Diagnostic Value of Cell-free Circulating MicroRNAs for Obesity and Type 2 Diabetes: A Meta-analysis

Type 2 diabetes mellitus (T2DM) is the most common metabolic disorder worldwide. Because of population aging and increasing trends toward obesity and sedentary lifestyles, the number of affected individuals is increasing at worrisome rates. While both environmental and genetic factors are known to c...

Celý popis

Uloženo v:
Podrobná bibliografie
Vydáno v:Journal of molecular biomarkers & diagnosis Ročník 6; číslo 6
Hlavní autoři: Villard, Audrey, Marchand, Lucien, Thivolet, Charles, Rome, Sophie
Médium: Journal Article
Jazyk:angličtina
Vydáno: United States 01.11.2015
Témata:
ISSN:2155-9929
On-line přístup:Zjistit podrobnosti o přístupu
Tagy: Přidat tag
Žádné tagy, Buďte první, kdo vytvoří štítek k tomuto záznamu!
Abstract Type 2 diabetes mellitus (T2DM) is the most common metabolic disorder worldwide. Because of population aging and increasing trends toward obesity and sedentary lifestyles, the number of affected individuals is increasing at worrisome rates. While both environmental and genetic factors are known to contribute to the development of T2DM, continuous research is needed to identify specific biomarkers that could aid both in prevention of the disease and development of newer therapeutic options. Circulating miRNAs are considered as potential biomarkers because they are stable and resistant to degradation by blood RNAses and are modified under different pathophysiological conditions. In this study we carried out a systematic electronic search on PubMed to retrieve all articles that have investigated circulating miRNAs for diagnosing obesity andT2DM in human. We also included lifestyle intervention studies known to be highly effective in delaying onset of diabetes, and studies analyzing the effect of bariatric surgery and anti-diabetic treatment. A total of 26 studies were enrolled in the global meta-analysis. Candidate miRNAs were defined as those reported in at least 2 studies with same direction of differential expression. Ten miRNAs altered in blood of patients suffering fromT2DM were identified (increased: miR-320a, miR-142-3p, miR-222, miR-29a, miR-27a, miR-375; decreased: miR-197, miR-20b, miR-17, miR-652) and 7 miRNAs in blood of obese subjects were identified (increased: miR-142-3p, miR-140-5p, miR-222; decreased:miR-21-5p, miR-221-3p, miR-125-5p, mir-103-5p). Both obese and T2DM patients had elevated concentrations of miR-142-3p and miR-222. MiRNAs target genes were predicted and their cellular functions are discussed in relation with the pathologies. Although a significant number of studies were taken into account in this review, we found a strong discrepancy between miRNA detection and quantification indicating that many of pre-analytical variables have yet to be normalized. Pre-analytical and analytical challenges are also discussed.
AbstractList Type 2 diabetes mellitus (T2DM) is the most common metabolic disorder worldwide. Because of population aging and increasing trends toward obesity and sedentary lifestyles, the number of affected individuals is increasing at worrisome rates. While both environmental and genetic factors are known to contribute to the development of T2DM, continuous research is needed to identify specific biomarkers that could aid both in prevention of the disease and development of newer therapeutic options. Circulating miRNAs are considered as potential biomarkers because they are stable and resistant to degradation by blood RNAses and are modified under different pathophysiological conditions. In this study we carried out a systematic electronic search on PubMed to retrieve all articles that have investigated circulating miRNAs for diagnosing obesity andT2DM in human. We also included lifestyle intervention studies known to be highly effective in delaying onset of diabetes, and studies analyzing the effect of bariatric surgery and anti-diabetic treatment. A total of 26 studies were enrolled in the global meta-analysis. Candidate miRNAs were defined as those reported in at least 2 studies with same direction of differential expression. Ten miRNAs altered in blood of patients suffering fromT2DM were identified (increased: miR-320a, miR-142-3p, miR-222, miR-29a, miR-27a, miR-375; decreased: miR-197, miR-20b, miR-17, miR-652) and 7 miRNAs in blood of obese subjects were identified (increased: miR-142-3p, miR-140-5p, miR-222; decreased:miR-21-5p, miR-221-3p, miR-125-5p, mir-103-5p). Both obese and T2DM patients had elevated concentrations of miR-142-3p and miR-222. MiRNAs target genes were predicted and their cellular functions are discussed in relation with the pathologies. Although a significant number of studies were taken into account in this review, we found a strong discrepancy between miRNA detection and quantification indicating that many of pre-analytical variables have yet to be normalized. Pre-analytical and analytical challenges are also discussed.
