Suchergebnisse - "etiology [Cognition Disorders]"

Cerebral glucose metabolic correlates of cognitive and behavioural impairments in amyotrophic lateral sclerosis

Autoren: Annaliis Lehto Julia Schumacher Elisabeth Kasper et al.

Quelle: J Neurol
Journal of neurology 271(8), 5290-5300 (2024). doi:10.1007/s00415-024-12388-z

Schlagwörter: Male, Grey matter volume, Neuropsychological Tests, diagnostic imaging [Cognition Disorders], Cerebral glucose metabolism, Cognition, Mental Disorders/metabolism [MeSH], Aged [MeSH], Magnetic Resonance Imaging [MeSH], Male [MeSH], Positron-Emission Tomography [MeSH], Glucose/metabolism [MeSH], Cognition Disorders/diagnostic imaging [MeSH], Female [MeSH], Amyotrophic lateral sclerosis, Brain/diagnostic imaging [MeSH], Fluorodeoxyglucose F18/metabolism [MeSH], Cerebral Cortex/metabolism [MeSH], Humans [MeSH], Middle Aged [MeSH], Amyotrophic Lateral Sclerosis/metabolism [MeSH], Amyotrophic Lateral Sclerosis/diagnostic imaging [MeSH], FDG-PET, Cognition Disorders/metabolism [MeSH], Cerebral Cortex/diagnostic imaging [MeSH], Brain/metabolism [MeSH], Mental Disorders/diagnostic imaging [MeSH], Cognition Disorders/etiology [MeSH], Original Communication, Neuropsychological Tests [MeSH], diagnostic imaging [Amyotrophic Lateral Sclerosis], Fluorodeoxyglucose F18, metabolism [Cognition Disorders], diagnostic imaging [Cerebral Cortex], metabolism [Fluorodeoxyglucose F18], Humans, ddc:610, diagnostic imaging [Brain], Aged, Cerebral Cortex, metabolism [Amyotrophic Lateral Sclerosis], metabolism [Cerebral Cortex], Mental Disorders, Amyotrophic Lateral Sclerosis, etiology [Cognition Disorders], Brain, Middle Aged, Magnetic Resonance Imaging, 3. Good health, metabolism [Glucose], Glucose, metabolism [Brain], Positron-Emission Tomography, Female, Cognition Disorders, metabolism [Mental Disorders], diagnostic imaging [Mental Disorders]

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38861034
https://repository.publisso.de/resource/frl:6522605

Risk factors for domain-specific neurocognitive outcome in pediatric survivors of a brain tumor in the posterior fossa—Results of the HIT 2000 trial

Autoren: Martin Mynarek Anne Rossius Anika Guiard et al.

Quelle: Neuro-Oncology 26(11), 2113-2124 (2024). doi:10.1093/neuonc/noae092
Mynarek, M, Rossius, A, Guiard, A, Ottensmeier, H, von Hoff, K, Obrecht-Sturm, D, Bußenius, L, Friedrich, C, von Bueren, A O, Gerber, N U, Traunwieser, T, Kortmann, R-D, Warmuth-Metz, M, Bison, B, Thomale, U-W, Krauss, J, Pietsch, T, Clifford, S C, Pfister, S M, Sturm, D, Sahm, F, Tischler, T & Rutkowski, S 2024, ' Risk factors for domain-specific neurocognitive outcome in pediatric survivors of a brain tumor in the posterior fossa-Results of the HIT 2000 trial ', Neuro-Oncology . https://doi.org/10.1093/neuonc/noae092

Schlagwörter: Male, Mutism / etiology, quality of survival, ependymoma, Adolescent, Mutism, Cancer Survivors / psychology, etiology [Hydrocephalus], etiology [Mutism], Infratentorial Neoplasms, Neuropsychological Tests, Cognition Disorders / etiology, medulloblastoma, Cancer Survivors, Risk Factors, Infratentorial Neoplasms / therapy, Humans, ddc:610, Child, statistics & numerical data [Cancer Survivors], Ependymoma / therapy, 2. Zero hunger, neuropsychological late effects, Infant, etiology [Cognition Disorders], Neuropsychological late effects, Hydrocephalus / etiology, therapy [Medulloblastoma], Prognosis, Quality of survival, infant, 3. Good health, Ependymoma, Child, Preschool, therapy [Infratentorial Neoplasms], psychology [Cancer Survivors], Medulloblastoma / therapy, Female, Cancer Survivors / statistics & numerical data, Cognition Disorders, therapy [Ependymoma], Medulloblastoma, Follow-Up Studies, Hydrocephalus

Dateibeschreibung: application/pdf

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38835160

The myth of brain damage: no change of neurofilament light chain during transient cognitive side-effects of ECT

Autoren: Matthias Besse Michael Belz Claudia Bartels et al.

Weitere Verfasser: Matthias Besse Michael Belz Claudia Bartels et al.

