Revisiting the relationship between autism and schizophrenia: toward an integrated neurobiology

Schizophrenia and autism have been linked since their earliest descriptions. Both are disorders of cerebral specialization originating in the embryonic period. Genetic, molecular, and cytologic research highlights a variety of shared contributory mechanisms that may lead to patterns of abnormal conn...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:Annual review of clinical psychology Jg. 9; S. 555
Hauptverfasser: de Lacy, Nina, King, Bryan H
Format: Journal Article
Sprache:Englisch
Veröffentlicht: United States 01.01.2013
Schlagworte:
ISSN:1548-5951, 1548-5951
Online-Zugang:Weitere Angaben
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
Abstract Schizophrenia and autism have been linked since their earliest descriptions. Both are disorders of cerebral specialization originating in the embryonic period. Genetic, molecular, and cytologic research highlights a variety of shared contributory mechanisms that may lead to patterns of abnormal connectivity arising from altered development and topology. Overt behavioral pathology likely emerges during or after neurosensitive periods in which resource demands overwhelm system resources and the individual's ability to compensate using interregional activation fails. We are at the threshold of being able to chart autism and schizophrenia from the inside out. In so doing, the door is opened to the consideration of new therapeutics that are developed based upon molecular, synaptic, and systems targets common to both disorders.
AbstractList Schizophrenia and autism have been linked since their earliest descriptions. Both are disorders of cerebral specialization originating in the embryonic period. Genetic, molecular, and cytologic research highlights a variety of shared contributory mechanisms that may lead to patterns of abnormal connectivity arising from altered development and topology. Overt behavioral pathology likely emerges during or after neurosensitive periods in which resource demands overwhelm system resources and the individual's ability to compensate using interregional activation fails. We are at the threshold of being able to chart autism and schizophrenia from the inside out. In so doing, the door is opened to the consideration of new therapeutics that are developed based upon molecular, synaptic, and systems targets common to both disorders.
Schizophrenia and autism have been linked since their earliest descriptions. Both are disorders of cerebral specialization originating in the embryonic period. Genetic, molecular, and cytologic research highlights a variety of shared contributory mechanisms that may lead to patterns of abnormal connectivity arising from altered development and topology. Overt behavioral pathology likely emerges during or after neurosensitive periods in which resource demands overwhelm system resources and the individual's ability to compensate using interregional activation fails. We are at the threshold of being able to chart autism and schizophrenia from the inside out. In so doing, the door is opened to the consideration of new therapeutics that are developed based upon molecular, synaptic, and systems targets common to both disorders.Schizophrenia and autism have been linked since their earliest descriptions. Both are disorders of cerebral specialization originating in the embryonic period. Genetic, molecular, and cytologic research highlights a variety of shared contributory mechanisms that may lead to patterns of abnormal connectivity arising from altered development and topology. Overt behavioral pathology likely emerges during or after neurosensitive periods in which resource demands overwhelm system resources and the individual's ability to compensate using interregional activation fails. We are at the threshold of being able to chart autism and schizophrenia from the inside out. In so doing, the door is opened to the consideration of new therapeutics that are developed based upon molecular, synaptic, and systems targets common to both disorders.