Type 2 diabetes mellitus (T2DM) is the most common metabolic disorder worldwide. Because of population aging and increasing trends toward obesity and sedentary lifestyles, the number of affected individuals is increasing at worrisome rates. While both environmental and genetic factors are known to contribute to the development of T2DM, continuous research is needed to identify specific biomarkers that could aid both in prevention of the disease and development of newer therapeutic options. Circulating miRNAs are considered as potential biomarkers because they are stable and resistant to degradation by blood RNAses and are modified under different pathophysiological conditions. In this study we carried out a systematic electronic search on PubMed to retrieve all articles that have investigated circulating miRNAs for diagnosing obesity andT2DM in human. We also included lifestyle intervention studies known to be highly effective in delaying onset of diabetes, and studies analyzing the effect of bariatric surgery and anti-diabetic treatment. A total of 26 studies were enrolled in the global meta-analysis. Candidate miRNAs were defined as those reported in at least 2 studies with same direction of differential expression. Ten miRNAs altered in blood of patients suffering fromT2DM were identified (increased: miR-320a, miR-142-3p, miR-222, miR-29a, miR-27a, miR-375; decreased: miR-197, miR-20b, miR-17, miR-652) and 7 miRNAs in blood of obese subjects were identified (increased: miR-142-3p, miR-140-5p, miR-222; decreased:miR-21-5p, miR-221-3p, miR-125-5p, mir-103-5p). Both obese and T2DM patients had elevated concentrations of miR-142-3p and miR-222. MiRNAs target genes were predicted and their cellular functions are discussed in relation with the pathologies. Although a significant number of studies were taken into account in this review, we found a strong discrepancy between miRNA detection and quantification indicating that many of pre-analytical variables have yet to be normalized. Pre-analytical and analytical challenges are also discussed.Type 2 diabetes mellitus (T2DM) is the most common metabolic disorder worldwide. Because of population aging and increasing trends toward obesity and sedentary lifestyles, the number of affected individuals is increasing at worrisome rates. While both environmental and genetic factors are known to contribute to the development of T2DM, continuous research is needed to identify specific biomarkers that could aid both in prevention of the disease and development of newer therapeutic options. Circulating miRNAs are considered as potential biomarkers because they are stable and resistant to degradation by blood RNAses and are modified under different pathophysiological conditions. In this study we carried out a systematic electronic search on PubMed to retrieve all articles that have investigated circulating miRNAs for diagnosing obesity andT2DM in human. We also included lifestyle intervention studies known to be highly effective in delaying onset of diabetes, and studies analyzing the effect of bariatric surgery and anti-diabetic treatment. A total of 26 studies were enrolled in the global meta-analysis. Candidate miRNAs were defined as those reported in at least 2 studies with same direction of differential expression. Ten miRNAs altered in blood of patients suffering fromT2DM were identified (increased: miR-320a, miR-142-3p, miR-222, miR-29a, miR-27a, miR-375; decreased: miR-197, miR-20b, miR-17, miR-652) and 7 miRNAs in blood of obese subjects were identified (increased: miR-142-3p, miR-140-5p, miR-222; decreased:miR-21-5p, miR-221-3p, miR-125-5p, mir-103-5p). Both obese and T2DM patients had elevated concentrations of miR-142-3p and miR-222. MiRNAs target genes were predicted and their cellular functions are discussed in relation with the pathologies. Although a significant number of studies were taken into account in this review, we found a strong discrepancy between miRNA detection and quantification indicating that many of pre-analytical variables have yet to be normalized. Pre-analytical and analytical challenges are also discussed.