Quelle: Eur Arch Psychiatry Clin Neurosci
European archives of psychiatry and clinical neuroscience 274(5), 1187-1195 (2024). doi:10.1007/s00406-023-01686-8

Schlagwörter: Male, Adult, Biomarkers, Female [MeSH], Depressive Disorder, Major/therapy [MeSH], Aged [MeSH], Cognition Disorders/blood [MeSH], Adult [MeSH], Biomarkers/blood [MeSH], Humans [MeSH], Electroconvulsive therapy (ECT), Middle Aged [MeSH], Electroconvulsive Therapy/adverse effects [MeSH], Neurofilament light chain (NfL), Male [MeSH], Neurofilament Proteins/blood [MeSH], Neuropsychology, Depressive Disorder, Major/blood [MeSH], Original Paper, Brain damage, Cognition Disorders/etiology [MeSH], Cognition, Neuropsychological Tests [MeSH], blood [Neurofilament Proteins], blood [Depressive Disorder, Major], Neuropsychological Tests, Neurofilament Proteins, Humans, ddc:610, neurofilament protein L, Electroconvulsive Therapy, Aged, Depressive Disorder, Major, blood [Biomarkers], therapy [Depressive Disorder, Major], etiology [Cognition Disorders], Middle Aged, 3. Good health, Female, adverse effects [Electroconvulsive Therapy], Cognition Disorders, blood [Cognition Disorders]

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/37656172
https://resolver.sub.uni-goettingen.de/purl?gro-2/133143
https://repository.publisso.de/resource/frl:6494102

Cerebrospinal Fluid Levels of Kininogen‐1 Indicate Early Cognitive Impairment in Parkinson's Disease

Autoren: Anna Månberg Wassilios G. Meissner Jean-Christophe Corvol et al.

Weitere Verfasser: Anna Månberg Wassilios G. Meissner Jean-Christophe Corvol et al.

Quelle: Movement disorders 35(11), 2101-2106 (2020). doi:10.1002/mds.28192

Schlagwörter: cognition, Kininogens, Parkinson's disease, neurodegeneration, etiology [Cognition Disorders], Parkinson Disease, Neuropsychological Tests, Mental Status and Dementia Tests, etiology [Cognitive Dysfunction], 3. Good health, [SDV] Life Sciences [q-bio], kininogen-1, 03 medical and health sciences, 0302 clinical medicine, Humans, Cognitive Dysfunction, ddc:610, complications [Parkinson Disease], Cognition Disorders, diagnosis [Cognition Disorders]

Dateibeschreibung: application/pdf

Zugangs-URL: https://movementdisorders.onlinelibrary.wiley.com/doi/pdfdirect/10.1002/mds.28192
https://pubmed.ncbi.nlm.nih.gov/33179332
https://www.ncbi.nlm.nih.gov/pubmed/33179332
https://movementdisorders.onlinelibrary.wiley.com/doi/10.1002/mds.28192
https://www.mysciencework.com/publication/show/cerebrospinal-fluid-levels-kininogen1-indicate-early-cognitive-impairment-parkinsons-disease-47138b78
https://hal.inria.fr/hal-03212590v1
https://onlinelibrary.wiley.com/doi/full/10.1002/mds.28192
https://pubmed.ncbi.nlm.nih.gov/33179332/
https://pub.dzne.de/record/154382
https://hal.sorbonne-universite.fr/hal-03212590v1
https://hal.sorbonne-universite.fr/hal-03212590v1/document
https://doi.org/10.1002/mds.28192

Impact of atrial fibrillation burden on cognitive function after left atrial ablation – Results of the MACPAF study

Autoren: Herm, Juliane Schirdewan, Alexander Koch, Lydia et al.

Quelle: Journal of clinical neuroscience 73, 168-172 (2020). doi:10.1016/j.jocn.2019.12.030

Schlagwörter: Male, complications [Atrial Fibrillation], epidemiology [Cognition Disorders], surgery [Atrial Fibrillation], etiology [Cognition Disorders], Middle Aged, physiopathology [Atrial Fibrillation], 03 medical and health sciences, Cognition, Treatment Outcome, 0302 clinical medicine, Atrial Fibrillation, Catheter Ablation, Humans, Female, ddc:610, Prospective Studies, Cognition Disorders, methods [Catheter Ablation], Aged

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/31992513
https://pubmed.ncbi.nlm.nih.gov/31992513/
https://europepmc.org/article/MED/31992513
https://www.sciencedirect.com/science/article/pii/S0967586819317552
https://www.ncbi.nlm.nih.gov/pubmed/31992513
https://pub.dzne.de/record/144990

Bilateral contemporaneous posteroventral pallidotomy for the treatment of Parkinson's disease: neuropsychological and neurological side effects. Report of four cases and review of the literature

Autoren: Ghika, J. Ghika-Schmid, F. Fankhauser, H. et al.

Schlagwörter: Activities of Daily Living Blepharospasm/etiology Cognition Disorders/*etiology Decision Making Deglutition Disorders/etiology Depression/etiology Disease Progression Dysarthria/etiology Fatigue/etiology Female Globus Pallidus/*surgery Humans Male Mental Disorders/*etiology Middle Aged Mood Disorders/*etiology Motor Activity/physiology Movement Disorders/surgery Neuropsychology Obsessive-Compulsive Disorder/etiology Parkinson Disease/physiopathology/psychology/*surgery Personality Disorders/etiology *Postoperative Complications Sialorrhea/etiology

Relation: Journal of Neurosurgery; https://iris.unil.ch/handle/iris/198298; serval:BIB_CF30D349F36C; 000081681800019

Verfügbarkeit: https://iris.unil.ch/handle/iris/198298
https://doi.org/10.3171/jns.1999.91.2.0313

Non-motor symptoms are relevant and possibly treatable in hereditary spastic paraplegia type 4 (SPG4)

Autoren: Tim W. Rattay Andreas Boldt Maximilian Völker et al.

