Suchergebnisse - "Receptors, Androgen/genetics [MeSH]"

Targeted Therapy in Salivary Gland Cancer: Prevalence of a Selected Panel of Actionable Molecular Alterations in a German Tertiary Referral Center Patient Cohort

Autoren: Maximilian Linxweiler Silke Wemmert Felix Leon Braun et al.

Quelle: Mol Diagn Ther

Schlagwörter: Male, Adult, Aged, 80 and over, ddc:610, Female [MeSH], Aged, 80 and over [MeSH], Aged [MeSH], Adult [MeSH], Receptor, ErbB-2/metabolism [MeSH], Humans [MeSH], Tertiary Care Centers [MeSH], Salivary Gland Neoplasms/metabolism [MeSH], Receptors, Androgen/metabolism [MeSH], Receptor, ErbB-2/genetics [MeSH], Middle Aged [MeSH], Receptors, Androgen/genetics [MeSH], Germany/epidemiology [MeSH], Immunohistochemistry [MeSH], Molecular Targeted Therapy [MeSH], Male [MeSH], Salivary Gland Neoplasms/genetics [MeSH], Biomarkers, Tumor/genetics [MeSH], Original Research Article, Salivary Gland Neoplasms/drug therapy [MeSH], Salivary Gland Neoplasms/pathology [MeSH], Salivary Gland Neoplasms/epidemiology [MeSH], In Situ Hybridization, Fluorescence [MeSH], Middle Aged, Salivary Gland Neoplasms, Immunohistochemistry, Tertiary Care Centers, Germany, Biomarkers, Tumor, Humans, Female, Molecular Targeted Therapy, In Situ Hybridization, Fluorescence, Aged

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/39485665
https://repository.publisso.de/resource/frl:6498163

Emerging frontiers in androgen receptor research for prostate Cancer: insights from the 2nd international androgen receptor Symposium

Autoren: Israel, JS Marcelin, L Thomas, C et al.

Weitere Verfasser: Israel, JS Marcelin, L Thomas, C et al.

Quelle: J Exp Clin Cancer Res
Journal of Experimental & Clinical Cancer Research, Vol 43, Iss 1, Pp 1-14 (2024)
Journal of experimental & clinical cancer research, 43(1):194

Schlagwörter: AR, Androgen deprivation therapy, Meeting Report, NR3C4, Humans [MeSH], Prostatic Neoplasms/metabolism [MeSH], Receptors, Androgen/metabolism [MeSH], PSMA, Receptors, Androgen/genetics [MeSH], PCa, Prostatic Neoplasms/genetics [MeSH], Male [MeSH], Prostatic Neoplasms/therapy [MeSH], 0301 basic medicine, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, 3. Good health, 03 medical and health sciences, 0302 clinical medicine, Cancers, RC254-282

Dateibeschreibung: application/pdf

Zugangs-URL: https://doaj.org/article/39e0ffdb610b4782878eda81fbb46bd2
http://hdl.handle.net/10138/584594
https://repository.publisso.de/resource/frl:6506879

Transcriptomic response of prostate cancer cells to carbon ion and photon irradiation with focus on androgen receptor and TP53 signaling

Autoren: Jörg Hänze Lilly M. Mengen Marco Mernberger et al.

Weitere Verfasser: Jörg Hänze Lilly M. Mengen Marco Mernberger et al.

Quelle: Radiat Oncol
Radiation Oncology, Vol 19, Iss 1, Pp 1-16 (2024)

Schlagwörter: Male, 0301 basic medicine, DNA Repair, Medizin, R895-920, Heavy Ion Radiotherapy, Photon irradiation, Androgen receptor, TP53, Prostate cancer, Carbon ion irradiation, Medical physics. Medical radiology. Nuclear medicine, 03 medical and health sciences, Cell Line, Tumor, Humans, ddc:610, Cell Line, Tumor [MeSH], DNA Repair [MeSH], Heavy Ion Radiotherapy [MeSH], Humans [MeSH], Prostatic Neoplasms/metabolism [MeSH], Receptors, Androgen/metabolism [MeSH], Transcriptome/radiation effects [MeSH], Signal Transduction/radiation effects [MeSH], Prostatic Neoplasms/pathology [MeSH], Tumor Suppressor Protein p53/metabolism [MeSH], Prostatic Neoplasms/radiotherapy [MeSH], Receptors, Androgen/genetics [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Photons [MeSH], Carbon [MeSH], DNA Damage/radiation effects [MeSH], Male [MeSH], Gene Expression Regulation, Neoplastic/radiation effects [MeSH], Research, RC254-282, Photons, 0303 health sciences, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Prostatic Neoplasms, Medical sciences Medicine, Carbon, 3. Good health, Gene Expression Regulation, Neoplastic, Receptors, Androgen, Tumor Suppressor Protein p53, Transcriptome, DNA Damage, Signal Transduction

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38956684
https://doaj.org/article/fad8adc82b6c4138974260b87cd5e500
https://repository.publisso.de/resource/frl:6503499

The clock gene BHLHE40 and atypical CCNG2 control androgen-induced cellular senescence as a novel tumor suppressive pathway in prostate cancer

Autoren: Mehdi Heidari Horestani Golnaz Atri Roozbahani Aria Baniahmad

Quelle: J Exp Clin Cancer Res
Journal of Experimental & Clinical Cancer Research, Vol 43, Iss 1, Pp 1-19 (2024)

