Perfluorinated Chemicals as Emerging Environmental Threats to Kidney Health: A Scoping Review

Per- and polyfluoroalkyl substances (PFASs) are a large group of manufactured nonbiodegradable compounds. Despite increasing awareness as global pollutants, the impact of PFAS exposure on human health is not well understood, and there are growing concerns for adverse effects on kidney function. Ther...

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Published in:Clinical journal of the American Society of Nephrology Vol. 13; no. 10; p. 1479
Main Authors: Stanifer, John W, Stapleton, Heather M, Souma, Tomokazu, Wittmer, Ashley, Zhao, Xinlu, Boulware, L Ebony
Format: Journal Article
Language:English
Published: United States 08.10.2018
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ISSN:1555-905X, 1555-905X
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Abstract Per- and polyfluoroalkyl substances (PFASs) are a large group of manufactured nonbiodegradable compounds. Despite increasing awareness as global pollutants, the impact of PFAS exposure on human health is not well understood, and there are growing concerns for adverse effects on kidney function. Therefore, we conducted a scoping review to summarize and identify gaps in the understanding between PFAS exposure and kidney health. We systematically searched PubMed, EMBASE, EBSCO Global Health, World Health Organization Global Index, and Web of Science for studies published from 1990 to 2018. We included studies on the epidemiology, pharmacokinetics, or toxicology of PFAS exposure and kidney-related health, including clinical, histologic, molecular, and metabolic outcomes related to kidney disease, or outcomes related to the pharmacokinetic role of the kidneys. We identified 74 studies, including 21 epidemiologic, 13 pharmacokinetic, and 40 toxicological studies. Three population-based epidemiologic studies demonstrated associations between PFAS exposure and lower kidney function. Along with toxicology studies ( =10) showing tubular histologic and cellular changes from PFAS exposure, pharmacokinetic studies ( =5) demonstrated the kidneys were major routes of elimination, with active proximal tubule transport. In several studies ( =17), PFAS exposure altered several pathways linked to kidney disease, including oxidative stress pathways, peroxisome proliferators-activated receptor pathways, NF-E2-related factor 2 pathways, partial epithelial mesenchymal transition, and enhanced endothelial permeability through actin filament modeling. A growing body of evidence portends PFASs are emerging environmental threats to kidney health; yet several important gaps in our understanding still exist.
AbstractList Per- and polyfluoroalkyl substances (PFASs) are a large group of manufactured nonbiodegradable compounds. Despite increasing awareness as global pollutants, the impact of PFAS exposure on human health is not well understood, and there are growing concerns for adverse effects on kidney function. Therefore, we conducted a scoping review to summarize and identify gaps in the understanding between PFAS exposure and kidney health. We systematically searched PubMed, EMBASE, EBSCO Global Health, World Health Organization Global Index, and Web of Science for studies published from 1990 to 2018. We included studies on the epidemiology, pharmacokinetics, or toxicology of PFAS exposure and kidney-related health, including clinical, histologic, molecular, and metabolic outcomes related to kidney disease, or outcomes related to the pharmacokinetic role of the kidneys. We identified 74 studies, including 21 epidemiologic, 13 pharmacokinetic, and 40 toxicological studies. Three population-based epidemiologic studies demonstrated associations between PFAS exposure and lower kidney function. Along with toxicology studies ( =10) showing tubular histologic and cellular changes from PFAS exposure, pharmacokinetic studies ( =5) demonstrated the kidneys were major routes of elimination, with active proximal tubule transport. In several studies ( =17), PFAS exposure altered several pathways linked to kidney disease, including oxidative stress pathways, peroxisome proliferators-activated receptor pathways, NF-E2-related factor 2 pathways, partial epithelial mesenchymal transition, and enhanced endothelial permeability through actin filament modeling. A growing body of evidence portends PFASs are emerging environmental threats to kidney health; yet several important gaps in our understanding still exist.