Quelle: Journal of neurology 267(2), 369-379 (2020). doi:10.1007/s00415-019-09573-w

Schlagwörter: Adult, Male, etiology [Fatigue], physiopathology [Paraplegia], etiology [Depression], therapy [Spastic Paraplegia, Hereditary], psychology [Cognition Disorders], etiology [Urinary Bladder Diseases], psychology [Fatigue], etiology [Restless Legs Syndrome], Pain, etiology [Sexual Dysfunction, Physiological], Young Adult, 03 medical and health sciences, 0302 clinical medicine, physiopathology [Spastic Paraplegia, Hereditary], psychology [Spastic Paraplegia, Hereditary], Restless Legs Syndrome, psychology [Paraplegia], Humans, etiology [Fecal Incontinence], ddc:610, therapy [Paraplegia], Fatigue, Aged, Paraplegia, Memory Disorders, Depression, Spastic Paraplegia, Hereditary, Urinary Bladder Diseases, etiology [Cognition Disorders], Middle Aged, Mental Status and Dementia Tests, etiology [Pain], etiology [Memory Disorders], Sexual Dysfunction, Physiological, psychology [Depression], psychology [Restless Legs Syndrome], Quality of Life, Female, Self Report, Cognition Disorders, Fecal Incontinence

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/31646384
https://link.springer.com/article/10.1007/s00415-019-09573-w
https://europepmc.org/article/MED/31646384
https://www.ncbi.nlm.nih.gov/pubmed/31646384
https://pubmed.ncbi.nlm.nih.gov/31646384/
https://link.springer.com/10.1007/s00415-019-09573-w
https://pub.dzne.de/record/141598

A mouse model for intellectual disability caused by mutations in the X-linked 2′‑O‑methyltransferase Ftsj1 gene

Autoren: Martin B. Delatycki Diego J. Walther Helmut Fuchs et al.

Quelle: Biochimica et biophysica acta / Molecular basis of disease 1865(9), 2083-2093 (2019). doi:10.1016/j.bbadis.2018.12.011
Biochim Biophys Acta Mol Basis Dis
Biochimica et biophysica acta : molecular basis of disease

Schlagwörter: Male, 0301 basic medicine, Intellectual disability, Nociceptive Pain, Mice, Ecology,Evolution & Ethology, Ftsj1, Mice, Knockout, Chemical Biology & High Throughput, tRNA Methyltransferases, 0303 health sciences, Behavior, Animal, Physics, tRNA methyltransferase, Nuclear Proteins, genetics [Mental Retardation, X-Linked], genetics [Nuclear Proteins], genetics [tRNA Methyltransferases], ddc, Chemistry, Female, Synthetic Biology, etiology [Intellectual Disability], metabolism [Nuclear Proteins], pathology [Nociceptive Pain], pathology [Intellectual Disability], Mouse model, 03 medical and health sciences, physiology [Methyltransferases], Intellectual Disability, X-Linked Intellectual Disability, Animals, metabolism [tRNA Methyltransferases], Family, ddc:610, Biology, metabolism [Methyltransferases], physiology [tRNA Methyltransferases], Computational & Systems Biology, X-linked, etiology [Cognition Disorders], Methyltransferases, Mice, Inbred C57BL, Disease Models, Animal, Metabolism, etiology [Nociceptive Pain], genetics [Methyltransferases], Mutation, pathology [Cognition Disorders], Human medicine, Cognition Disorders

Dateibeschreibung: application/pdf; pdf

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/30557699
https://portal.sahmriresearch.org/en/publications/a-mouse-model-for-intellectual-disability-caused-by-mutations-in-
https://www.ncbi.nlm.nih.gov/pubmed/30557699
https://europepmc.org/abstract/MED/30557699
https://findanexpert.unimelb.edu.au/scholarlywork/1362936-a-mouse-model-for-intellectual-disability-caused-by-mutations-in-the-x-linked-2-'-o-methyltransferase-ftsj1-gene
http://www.ncbi.nlm.nih.gov/pubmed/30557699
https://mediatum.ub.tum.de/1471369
https://pub.dzne.de/record/140836
http://hdl.handle.net/21.11116/0000-0003-587A-2
http://hdl.handle.net/21.11116/0000-0003-587C-0
https://hdl.handle.net/10067/1618000151162165141
https://repository.uantwerpen.be/docman/irua/5dda27/161800.pdf
https://mediatum.ub.tum.de/1538417

Preferential degradation of cognitive networks differentiates Alzheimer’s disease from ageing

Autoren: Virginia Buckles Robert A. Koeppe Daniel S. Marcus et al.

Weitere Verfasser: Virginia Buckles Robert A. Koeppe Daniel S. Marcus et al.

