Lupus érythémateux systémique grave induit par l’isoniazide

Le lupus érythémateux systémique est une maladie auto-immune caractérisée par la présence d’anticorps antinucléaires (anti-ADN natif). Nous rapportons un cas rare de lupus érythémateux systémique grave avec atteinte rénale et neurologique induit par l’isoniazide. La patiente, âgée de 23 ans, a prése...

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Published in:Revue de pneumologie clinique Vol. 65; no. 6; pp. 361 - 364
Main Authors: Rakotoson, J.L., Randriamanana, D., Rakotomizao, J.R., Andrianasolo, R., Rakotoarivelo, R., Andrianarisoa, A.C.F.
Format: Journal Article
Language:French
Published: Paris Elsevier Masson SAS 01.12.2009
Masson
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ISSN:0761-8417
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Abstract Le lupus érythémateux systémique est une maladie auto-immune caractérisée par la présence d’anticorps antinucléaires (anti-ADN natif). Nous rapportons un cas rare de lupus érythémateux systémique grave avec atteinte rénale et neurologique induit par l’isoniazide. La patiente, âgée de 23 ans, a présenté un lupus induit par l’isoniazide un mois après le traitement d’une tuberculose pleurale. Les anticorps antinucléaires, anti-ADN natif, anti-ENA, anti-Sm, anti-SSa, anti-SSb, anti-histone étaient présents dans le sérum de notre malade. La maladie s’est manifestée par des arthralgies, éruptions cutanées, fièvre, anémie, pleurésie, péricardite et un anasarque. Elle présentait une atteinte rénale et neurologique qui fait la gravité de la maladie. L’arrêt de l’isoniazide et un bolus de méthyl-prednisolone pendant trois jours relayés par une corticothérapie per os constituaient la base du traitement. L’évolution était favorable avec arrêt de la corticothérapie au bout de huit mois. The authors report a rare case of low register systemic lupus erythematosus with renal attack and neurological armature by isoniazid. The patient was a 23-year-old woman presenting a lupus induced by isoniazid 1 month after the treatment of pleural tuberculosis. Antinuclear antibodies, anti-native DNA, anti-ENA, anti-Sm, anti-SSa, anti-SSb and antihistone were present. The symptoms included arthralgia, fever, anaemia, pleural effusion, pericarditis and anasarca. She presented a renal and neurological attack, accounting for the gravity of the disease. The treatment consisted of the interruption of the isoniazid and a bolus of methyl-prednisolone during 3 days relayed by an oral corticosteroid. The evolution was favourable after 8 months of corticosteroids.
AbstractList Le lupus érythémateux systémique est une maladie auto-immune caractérisée par la présence d’anticorps antinucléaires (anti-ADN natif). Nous rapportons un cas rare de lupus érythémateux systémique grave avec atteinte rénale et neurologique induit par l’isoniazide. La patiente, âgée de 23 ans, a présenté un lupus induit par l’isoniazide un mois après le traitement d’une tuberculose pleurale. Les anticorps antinucléaires, anti-ADN natif, anti-ENA, anti-Sm, anti-SSa, anti-SSb, anti-histone étaient présents dans le sérum de notre malade. La maladie s’est manifestée par des arthralgies, éruptions cutanées, fièvre, anémie, pleurésie, péricardite et un anasarque. Elle présentait une atteinte rénale et neurologique qui fait la gravité de la maladie. L’arrêt de l’isoniazide et un bolus de méthyl-prednisolone pendant trois jours relayés par une corticothérapie per os constituaient la base du traitement. L’évolution était favorable avec arrêt de la corticothérapie au bout de huit mois. The authors report a rare case of low register systemic lupus erythematosus with renal attack and neurological armature by isoniazid. The patient was a 23-year-old woman presenting a lupus induced by isoniazid 1 month after the treatment of pleural tuberculosis. Antinuclear antibodies, anti-native DNA, anti-ENA, anti-Sm, anti-SSa, anti-SSb and antihistone were present. The symptoms included arthralgia, fever, anaemia, pleural effusion, pericarditis and anasarca. She presented a renal and neurological attack, accounting for the gravity of the disease. The treatment consisted of the interruption of the isoniazid and a bolus of methyl-prednisolone during 3 days relayed by an oral corticosteroid. The evolution was favourable after 8 months of corticosteroids.
Author Andrianasolo, R.
Rakotoarivelo, R.
Andrianarisoa, A.C.F.
Rakotomizao, J.R.
Randriamanana, D.
Rakotoson, J.L.
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Issue 6
Keywords Systemic lupus erythematosus
Tuberculosis
Lupus érythémateux disséminé
Pleurisy
Isoniazide
Tuberculose
Pleurésie
Isoniazid
Immunopathologie
Maladie de système
Bactériose
Antituberculeux
Lupus érythémateux
Pathologie de l'appareil respiratoire
Lupus érythémateux dissémine
Mycobactériose
Pathologie de la plèvre
Induit
Infection
Maladie autoimmune
Antibactérien
Pathologie du tissu conjonctif
Pneumologie
Grave
Pathologie de la peau
Antibiotique
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Snippet Le lupus érythémateux systémique est une maladie auto-immune caractérisée par la présence d’anticorps antinucléaires (anti-ADN natif). Nous rapportons un cas...
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SubjectTerms Antibactériens
Antibiotiques. Antiinfectieux. Antiparasitaires
Appareil respiratoire : syndromes et maladies diverses
Bactérioses
Bactérioses humaines
Isoniazid
Isoniazide
Lupus érythémateux disséminé
Pathologie infectieuse
Pharmacologie. Traitements medicamenteux
Pleurisy
Pleurésie
Pneumologie
Sarcoidoses. Granulomatoses d'etiologie indeterminee. Maladies du tissu conjonctif. Maladies du tissu elastique. Vascularites
Sciences biologiques et medicales
Sciences medicales
Systemic lupus erythematosus
Tuberculose
Tuberculose et mycobactérioses atypiques
Tuberculosis
Title Lupus érythémateux systémique grave induit par l’isoniazide
URI https://dx.doi.org/10.1016/j.pneumo.2009.08.006
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