Suchergebnisse - "Wnt11"

Rewiring immune evasion in liver metastases: WNT11 as a central node — a mini review

Autoren: Xiaoling Wang Youcai Huang Tingting Luo et al.

Quelle: Frontiers in Oncology, Vol 15 (2025)

Schlagwörter: liver metastasis, Wnt11, immune checkpoint blockade resistance, CaMKII signaling, tumor immune microenvironment, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, RC254-282

Dateibeschreibung: electronic resource

Relation: https://www.frontiersin.org/articles/10.3389/fonc.2025.1666889/full; https://doaj.org/toc/2234-943X

Zugangs-URL: https://doaj.org/article/4a81935e4cd545ecae26c14513f0f0a4

A de novo SALL4 mutation causes unilateral renal agenesis by misregulating genes involved in kidney development

Autoren: Rong Zhao Yali Fan Jieyan Li et al.

Quelle: Orphanet Journal of Rare Diseases, Vol 20, Iss 1, Pp 1-9 (2025)

Schlagwörter: Renal agenesis, SALL4, Fetal kidney ureteric bud cells, WNT11, PAX2, Medicine

Dateibeschreibung: electronic resource

Relation: https://doaj.org/toc/1750-1172

Zugangs-URL: https://doaj.org/article/dc74220a295749d6982d5b0b17d53f10

Additional file 1 of A de novo SALL4 mutation causes unilateral renal agenesis by misregulating genes involved in kidney development

Autoren: Rong Zhao Yali Fan Jieyan Li et al.

Schlagwörter: Genetics, Renal agenesis, SALL4, Fetal kidney ureteric bud cells, WNT11, PAX2

Relation: https://figshare.com/articles/presentation/Additional_file_1_of_A_de_novo_SALL4_mutation_causes_unilateral_renal_agenesis_by_misregulating_genes_involved_in_kidney_development/29263870

Verfügbarkeit: https://doi.org/10.6084/m9.figshare.29263870.v1
https://figshare.com/articles/presentation/Additional_file_1_of_A_de_novo_SALL4_mutation_causes_unilateral_renal_agenesis_by_misregulating_genes_involved_in_kidney_development/29263870

Additional file 2 of A de novo SALL4 mutation causes unilateral renal agenesis by misregulating genes involved in kidney development

Autoren: Rong Zhao Yali Fan Jieyan Li et al.

Schlagwörter: Genetics, Renal agenesis, SALL4, Fetal kidney ureteric bud cells, WNT11, PAX2

Relation: https://figshare.com/articles/journal_contribution/Additional_file_2_of_A_de_novo_SALL4_mutation_causes_unilateral_renal_agenesis_by_misregulating_genes_involved_in_kidney_development/29263873

Verfügbarkeit: https://doi.org/10.6084/m9.figshare.29263873.v1
https://figshare.com/articles/journal_contribution/Additional_file_2_of_A_de_novo_SALL4_mutation_causes_unilateral_renal_agenesis_by_misregulating_genes_involved_in_kidney_development/29263873

Gollop-Wolfgang Complex Is Associated with a Monoallelic Variation in WNT11

Autoren: Odrzywolski, Adrian Tuysuz, Beyhan Debeer, Philippe et al.

Quelle: ISSN:2073-4425 ; Genes, vol. 15 (1), Art.No. ARTN 129.

Schlagwörter: Science & Technology, Life Sciences & Biomedicine, Genetics & Heredity, Gollop-Wolfgang complex, whole genome sequencing, split-hand/foot malformation, WNT11, LONG-BONE DEFICIENCY, STRUCTURAL VARIATION, DLX5 MUTATION, FAMILY, VARIANTS, APLASIA, TIBIA, HAND, BIFURCATION, Gollop–Wolfgang complex, Abnormalities, Multiple, Humans, Femur, Hand Deformities, Congenital, 0604 Genetics, 3105 Genetics