Quelle: Brain 141(5), 1486-1500 (2018). doi:10.1093/brain/awy053

Schlagwörter: Male, Aging, 2-(4'-(methylamino)phenyl)-6-hydroxybenzothiazole, 32 Biomedical and Clinical Sciences, genetics [Alzheimer Disease], Neurodegenerative, Alzheimer's Disease, diagnostic imaging [Cognition Disorders], Functional connectivity, 0302 clinical medicine, pharmacokinetics [Thiazoles], Models, Neural Pathways, Medicine and Health Sciences, 80 and over, 2.1 Biological and endogenous factors, Aged, 80 and over, Brain Mapping, anzsrc-for: 42 Health Sciences, Aniline Compounds, complications [Alzheimer Disease], Middle Aged, Magnetic Resonance Imaging, Alzheimer’s disease, diagnostic imaging [Neural Pathways], 3. Good health, 52 Psychology, Neurological, Biomedical Imaging, pharmacokinetics [Fluorodeoxyglucose F18], Female, amyloid imaging, Alzheimer's disease, Adult, Models, Neurological, Amyloid imaging, anzsrc-for: 52 Psychology, 03 medical and health sciences, anzsrc-for: 32 Biomedical and Clinical Sciences, Clinical Research, Alzheimer Disease, Fluorodeoxyglucose F18, Acquired Cognitive Impairment, Humans, ddc:610, anzsrc-for: 17 Psychology and Cognitive Sciences, Aged, drug effects [Neural Pathways], Prevention, functional connectivity, Neurosciences, 42 Health Sciences, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), etiology [Cognition Disorders], Brain Disorders, 4.1 Discovery and preclinical testing of markers and technologies, pharmacokinetics [Aniline Compounds], Thiazoles, anzsrc-for: 11 Medical and Health Sciences, Positron-Emission Tomography, Dominantly Inherited Alzheimer Network, Dementia, Cognition Disorders, diagnostic imaging [Alzheimer Disease], dementia

Dateibeschreibung: application/pdf

Zugangs-URL: https://academic.oup.com/brain/article-pdf/141/5/1486/25083148/awy053.pdf
https://pubmed.ncbi.nlm.nih.gov/29522171
https://pub.dzne.de/record/139950

The association between objectively measured physical activity, depression, cognition, and health-related quality of life in Parkinson's disease

Autoren: Jos Prinzen Walter Maetzler Bernhard Cerff et al.

Quelle: Parkinsonism & related disorders 48, 74-81 (2018). doi:10.1016/j.parkreldis.2017.12.023

Schlagwörter: Adult, Male, etiology [Depression], Neuropsychological Tests, Severity of Illness Index, 03 medical and health sciences, 0302 clinical medicine, Surveys and Questionnaires, Accelerometry, psychology [Quality of Life], Humans, ddc:610, 10. No inequality, Exercise, Aged, Aged, 80 and over, Psychiatric Status Rating Scales, 2. Zero hunger, Depression, etiology [Cognition Disorders], Parkinson Disease, Middle Aged, Cross-Sectional Studies, Quality of Life, Female, complications [Parkinson Disease], psychology [Parkinson Disease], Cognition Disorders

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/29307560
https://pubmed.ncbi.nlm.nih.gov/29307560/
https://pub.dzne.de/record/139865
https://europepmc.org/article/MED/29307560
https://www.ncbi.nlm.nih.gov/pubmed/29307560
https://www.sciencedirect.com/science/article/pii/S1353802017308611

In vivo cholinergic basal forebrain atrophy predicts cognitive decline in de novo Parkinson’s disease

Autoren: Ray, Nicola J Bradburn, Steven Murgatroyd, Christopher et al.

Quelle: Brain
RISalud-ANDALUCIA. Repositorio Institucional de Salud de Andalucía
instname
Brain 141(1), 165-176 (2017). doi:10.1093/brain/awx310

Schlagwörter: Male, diagnostic imaging [Basal Forebrain], Núcleo Basal de Meynert, Basal Forebrain, Cholinergic Agents, Motor Activity, Neuropsychological Tests, Síntomas prodrómicos, 17 Psychology And Cognitive Sciences, mild cognitive impairment, metabolism [Cholinergic Agents], Enfermedad de Alzheimer, Image Processing, Computer-Assisted, Memoria, Humans, ddc:610, Longitudinal Studies, structural MRI, diagnosis [Cognition Disorders], Aged, Brain Mapping, Neurology & Neurosurgery, Prosencéfalo basal, Comprensión, etiology [Cognition Disorders], Parkinson Disease, 11 Medical And Health Sciences, Original Articles, Middle Aged, Magnetic Resonance Imaging, Biomarcadores, ROC Curve, Enfermedad de Parkinson, Parkinson's disease, Disease Progression, pathology [Cognition Disorders], metabolism [Basal Forebrain], Disfunción cognitiva, Female, complications [Parkinson Disease], Atrophy, Cognition Disorders, Desnervación, dementia

Dateibeschreibung: application/pdf

Zugangs-URL: https://academic.oup.com/brain/article-pdf/141/1/165/24175653/awx310.pdf
https://pubmed.ncbi.nlm.nih.gov/29228203
http://hdl.handle.net/10668/11893
https://eprints.whiterose.ac.uk/126219/1/Ray_et_al_2018_Brain.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5837422/
https://pubmed.ncbi.nlm.nih.gov/29228203/
https://eprints.whiterose.ac.uk/126219/
https://e-space.mmu.ac.uk/619467/
https://europepmc.org/article/MED/29228203
https://academic.oup.com/brain/article/141/1/165/4712018
https://pub.dzne.de/record/139720
https://hdl.handle.net/10668/11893
https://e-space.mmu.ac.uk/619467/

Cognitive impairment in Glucocerebrosidase (GBA)‐associated PD: Not primarily associated with cerebrospinal fluid Abeta and Tau profiles

Autoren: Ilona Csoti Karin Srulijes Andrea Pilotto et al.

