Omentin-1 attenuates glucocorticoid-induced cardiac injury by phosphorylating GSK3β

Glucocorticoid excess often causes a variety of cardiovascular complications, including hypertension, atherosclerosis, and cardiac hypertrophy. To abrogate its cardiac side effects, it is necessary to fully disclose the pathophysiological role of glucocorticoid in cardiac remodelling. Previous clini...

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Vydáno v:	Journal of molecular endocrinology Ročník 66; číslo 4; s. 273
Hlavní autoři:	Jin, Zhousheng, Xia, Fangfang, Dong, Jiaojiao, Lin, Tingting, Cai, Yaoyao, Chen, Jiali, Chen, Xixi, Huang, Zhenyang, Wang, Quanguang, Chen, Hongfei, Zhang, Junkai
Médium:	Journal Article
Jazyk:	angličtina
Vydáno:	England 01.05.2021
Témata:	Animals Cardiomegaly - chemically induced Cardiomegaly - genetics Cardiomegaly - therapy Cushing Syndrome - blood Cushing Syndrome - genetics Cushing Syndrome - pathology Cytokines - genetics Female Glucocorticoids - blood Glucocorticoids - toxicity Glycogen Synthase Kinase 3 beta - genetics Healthy Volunteers Humans Lectins - genetics Male Mitochondria - genetics Phosphorylation - genetics Rats Signal Transduction - genetics glucocorticoid excess cardiac remodelling glycogen synthase kinase 3β omentin-1 mitochondria
ISSN:	1479-6813, 1479-6813
On-line přístup:	Zjistit podrobnosti o přístupu
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