Author Villard, Audrey
Marchand, Lucien
Thivolet, Charles
Rome, Sophie
Author_xml – sequence: 1
  givenname: Audrey
  surname: Villard
  fullname: Villard, Audrey
  organization: CarMen laboratory (UMR INSERM 1060, INRA 1397, INSA), University of Lyon, Faculty of Medicine Lyon-Sud, Chemin du Grand Revoyet, 69600 Oullins, France
– sequence: 2
  givenname: Lucien
  surname: Marchand
  fullname: Marchand, Lucien
  organization: Hospices Civils de Lyon, Service d'Endocrinologie Diabète Nutrition, Lyon, France
– sequence: 3
  givenname: Charles
  surname: Thivolet
  fullname: Thivolet, Charles
  organization: CarMen laboratory (UMR INSERM 1060, INRA 1397, INSA), University of Lyon, Faculty of Medicine Lyon-Sud, Chemin du Grand Revoyet, 69600 Oullins, France; Hospices Civils de Lyon, Service d'Endocrinologie Diabète Nutrition, Lyon, France
– sequence: 4
  givenname: Sophie
  surname: Rome
  fullname: Rome, Sophie
  organization: CarMen laboratory (UMR INSERM 1060, INRA 1397, INSA), University of Lyon, Faculty of Medicine Lyon-Sud, Chemin du Grand Revoyet, 69600 Oullins, France
BackLink https://www.ncbi.nlm.nih.gov/pubmed/27308097$$D View this record in MEDLINE/PubMed
BookMark eNo9kD1rwzAURTWkNGmaH9ClaOziVHq2LKubcT8haaCkXY0kPwWBY6eWPfjf19C004HD5cK9V2TWtA0ScsPZOuES7oELESkFas0ZYyD4jCz-3ZysQvCGgRQcZBxfkvkEljElF6R69PrQtKH3ln7pekDaOlpgXUeuQ6SF7-xQ6943B7r1tms_3vNAXdvRncHg-5HqpqL78YQU6FRlsMfwQHO6xV5HutH1GHy4JhdO1wFXZy7J5_PTvniNNruXtyLfRDaWkkcWROqAWwFxholUMk20cLEVMmHGQOUSngoVo9RZJYBx0JNVaJ00ymSoYUnufntPXfs9YOjLow92GqMbbIdQ8gxSyQCSdIrenqODOWJVnjp_1N1Y_j0DPwlZZMU
CitedBy_id crossref_primary_10_1038_s41598_019_55076_z
crossref_primary_10_1038_s41598_023_46516_y
crossref_primary_10_1016_j_atherosclerosis_2020_02_004
crossref_primary_10_1186_s12933_018_0767_z
crossref_primary_10_1371_journal_pone_0247556
crossref_primary_10_1093_cvr_cvy049
crossref_primary_10_1210_endocr_bqab241
crossref_primary_10_1155_2021_6626845
crossref_primary_10_1007_s11010_022_04362_y
crossref_primary_10_3390_nu14224889
crossref_primary_10_1111_cob_12192
crossref_primary_10_1007_s00592_019_01430_6
crossref_primary_10_3389_fendo_2021_750261
crossref_primary_10_1007_s12291_022_01069_1
crossref_primary_10_1016_j_semcancer_2023_05_001
crossref_primary_10_1016_j_numecd_2017_10_015
crossref_primary_10_1038_s41598_025_04062_9
crossref_primary_10_3390_cells9061557
crossref_primary_10_1038_s41598_018_32274_9
crossref_primary_10_1002_mnfr_70027
crossref_primary_10_3390_cells10123331
crossref_primary_10_3390_ijms21103485
crossref_primary_10_1177_1535370217731291
crossref_primary_10_3390_cells9112489
crossref_primary_10_3390_ijms20235998
crossref_primary_10_3389_fphys_2018_00883
crossref_primary_10_4093_dmj_2022_0226
crossref_primary_10_1172_JCI129187
crossref_primary_10_3390_cancers15174317
crossref_primary_10_1002_oby_23330
crossref_primary_10_7717_peerj_19221
crossref_primary_10_3389_fgene_2020_563166
crossref_primary_10_3389_fgene_2021_620744
crossref_primary_10_3389_fendo_2022_917041
crossref_primary_10_3390_ijms21186902
crossref_primary_10_3389_fendo_2022_971354
crossref_primary_10_1139_cjpp_2016_0580
crossref_primary_10_3390_jcm12165265
crossref_primary_10_1007_s12013_020_00932_3
crossref_primary_10_3390_ijms24076186