Quelle: Movement disorders 32(12), 1780-1783 (2017). doi:10.1002/mds.27199
Movement Disorders

Schlagwörter: Adult, Male, Adolescent, CSF, genetics [Mutation], tau Proteins, cerebrospinal fluid [Amyloid beta-Peptides], Neuropsychological Tests, Abeta, GBA, Parkinson, Tau, alpha-synuclein, Severity of Illness Index, genetics [Glucosylceramidase], Statistics, Nonparametric, Young Adult, 03 medical and health sciences, 0302 clinical medicine, genetics [Parkinson Disease], cerebrospinal fluid [Parkinson Disease], Humans, ddc:610, cerebrospinal fluid [Peptide Fragments], 10. No inequality, Aged, Retrospective Studies, Aged, 80 and over, Amyloid beta-Peptides, etiology [Cognition Disorders], Parkinson Disease, Middle Aged, amyloid beta-protein (1-42), Peptide Fragments, 3. Good health, alpha‐synuclein, cerebrospinal fluid [tau Proteins], Mutation, Glucosylceramidase, Female, complications [Parkinson Disease], Cognition Disorders

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/29094781
https://pubmed.ncbi.nlm.nih.gov/29094781/
https://iris.unibs.it/handle/11379/498935
https://www.ncbi.nlm.nih.gov/pubmed/29094781
https://core.ac.uk/display/148039682
https://movementdisorders.onlinelibrary.wiley.com/doi/full/10.1002/mds.27199
https://onlinelibrary.wiley.com/doi/10.1002/mds.27199
https://pub.dzne.de/record/139678
https://hdl.handle.net/11379/498935
https://doi.org/10.1002/mds.27199
https://discovery-pp.ucl.ac.uk/id/eprint/10057927/

High prevalence of neuronal surface autoantibodies associated with cognitive deficits in cancer patients

Autoren: Lutz Harms Harald Prüss Carsten Finke et al.

Quelle: Journal of neurology 264(9), 1968-1977 (2017). doi:10.1007/s00415-017-8582-0

Schlagwörter: Adult, Male, 0301 basic medicine, epidemiology [Cognition Disorders], Adolescent, immunology [Nerve Tissue Proteins], metabolism [Serum Albumin], Nerve Tissue Proteins, cerebrospinal fluid [Albumins], Neuropsychological Tests, immunology [Antigens, Surface], Statistics, Nonparametric, Young Adult, 03 medical and health sciences, 0302 clinical medicine, Albumins, Neoplasms, Prevalence, Humans, ddc:610, Child, classification [Neoplasms], Serum Albumin, Aged, Autoantibodies, Aged, 80 and over, epidemiology [Neoplasms], complications [Neoplasms], etiology [Cognition Disorders], Middle Aged, cerebrospinal fluid [Cognition Disorders], 3. Good health, cerebrospinal fluid [Autoantibodies], Case-Control Studies, Antigens, Surface, blood [Autoantibodies], Female, Cognition Disorders, blood [Cognition Disorders]

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/28785798
https://europepmc.org/article/MED/28785798
https://link.springer.com/content/pdf/10.1007%2Fs00415-017-8582-0.pdf
https://pubmed.ncbi.nlm.nih.gov/28785798/
https://www.ncbi.nlm.nih.gov/pubmed/28785798
https://link.springer.com/article/10.1007/s00415-017-8582-0

Prion Protein Interactome: Identifying Novel Targets in Slowly and Rapidly Progressive Forms of Alzheimer’s Disease

Autoren: Inga Zerr Matthias Schmitz Mohsin Shafiq et al.

Weitere Verfasser: Inga Zerr Matthias Schmitz Mohsin Shafiq et al.

Quelle: Articles publicats en revistes (Patologia i Terapèutica Experimental)
Dipòsit Digital de la UB
instname
Universidad de Barcelona
Journal of Alzheimer's disease 59(1), 265-275 (2017). doi:10.3233/JAD-170237

Schlagwörter: Male, metabolism [Histones], Creutzfeldt-Jakob Syndrome, Prion Proteins, Histones, pathology [Alzheimer Disease], 03 medical and health sciences, 0302 clinical medicine, complications [Creutzfeldt-Jakob Syndrome], metabolism [Prion Proteins], Adipokines, Alzheimer Disease, Pathology, Malaltia de Creutzfeldt-Jakob, Humans, metabolism [Glycoproteins], ddc:610, Cervell, Aged, Glycoproteins, AZGP1 protein, human, Aged, 80 and over, pathology [Creutzfeldt-Jakob Syndrome], etiology [Cognition Disorders], Brain, complications [Alzheimer Disease], Alzheimer's disease, Middle Aged, Patologia, Metabolisme, Creutzfeldt-Jakob disease, 3. Good health, histone H2B type 1-A, Malaltia d'Alzheimer, Metabolism, metabolism [Brain], Disease Progression, pathology [Cognition Disorders], Female, Carrier Proteins, Cognition Disorders, metabolism [Carrier Proteins]

Dateibeschreibung: application/pdf

Zugangs-URL: http://diposit.ub.edu/dspace/bitstream/2445/154539/1/690168.pdf
https://pubmed.ncbi.nlm.nih.gov/28671123
https://hdl.handle.net/2445/154539
http://hdl.handle.net/2445/154539
https://europepmc.org/article/MED/28671123
https://www.ncbi.nlm.nih.gov/pubmed/28671123
https://www.medra.org/servlet/aliasResolver?alias=iospress&doi=10.3233/JAD-170237
http://diposit.ub.edu/dspace/handle/2445/154539
http://diposit.ub.edu/dspace/bitstream/2445/154539/1/690168.pdf
https://pubmed.ncbi.nlm.nih.gov/28671123/
https://resolver.sub.uni-goettingen.de/purl?gro-2/78355

Biomarkers and Functional Decline in Prodromal Alzheimer’s Disease

Autoren: Catherine Robb Jakob Schöpe Chinedu T. Udeh-Momoh et al.