Dateibeschreibung: application/pdf

Relation: https://lirias.kuleuven.be/handle/20.500.12942/736916; https://doi.org/10.3390/genes15010129; https://pubmed.ncbi.nlm.nih.gov/38275609

Verfügbarkeit: https://lirias.kuleuven.be/handle/20.500.12942/736916
https://hdl.handle.net/20.500.12942/736916
https://lirias.kuleuven.be/retrieve/61af055b-df9e-432b-a18e-05db89ff8283
https://doi.org/10.3390/genes15010129
https://pubmed.ncbi.nlm.nih.gov/38275609

Tissue specific requirements for WNT11 in developing outflow tract and dorsal mesenchymal protrusion

Autoren: van Vliet, Patrick P Lin, Lizhu Boogerd, Cornelis J et al.

Quelle: Developmental Biology. 429(1)

Schlagwörter: Biomedical and Clinical Sciences, Cardiovascular Medicine and Haematology, Genetics, Congenital Structural Anomalies, Cardiovascular, Pediatric, Heart Disease, 1.1 Normal biological development and functioning, Animals, Apoptosis, Cell Differentiation, Cell Proliferation, Embryo, Mammalian, Gene Deletion, Germ Cells, Heart, Hedgehog Proteins, Homeodomain Proteins, Integrases, LIM-Homeodomain Proteins, Mesoderm, Mice, Knockout, Myocardium, Organogenesis, Phenotype, Signal Transduction, Transcription Factors, Wnt Proteins, Homeobox Protein PITX2, Wnt11, Outflow tract, Dorsal mesenchymal protrusion, Biological Sciences, Medical and Health Sciences, Developmental Biology, Biological sciences, Biomedical and clinical sciences, Health sciences

Dateibeschreibung: application/pdf

Zugangs-URL: https://escholarship.org/uc/item/3qt3c9bx
https://escholarship.org/content/qt3qt3c9bx/qt3qt3c9bx.pdf

WNT11/ROR2 signaling is associated with tumor invasion and poor survival in breast cancer

Autoren: Kerstin Menck Saskia Heinrichs Darius Wlochowitz et al.

Quelle: Journal of Experimental & Clinical Cancer Research, Vol 40, Iss 1, Pp 1-18 (2021)

Schlagwörter: Breast cancer, Metastasis, ROR2, WNT11, BRCAness, Network analysis, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, RC254-282

Dateibeschreibung: electronic resource

Relation: https://doaj.org/toc/1756-9966

Zugangs-URL: https://doaj.org/article/d0a5a0c967764391a19be071ba9a9dca

Table 3_Genome-wide CRISPR/Cas9 screening identifies key profibrotic regulators of TGF-β1-induced epithelial-mesenchymal transformation and pulmonary fibrosis.xls

Autoren: Chunjiang Tan Juan Wang Xiangrong Ye et al.

Schlagwörter: Molecular Biology, idiopathic pulmonary fibrosis, epithelial-mesenchymal transformation, extracellular matrix, genome-wide CRISPR/Cas9 knockout screening, TGF-β1, COL20A1, COL27A1, WNT11

Verfügbarkeit: https://doi.org/10.3389/fmolb.2025.1507163.s003
https://figshare.com/articles/dataset/Table_3_Genome-wide_CRISPR_Cas9_screening_identifies_key_profibrotic_regulators_of_TGF-_1-induced_epithelial-mesenchymal_transformation_and_pulmonary_fibrosis_xls/28427033

Table 2_Genome-wide CRISPR/Cas9 screening identifies key profibrotic regulators of TGF-β1-induced epithelial-mesenchymal transformation and pulmonary fibrosis.xls

Autoren: Chunjiang Tan Juan Wang Xiangrong Ye et al.

