnoq4zrsZ-IFKvUVavrFa98SA-Pa4yu4CfyoXN9wbPn95T8_X69urpZ3P7-8fPq2-0CamqGhYFOaO7qlnIwddeA0U3tsQUqKZWNM17w1njVMQHGc1MjSIpatcDBeW3EKbnc--7GdoseMA7Z9XaXw9blR5tcsO83Mfyz63RvGRdSSqYmhy_PDjndjVgGuw0FsO9dxDQWK7jQWolGzWHnb8NeU16uOAF8D0BOpWTsXhFG7dyW3di5LTu3ZfdtTaKLvQinO90HzLZAwAjoQ0YYrE_hf_InHpGeTw
Cites_doi 10.1021/ed101169t
10.1021/acschembio.3c00619
10.1016/j.cell.2013.11.020
10.1038/s41580-018-0029-7
10.1016/j.cmet.2012.06.001
10.1016/j.drudis.2011.12.016
10.1038/nrg.2017.33
10.1016/j.trecan.2017.12.005
10.1016/j.ccell.2018.07.014
10.1021/ja4028346
10.1016/j.cmet.2025.01.019
ContentType Journal Article
Copyright 2025 The Author(s)
Copyright © 2025 The Author(s). Published by Elsevier Inc. All rights reserved.
2025 The Author(s) 2025
Copyright_xml – notice: 2025 The Author(s)
– notice: Copyright © 2025 The Author(s). Published by Elsevier Inc. All rights reserved.
– notice: 2025 The Author(s) 2025
DBID 6I.
AAFTH
AAYXX
CITATION
CGR
CUY
CVF
ECM
EIF
NPM
7X8
5PM
DOI 10.1016/j.xpro.2025.104015
DatabaseName ScienceDirect Open Access Titles
Elsevier:ScienceDirect:Open Access
CrossRef
Medline
MEDLINE
MEDLINE (Ovid)
MEDLINE
MEDLINE
PubMed
MEDLINE - Academic
PubMed Central (Full Participant titles)
DatabaseTitle CrossRef
MEDLINE
Medline Complete
MEDLINE with Full Text
PubMed
MEDLINE (Ovid)
MEDLINE - Academic
DatabaseTitleList MEDLINE

MEDLINE - Academic

Database_xml – sequence: 1
  dbid: NPM
  name: PubMed
  url: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 2
  dbid: 7X8
  name: MEDLINE - Academic
  url: https://search.proquest.com/medline
  sourceTypes: Aggregation Database
DeliveryMethod fulltext_linktorsrc
EISSN 2666-1667
ExternalDocumentID PMC12355516
40773351
10_1016_j_xpro_2025_104015
S2666166725004216
Genre Journal Article
GroupedDBID 0R~
53G
6I.
AAEDW
AAFTH
AALRI
AAMRU
AAXUO
AAYWO
ACVFH
ADCNI
ADVLN
AEUPX
AEXQZ
AFPUW
AIGII
AITUG
AKBMS
AKRWK
AKYEP
ALMA_UNASSIGNED_HOLDINGS
AMRAJ
APXCP
EBS
FDB
GROUPED_DOAJ
M41
M~E
OK1
ROL
RPM
AAYXX
CITATION
CGR
CUY
CVF
ECM
EIF
NPM
7X8
5PM
ID FETCH-LOGICAL-c409t-9cf372a4b02c94f8c9784debc050058a9d32b9d6f13c9d294ec50e76bc2cad793
ISICitedReferencesCount 0
ISICitedReferencesURI http://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=Summon&SrcAuth=ProQuest&DestLinkType=CitingArticles&DestApp=WOS_CPL&KeyUT=001547197700001&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
ISSN 2666-1667
IngestDate Tue Sep 30 17:02:40 EDT 2025
Thu Oct 02 22:08:27 EDT 2025
Wed Sep 24 03:07:05 EDT 2025
Sat Nov 29 07:27:05 EST 2025
Sun Oct 19 01:40:33 EDT 2025
IsDoiOpenAccess true
IsOpenAccess true
IsPeerReviewed true
IsScholarly true
Issue 3
Keywords Protein Biochemistry
NMR
Molecular Biology
Protein expression and purification
Cell Biology
Language English
License This is an open access article under the CC BY-NC-ND license.
Copyright © 2025 The Author(s). Published by Elsevier Inc. All rights reserved.
This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).
