Suchergebnisse - "Aubert, Incarnation"
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Increased D1 dopamine receptor signaling in levodopa-induced dyskinesia
ISSN: 0364-5134, 1531-8249Veröffentlicht: Hoboken Wiley Subscription Services, Inc., A Wiley Company 01.01.2005Veröffentlicht in Annals of neurology (01.01.2005)“… Involuntary movements, or dyskinesia, represent a debilitating complication of levodopa therapy for Parkinson's disease. Although changes affecting D1 and D2 …”
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Neuroanatomical Study of the A11 Diencephalospinal Pathway in the Non-Human Primate
ISSN: 1932-6203, 1932-6203Veröffentlicht: United States Public Library of Science 13.10.2010Veröffentlicht in PloS one (13.10.2010)“… The A11 diencephalospinal pathway is crucial for sensorimotor integration and pain control at the spinal cord level. When disrupted, it is thought to be …”
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Phenotypical characterization of the neurons expressing the D1 and D2 dopamine receptors in the monkey striatum
ISSN: 0021-9967, 1096-9861Veröffentlicht: New York John Wiley & Sons, Inc 28.02.2000Veröffentlicht in Journal of comparative neurology (1911) (28.02.2000)“… The striatum is regulated by dopaminergic inputs from the substantia nigra. Several anatomical studies using in situ hybridization have demonstrated that in …”
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High frequency stimulation of the entopeduncular nucleus sets the cortico-basal ganglia network to a new functional state in the dystonic hamster
ISSN: 0969-9961, 1095-953X, 1095-953XVeröffentlicht: United States Elsevier Inc 01.09.2009Veröffentlicht in Neurobiology of disease (01.09.2009)“… High frequency stimulation (HFS) of the internal pallidum is effective for the treatment of dystonia. Only few studies have investigated the effects of …”
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Lentiviral overexpression of GRK6 alleviates L-dopa-induced dyskinesia in experimental Parkinson's disease
ISSN: 1946-6242, 1946-6242Veröffentlicht: United States 21.04.2010Veröffentlicht in Science translational medicine (21.04.2010)“… Parkinson's disease is caused primarily by degeneration of brain dopaminergic neurons in the substantia nigra and the consequent deficit of dopamine in the …”
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Increased D 1 dopamine receptor signaling in levodopa‐induced dyskinesia
ISSN: 0364-5134, 1531-8249Veröffentlicht: 01.01.2005Veröffentlicht in Annals of neurology (01.01.2005)“… Involuntary movements, or dyskinesia, represent a debilitating complication of levodopa therapy for Parkinson's disease. Although changes affecting D 1 and D 2 …”
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Striatal Overexpression of ΔJunD Resets L-DOPA-Induced Dyskinesia in a Primate Model of Parkinson Disease
ISSN: 0006-3223, 1873-2402Veröffentlicht: New York, NY Elsevier Inc 2009Veröffentlicht in Biological psychiatry (1969) (2009)“… Involuntary movements, or dyskinesia, represent a debilitating complication of dopamine replacement therapy for Parkinson disease (PD). The transcription …”
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Antagonizing L-type Ca2+ Channel Reduces Development of Abnormal Involuntary Movement in the Rat Model of L-3,4-Dihydroxyphenylalanine-Induced Dyskinesia
ISSN: 0006-3223, 1873-2402, 1873-2402Veröffentlicht: New York, NY Elsevier 15.03.2009Veröffentlicht in Biological psychiatry (1969) (15.03.2009)“… Chronic L-3,4-dihydroxyphenylalanine (L-DOPA) treatment of Parkinson's disease (PD) leads to debilitating involuntary movements, termed L-DOPA-induced …”
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Enhanced Preproenkephalin-B–Derived Opioid Transmission in Striatum and Subthalamic Nucleus Converges Upon Globus Pallidus Internalis in L-3,4-dihydroxyphenylalanine–Induced Dyskinesia
ISSN: 0006-3223, 1873-2402Veröffentlicht: New York, NY Elsevier Inc 01.04.2007Veröffentlicht in Biological psychiatry (1969) (01.04.2007)“… A role for enhanced opioid peptide transmission has been suggested in the genesis of levodopa-induced dyskinesia. However, basal ganglia nuclei other than the …”
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Involvement of sensorimotor, limbic, and associative basal ganglia domains in L-3,4-dihydroxyphenylalanine-induced dyskinesia
ISSN: 1529-2401, 1529-2401Veröffentlicht: United States 23.02.2005Veröffentlicht in The Journal of neuroscience (23.02.2005)“… Dyskinesia represents a debilitating complication of L-3,4-dihydroxyphenylalanine (L-dopa) therapy for Parkinson's disease. Such motor manifestations are …”