crossref_primary_10_1080_07435800_2021_1914079
crossref_primary_10_2147_DMSO_S304055
crossref_primary_10_3389_fonc_2020_01567
crossref_primary_10_3390_diagnostics15020169
crossref_primary_10_1111_ijpo_12247
crossref_primary_10_3390_ijms231810635
crossref_primary_10_1111_cpr_12877
crossref_primary_10_1155_2016_1869082
crossref_primary_10_3390_jcm11185271
crossref_primary_10_1111_jcmm_14920
crossref_primary_10_3390_ijms20122922
crossref_primary_10_3390_ijms25094827
crossref_primary_10_1016_j_molmet_2017_08_007
crossref_primary_10_3390_pr10122551
crossref_primary_10_3390_ijms21249509
crossref_primary_10_1007_s13668_024_00549_5
crossref_primary_10_1111_jdi_14416
crossref_primary_10_33678_cor_2022_050
crossref_primary_10_3389_fgene_2020_00367
crossref_primary_10_3390_ijms22157855
crossref_primary_10_3390_ijms26136006
crossref_primary_10_1038_s41598_019_43793_4
crossref_primary_10_3390_cells11091553
crossref_primary_10_1016_j_yexcr_2017_03_060
crossref_primary_10_1371_journal_pone_0208131
crossref_primary_10_1016_j_genrep_2020_100954
crossref_primary_10_1016_j_jnutbio_2024_109714
crossref_primary_10_1210_endrev_bnac009
crossref_primary_10_3390_cimb46040212
crossref_primary_10_1016_j_gene_2019_05_012
crossref_primary_10_22270_jddt_v15i8_7336
crossref_primary_10_1111_jcmm_13885
crossref_primary_10_3390_ijms21072437
crossref_primary_10_1016_j_freeradbiomed_2021_05_004
crossref_primary_10_1016_j_crvi_2016_12_001
crossref_primary_10_1111_1753_0407_13431
crossref_primary_10_3390_biomedicines10061292
crossref_primary_10_1002_jcp_28649
ContentType Journal Article
DBID NPM
7X8
DOI 10.4172/2155-9929.1000251
DatabaseName PubMed
MEDLINE - Academic
DatabaseTitle PubMed
MEDLINE - Academic
DatabaseTitleList PubMed
MEDLINE - Academic
Database_xml – sequence: 1
  dbid: NPM
  name: PubMed
  url: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 2
  dbid: 7X8
  name: MEDLINE - Academic
  url: https://search.proquest.com/medline
  sourceTypes: Aggregation Database
DeliveryMethod no_fulltext_linktorsrc
ExternalDocumentID 27308097
Genre Journal Article
GrantInformation_xml – fundername: NIAID NIH HHS
  grantid: P01 AI042288
GroupedDBID ALMA_UNASSIGNED_HOLDINGS
KQ8
NPM
7X8
ID FETCH-LOGICAL-c3771-c256f21c5238e479764a5f3c5740bb2df416593e7a8d52012abb29ecf7b9b8ea2
IEDL.DBID 7X8
ISSN 2155-9929
IngestDate Fri Jul 11 08:44:27 EDT 2025
Sat Sep 18 05:37:59 EDT 2021
IsDoiOpenAccess false
IsOpenAccess true
IsPeerReviewed false
IsScholarly true
Issue 6
Keywords Type 2 diabetes
Obesity
Circulating miRNAs
Meta-analysis
Language English
LinkModel DirectLink
MergedId FETCHMERGED-LOGICAL-c3771-c256f21c5238e479764a5f3c5740bb2df416593e7a8d52012abb29ecf7b9b8ea2
Notes ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
content type line 23
OpenAccessLink http://doi.org/10.4172/2155-9929.1000251
PMID 27308097
PQID 1826702246
PQPubID 23479
ParticipantIDs proquest_miscellaneous_1826702246
pubmed_primary_27308097
PublicationCentury 2000
PublicationDate 2015-Nov
PublicationDateYYYYMMDD 2015-11-01
PublicationDate_xml – month: 11
  year: 2015
  text: 2015-Nov
PublicationDecade 2010
PublicationPlace United States
PublicationPlace_xml – name: United States
PublicationTitle Journal of molecular biomarkers & diagnosis
