Search Results - "Prostatic Neoplasms metabolism"

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    Source: Mol Cancer
    Molecular Cancer, Vol 24, Iss 1, Pp 1-26 (2025)

    File Description: application/pdf; application/zip; text/xml

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    Source: Mol Imaging Biol
    Mamlins, Eduards; Scharbert, Lara; Cardinale, Jens; Krotov, Maria; Winter, Erik; Rathke, Hendrik; Strodel, Birgit; Ankrah, Alfred O; Sathekge, Mike; Haberkorn, Uwe; Kratochwil, Clemens; Giesel, Frederik L (2024). The Theranostic Optimization of PSMA-GCK01 Does Not Compromise the Imaging Characteristics of [99mTc]Tc-PSMA-GCK01 Compared to Dedicated Diagnostic [99mTc]Tc-EDDA/HYNIC-iPSMA in Prostate Cancer. Molecular imaging and biology, 26(1), pp. 81-89. Springer-Verlag 10.1007/s11307-023-01881-y <http://dx.doi.org/10.1007/s11307-023-01881-y>
    Molecular imaging & biology 26(1), 81-89 (2024). doi:10.1007/s11307-023-01881-y

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    Source: J Exp Clin Cancer Res
    Journal of Experimental & Clinical Cancer Research, Vol 43, Iss 1, Pp 1-21 (2024)

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    Source: Mol Cancer
    Molecular Cancer, Vol 23, Iss 1, Pp 1-22 (2024)
    Molecular Cancer

    Subject Terms: Male, STAT3 Transcription Factor, Cytotoxic T-Cells, IL6ST/STAT3 Signaling, Senescence, Gene Expression Regulation, Neoplastic [MeSH], Immune cell infiltration, Cell Line, Tumor [MeSH], STAT3 Transcription Factor/metabolism [MeSH], Cytotoxic T-cells, Prostatic Neoplasms/metabolism [MeSH], IL6ST/STAT3 signaling, Prostatic Neoplasms/pathology [MeSH], Tumor Suppressor Protein p53/metabolism [MeSH], Tumor microenvironment, Senescence-associated secretory phenotype, Male [MeSH], Disease Models, Animal [MeSH], Prostate cancer, Cyclin-Dependent Kinase Inhibitor p16/metabolism [MeSH], Cellular Senescence [MeSH], Humans [MeSH], Animals [MeSH], Tumor Suppressor Protein p53/genetics [MeSH], Cyclin-Dependent Kinase Inhibitor p16/genetics [MeSH], Prostatic Neoplasms/genetics [MeSH], Mice [MeSH], L-gp130, Research, Signal Transduction [MeSH], Tumor Microenvironment [MeSH], Mice, SDG 3 - Good Health and Well-being, Cell Line, Tumor, Tumor Microenvironment, Animals, Humans, 302055 Oncology, RC254-282, Cellular Senescence, Cyclin-Dependent Kinase Inhibitor p16, Cancer och onkologi, Immune Cell Infiltration, Prostate Cancer, 302055 Onkologie, 106018 Human biology, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Prostatic Neoplasms, 106018 Humanbiologie, Gene Expression Regulation, Neoplastic, Disease Models, Animal, Cancer and Oncology, SDG 3 – Gesundheit und Wohlergehen, Senescence-associated Secretory Phenotype, Tumor Suppressor Protein p53, Signal Transduction

    File Description: application/pdf

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    Source: Radiat Oncol
    Radiation Oncology, Vol 19, Iss 1, Pp 1-11 (2024)

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    Source: Radiat Oncol
    Radiation Oncology, Vol 19, Iss 1, Pp 1-16 (2024)

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    Source: J Exp Clin Cancer Res
    Journal of Experimental & Clinical Cancer Research, Vol 43, Iss 1, Pp 1-19 (2024)

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    Source: Mol Cancer
    Molecular Cancer, Vol 23, Iss 1, Pp 1-21 (2024)
    Molecular Cancer

    Subject Terms: Male, 0301 basic medicine, Cancer Research, Prostatic Neoplasmspathologygeneticsmetabolism, Animals, Mice, Humans, PTEN Phosphohydrolasegeneticsmetabolism, Disease Progression, Tumor Microenvironmentimmunology, Senescence-Associated Secretory Phenotype, Proto-Oncogene Proteins c-junmetabolism, Gene Expression Regulation, Neoplastic, Cell Line, Tumor, Gene Expression Profiling, Cellular Senescencegenetics, Disease Models, Animal, Proto-Oncogene Proteins c-jun, AP-1 transcription factors, Senescence, SASP, Gene Expression Regulation, Neoplastic [MeSH], Cell Line, Tumor [MeSH], Disease Progression [MeSH], Immune infiltration, Prostatic Neoplasms/metabolism [MeSH], Prostatic Neoplasms/pathology [MeSH], Tumor Microenvironment/immunology [MeSH], Male [MeSH], Cellular Senescence/genetics [MeSH], Disease Models, Animal [MeSH], Prostate cancer, Humans [MeSH], PTEN Phosphohydrolase/genetics [MeSH], Animals [MeSH], Prostatic Neoplasms/genetics [MeSH], PTEN Phosphohydrolase/metabolism [MeSH], Proto-Oncogene Proteins c-jun/metabolism [MeSH], Mice [MeSH], Research, JUN, Gene Expression Profiling [MeSH], Senescence-Associated Secretory Phenotype [MeSH], 03 medical and health sciences, 106023 Molekularbiologie, SDG 3 - Good Health and Well-being, Tumor Microenvironment, 302055 Oncology, RC254-282, Cellular Senescence, Cancer och onkologi, 0303 health sciences, 302055 Onkologie, PTEN Phosphohydrolase, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Prostatic Neoplasms, 106023 Molecular biology, 3. Good health, Cancer and Oncology, SDG 3 – Gesundheit und Wohlergehen, Integrative Biomedicine [Topic 3]

    File Description: application/pdf

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    Contributors: Emma Scott Kirsty Hodgson Beatriz Calle et al.

    Source: Oncogene
    Zaguán. Repositorio Digital de la Universidad de Zaragoza
    Universidad de Zaragoza
    Scott, E, Hodgson, K, Calle, B, Turner, H, Cheung, K, Bermudez, A, Marques, F J G, Pye, H, Yo, E C, Islam, K, Oo, H Z, McClurg, U L, Wilson, L, Thomas, H, Frame, F M, Orozco-Moreno, M, Bastian, K, Arredondo, H M, Roustan, C, Gray, M A, Kelly, L, Tolson, A, Mellor, E, Hysenaj, G, Goode, E A, Garnham, R, Duxfield, A, Heavey, S, Stopka-Farooqui, U, Haider, A, Freeman, A, Singh, S, Johnston, E W, Punwani, S, Knight, B, McCullagh, P, McGrath, J, Crundwell, M, Harries, L, Bogdan, D, Westaby, D, Fowler, G, Flohr, P, Yuan, W, Sharp, A, de Bono, J, Maitland, N J, Wisnovsky, S, Bertozzi, C R, Heer, R, Guerrero, R H, Daugaard, M, Leivo, J, Whitaker, H, Pitteri, S, Wang, N, Elliott, D J, Schumann, B & Munkley, J 2023, ' Upregulation of GALNT7 in prostate cancer modifies O-glycosylation and promotes tumour growth ', Oncogene, vol. 42, pp. 926–937 . https://doi.org/10.1038/s41388-023-02604-x

    File Description: application/pdf; Print-Electronic

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    Source: J Transl Med
    Journal of Translational Medicine, Vol 22, Iss 1, Pp 1-8 (2024)

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