LinkModel OpenURL
MergedId FETCHMERGED-LOGICAL-c409t-9cf372a4b02c94f8c9784debc050058a9d32b9d6f13c9d294ec50e76bc2cad793
Notes ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
content type line 23
Technical contact
These authors contributed equally
Lead contact
OpenAccessLink http://dx.doi.org/10.1016/j.xpro.2025.104015
PMID 40773351
PQID 3237763869
PQPubID 23479
ParticipantIDs pubmedcentral_primary_oai_pubmedcentral_nih_gov_12355516
proquest_miscellaneous_3237763869
pubmed_primary_40773351
crossref_primary_10_1016_j_xpro_2025_104015
elsevier_sciencedirect_doi_10_1016_j_xpro_2025_104015
PublicationCentury 2000
PublicationDate 2025-09-19
PublicationDateYYYYMMDD 2025-09-19
PublicationDate_xml – month: 09
  year: 2025
  text: 2025-09-19
  day: 19
PublicationDecade 2020
PublicationPlace United States
PublicationPlace_xml – name: United States
PublicationTitle STAR protocols
PublicationTitleAlternate STAR Protoc
PublicationYear 2025
Publisher Elsevier Inc
Elsevier
Publisher_xml – name: Elsevier Inc
– name: Elsevier
References Cheng, Zhang, Wang, Song, Gao, Song, Wang, Zhang, Zhou, Shan (bib1) 2025; 37
Shenoy, Creagan, Witzig, Levine (bib6) 2018; 34
Yin, Mao, Zhao, Chong, Yang, Zhao, Zhang, Huang, Gao, Li (bib7) 2013; 135
Ye, Guan, Xiong (bib5) 2018; 4
Viegas, Manso, Nobrega, Cabrita (bib11) 2011; 88
Lu, Thompson (bib3) 2012; 16
Li, Egervari, Wang, Berger, Lu (bib2) 2018; 19
Bhunia, Bhattacharjya, Chatterjee (bib10) 2012; 17
Treadway, Boyer, Yang, Yang, Liu, Li, Cheng, Marzluff, Ye, Xiong (bib9) 2024; 19
Hu, Li, Cheng, Rao, Gong, Liu, Shi, Zhu, Wang, Xu (bib8) 2013; 155
Wu, Zhang (bib4) 2017; 18
Ye (10.1016/j.xpro.2025.104015_bib5) 2018; 4
Cheng (10.1016/j.xpro.2025.104015_bib1) 2025; 37
Shenoy (10.1016/j.xpro.2025.104015_bib6) 2018; 34
Hu (10.1016/j.xpro.2025.104015_bib8) 2013; 155
Wu (10.1016/j.xpro.2025.104015_bib4) 2017; 18
Treadway (10.1016/j.xpro.2025.104015_bib9) 2024; 19
Yin (10.1016/j.xpro.2025.104015_bib7) 2013; 135
Bhunia (10.1016/j.xpro.2025.104015_bib10) 2012; 17
Lu (10.1016/j.xpro.2025.104015_bib3) 2012; 16
Viegas (10.1016/j.xpro.2025.104015_bib11) 2011; 88
Li (10.1016/j.xpro.2025.104015_bib2) 2018; 19
References_xml – volume: 34
  start-page: 700
  year: 2018
  end-page: 706
  ident: bib6
  article-title: Ascorbic Acid in Cancer Treatment: Let the Phoenix Fly
  publication-title: Cancer Cell
– volume: 155
  start-page: 1545
  year: 2013
  end-page: 1555
  ident: bib8
  article-title: Crystal structure of TET2-DNA complex: insight into TET-mediated 5mC oxidation
  publication-title: Cell
– volume: 17
  start-page: 505
  year: 2012
  end-page: 513
  ident: bib10
  article-title: Applications of saturation transfer difference NMR in biological systems
  publication-title: Drug Discov. Today
– volume: 37
  start-page: 1137
  year: 2025
  end-page: 1151.e5
  ident: bib1
  article-title: Pathogen-derived glyoxylate inhibits Tet2 DNA dioxygenase to facilitate bacterial persister formation
  publication-title: Cell Metab.
– volume: 18
  start-page: 517
  year: 2017
  end-page: 534
  ident: bib4
  article-title: TET-mediated active DNA demethylation: mechanism, function and beyond
  publication-title: Nat. Rev. Genet.
– volume: 88
  start-page: 990
  year: 2011
  end-page: 994
  ident: bib11
  article-title: Saturation-Transfer Difference (STD) NMR: A Simple and Fast Method for Ligand Screening and Characterization of Protein Binding
  publication-title: J. Chem. Educ.
– volume: 19
  start-page: 563
  year: 2018
  end-page: 578
  ident: bib2
  article-title: Regulation of chromatin and gene expression by metabolic enzymes and metabolites
  publication-title: Nat. Rev. Mol. Cell Biol.