PublicationTitleAlternate J Mol Biomark Diagn
PublicationYear 2015
References 26233309 - Genes Nutr. 2015 Sep;10(5):483
22510765 - Nat Rev Genet. 2012 Apr 18;13(5):358-69
24361879 - Biochem Biophys Res Commun. 2014 Jan 17;443(3):980-6
20615901 - Nucleic Acids Res. 2010 Nov;38(20):7248-59
22265214 - Lancet Neurol. 2012 Feb;11(2):189-200
21609964 - Nucleic Acids Res. 2011 Sep 1;39(16):7223-33
19246379 - Proc Natl Acad Sci U S A. 2009 Mar 17;106(11):4402-7
22224773 - Annu Rev Immunol. 2012;30:295-312
25976503 - Tumour Biol. 2015 Sep;36(10):8065-73
19745058 - Clin Chem. 2009 Nov;55(11):1977-83
22155483 - Exp Neurol. 2012 Jun;235(2):491-6
22293113 - J Genet Genomics. 2012 Jan;39(1):11-8
24455723 - Biomed Res Int. 2013;2013:761617
22958478 - BMC Mol Biol. 2012 Sep 07;13:27
18663219 - Proc Natl Acad Sci U S A. 2008 Jul 29;105(30):10513-8
24037889 - J Clin Endocrinol Metab. 2013 Nov;98(11):E1835-44
24478399 - Diabetes Care. 2014;37(5):1375-83
23928666 - J Clin Endocrinol Metab. 2013 Oct;98(10):E1655-60
22652795 - Front Biosci (Landmark Ed). 2012 Jun 01;17:2508-40
22158052 - Cancer Prev Res (Phila). 2012 Mar;5(3):492-7
23635652 - Mol Cancer Ther. 2013 Jul;12(7):1266-75
23834149 - Curr Drug Targets. 2013 Sep;14(10):1110-7
21829658 - PLoS One. 2011;6(8):e22839
24927146 - Int J Mol Sci. 2014 Jun 12;15(6):10567-77
16969338 - Mol Syst Biol. 2006;2:46
25677225 - Diabetologia. 2015 May;58(5):900-11
18773077 - PLoS One. 2008 Sep 05;3(9):e3148
23150891 - Eur J Prev Cardiol. 2014 Apr;21(4):484-91
26271186 - Anal Biochem. 2015 Nov 1;488:28-35
25726255 - Metabolism. 2015 Apr;64(4):489-97
24955210 - Oxid Med Cell Longev. 2014;2014:729194
20651284 - Circ Res. 2010 Sep 17;107(6):810-7
21887347 - PLoS One. 2011;6(8):e23925
24937531 - J Clin Endocrinol Metab. 2014 Sep;99(9):E1681-5
14744438 - Cell. 2004 Jan 23;116(2):281-97
19800254 - Trends Endocrinol Metab. 2009 Nov;20(9):452-9
25532038 - J Clin Endocrinol Metab. 2015 Mar;100(3):E407-15
26136647 - Front Neurosci. 2015 Jun 16;9:204
17486113 - Nat Cell Biol. 2007 Jun;9(6):654-9
25597340 - Eur J Neurol. 2015 Nov;22(11):1419-20
26116273 - Mol Aspects Med. 2015 Jun-Oct;43-44:66-76
22859996 - PLoS One. 2012;7(7):e41561
24363306 - Am J Physiol Heart Circ Physiol. 2014 Feb 15;306(4):H557-63
24385703 - Chin J Cancer Res. 2013 Dec;25(6):743-8
25923429 - PLoS One. 2015 Apr 29;10(4):e0122991
18318758 - Br J Haematol. 2008 May;141(5):672-5
18766170 - Cell Res. 2008 Oct;18(10):997-1006
23680784 - Nucleic Acids Res. 2013 Jul;41(Web Server issue):W169-73
23472110 - PLoS One. 2013;8(3):e57841
24436293 - J Appl Physiol (1985). 2014 Mar 1;116(5):522-31
26020947 - PLoS One. 2015 May 28;10(5):e0128372
20587728 - Diabetes Care. 2010 Jul;33(7):1674-85
24023848 - PLoS One. 2013 Sep 02;8(9):e73272
21367929 - J Clin Endocrinol Metab. 2011 May;96(5):E846-50
25977294 - Nucleic Acids Res. 2015 Jul 1;43(W1):W460-6
23032062 - J Clin Endocrinol Metab. 2012 Dec;97(12):E2271-6
23036329 - Methods. 2013 Jan;59(1):S1-6
20370587 - Expert Rev Mol Diagn. 2010 Apr;10(3):297-308
24497980 - PLoS One. 2014 Jan 30;9(1):e86792
22064828 - Aging (Albany NY). 2011 Oct;3(10):985-1002
16337999 - Cell. 2005 Dec 16;123(6):1133-46
24586621 - PLoS One. 2014 Feb 19;9(2):e89237