Weitere Verfasser: Catherine Robb Jakob Schöpe Chinedu T. Udeh-Momoh et al.

Quelle: Journal of Alzheimer's disease 58(1), 69-78 (2017). doi:10.3233/JAD-161162

Schlagwörter: NATIONAL INSTITUTE, Male, positron emission tomography, Time Factors, cerebrospinal fluid [Amyloid beta-Peptides], Neuropsychological Tests, diagnostic imaging [Cognition Disorders], RECOMMENDATIONS, 0302 clinical medicine, Activities of Daily Living, magnetic resonance imaging, Longitudinal Studies, ELDERLY-PATIENTS, Aged, 80 and over, DEMENTIA, Activities of daily living, complications [Alzheimer Disease], Alzheimer's disease, Middle Aged, 3. Good health, INSTRUMENTAL ACTIVITIES, biomarker, Female, MILD-COGNITIVE-IMPAIRMENT, ASSOCIATION WORKGROUPS, Life Sciences & Biomedicine, metabolism [Alzheimer Disease], early diagnosis, metabolism [Biomarkers], Prodromal Symptoms, cerebrospinal fluid, EVERYDAY FUNCTION, 03 medical and health sciences, Alzheimer Disease, Fluorodeoxyglucose F18, Humans, ddc:610, pharmacology [Fluorodeoxyglucose F18], Aged, Science & Technology, Neurology & Neurosurgery, Amyloid beta-Peptides, Neurosciences, etiology [Cognition Disorders], 1103 Clinical Sciences, prediction, 1702 Cognitive Science, CONVERSION, Positron-Emission Tomography, DIAGNOSTIC GUIDELINES, Neurosciences & Neurology, 1109 Neurosciences, Cognition Disorders, Mental Status Schedule, diagnostic imaging [Alzheimer Disease], Biomarkers

Zugangs-URL: http://spiral.imperial.ac.uk/bitstream/10044/1/48871/6/Robb%20et%20al.%20JAD%20accepted%20version.pdf
https://pubmed.ncbi.nlm.nih.gov/28372331
https://content.iospress.com/articles/journal-of-alzheimers-disease/jad161162
https://epub.ub.uni-muenchen.de/50488/index.html
https://pubmed.ncbi.nlm.nih.gov/28372331/
https://www.ncbi.nlm.nih.gov/pubmed/28372331
https://mediatum.ub.tum.de/1448700
https://www.medra.org/servlet/aliasResolver?alias=iospress&doi=10.3233/JAD-161162
http://hdl.handle.net/10044/1/48871

Cognitive subtypes of probable Alzheimer's disease robustly identified in four cohorts

Autoren: Scheltens, NME Tijms, BM Koene, T et al.

Quelle: Scheltens, N M E, Tijms, B M, Koene, T, Barkhof, F, Teunissen, C E, Wolfsgruber, S, Wagner, M, Kornhuber, J, Peters, O, Cohn-Sheehy, B I, Rabinovici, G D, Miller, B L, Kramer, J H, Scheltens, P, van der Flier, W M, Alzheimer's Disease Neuroimaging Initiative, German Dementia Competence Network, University of California San Francisco Memory and Aging Center & Amsterdam Dementia Cohort 2017, 'Cognitive subtypes of probable Alzheimer's disease robustly identified in four cohorts', Alzheimer's and Dementia, vol. 13, no. 11, pp. 1226-1236. https://doi.org/10.1016/j.jalz.2017.03.002
Alzheimer's and dementia 13(11), 1226-1236 (2017). doi:10.1016/j.jalz.2017.03.002
Alzheimer's & Dementia, vol 13, iss 11

Schlagwörter: Male, Aging, Denmark, Biological Psychology, Apolipoprotein E4, Clinical sciences, genetics [Alzheimer Disease], cerebrospinal fluid [Amyloid beta-Peptides], Neurodegenerative, Neuropsychological Tests, Alzheimer's Disease, diagnostic imaging [Cognition Disorders], Cohort Studies, Cognition, 0302 clinical medicine, Germany, 80 and over, Psychology, 2.1 Biological and endogenous factors, Cluster Analysis, genetics [Apolipoprotein E4], Subtypes, Aged, 80 and over, University of California San Francisco Memory and Aging Center, complications [Alzheimer Disease], Alzheimer's disease, Middle Aged, amyloid beta-protein (1-42), 16. Peace & justice, classification [Cognition Disorders], Magnetic Resonance Imaging, 3. Good health, cerebrospinal fluid [Alzheimer Disease], Neurological, Disease Progression, Female, Clinical Sciences, German Dementia Competence Network, Amsterdam Dementia Cohort, 03 medical and health sciences, Neuropsychology, Alzheimer Disease, Behavioral and Social Science, Acquired Cognitive Impairment, Humans, ddc:610, cerebrospinal fluid [Peptide Fragments], Aged, Amyloid beta-Peptides, Neurosciences, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), etiology [Cognition Disorders], Peptide Fragments, United States, Brain Disorders, Geriatrics, Biological psychology, Dementia, Heterogeneity, Cognition Disorders, Mental Status Schedule, diagnostic imaging [Alzheimer Disease], Atypical