Author de Lacy, Nina
King, Bryan H
Author_xml – sequence: 1
  givenname: Nina
  surname: de Lacy
  fullname: de Lacy, Nina
  organization: University of Washington and Seattle Children's Hospital, Seattle, Washington 98195, USA
– sequence: 2
  givenname: Bryan H
  surname: King
  fullname: King, Bryan H
BackLink https://www.ncbi.nlm.nih.gov/pubmed/23537488$$D View this record in MEDLINE/PubMed
BookMark eNpNkEtLxDAYRYOMOA_9C5KN4KaaV9vUnQy-YEAQXZc0_TKNdJKapDOMv94BR3B1D9zDXdw5mjjvAKErSm4oFcWtcm4MsM10b90Q9xnJCaMsozIvWHmCZjQXMsurnE7-8RTNY_wkhHORszM0ZTznpZByhuo32Npok3VrnDrAAXqVrHexswNuIO0AHFZjsnGDlWtx1J399kMXwFl1h5PfqdAeGmxdgnVQCVrsYAy-sb736_05OjWqj3BxzAX6eHx4Xz5nq9enl-X9KlNC8pRppRTVTcOglJwYVTEiNTea6bJqmYQcSFlIgFIYZhqmiWiNqYRihRFcGsoW6Pp3dwj-a4SY6o2NGvpeOfBjrClntKw4ofKgXh7VsdlAWw_BblTY13-nsB-I323p
CitedBy_id crossref_primary_10_1016_j_jpsychires_2014_05_017
crossref_primary_10_1016_j_neuro_2015_12_014
crossref_primary_10_9758_cpn_25_1296
crossref_primary_10_3389_fpsyt_2022_768586
crossref_primary_10_1093_cercor_bhw364
crossref_primary_10_1017_S0033291718000600
crossref_primary_10_1016_j_bpsc_2020_03_008
crossref_primary_10_1080_15622975_2021_1939155
crossref_primary_10_3390_ijms22179464
crossref_primary_10_26508_lsa_202302142
crossref_primary_10_3389_fnhum_2014_00283
crossref_primary_10_1016_j_schres_2024_06_009
crossref_primary_10_1016_j_nbd_2017_08_013
crossref_primary_10_1002_aur_1759
crossref_primary_10_1007_s41252_019_00147_5
crossref_primary_10_1371_journal_pone_0122711
crossref_primary_10_1016_j_brainres_2024_149198
crossref_primary_10_1016_j_neuron_2015_11_023
crossref_primary_10_1371_journal_pone_0177820
crossref_primary_10_1002_hbm_23952
crossref_primary_10_1016_j_schres_2024_12_007
crossref_primary_10_1016_j_neuroimage_2018_06_003
crossref_primary_10_1038_nature18637
crossref_primary_10_1002_ajmg_b_32406
crossref_primary_10_1016_j_pnpbp_2019_109648
crossref_primary_10_1080_13651501_2025_2472672
crossref_primary_10_1016_j_biopsych_2019_07_030
crossref_primary_10_3389_fpsyt_2017_00069
crossref_primary_10_1016_j_chc_2013_07_001
crossref_primary_10_1186_s12864_016_2475_y
crossref_primary_10_1002_aur_2659
crossref_primary_10_1016_j_schres_2020_08_022
crossref_primary_10_3389_fpsyt_2022_934005
crossref_primary_10_1016_j_ajp_2024_104345
crossref_primary_10_1002_dneu_22795
crossref_primary_10_1089_cap_2016_29120_bjc
crossref_primary_10_1038_s41598_023_39881_1
crossref_primary_10_3389_fpsyt_2023_1205138
crossref_primary_10_1016_j_psychres_2018_10_074
crossref_primary_10_1007_s10803_021_05035_3
crossref_primary_10_1038_tp_2015_112
crossref_primary_10_1016_j_expneurol_2013_12_015
crossref_primary_10_1111_acps_12579
crossref_primary_10_1097_YCO_0000000000000040
crossref_primary_10_1038_s41398_020_01001_2
crossref_primary_10_3390_ijerph19095616
crossref_primary_10_1177_0269881114544778
crossref_primary_10_1016_j_anpsic_2018_11_001
crossref_primary_10_1016_j_ejmg_2017_12_005
crossref_primary_10_1186_s12888_014_0302_z
crossref_primary_10_1016_j_neubiorev_2017_01_030
crossref_primary_10_1155_2015_167308
crossref_primary_10_4103_1673_5374_293133
crossref_primary_10_3389_fnbeh_2015_00038
crossref_primary_10_3389_fnins_2023_1135559
crossref_primary_10_1007_s10803_017_3065_9
crossref_primary_10_1016_j_mehy_2019_03_015
crossref_primary_10_3390_biomedicines11020289
crossref_primary_10_1038_mp_2014_172
crossref_primary_10_1192_j_eurpsy_2020_99
crossref_primary_10_1093_schbul_sbu188
crossref_primary_10_1002_ajmg_b_32363
crossref_primary_10_1007_s10577_018_9587_y
crossref_primary_10_1177_1359104517713241
crossref_primary_10_1080_13569783_2018_1561256
crossref_primary_10_1111_eip_13031
crossref_primary_10_1089_cap_2018_29159_bjc
crossref_primary_10_1016_j_celrep_2023_112430
crossref_primary_10_1016_j_neubiorev_2018_01_014
ContentType Journal Article
DBID CGR
CUY
CVF
ECM
EIF
NPM
7X8
DOI 10.1146/annurev-clinpsy-050212-185627
DatabaseName Medline
MEDLINE
MEDLINE (Ovid)
MEDLINE
MEDLINE
PubMed
MEDLINE - Academic
DatabaseTitle MEDLINE
Medline Complete
MEDLINE with Full Text
PubMed
MEDLINE (Ovid)
MEDLINE - Academic
DatabaseTitleList MEDLINE
MEDLINE - Academic
Database_xml – sequence: 1
  dbid: NPM
  name: PubMed
  url: http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed
  sourceTypes: Index Database
– sequence: 2
  dbid: 7X8
  name: MEDLINE - Academic
  url: https://search.proquest.com/medline
  sourceTypes: Aggregation Database
DeliveryMethod no_fulltext_linktorsrc
EISSN 1548-5951
ExternalDocumentID 23537488
Genre Journal Article
Review
GroupedDBID ---
-QD
-QH
0R~
1KX
23M
4.4
51A
53G
5FA
5FB
5FC
5FD
5FE
5FF
5FG
5FH
5GY
6J9
70K
70N
70Q
70S
70W
70X
79.