21423178 - Nat Cell Biol. 2011 Apr;13(4):423-33
23317344 - Ann N Y Acad Sci. 2013 Apr;1281:51-63
26182553 - Nurs Times. 2015 Mar 18-24;111(12 ):17-9
25150024 - Mutagenesis. 2014 Sep;29(5):385-91
20857148 - Acta Diabetol. 2011 Mar;48(1):61-9
24498934 - Cardiovasc Diabetol. 2014 Feb 05;13:37
25815110 - Oxid Med Cell Longev. 2015;2015:985845
23716129 - J Cardiovasc Transl Res. 2013 Aug;6(4):458-70
23396142 - Clin Chem. 2013 May;59(5):781-92
24376572 - PLoS One. 2013 Dec 23;8(12):e82753
23619912 - J Mol Med (Berl). 2013 Aug;91(8):989-1000
26131326 - World J Diabetes. 2015 Jun 25;6(6):850-67
20847327 - Clin Chem. 2010 Nov;56(11):1733-41
25861754 - Tumour Biol. 2015 Sep;36(9):6985-90
24073250 - PLoS One. 2013 Sep 20;8(9):e75184
25515554 - Eur J Endocrinol. 2015 Mar;172(3):291-300
21383194 - Proc Natl Acad Sci U S A. 2011 Mar 22;108(12):5003-8
25331995 - Sheng Li Xue Bao. 2014 Oct 25;66(5):497-510
23755214 - PLoS One. 2013 Jun 05;8(6):e65300
24093444 - J Biomed Sci. 2013 Oct 04;20:72
23358195 - Toxicol Sci. 2013 Apr;132(2):478-92
21909417 - PLoS One. 2011;6(9):e24145
25995320 - Circulation. 2015 Jun 23;131(25):2202-16
24803022 - Cell Physiol Biochem. 2014;33(5):1329-39
26084689 - Physiol Rev. 2015 Jul;95(3):727-48
21690193 - J Physiol. 2011 Aug 15;589(Pt 16):3983-94
26063956 - Dis Markers. 2015;2015:435656
22849433 - BMC Genomics. 2012 Aug 01;13:357
23923026 - PLoS One. 2013 Jul 29;8(7):e70823
21057646 - Commun Integr Biol. 2010 Sep;3(5):478-81
24204780 - PLoS One. 2013 Oct 15;8(10):e77251
21525457 - Diabetes Care. 2011 May;34 Suppl 2:S210-4
19996457 - Sci Signal. 2009 Dec 08;2(100):ra81
22783422 - Oncol Lett. 2012 Jun;3(6):1221-1226
References_xml – reference: 19745058 - Clin Chem. 2009 Nov;55(11):1977-83
– reference: 24363306 - Am J Physiol Heart Circ Physiol. 2014 Feb 15;306(4):H557-63
– reference: 20615901 - Nucleic Acids Res. 2010 Nov;38(20):7248-59
– reference: 24436293 - J Appl Physiol (1985). 2014 Mar 1;116(5):522-31
– reference: 22155483 - Exp Neurol. 2012 Jun;235(2):491-6
– reference: 16969338 - Mol Syst Biol. 2006;2:46
– reference: 24955210 - Oxid Med Cell Longev. 2014;2014:729194
– reference: 26116273 - Mol Aspects Med. 2015 Jun-Oct;43-44:66-76
– reference: 26020947 - PLoS One. 2015 May 28;10(5):e0128372
– reference: 24803022 - Cell Physiol Biochem. 2014;33(5):1329-39
– reference: 23635652 - Mol Cancer Ther. 2013 Jul;12(7):1266-75
– reference: 26271186 - Anal Biochem. 2015 Nov 1;488:28-35
– reference: 24455723 - Biomed Res Int. 2013;2013:761617
– reference: 24586621 - PLoS One. 2014 Feb 19;9(2):e89237
– reference: 23834149 - Curr Drug Targets. 2013 Sep;14(10):1110-7
– reference: 21525457 - Diabetes Care. 2011 May;34 Suppl 2:S210-4
– reference: 26233309 - Genes Nutr. 2015 Sep;10(5):483
– reference: 20651284 - Circ Res. 2010 Sep 17;107(6):810-7
– reference: 25726255 - Metabolism. 2015 Apr;64(4):489-97
– reference: 22783422 - Oncol Lett. 2012 Jun;3(6):1221-1226
– reference: 23150891 - Eur J Prev Cardiol. 2014 Apr;21(4):484-91
– reference: 23317344 - Ann N Y Acad Sci. 2013 Apr;1281:51-63
– reference: 23619912 - J Mol Med (Berl). 2013 Aug;91(8):989-1000
– reference: 22224773 - Annu Rev Immunol. 2012;30:295-312