Schlagwörter: Male, 0301 basic medicine, Mice, 03 medical and health sciences, Cell Line, Tumor, Basic Helix-Loop-Helix Transcription Factors, Clock gene, Humans, Animals, RC254-282, Cellular Senescence, Bipolar androgen therapy, Gene Expression Regulation, Neoplastic [MeSH], Cell Line, Tumor [MeSH], Prostatic Neoplasms/metabolism [MeSH], Receptors, Androgen/metabolism [MeSH], Prostatic Neoplasms/drug therapy [MeSH], Prostatic Neoplasms/pathology [MeSH], Androgens/metabolism [MeSH], Receptors, Androgen/genetics [MeSH], Male [MeSH], Xenograft Model Antitumor Assays [MeSH], Basic Helix-Loop-Helix Transcription Factors/metabolism [MeSH], Prostate cancer, Androgens/pharmacology [MeSH], Cellular Senescence [MeSH], Humans [MeSH], Homeodomain Proteins/genetics [MeSH], Androgen-induced cellular senescence, Animals [MeSH], Prostatic Neoplasms/genetics [MeSH], Mice [MeSH], Basic Helix-Loop-Helix Transcription Factors/genetics [MeSH], Homeodomain Proteins/metabolism [MeSH], Research, Androgen receptor, Homeodomain Proteins, 0303 health sciences, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Prostatic Neoplasms, Xenograft Model Antitumor Assays, 3. Good health, Gene Expression Regulation, Neoplastic, Receptors, Androgen, Androgens

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38902772
https://doaj.org/article/1a795d9f667a48d68b5166f2f0ec8f96
https://repository.publisso.de/resource/frl:6497081

Detecting androgen receptor (AR), AR variant 7 (AR-V7), prostate-specific membrane antigen (PSMA), and prostate-specific antigen (PSA) gene expression in CTCs and plasma exosome-derived cfRNA in patients with metastatic castration-resistant prostate cancer (mCRPC) by integrating the VTX-1 CTC isolation system with the QIAGEN AdnaTest

Autoren: Liu, Haiyan E. Vuppalapaty, Meghah Hoerner, Christian R. et al.

Quelle: BMC Cancer
BMC Cancer, Vol 24, Iss 1, Pp 1-14 (2024)

Schlagwörter: Male, 0301 basic medicine, Castration resistance, Exosomes, 03 medical and health sciences, 0302 clinical medicine, PSMA, Androgen Receptor Antagonists, Biomarkers, Tumor, Humans, Protein Isoforms, RNA, Messenger, RC254-282, Prostate cancer, Research, Prostate, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Therapeutic resistance, Prostate-Specific Antigen, Neoplastic Cells, Circulating, 3. Good health, Prostatic Neoplasms, Castration-Resistant, Receptors, Androgen, Androgen receptor inhibitors, AR-V7, Androgen Receptor Antagonists/therapeutic use [MeSH], Prostate-Specific Antigen [MeSH], Receptors, Androgen/metabolism [MeSH], RNA, Messenger/genetics [MeSH], Receptors, Androgen/genetics [MeSH], Male [MeSH], Androgen Receptor Antagonists/pharmacology [MeSH], Prostate/pathology [MeSH], Cell-Free Nucleic Acids/metabolism [MeSH], Circulating tumor cells, Cell-Free Nucleic Acids/genetics [MeSH], Neoplastic Cells, Circulating/pathology [MeSH], Humans [MeSH], Prostatic Neoplasms, Castration-Resistant/drug therapy [MeSH], Exosomes/metabolism [MeSH], Protein Isoforms/genetics [MeSH], Cell-free RNA, Biomarkers, Tumor/genetics [MeSH], Exosomes/genetics [MeSH], Cell-Free Nucleic Acids

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38627648
https://doaj.org/article/f147876cb7db462d84fa5bc412a8c4d4
https://repository.publisso.de/resource/frl:6520065

NanoLuc Binary Technology as a Methodological Approach: An Important New Tool for Studying the Localization of Androgen Receptor and Androgen Receptor Splice Variant V7 Homo- and Heterodimers

Autoren: Juan Guzman Katrin Weigelt Angela Neumann et al.

Quelle: BMC Cancer
BMC Cancer, Vol 24, Iss 1, Pp 1-13 (2024)

Schlagwörter: Male, Prostate cancer, NanoLuc Binary Technology, Research, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Prostatic Neoplasms, Androgen Antagonists, 3. Good health, Androgen receptor, Prostatic Neoplasms, Castration-Resistant, HEK293 Cells, Receptors, Androgen, Prostatic Neoplasms, Castration-Resistant/pathology [MeSH], Humans [MeSH], Homodimer, Receptors, Androgen/metabolism [MeSH], Heterodimer, Prostatic Neoplasms/pathology [MeSH], Androgen Antagonists/pharmacology [MeSH], Receptors, Androgen/genetics [MeSH], Androgen Antagonists/therapeutic use [MeSH], Protein Isoforms/genetics [MeSH], Luciferases [MeSH], Male [MeSH], Androgen receptor variant 7, HEK293 Cells [MeSH], Androgens [MeSH], Androgens, Humans, Protein Isoforms, Luciferases, RC254-282

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38500100
https://doaj.org/article/540315149f484863b24ccb2561a69445
https://repository.publisso.de/resource/frl:6492857