7A.
7B-
7X7
88E
8AO
8FI
8FJ
8NG
8R4
8R5
AABJL
AAGWO
AAKOE
AALHT
AALUV
AAOHI
AAQMF
AARJV
AAVPX
AAWJP
AAWTL
AAXSQ
AAYIS
AAZCL
ABBTB
ABDOG
ABGCZ
ABGRM
ABIPL
ABIVO
ABJNI
ABKGM
ABMRD
ABUWG
ABVYV
ABZNY
ACAHA
ACDVT
ACGFO
ACGFS
ACHQT
ACJYF
ACKHT
ACMXS
ACPHO
ACQCJ
ACRLM
ACSOE
ADBBV
ADEJD
ADGWB
ADHEY
ADLON
ADNJN
ADSVE
AEAIQ
AEKBM
AENEX
AEPIK
AEWNI
AFCZG
AFERR
AFKDQ
AFKEJ
AFKRA
AFONB
AHIXL
AHKZM
AHVNO
AICBU
AIDEK
AIJFW
AJAAW
AJOTX
ALAFQ
ALIPV
ALMA_UNASSIGNED_HOLDINGS
AMTJG
AONFT
AOUBY
AQQLW
ATAUN
AZQEC
B9D
B9E
B9F
B9G
B9H
B9L
B9M
B9N
BCFVH
BENPR
BJPMW
BMYRD
BPHCQ
BVIZK
BVXVI
CCPQU
CGR
CS3
CUY
CVF
DWQXO
EBS
ECM
EIF
EJD
F-Q
F-S
F-V
F-X
F-Y
F-Z
F.1
F5P
FIWKU
FIXEU
FMZAJ
FQMFW
FT0
FU.
FUEKT
FW-
FXG
FYUFA
GJQJI
GLOEX
GNDDA
GNUQQ
GOAVI
GQXMV
GURLG
H13
HMCUK
HZ~
H~9
J1V
M1P
M22
M2M
N9A
NEJ
NPM
O9-
P0P
P2P
PHGZT
PQQKQ
PROAC
PSQYO
PSYQQ
Q2X
RAR
RAV
UKHRP
ZYWBE
7X8
ACQLW
RIG
ID FETCH-LOGICAL-a483t-caaa1cbb2e7830fa9208c3fc2c79d28e5e0768ee74f2fb2c04dff94a26f438f12
IEDL.DBID 7X8
ISICitedReferencesCount 82
ISICitedReferencesURI http://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=Summon&SrcAuth=ProQuest&DestLinkType=CitingArticles&DestApp=WOS_CPL&KeyUT=000321742100022&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
ISSN 1548-5951
IngestDate Wed Oct 01 13:25:27 EDT 2025
Thu Apr 03 06:57:08 EDT 2025
IsPeerReviewed true
IsScholarly true
Language English
LinkModel DirectLink
MergedId FETCHMERGED-LOGICAL-a483t-caaa1cbb2e7830fa9208c3fc2c79d28e5e0768ee74f2fb2c04dff94a26f438f12
Notes ObjectType-Article-1
SourceType-Scholarly Journals-1
ObjectType-Feature-2
ObjectType-Review-3
content type line 23
PMID 23537488
PQID 1321793018
PQPubID 23479
ParticipantIDs proquest_miscellaneous_1321793018
pubmed_primary_23537488
PublicationCentury 2000
PublicationDate 2013-01-01
PublicationDateYYYYMMDD 2013-01-01
PublicationDate_xml – month: 01
  year: 2013
  text: 2013-01-01
  day: 01
PublicationDecade 2010
PublicationPlace United States
PublicationPlace_xml – name: United States
PublicationTitle Annual review of clinical psychology
PublicationTitleAlternate Annu Rev Clin Psychol
PublicationYear 2013
SSID ssj0033452
Score 2.3296828
SecondaryResourceType review_article
Snippet Schizophrenia and autism have been linked since their earliest descriptions. Both are disorders of cerebral specialization originating in the embryonic period....