Schlagwörter: Molecular Biology, idiopathic pulmonary fibrosis, epithelial-mesenchymal transformation, extracellular matrix, genome-wide CRISPR/Cas9 knockout screening, TGF-β1, COL20A1, COL27A1, WNT11

Verfügbarkeit: https://doi.org/10.3389/fmolb.2025.1507163.s002
https://figshare.com/articles/dataset/Table_2_Genome-wide_CRISPR_Cas9_screening_identifies_key_profibrotic_regulators_of_TGF-_1-induced_epithelial-mesenchymal_transformation_and_pulmonary_fibrosis_xls/28427039

Table 1_Genome-wide CRISPR/Cas9 screening identifies key profibrotic regulators of TGF-β1-induced epithelial-mesenchymal transformation and pulmonary fibrosis.xls

Autoren: Chunjiang Tan Juan Wang Xiangrong Ye et al.

Schlagwörter: Molecular Biology, idiopathic pulmonary fibrosis, epithelial-mesenchymal transformation, extracellular matrix, genome-wide CRISPR/Cas9 knockout screening, TGF-β1, COL20A1, COL27A1, WNT11

Verfügbarkeit: https://doi.org/10.3389/fmolb.2025.1507163.s001
https://figshare.com/articles/dataset/Table_1_Genome-wide_CRISPR_Cas9_screening_identifies_key_profibrotic_regulators_of_TGF-_1-induced_epithelial-mesenchymal_transformation_and_pulmonary_fibrosis_xls/28427042

Wnt11 in regulation of physiological and pathological cardiac growth

Autoren: Eveliina Halmetoja Irina Nagy Zoltan Szabo et al.

Quelle: The FASEB Journal. 36

Schlagwörter: 0301 basic medicine, left ventricle remodeling, 0303 health sciences, cardiac hypertrophy, Myocardium, Wnt11 protein, Cardiomegaly, Heart, pressure overload, 3. Good health, Wnt Proteins, Mice, 03 medical and health sciences, Animals, Myocytes, Cardiac, Cell Proliferation

Dateibeschreibung: application/pdf

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/36098469
http://urn.fi/urn:nbn:fi-fe2022122173167

Wnt11 acts on dermomyotome cells to guide epaxial myotome morphogenesis

Autoren: Ann Kathrin Heilig Ryohei Nakamura Atsuko Shimada et al.

Quelle: eLife, Vol 11 (2022)

Schlagwörter: dermomyotome, myotome, somite, Zic1, Wnt11, dorsalization, Medicine, Science, Biology (General), QH301-705.5

Dateibeschreibung: electronic resource

Relation: https://elifesciences.org/articles/71845; https://doaj.org/toc/2050-084X

Zugangs-URL: https://doaj.org/article/da4aaf07999c4e55a65263b48060c302

Cited2 in cardiac development: an inside and outside job

Autoren: Santos, João Miguel Almeida Bragança, José Futschik, Matthias E. et al.

Weitere Verfasser: Santos, João Miguel Almeida Bragança, José Futschik, Matthias E. et al.

Quelle: urn:tid:101480008

Schlagwörter: CITED2, WNT5a, WNT11, Defeitos Cardíacos, Células estaminais

Dateibeschreibung: application/pdf

Verfügbarkeit: http://hdl.handle.net/10400.1/13605

Maternal Wnt11b regulates cortical rotation during Xenopus axis formation: analysis of maternal-effect wnt11b mutants

Autoren: Houston, Douglas W. Elliott, Karen L. Coppenrath, Kelsey et al.

Quelle: Houston, D. W., Elliott, K. L., Coppenrath, K., Wlizla, M., & Horb, M. E. (2022). Maternal Wnt11b regulates cortical rotation during Xenopus axis formation: analysis of maternal-effect wnt11b mutants. Development, 149(17), dev.200552. ; doi:10.1242/dev.200552

Schlagwörter: Xenopus, Dorsal axis, Wnt11, Wnt11b, Microtubules, Cortical rotation, CRISPR/Cas9

Relation: https://doi.org/10.1242/dev.200552; Houston, D. W., Elliott, K. L., Coppenrath, K., Wlizla, M., & Horb, M. E. (2022). Maternal Wnt11b regulates cortical rotation during Xenopus axis formation: analysis of maternal-effect wnt11b mutants. Development, 149(17), dev.200552.; https://hdl.handle.net/1912/29680

Verfügbarkeit: https://hdl.handle.net/1912/29680

Wnt11 in regulation of physiological and pathological cardiac growth

Autoren: Halmetoja, E. (Eveliina) Nagy, I. (Irina) Szabo, Z. (Zoltan) et al.