Dateibeschreibung: application/pdf

Zugangs-URL: https://europepmc.org/articles/pmc5857387?pdf=render
https://pubmed.ncbi.nlm.nih.gov/28427934
http://alzheimersanddementiajournal.net/article/S1552-5260(17)30137-1/abstract
https://www.alzheimersanddementia.com/article/S1552-5260(17)30137-1/fulltext
http://www.sciencedirect.com/science/article/pii/S1552526017301371
https://core.ac.uk/display/111022140
https://alzheimersanddementiajournal.org/article/S1552-5260(17)30137-1/abstract
https://alzheimersanddementiajournal.net/article/S1552-5260(17)30137-1/references
https://research.vumc.nl/en/publications/f4648f45-f90a-4b96-bfa5-8d125bd95d5b
https://pub.dzne.de/record/139593
https://discovery-pp.ucl.ac.uk/id/eprint/1564464/
https://escholarship.org/content/qt2t53h3h0/qt2t53h3h0.pdf
https://escholarship.org/uc/item/2t53h3h0

Left frontal cortex connectivity underlies cognitive reserve in prodromal Alzheimer disease

Autoren: Franzmeier, Nicolai Duering, Marco Weiner, Michael et al.

Quelle: Neurology 88(11), 1054-1061 (2017). doi:10.1212/WNL.0000000000003711
Neurology, vol 88, iss 11

Schlagwörter: Male, Aging, pathology [Nerve Net], Neurodegenerative, pathology [Frontal Lobe], Neuropsychological Tests, Alzheimer's Disease, Functional Laterality, pathology [Alzheimer Disease], 0302 clinical medicine, Cognitive Reserve, 2.1 Biological and endogenous factors, Aetiology, etiology [Epilepsy], 10. No inequality, 2. Zero hunger, complications [Alzheimer Disease], Magnetic Resonance Imaging, Frontal Lobe, 3. Good health, blood [Oxygen], Neurological, Biomedical Imaging, diagnostic imaging [Epilepsy], Cognitive Sciences, Female, Clinical Sciences, Prodromal Symptoms, 03 medical and health sciences, diagnostic imaging [Frontal Lobe], Clinical Research, Alzheimer Disease, Fluorodeoxyglucose F18, Acquired Cognitive Impairment, metabolism [Fluorodeoxyglucose F18], Humans, ddc:610, Neurology & Neurosurgery, Chi-Square Distribution, Epilepsy, Prevention, Neurosciences, Alzheimer's Disease Neuroimaging Initiative, physiology [Functional Laterality], Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), etiology [Cognition Disorders], Brain Disorders, physiology [Cognitive Reserve], Oxygen, Positron-Emission Tomography, Dementia, Nerve Net, Cognition Disorders, diagnostic imaging [Alzheimer Disease]

Dateibeschreibung: application/pdf

Zugangs-URL: https://europepmc.org/articles/pmc5384837?pdf=render
https://pubmed.ncbi.nlm.nih.gov/28188306
http://europepmc.org/articles/PMC5384837
https://n.neurology.org/content/88/11/1054
https://n.neurology.org/content/88/11/1054.full.pdf
http://n.neurology.org/content/88/11/1054
https://epub.ub.uni-muenchen.de/52436/index.html
https://www.neurology.org/lookup/doi/10.1212/WNL.0000000000003711
https://pub.dzne.de/record/139148
https://epub.ub.uni-muenchen.de/52436/
https://escholarship.org/uc/item/7k9921t8

Loss-of-function mutations in theATP13A2/PARK9 gene cause complicated hereditary spastic paraplegia (SPG78)

Autoren: Teodora Chamova Ludger Schöls Ivailo Tournev et al.

Quelle: Brain 140(2), 287-305 (2017). doi:10.1093/brain/aww307
Brain
Brain; a journal of neurology

Schlagwörter: Adult, Male, Leupeptins, benzyloxycarbonylleucyl-leucyl-leucine aldehyde, Medizin, pharmacology [Enzyme Inhibitors], genetics [Mutation], drug effects [Gene Expression Regulation], genetics [Cognition Disorders], Neuropsychological Tests, genetics [Gene Expression Regulation], genetics [Mental Disorders], metabolism [Lysosomes], ultrastructure [Mitochondria], genetics [Spastic Paraplegia, Hereditary], Chlorocebus aethiops, Animals, Humans, Genetic Predisposition to Disease, ddc:610, genetics [Genetic Predisposition to Disease], Enzyme Inhibitors, drug effects [Lysosomes], Cells, Cultured, diagnostic imaging [Spastic Paraplegia, Hereditary], Family Health, Psychiatric Status Rating Scales, genetics [Proton-Translocating ATPases], Mental Disorders, etiology [Cognition Disorders], drug effects [Mitochondria], cytology [Cells, Cultured], ultrastructure [Lysosomes], Middle Aged, ultrastructure [Cells, Cultured], metabolism [Mitochondria], Mitochondria, 3. Good health, pharmacology [Leupeptins], Proton-Translocating ATPases, Gene Expression Regulation, ATP13A2 protein, human, Mutation, Human medicine, complications [Spastic Paraplegia, Hereditary], Cognition Disorders, Lysosomes, etiology [Mental Disorders]