SourceID proquest
pubmed
SourceType Aggregation Database
Index Database
StartPage 555
SubjectTerms Autistic Disorder - etiology
Autistic Disorder - genetics
Autistic Disorder - physiopathology
Brain - growth & development
Brain - metabolism
Brain - physiopathology
Humans
Schizophrenia - etiology
Schizophrenia - genetics
Schizophrenia - physiopathology
Title Revisiting the relationship between autism and schizophrenia: toward an integrated neurobiology
URI https://www.ncbi.nlm.nih.gov/pubmed/23537488
https://www.proquest.com/docview/1321793018
Volume 9
WOSCitedRecordID wos000321742100022&url=https%3A%2F%2Fcvtisr.summon.serialssolutions.com%2F%23%21%2Fsearch%3Fho%3Df%26include.ft.matches%3Dt%26l%3Dnull%26q%3D
hasFullText
inHoldings 1
isFullTextHit
isPrint
link http://cvtisr.summon.serialssolutions.com/2.0.0/link/0/eLvHCXMwpV1LS8QwEA7qinjxga_1RQQ9BtskbVIvIuLiQZdFVPZW0jzQg926XQX_vZNsVr0IgpdeSqFMv2a-yUy-D6HjrCgqUemEeOl0wlNhicpz-CBQMFeQD1IVxKofb0S_L4fDYhA33No4VjlbE8NCbUba75GfQtXksZSk8rx5Jd41yndXo4XGPOowoDIe1WL41UVgjAfHHc_KSQZUYgmdRHXcmckL8ecPm_aDJJnXOieQvXIqfmebIev0Vv_7vmtoJfJNfDEFyDqas_UGKu_CmXI_8YyBAOLxbCLu6bnBcXILK4Bk-4JVbXD7czLvDE_CqC3cwV9iEwYHYcwo6bSJHnpX95fXJDotEMUlmxCtlEp1VVErJEucKmgiNXOaalEYKm1mfcPOWsEddRXVCTfOFVzR3HEmXUq30EI9qu0OwlwYmeTGOKUynkM955zIbGG1hULPMNpFR7N4lYBk355QtR29teV3xLpoexr0splKbpSUZV4nR-7-4ek9tEyDZ4XfJ9lHHQf_sT1Ai_odwjY-DBCBa39w-wmEvcj7
linkProvider ProQuest
openUrl ctx_ver=Z39.88-2004&ctx_enc=info%3Aofi%2Fenc%3AUTF-8&rfr_id=info%3Asid%2Fsummon.serialssolutions.com&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Revisiting+the+relationship+between+autism+and+schizophrenia%3A+toward+an+integrated+neurobiology&rft.jtitle=Annual+review+of+clinical+psychology&rft.au=de+Lacy%2C+Nina&rft.au=King%2C+Bryan+H&rft.date=2013-01-01&rft.eissn=1548-5951&rft.volume=9&rft.spage=555&rft_id=info:doi/10.1146%2Fannurev-clinpsy-050212-185627&rft_id=info%3Apmid%2F23537488&rft_id=info%3Apmid%2F23537488&rft.externalDocID=23537488
thumbnail_l http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/lc.gif&issn=1548-5951&client=summon
thumbnail_m http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/mc.gif&issn=1548-5951&client=summon
thumbnail_s http://covers-cdn.summon.serialssolutions.com/index.aspx?isbn=/sc.gif&issn=1548-5951&client=summon