– volume: 4
  start-page: 151
  year: 2018
  end-page: 165
  ident: bib5
  article-title: Metabolism, Activity, and Targeting of D- and L-2-Hydroxyglutarates
  publication-title: Trends Cancer
– volume: 19
  start-page: 15
  year: 2024
  end-page: 21
  ident: bib9
  article-title: Using NMR to Monitor TET-Dependent Methylcytosine Dioxygenase Activity and Regulation
  publication-title: ACS Chem. Biol.
– volume: 16
  start-page: 9
  year: 2012
  end-page: 17
  ident: bib3
  article-title: Metabolic regulation of epigenetics
  publication-title: Cell Metab.
– volume: 135
  start-page: 10396
  year: 2013
  end-page: 10403
  ident: bib7
  article-title: Ascorbic acid enhances Tet-mediated 5-methylcytosine oxidation and promotes DNA demethylation in mammals
  publication-title: J. Am. Chem. Soc.
– volume: 88
  start-page: 990
  year: 2011
  ident: 10.1016/j.xpro.2025.104015_bib11
  article-title: Saturation-Transfer Difference (STD) NMR: A Simple and Fast Method for Ligand Screening and Characterization of Protein Binding
  publication-title: J. Chem. Educ.
  doi: 10.1021/ed101169t
– volume: 19
  start-page: 15
  year: 2024
  ident: 10.1016/j.xpro.2025.104015_bib9
  article-title: Using NMR to Monitor TET-Dependent Methylcytosine Dioxygenase Activity and Regulation
  publication-title: ACS Chem. Biol.
  doi: 10.1021/acschembio.3c00619
– volume: 155
  start-page: 1545
  year: 2013
  ident: 10.1016/j.xpro.2025.104015_bib8
  article-title: Crystal structure of TET2-DNA complex: insight into TET-mediated 5mC oxidation
  publication-title: Cell
  doi: 10.1016/j.cell.2013.11.020
– volume: 19
  start-page: 563
  year: 2018
  ident: 10.1016/j.xpro.2025.104015_bib2
  article-title: Regulation of chromatin and gene expression by metabolic enzymes and metabolites
  publication-title: Nat. Rev. Mol. Cell Biol.
  doi: 10.1038/s41580-018-0029-7
– volume: 16
  start-page: 9
  year: 2012
  ident: 10.1016/j.xpro.2025.104015_bib3
  article-title: Metabolic regulation of epigenetics
  publication-title: Cell Metab.
  doi: 10.1016/j.cmet.2012.06.001
– volume: 17
  start-page: 505
  year: 2012
  ident: 10.1016/j.xpro.2025.104015_bib10
  article-title: Applications of saturation transfer difference NMR in biological systems
  publication-title: Drug Discov. Today
  doi: 10.1016/j.drudis.2011.12.016
– volume: 18
  start-page: 517
  year: 2017
  ident: 10.1016/j.xpro.2025.104015_bib4
  article-title: TET-mediated active DNA demethylation: mechanism, function and beyond
  publication-title: Nat. Rev. Genet.
  doi: 10.1038/nrg.2017.33
– volume: 4
  start-page: 151
  year: 2018
  ident: 10.1016/j.xpro.2025.104015_bib5
  article-title: Metabolism, Activity, and Targeting of D- and L-2-Hydroxyglutarates
  publication-title: Trends Cancer
  doi: 10.1016/j.trecan.2017.12.005
– volume: 34
  start-page: 700
  year: 2018
  ident: 10.1016/j.xpro.2025.104015_bib6
  article-title: Ascorbic Acid in Cancer Treatment: Let the Phoenix Fly
  publication-title: Cancer Cell
  doi: 10.1016/j.ccell.2018.07.014
– volume: 135
  start-page: 10396
  year: 2013
  ident: 10.1016/j.xpro.2025.104015_bib7
  article-title: Ascorbic acid enhances Tet-mediated 5-methylcytosine oxidation and promotes DNA demethylation in mammals
  publication-title: J. Am. Chem. Soc.
  doi: 10.1021/ja4028346
– volume: 37
  start-page: 1137
  year: 2025
  ident: 10.1016/j.xpro.2025.104015_bib1
  article-title: Pathogen-derived glyoxylate inhibits Tet2 DNA dioxygenase to facilitate bacterial persister formation
  publication-title: Cell Metab.
  doi: 10.1016/j.cmet.2025.01.019
SSID ssj0002512418
Score 2.303878
Snippet Epigenetic enzyme activity is coupled to cellular metabolism through their reliance on metabolic cofactors and substrates. Here, we describe steps for...