Schlagwörter: Wnt11 protein, cardiac hypertrophy, left ventricle remodeling, pressure overload

Dateibeschreibung: application/pdf

Verfügbarkeit: http://urn.fi/urn:nbn:fi-fe2022122173167

Maternal Wnt11b regulates cortical rotation duringXenopusaxis formation: analysis of maternal-effectwnt11bmutants

Autoren: Douglas W. Houston Karen L. Elliott Kelsey Coppenrath et al.

Quelle: Development

Schlagwörter: 0301 basic medicine, 0303 health sciences, Embryo, Nonmammalian, Xenopus, Dorsal axis, Embryonic Development, Xenopus Proteins, Ligands, Microtubules, Wnt Proteins, Cortical rotation, Xenopus laevis, 03 medical and health sciences, Animals, CRISPR/Cas9, Wnt Signaling Pathway, Wnt11b, beta Catenin, Wnt11, Research Article, Body Patterning

Zugangs-URL: https://www.biorxiv.org/content/biorxiv/early/2022/02/04/2022.02.02.478872.full.pdf
https://pubmed.ncbi.nlm.nih.gov/35946588

The CapZ interacting protein Rcsd1 is required for cardiogenesis downstream of Wnt11a in Xenopus laevis

Autoren: Hempel, Annemarie Kühl, Susanne J Rothe, Melanie et al.

Quelle: Developmental Biology. 424:28-39

Schlagwörter: 0301 basic medicine, Cytoplasm, CAPPING PROTEIN, Organogenesis, Rcsd1, Amino Acid Motifs, Embryonic Development, Xenopus Proteins, ACTIVATION, Mice, Xenopus laevis, 03 medical and health sciences, Animals, Humans, LIVING CELLS, SPECIFICATION, Molecular Biology, 0303 health sciences, Science & Technology, Cardiogenesis, Myocardium, WNT-11, Intracellular Signaling Peptides and Proteins, Cell Differentiation, Cell Biology, Wnt Proteins, DIFFERENTIATION, HEK293 Cells, Phenotype, CARDIOMYOCYTE DEVELOPMENT, MORPHOGENESIS, NIH 3T3 Cells, HEART, EMBRYONIC STEM-CELLS, Life Sciences & Biomedicine, Gene Deletion, Wnt11, Developmental Biology, Protein Binding, Signal Transduction

Dateibeschreibung: application/pdf

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/28237811
https://www.ncbi.nlm.nih.gov/pubmed/28237811
https://dukespace.lib.duke.edu/dspace/handle/10161/17292
https://www.sciencedirect.com/science/article/pii/S001216061630865X
https://www.sciencedirect.com/science/article/abs/pii/S001216061630865X
http://jultika.oulu.fi/files/nbnfi-fe201707067658.pdf
https://europepmc.org/abstract/MED/28237811
http://urn.fi/urn:nbn:fi-fe201707067658

WNT11-Conditioned Medium Promotes Angiogenesis through the Activation of Non-Canonical WNT-PKC-JNK Signaling Pathway

Autoren: Jingcai Wang Min Gong Shi Zuo et al.

Quelle: Genes, Vol 11, Iss 1277, p 1277 (2020)

Schlagwörter: Wnt11, mesenchymal stem cells, angiogenesis, ischemic myocardium, Wnt-PKC-JNK pathways, Genetics, QH426-470

Relation: https://www.mdpi.com/2073-4425/11/11/1277; https://doaj.org/toc/2073-4425; https://doaj.org/article/1154fc4296e7468ba4ac10f35df6522e

Verfügbarkeit: https://doi.org/10.3390/genes11111277
https://doaj.org/article/1154fc4296e7468ba4ac10f35df6522e

Wnt11/Fgfr1b cross-talk modulates the fate of cells in palate development

Autoren: Jong Min Lee Jinglei Cai Kyoung Won Cho et al.

