Výsledky vyhľadávania - "VPC-13566"

Antihormone treatment differentially regulates PSA secretion, PSMA expression and 68Ga–PSMA uptake in LNCaP cells

Autori: C. S. Mathy T. Mayr S. Kürpig a ďalší

Zdroj: J Cancer Res Clin Oncol

Predmety: Glutamate Carboxypeptidase II, Male, Secretory Pathway, Edetic Acid/analogs, Cell Line, Tumor [MeSH], Glutamate Carboxypeptidase II/metabolism [MeSH], Prostate-Specific Antigen/drug effects [MeSH], Prostatic Neoplasms/metabolism [MeSH], Prostatic Neoplasms/drug therapy [MeSH], Prostatic Neoplasms/pathology [MeSH], Androgen Antagonists/therapeutic use [MeSH], Adenocarcinoma/metabolism [MeSH], Male [MeSH], Adenocarcinoma/drug therapy [MeSH], Adenocarcinoma/pathology [MeSH], Edetic Acid/pharmacokinetics [MeSH], Antigens, Surface/genetics [MeSH], Positron Emission Tomography Computed Tomography/methods [MeSH], [, Glutamate Carboxypeptidase II/genetics [MeSH], Prostate cancer, Original Article – Cancer Research, PC-3 Cells [MeSH], Humans [MeSH], VPC-13566, Androstenes/therapeutic use [MeSH], Secretory Pathway/drug effects [MeSH], Prostate-specific membrane antigen, Antigens, Surface/metabolism [MeSH], Androgen Antagonists/pharmacology [MeSH], Androgen antagonist, Gene Expression Regulation, Neoplastic/drug effects [MeSH], Androstenes/pharmacology [MeSH], Oligopeptides/pharmacokinetics [MeSH], Adenocarcinoma/diagnosis [MeSH], Prostate-Specific Antigen/metabolism [MeSH], Prostatic Neoplasms/diagnosis [MeSH], Abiraterone, Prostatic Neoplasms, Androgen Antagonists, Gallium Radioisotopes, Adenocarcinoma, Prostate-Specific Antigen, 3. Good health, Gene Expression Regulation, Neoplastic, 03 medical and health sciences, 0302 clinical medicine, Cell Line, Tumor, Positron Emission Tomography Computed Tomography, Antigens, Surface, PC-3 Cells, Humans, Androstenes, Oligopeptides, Edetic Acid, Gallium Isotopes

Prístupová URL adresa: https://link.springer.com/content/pdf/10.1007/s00432-021-03583-w.pdf
https://pubmed.ncbi.nlm.nih.gov/33760944
https://europepmc.org/article/PMC/PMC8076114
https://www.ncbi.nlm.nih.gov/pubmed/33760944
https://link.springer.com/content/pdf/10.1007/s00432-021-03583-w.pdf
https://link.springer.com/article/10.1007/s00432-021-03583-w
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8076114
https://pubmed.ncbi.nlm.nih.gov/33760944/
https://repository.publisso.de/resource/frl:6449904

Development of Novel Drugs That Target Coactivation Sites of the Androgen Receptor for Treatment of Antiandrogen-Resistant Prostate Cancer

Autori: BRITISH COLUMBIA UNIV VANCOUVER Cherkasov, Artem Rennie, Paul

Zdroj: DTIC

Témy: Biochemistry, Biology, Medicine and Medical Research, Pharmacology, ANDROGENS, DRUGS, PROSTATE CANCER, RESISTANCE(BIOLOGY), ASSAYING, CELLS(BIOLOGY), COMPUTER AIDED DESIGN, GENOMICS, INHIBITION, INHIBITORS, INTERFEROMETRY, MICROSOMES, MODELS, MOLECULES, NEOPLASMS, PROTEINS, RECEPTOR SITES(PHYSIOLOGY), SYNTHESIS(CHEMISTRY), TRANSCRIPTION(GENETICS), AR(ANDROGEN RECEPTORS), CHEMICAL GENOMICS, COMPUTER-AIDED DRUG DESIGN, DRUG RESISTANCE, HORMONE RESISTANCE, SMALL MOLECULE DRUGS, ANTI-ANDROGENS, BF3(BINDING FUNCTION 3), COMPOUND VPC-13566, PCA(PROSTATE CANCER), ENZALUTAMIDE RESISTANT CELLS, LNCAP CELLS, MTS ASSAYS, BLI(BIOLAYER INTERFEROMETRY), XENOGRAFT MODELS, Text

URL: https://apps.dtic.mil/docs/citations/ADA611450