– reference: 21609964 - Nucleic Acids Res. 2011 Sep 1;39(16):7223-33
– reference: 24093444 - J Biomed Sci. 2013 Oct 04;20:72
– reference: 23680784 - Nucleic Acids Res. 2013 Jul;41(Web Server issue):W169-73
– reference: 18663219 - Proc Natl Acad Sci U S A. 2008 Jul 29;105(30):10513-8
– reference: 25532038 - J Clin Endocrinol Metab. 2015 Mar;100(3):E407-15
– reference: 23928666 - J Clin Endocrinol Metab. 2013 Oct;98(10):E1655-60
– reference: 20857148 - Acta Diabetol. 2011 Mar;48(1):61-9
– reference: 21909417 - PLoS One. 2011;6(9):e24145
– reference: 22064828 - Aging (Albany NY). 2011 Oct;3(10):985-1002
– reference: 20847327 - Clin Chem. 2010 Nov;56(11):1733-41
– reference: 16337999 - Cell. 2005 Dec 16;123(6):1133-46
– reference: 23032062 - J Clin Endocrinol Metab. 2012 Dec;97(12):E2271-6
– reference: 21383194 - Proc Natl Acad Sci U S A. 2011 Mar 22;108(12):5003-8
– reference: 21367929 - J Clin Endocrinol Metab. 2011 May;96(5):E846-50
– reference: 22859996 - PLoS One. 2012;7(7):e41561
– reference: 24937531 - J Clin Endocrinol Metab. 2014 Sep;99(9):E1681-5
– reference: 22265214 - Lancet Neurol. 2012 Feb;11(2):189-200
– reference: 22652795 - Front Biosci (Landmark Ed). 2012 Jun 01;17:2508-40
– reference: 24497980 - PLoS One. 2014 Jan 30;9(1):e86792
– reference: 21057646 - Commun Integr Biol. 2010 Sep;3(5):478-81
– reference: 25976503 - Tumour Biol. 2015 Sep;36(10):8065-73
– reference: 19800254 - Trends Endocrinol Metab. 2009 Nov;20(9):452-9
– reference: 22293113 - J Genet Genomics. 2012 Jan;39(1):11-8
– reference: 18766170 - Cell Res. 2008 Oct;18(10):997-1006
– reference: 26131326 - World J Diabetes. 2015 Jun 25;6(6):850-67
– reference: 24204780 - PLoS One. 2013 Oct 15;8(10):e77251
– reference: 25977294 - Nucleic Acids Res. 2015 Jul 1;43(W1):W460-6
– reference: 24023848 - PLoS One. 2013 Sep 02;8(9):e73272
– reference: 22158052 - Cancer Prev Res (Phila). 2012 Mar;5(3):492-7
– reference: 24498934 - Cardiovasc Diabetol. 2014 Feb 05;13:37
– reference: 21423178 - Nat Cell Biol. 2011 Apr;13(4):423-33
– reference: 18773077 - PLoS One. 2008 Sep 05;3(9):e3148
– reference: 20370587 - Expert Rev Mol Diagn. 2010 Apr;10(3):297-308
– reference: 24361879 - Biochem Biophys Res Commun. 2014 Jan 17;443(3):980-6
– reference: 25597340 - Eur J Neurol. 2015 Nov;22(11):1419-20
– reference: 26136647 - Front Neurosci. 2015 Jun 16;9:204
– reference: 25861754 - Tumour Biol. 2015 Sep;36(9):6985-90
– reference: 24073250 - PLoS One. 2013 Sep 20;8(9):e75184
– reference: 22849433 - BMC Genomics. 2012 Aug 01;13:357
– reference: 18318758 - Br J Haematol. 2008 May;141(5):672-5
– reference: 26182553 - Nurs Times. 2015 Mar 18-24;111(12 ):17-9
– reference: 19246379 - Proc Natl Acad Sci U S A. 2009 Mar 17;106(11):4402-7
– reference: 24037889 - J Clin Endocrinol Metab. 2013 Nov;98(11):E1835-44
– reference: 23923026 - PLoS One. 2013 Jul 29;8(7):e70823
– reference: 24478399 - Diabetes Care. 2014;37(5):1375-83
– reference: 23396142 - Clin Chem. 2013 May;59(5):781-92
– reference: 23755214 - PLoS One. 2013 Jun 05;8(6):e65300
– reference: 26063956 - Dis Markers. 2015;2015:435656
– reference: 26084689 - Physiol Rev. 2015 Jul;95(3):727-48
– reference: 20587728 - Diabetes Care. 2010 Jul;33(7):1674-85
– reference: 21829658 - PLoS One. 2011;6(8):e22839