Weitere Verfasser: Jong Min Lee Jinglei Cai Kyoung Won Cho et al.

Quelle: Developmental Biology. 314:341-350

Schlagwörter: 0301 basic medicine, Aging, Fibroblast Growth Factor, Maxilla - Cytology - Growth & Development, z-VAD-fmk, Apoptosis, Polymerase Chain Reaction, Small Interfering - Genetics, Aging - Physiology, Mice, Palate/growth & development, Receptor, Fibroblast Growth Factor, Type 1 - Genetics - Physiology, Maxilla, Wnt Proteins/physiology, RNA, Small Interfering, Cell proliferation, In Situ Hybridization, Mice, Inbred Icr, Mice, Inbred ICR, 0303 health sciences, Palatal fusion, Wnt11 siRNA, Inbred ICR, Inbred Icr, Immunohistochemistry, Electroporation, Palatogenesis, Small Interfering/genetics, Wnt Proteins - Genetics - Physiology, Maxilla/growth & development, Receptor, Wnt11, Maxilla/cytology, 03 medical and health sciences, Organ Culture Techniques, Type 1/physiology, Animals, Receptor, Fibroblast Growth Factor, Type 1, Molecular Biology, Wnt Proteins/genetics, Fgfr1b, Palate - Cytology - Growth & Development, Palate, Type 1 - Genetics - Physiology, Receptor Cross-Talk/physiology, Cell Biology, Receptor Cross-Talk, Aging/physiology, Palatal growth, Wnt Proteins, Palate/cytology, Receptor Cross-Talk - Physiology, Rna, Small Interfering - Genetics, RNA, Rna, Type 1/genetics, Developmental Biology

Dateibeschreibung: 341~350

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/18191119
https://core.ac.uk/display/37994727
https://yonsei.pure.elsevier.com/en/publications/wnt11fgfr1b-cross-talk-modulates-the-fate-of-cells-in-palate-deve
https://hub.hku.hk/handle/10722/169546
https://www.ncbi.nlm.nih.gov/pubmed/18191119
https://www.sciencedirect.com/science/article/pii/S0012160607015679
https://www.sciencedirect.com/science/article/abs/pii/S0012160607015679
http://hdl.handle.net/10722/169546
http://hdl.handle.net/10722/169762

The Wnt11 Signaling Pathway in Potential Cellular EMT and Osteochondral Differentiation Progression in Nephrolithiasis Formation

Autoren: Deng He Yuchao Lu Henglong Hu et al.

Quelle: Int J Mol Sci
International Journal of Molecular Sciences
Volume 16
Issue 7
Pages 16313-16329

Schlagwörter: 0301 basic medicine, Epithelial-Mesenchymal Transition, Calcium Channels, L-Type, Blotting, Western, Intracellular Space, Fluorescent Antibody Technique, Cell Separation, Nephrolithiasis, Article, 03 medical and health sciences, Chondrocytes, TGF-β1, Animals, Cells, Cultured, 0303 health sciences, Cell Differentiation, Epithelial Cells, Calcium Channel Blockers, Disease Models, Animal, Kidney Tubules, 13. Climate action, Gene Knockdown Techniques, Disease Progression, calcium channel, Calcium, Chondrogenesis, Biomarkers, Wnt11, nephrolithiasis

Dateibeschreibung: application/pdf

Zugangs-URL: https://www.mdpi.com/1422-0067/16/7/16313/pdf
https://pubmed.ncbi.nlm.nih.gov/26193266
https://core.ac.uk/display/90626800
https://www.ncbi.nlm.nih.gov/pubmed/26193266
https://www.mdpi.com/1422-0067/16/7/16313
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4519952
https://www.mdpi.com/1422-0067/16/7/16313/pdf
https://europepmc.org/abstract/MED/26193266