SourceID pubmedcentral
proquest
pubmed
crossref
elsevier
SourceType Open Access Repository
Aggregation Database
Index Database
Publisher
StartPage 104015
SubjectTerms Cell Biology
Dioxygenases - metabolism
DNA-Binding Proteins - genetics
DNA-Binding Proteins - metabolism
Epigenesis, Genetic
Humans
Magnetic Resonance Spectroscopy - methods
Molecular Biology
NMR
Protein Binding
Protein Biochemistry
Protein expression and purification
Proto-Oncogene Proteins - metabolism
Protocol
Title Protocol for elucidating metabolite binding and regulation of TET2 dioxygenase
URI https://dx.doi.org/10.1016/j.xpro.2025.104015
https://www.ncbi.nlm.nih.gov/pubmed/40773351
https://www.proquest.com/docview/3237763869
https://pubmed.ncbi.nlm.nih.gov/PMC12355516
Volume 6
WOSCitedRecordID wos001547197700001&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
hasFullText 1
inHoldings 1
isFullTextHit
isPrint
journalDatabaseRights – providerCode: PRVAON
  databaseName: Directory of Open Access Journals (DOAJ)
  customDbUrl:
  eissn: 2666-1667
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0002512418
  issn: 2666-1667
  databaseCode: DOA
  dateStart: 20200101
  isFulltext: true
  titleUrlDefault: https://www.doaj.org/
  providerName: Directory of Open Access Journals
– providerCode: PRVHPJ
  databaseName: ROAD: Directory of Open Access Scholarly Resources
  customDbUrl:
  eissn: 2666-1667
  dateEnd: 99991231
  omitProxy: false
  ssIdentifier: ssj0002512418
  issn: 2666-1667
  databaseCode: M~E
  dateStart: 20200101
  isFulltext: true
  titleUrlDefault: https://road.issn.org
  providerName: ISSN International Centre
link http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwtV1Lj9MwELbYBaG9IBCv8qiMxG2VKo3T2D6uVos4QLWCIFVcIsdxaFeQVG2Duhd-OzOx3aRbLYIDh0aVk9rRfF8nM5MZDyFviwnP0a4OyjxRQYy5NfApMadBGWFYqRLVNpvg06mYzeSl67G5btsJ8KoS261c_leoYQzAxtLZf4B7NykMwHcAHY4AOxz_CvjLVb2pAV67mff3Ri-wgqH6hs2iAXGsOT7NF7aYpU0ut93oneGYXqQYkK2317CAf3PjjNfP6dknTOhqp9_Z4l3Ied5cNx3ZzufGjn-d103wYdFFZ11pez_gADBiwwSr1kyrmOCZngTjxLbR8Fo06ZGF9TQiuHuhLdg8UNY2bnA12sKdj3Cl0eHFINvljxYp8Dw5Y2532v0tsv2pI3I3Av8HM_s-_upCbWjCxWPhSqZsdt_NNU_IfT_LbRbKoQdyM5G2Z5mkD8kD51LQM0uFR-SOqR6TqacBBRrQHg1oRwPqaECBBrSjAa1LijSgPRo8IV_eXaTn7wPXOyPQ4LFvAqlLxiMV52GkZVwKLbmIC5PrcIKdJJUsWJTLIinHTMsikrHRk9DwJNeRVgUo7afkuKor85zQOGSKFUaNczDlciNELrgxEkQVl2EZxwNy6uWVLe0WKZnPHbzKUNAZCjqzgh6QiRdp5ow8a7xlQIs__u6Nl38GGhBfa6nK1M06YxHj8JQUiRyQZxaP3X14TAdE7CG1uwB3V98_Uy3m7S7rWESOb5Ff3DrpS3LS_T9ekePNqjGvyT39c7NYr4bkiM_EsI3xDFs-_ga90Jdu
linkProvider ISSN International Centre
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Protocol+for+elucidating+metabolite+binding+and+regulation+of+TET2+dioxygenase&rft.jtitle=STAR+protocols&rft.au=Zhang%2C+Shuyuan&rft.au=Cheng%2C+Zhou-Li&rft.au=Ye%2C+Dan&rft.date=2025-09-19&rft.eissn=2666-1667&rft.volume=6&rft.issue=3&rft.spage=104015&rft_id=info:doi/10.1016%2Fj.xpro.2025.104015&rft_id=info%3Apmid%2F40773351&rft.externalDocID=40773351
thumbnail_l http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=2666-1667&client=summon
thumbnail_m http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=2666-1667&client=summon
thumbnail_s http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=2666-1667&client=summon