Zugangs-URL: https://academic.oup.com/brain/article-pdf/140/2/287/24174630/aww307.pdf
https://pubmed.ncbi.nlm.nih.gov/28137957
https://academic.oup.com/brain/article/140/2/287/2962954
https://core.ac.uk/display/80811626
http://www.ncbi.nlm.nih.gov/pubmed/28137957
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5278306
https://miami.pure.elsevier.com/en/publications/loss-of-function-mutations-in-the-atp13a2park9-gene-cause-complic
https://europepmc.org/article/MED/28137957
https://pub.dzne.de/record/139142
https://hdl.handle.net/10067/1425590151162165141

In vivo Patterns of Tau Pathology, Amyloid-β Burden, and Neuronal Dysfunction in Clinical Variants of Alzheimer’s Disease

Autoren: Oezguer A. Onur Klaus Fliessbach Gérard N. Bischof et al.

Quelle: Journal of Alzheimer's disease 55(2), 465-471 (2017). doi:10.3233/JAD-160316

Schlagwörter: Male, metabolism [Amyloid beta-Peptides], tau Proteins, Neuropsychological Tests, diagnostic imaging [Cognition Disorders], pathology [Alzheimer Disease], Executive Function, 03 medical and health sciences, 0302 clinical medicine, pathology [Brain], Alzheimer Disease, Fluorodeoxyglucose F18, metabolism [Fluorodeoxyglucose F18], Humans, ddc:610, 10. No inequality, diagnostic imaging [Brain], etiology [Atrophy], 2. Zero hunger, Language Disorders, Amyloid beta-Peptides, pathology [Corticomedial Nuclear Complex], Corticomedial Nuclear Complex, etiology [Cognition Disorders], 7-(6-fluoropyridin-3-yl)-5H-pyrido(4,3-b)indole, Brain, complications [Alzheimer Disease], metabolism [Carbolines], metabolism [tau Proteins], 3. Good health, metabolism [Brain], Positron-Emission Tomography, Female, etiology [Language Disorders], Atrophy, Cognition Disorders, diagnostic imaging [Alzheimer Disease], Carbolines

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/27802224
https://juser.fz-juelich.de/record/825309
https://pubmed.ncbi.nlm.nih.gov/27802224/
https://content.iospress.com/articles/journal-of-alzheimers-disease/jad160316
https://www.medra.org/servlet/aliasResolver?alias=iospress&doi=10.3233/JAD-160316

Sirtuin 2 Inhibition Improves Cognitive Performance and Acts on Amyloid-β Protein Precursor Processing in Two Alzheimer’s Disease Mouse Models

Autoren: Gloria Biella Stefan F. Lichtenthaler Emanuele Nardo et al.

Quelle: Journal of Alzheimer's disease 53(3), 1193-1207 (2016). doi:10.3233/JAD-151135

Schlagwörter: 0301 basic medicine, metabolism [Sirtuin 2], pharmacology [Enzyme Inhibitors], genetics [Alzheimer Disease], drug effects [Gene Expression Regulation], pharmacology [Benzamides], genetics [Gene Expression Regulation], pathology [Alzheimer Disease], Amyloid beta-Protein Precursor, Mice, Sirtuin 2, pharmacology [Sulfonamides], metabolism [Amyloid beta-Protein Precursor], metabolism [Peptide Fragments], pharmacology [Quinolines], pharmacology [Furans], Enzyme Inhibitors, AGK2 compound, Sulfonamides, Microfilament Proteins, Brain, complications [Alzheimer Disease], Glioma, amyloid beta-protein (1-42), 3. Good health, genetics [Amyloid beta-Protein Precursor], therapeutic use [Enzyme Inhibitors], Benzamides, Quinolines, drug effects [Brain], metabolism [Amyloid beta-Peptides], Mice, Transgenic, 3-(1-azepanylsulfonyl)-N-(3-bromphenyl)benzamide, therapeutic use [Benzamides], therapeutic use [Sulfonamides], 03 medical and health sciences, drug effects [Phosphorylation], Alzheimer Disease, metabolism [Cognition Disorders], Cell Line, Tumor, Glial Fibrillary Acidic Protein, Animals, ddc:610, Furans, Amyloid beta-Peptides, Aif1 protein, mouse, Calcium-Binding Proteins, etiology [Cognition Disorders], metabolism [Glial Fibrillary Acidic Protein], drug therapy [Cognition Disorders], metabolism [Microfilament Proteins], amyloid beta-protein (1-40), metabolism [Calcium-Binding Proteins], therapeutic use [Furans], Peptide Fragments, Mice, Inbred C57BL, Disease Models, Animal, Gene Expression Regulation, metabolism [Brain], pathology [Cognition Disorders], Cognition Disorders, pathology [Glioma], therapeutic use [Quinolines]

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/27372638
https://www.ncbi.nlm.nih.gov/pubmed/27372638/
https://www.medra.org/servlet/aliasResolver?alias=iospress&doi=10.3233/JAD-151135
https://europepmc.org/article/MED/27372638
https://pubmed.ncbi.nlm.nih.gov/27372638/
https://content.iospress.com/articles/journal-of-alzheimers-disease/jad151135
https://moh-it.pure.elsevier.com/en/publications/sirtuin-2-inhibition-improves-cognitive-performance-and-acts-on-a
https://pub.dzne.de/record/138753