Per- and polyfluoroalkyl substances (PFASs) are a large group of manufactured nonbiodegradable compounds. Despite increasing awareness as global pollutants, the impact of PFAS exposure on human health is not well understood, and there are growing concerns for adverse effects on kidney function. Therefore, we conducted a scoping review to summarize and identify gaps in the understanding between PFAS exposure and kidney health.BACKGROUND AND OBJECTIVESPer- and polyfluoroalkyl substances (PFASs) are a large group of manufactured nonbiodegradable compounds. Despite increasing awareness as global pollutants, the impact of PFAS exposure on human health is not well understood, and there are growing concerns for adverse effects on kidney function. Therefore, we conducted a scoping review to summarize and identify gaps in the understanding between PFAS exposure and kidney health.We systematically searched PubMed, EMBASE, EBSCO Global Health, World Health Organization Global Index, and Web of Science for studies published from 1990 to 2018. We included studies on the epidemiology, pharmacokinetics, or toxicology of PFAS exposure and kidney-related health, including clinical, histologic, molecular, and metabolic outcomes related to kidney disease, or outcomes related to the pharmacokinetic role of the kidneys.DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTSWe systematically searched PubMed, EMBASE, EBSCO Global Health, World Health Organization Global Index, and Web of Science for studies published from 1990 to 2018. We included studies on the epidemiology, pharmacokinetics, or toxicology of PFAS exposure and kidney-related health, including clinical, histologic, molecular, and metabolic outcomes related to kidney disease, or outcomes related to the pharmacokinetic role of the kidneys.We identified 74 studies, including 21 epidemiologic, 13 pharmacokinetic, and 40 toxicological studies. Three population-based epidemiologic studies demonstrated associations between PFAS exposure and lower kidney function. Along with toxicology studies (n=10) showing tubular histologic and cellular changes from PFAS exposure, pharmacokinetic studies (n=5) demonstrated the kidneys were major routes of elimination, with active proximal tubule transport. In several studies (n=17), PFAS exposure altered several pathways linked to kidney disease, including oxidative stress pathways, peroxisome proliferators-activated receptor pathways, NF-E2-related factor 2 pathways, partial epithelial mesenchymal transition, and enhanced endothelial permeability through actin filament modeling.RESULTSWe identified 74 studies, including 21 epidemiologic, 13 pharmacokinetic, and 40 toxicological studies. Three population-based epidemiologic studies demonstrated associations between PFAS exposure and lower kidney function. Along with toxicology studies (n=10) showing tubular histologic and cellular changes from PFAS exposure, pharmacokinetic studies (n=5) demonstrated the kidneys were major routes of elimination, with active proximal tubule transport. In several studies (n=17), PFAS exposure altered several pathways linked to kidney disease, including oxidative stress pathways, peroxisome proliferators-activated receptor pathways, NF-E2-related factor 2 pathways, partial epithelial mesenchymal transition, and enhanced endothelial permeability through actin filament modeling.A growing body of evidence portends PFASs are emerging environmental threats to kidney health; yet several important gaps in our understanding still exist.CONCLUSIONSA growing body of evidence portends PFASs are emerging environmental threats to kidney health; yet several important gaps in our understanding still exist.
Author Stapleton, Heather M
Souma, Tomokazu
Zhao, Xinlu
Wittmer, Ashley
Stanifer, John W
Boulware, L Ebony
Author_xml – sequence: 1
  givenname: John W
  orcidid: 0000-0001-6379-300
  surname: Stanifer
  fullname: Stanifer, John W
  organization: Duke Global Health Institute
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  givenname: Heather M
  surname: Stapleton
  fullname: Stapleton, Heather M
  organization: Nicholas School of the Environment, and
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  givenname: Tomokazu
  surname: Souma
  fullname: Souma, Tomokazu
  organization: Division of Nephrology, Department of Medicine, Duke University School of Medicine, Durham, North Carolina; and
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  givenname: Ashley
  surname: Wittmer
  fullname: Wittmer, Ashley
  organization: Duke Global Health Institute
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  givenname: Xinlu
  surname: Zhao
  fullname: Zhao, Xinlu
  organization: Duke Global Health Institute
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  givenname: L Ebony
  surname: Boulware
  fullname: Boulware, L Ebony
  organization: Division of General Internal Medicine, Department of Medicine, Duke University, Durham, North Carolina
BackLink https://www.ncbi.nlm.nih.gov/pubmed/30213782$$D View this record in MEDLINE/PubMed
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Keywords Actin Cytoskeleton
Health Disparities
Kidney Diseases
Epidemiologic Studies
Peroxisome Proliferators
Global Health
Exposure
Permeability
kidney
Toxicology
Kidney Tubules, Proximal
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SubjectTerms Environmental Exposure - adverse effects
Environmental Pollutants - adverse effects
Fluorocarbon Polymers - adverse effects
Humans
Kidney Diseases - chemically induced
Title Perfluorinated Chemicals as Emerging Environmental Threats to Kidney Health: A Scoping Review
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