– reference: 25995320 - Circulation. 2015 Jun 23;131(25):2202-16
– reference: 14744438 - Cell. 2004 Jan 23;116(2):281-97
– reference: 21887347 - PLoS One. 2011;6(8):e23925
– reference: 23358195 - Toxicol Sci. 2013 Apr;132(2):478-92
– reference: 17486113 - Nat Cell Biol. 2007 Jun;9(6):654-9
– reference: 19996457 - Sci Signal. 2009 Dec 08;2(100):ra81
– reference: 25923429 - PLoS One. 2015 Apr 29;10(4):e0122991
– reference: 23472110 - PLoS One. 2013;8(3):e57841
– reference: 24385703 - Chin J Cancer Res. 2013 Dec;25(6):743-8
– reference: 25815110 - Oxid Med Cell Longev. 2015;2015:985845
– reference: 23716129 - J Cardiovasc Transl Res. 2013 Aug;6(4):458-70
– reference: 22510765 - Nat Rev Genet. 2012 Apr 18;13(5):358-69
– reference: 25677225 - Diabetologia. 2015 May;58(5):900-11
– reference: 24376572 - PLoS One. 2013 Dec 23;8(12):e82753
– reference: 25331995 - Sheng Li Xue Bao. 2014 Oct 25;66(5):497-510
– reference: 24927146 - Int J Mol Sci. 2014 Jun 12;15(6):10567-77
– reference: 25515554 - Eur J Endocrinol. 2015 Mar;172(3):291-300
– reference: 22958478 - BMC Mol Biol. 2012 Sep 07;13:27
– reference: 21690193 - J Physiol. 2011 Aug 15;589(Pt 16):3983-94
– reference: 23036329 - Methods. 2013 Jan;59(1):S1-6
– reference: 25150024 - Mutagenesis. 2014 Sep;29(5):385-91
SSID ssib027512733
ssib005568854
Score 1.9276683
Snippet Type 2 diabetes mellitus (T2DM) is the most common metabolic disorder worldwide. Because of population aging and increasing trends toward obesity and sedentary...
SourceID proquest
pubmed
SourceType Aggregation Database
Index Database
Title Diagnostic Value of Cell-free Circulating MicroRNAs for Obesity and Type 2 Diabetes: A Meta-analysis
URI https://www.ncbi.nlm.nih.gov/pubmed/27308097
https://www.proquest.com/docview/1826702246
Volume 6
hasFullText
inHoldings 1
isFullTextHit
isPrint
link http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwpZ1LSwMxEMeDWg9efOCrvhjBa2ib3W02XqRUi5cuRVR6K3mKUHZrt_XzO9lNrRdB8LKHWRZCMpv5JZnMn5AbZ7VTSmoqmNE05qpLFTOStgWTXaP927gSm-BZlo7HYhQ23MqQVrmaE6uJ2hTa75G3PAfzqvzZ3eyDetUof7oaJDQ2SSNClPFezcc_ylMl3TRdh0vGMbrxSl0eA11CBaJBfdAZYxhvfdt85oBH79-hswo-g73_Nnuf7AbshF7tJwdkw-aHxNzXeXZog1c5XVooHPTtdErd3Frov891Je6Vv8HQ5-09Zb0SEHIhqAmAzA34hSwwCJk15S30YGgXkspQ7eSIvAwenvuPNKguUB1x3qEaIcixjsYVampjjrgSy8RFOuFxW-E4OkS4RESWy9QkiA9MolXgoHIlVGolOyZbeZHbUwIiwhlBWt1WzsQdrQTXQkbOpCwyyBGqSa5XnTZBr_ZHFTK3xbKcrLutSU7qnp_M6vIbExxCxFzBz_7w9TnZwSYm9eXBC9Jw-E_bS7KtPxfv5fyqchd8ZqPhF9IMyAc
linkProvider ProQuest
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Diagnostic+Value+of+Cell-free+Circulating+MicroRNAs+for+Obesity+and+Type+2+Diabetes%3A+A+Meta-analysis&rft.jtitle=Journal+of+molecular+biomarkers+%26+diagnosis&rft.au=Villard%2C+Audrey&rft.au=Marchand%2C+Lucien&rft.au=Thivolet%2C+Charles&rft.au=Rome%2C+Sophie&rft.date=2015-11-01&rft.issn=2155-9929&rft.volume=6&rft.issue=6&rft_id=info:doi/10.4172%2F2155-9929.1000251&rft.externalDBID=NO_FULL_TEXT
thumbnail_l http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=2155-9929&client=summon
thumbnail_m http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=2155-9929&client=summon
thumbnail_s http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=2155-9929&client=summon