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Levodopa-induced dyskinesia in MPTP-treated macaques is not dependent on the extent and pattern of nigrostrial lesioning
ISSN: 0953-816X, 1460-9568Veröffentlicht: Oxford, UK Blackwell Science Ltd 01.07.2005Veröffentlicht in The European journal of neuroscience (01.07.2005)“… The extent of nigrostriatal denervation is presumed to play a role in the genesis of levodopa‐induced dyskinesia. Yet some parkinsonian patients who have been …”
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Striatal Overexpression of JunD Resets L-DOPA-Induced Dyskinesia in a Primate Model of Parkinson Disease
ISSN: 0006-3223Veröffentlicht: 15.09.2009Veröffentlicht in Biological psychiatry (1969) (15.09.2009)“… Involuntary movements, or dyskinesia, represent a debilitating complication of dopamine replacement therapy for Parkinson disease (PD). The transcription …”
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Antagonizing L-type Ca 2+ Channel Reduces Development of Abnormal Involuntary Movement in the Rat Model of L-3,4-Dihydroxyphenylalanine-Induced Dyskinesia
ISSN: 0006-3223, 1873-2402Veröffentlicht: Elsevier Inc 2009Veröffentlicht in Biological psychiatry (1969) (2009)“… Chronic L-3,4-dihydroxyphenylalanine (L-DOPA) treatment of Parkinson's disease (PD) leads to debilitating involuntary movements, termed L-DOPA-induced …”
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Cellular distribution of adenosine A2A receptor mrna in the primate striatum
ISSN: 0021-9967, 1096-9861Veröffentlicht: New York John Wiley & Sons, Inc 21.09.1998Veröffentlicht in Journal of comparative neurology (1911) (21.09.1998)“… The cellular expression of adenosine A2A receptor mRNA in the adult monkey and human striatum was examined by using single and double in situ hybridization …”
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SHORT COMMUNICATION: Levodopa-induced dyskinesia in MPTP-treated macaques is not dependent on the extent and pattern of nigrostrial lesioning
ISSN: 0953-816X, 1460-9568Veröffentlicht: 01.07.2005Veröffentlicht in The European journal of neuroscience (01.07.2005)“… The extent of nigrostriatal denervation is presumed to play a role in the genesis of levodopa-induced dyskinesia. Yet some parkinsonian patients who have been …”
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Cytoplasmic Expression of Human Telomerase Catalytic Protein (hTERT) in Neutrophils: An Immunoelectron Microscopy Study
ISSN: 0191-3123, 1521-0758, 1521-0758Veröffentlicht: England Informa UK Ltd 01.01.2008Veröffentlicht in Ultrastructural pathology (01.01.2008)“… Human telomerase comprises a catalytic protein subunit (hTERT) and an RNA subunit (hTR). Telomerase extends chromosome ends in compensation for the attrition …”
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Increased D sub(1) dopamine receptor signaling in levodopa-induced dyskinesia
ISSN: 0364-5134Veröffentlicht: 01.01.2005Veröffentlicht in Annals of neurology (01.01.2005)“… Involuntary movements, or dyskinesia, represent a debilitating complication of levodopa therapy for Parkinson's disease. Although changes affecting D sub(1) …”
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Toward a primate model of l-dopa-unresponsive parkinsonism mimicking striatonigral degeneration
ISSN: 0885-3185, 1531-8257Veröffentlicht: New York John Wiley & Sons, Inc 01.05.2000Veröffentlicht in Movement disorders (01.05.2000)“… We developed a primate model of striatonigral degeneration (SND), the neuropathology underlying levodopa‐unresponsive parkinsonism associated with multiple …”
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Striatal overexpression of DeltaJunD resets L-DOPA-induced dyskinesia in a primate model of Parkinson disease
ISSN: 1873-2402, 1873-2402Veröffentlicht: United States 15.09.2009Veröffentlicht in Biological psychiatry (1969) (15.09.2009)“… Involuntary movements, or dyskinesia, represent a debilitating complication of dopamine replacement therapy for Parkinson disease (PD). The transcription …”
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Mapping, Characterization, and Expression Analysis of the SM-20 Human Homologue, C1orf12, and Identification of a Novel Related Gene, SCAND2
ISSN: 0888-7543, 1089-8646Veröffentlicht: San Diego, CA Elsevier Inc 01.11.2000Veröffentlicht in Genomics (San Diego, Calif.) (01.11.2000)“… Major psychosis was shown to segregate with a balanced translocation (1q42.1; 11q14.3) in a multigenerational family. This study describes the identification …”
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