Výsledky vyhľadávania - "Myocardial Reperfusion Injury/metabolism [MeSH]"

Mitochondrial calcium in cardiac ischemia/reperfusion injury and cardioprotection

Autori: Edoardo Bertero Tudor-Alexandru Popoiu Christoph Maack

Zdroj: Basic Res Cardiol

Predmety: 0301 basic medicine, ddc:610, 0303 health sciences, Cardiotonic Agents, Mitochondrial Permeability Transition Pore, Cardiac myocytes, Mitochondria, Mitochondrial Membrane Transport Proteins/metabolism [MeSH], Myocardial Reperfusion Injury/pathology [MeSH], Calcium/metabolism [MeSH], Mitochondria, Heart/pathology [MeSH], Myocardial Reperfusion Injury/metabolism [MeSH], Humans [MeSH], Mitochondrial Permeability Transition Pore/metabolism [MeSH], Animals [MeSH], Mitochondria at the Heart of Cardioprotection, Calcium Channels/metabolism [MeSH], Mitochondria, Heart/metabolism [MeSH], Reactive oxygen species, Myocardial Reperfusion Injury/prevention, Calcium Signaling [MeSH], Calcium handling, Cardiotonic Agents/metabolism [MeSH], Myocardial infarction, Ischemia/reperfusion injury, Myocardial Reperfusion Injury, Mitochondrial Membrane Transport Proteins, Mitochondria, Heart, 03 medical and health sciences, Humans, Animals, Calcium, Calcium Signaling, Calcium Channels

Popis súboru: application/pdf

Prístupová URL adresa: https://pubmed.ncbi.nlm.nih.gov/38890208
https://repository.publisso.de/resource/frl:6500976
https://opus.bibliothek.uni-wuerzburg.de/frontdoor/index/index/docId/39277
https://doi.org/10.1007/s00395-024-01060-2
https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-392777
https://opus.bibliothek.uni-wuerzburg.de/files/39277/00395_2024_Article_1060.pdf

Ischemia does not provoke the full immune training repertoire in human cardiac fibroblasts

Autori: Constantin Mann Carolin van Alst Simone Gorressen a ďalší

Zdroj: Naunyn-Schmiedeberg's Archives of Pharmacology
Naunyn Schmiedebergs Arch Pharmacol

Predmety: Male, 0301 basic medicine, 0303 health sciences, L-Lactate Dehydrogenase, Research, Myocardium, TOR Serine-Threonine Kinases, Medizin, Myocardial Reperfusion Injury, Fibroblasts, Hypoxia-Inducible Factor 1, alpha Subunit, Mice, Inbred C57BL, 03 medical and health sciences, Humans, Animals, Cytokines, L-Lactate Dehydrogenase/metabolism [MeSH], Mice, Inbred C57BL [MeSH], Glycolysis, Myocardial Reperfusion Injury/metabolism [MeSH], Fibroblasts/drug effects [MeSH], Humans [MeSH], Cytokines/metabolism [MeSH], Animals [MeSH], Inflammasome, Ischemia, Male [MeSH], Cardiac fibroblasts, Hypoxia-Inducible Factor 1, alpha Subunit/metabolism [MeSH], Cells, Cultured [MeSH], Myocardial Reperfusion Injury/immunology [MeSH], TOR Serine-Threonine Kinases/metabolism [MeSH], Myocardium/immunology [MeSH], Myocardial, Fibroblasts/metabolism [MeSH], Trained immunity, Myocardium/metabolism [MeSH], Cells, Cultured

Prístupová URL adresa: https://pubmed.ncbi.nlm.nih.gov/38652279
https://repository.publisso.de/resource/frl:6493326

Insulin and glycolysis dependency of cardioprotection by nicotinamide riboside

Autori: Y. Xiao Q. Wang H. Zhang a ďalší

Zdroj: Basic Res Cardiol

Predmety: Niacinamide, 0301 basic medicine, 0303 health sciences, Cardiotonic Agents, Myocardium, Citric Acid Cycle, Isolated Heart Preparation, Myocardial Reperfusion Injury, Pyridinium Compounds, Original Contribution, PPP pathway, NAD, IR injury, Mice, Inbred C57BL, Mice, Disease Models, Animal, 03 medical and health sciences, Citric Acid Cycle/drug effects [MeSH], Glycolysis/drug effects [MeSH], Mice, Inbred C57BL [MeSH], Glycolysis, Myocardial Reperfusion Injury/metabolism [MeSH], Insulin, Niacinamide/pharmacology [MeSH], NAD/metabolism [MeSH], Animals [MeSH], Metabolomics [MeSH], Mice [MeSH], Myocardial Reperfusion Injury/prevention, Pyridinium Compounds/pharmacology [MeSH], Niacinamide/analogs, Cardiotonic Agents/pharmacology [MeSH], Disease Models, Animal [MeSH], Insulin/metabolism [MeSH], Isolated Heart Preparation [MeSH], Myocardium/metabolism [MeSH], Animals, Metabolomics

Prístupová URL adresa: https://pubmed.ncbi.nlm.nih.gov/38528175
https://pure.amsterdamumc.nl/en/publications/13aacc7c-b3f0-457f-830c-a629e56b5040
https://doi.org/10.1007/s00395-024-01042-4
https://repository.publisso.de/resource/frl:6507194

The Melatonin Receptor Agonist Ramelteon Induces Cardioprotection that Requires MT2 Receptor Activation and Release of Reactive Oxygen Species

Autori: Stroethoff, Martin Goetze, Lukas Torregroza, Carolin a ďalší

Zdroj: Cardiovasc Drugs Ther

Predmety: Male, 0301 basic medicine, Myocardial Infarction, Myocardial Reperfusion Injury, Cardioprotection, Mitochondrial Membrane Transport Proteins, Mitochondria, Heart, Ventricular Function, Left, 03 medical and health sciences, Myocardial Reperfusion Injury/physiopathology [MeSH], Pharmacology, Myocardial Infarction/pathology [MeSH], Indenes/pharmacology [MeSH], Myocardial Infarction/metabolism [MeSH], Original Article, Mitochondria, Heart/metabolism [MeSH], Male [MeSH], Myocardial Infarction/physiopathology [MeSH], Receptor, Melatonin, MT2/metabolism [MeSH], Myocytes, Cardiac/metabolism [MeSH], Myocytes, Cardiac/drug effects [MeSH], Disease Models, Animal [MeSH], Receptor, Melatonin, MT2/agonists [MeSH], Mitochondrial Membrane Transport Proteins/metabolism [MeSH], Myocardial Reperfusion Injury/pathology [MeSH], Mitochondria, Heart/pathology [MeSH], Myocardial Infarction/prevention, Myocardial Reperfusion Injury/metabolism [MeSH], Reactive Oxygen Species/metabolism [MeSH], Mitochondria, Heart/drug effects [MeSH], Animals [MeSH], Myocytes, Cardiac/pathology [MeSH], Rats, Wistar [MeSH], Myocardial Reperfusion Injury/prevention, Mitochondrial Permeability Transition Pore [MeSH], Ventricular Function, Left/drug effects [MeSH], Signal Transduction [MeSH], Melatonin receptor agonist, Cardiovascular Agents/pharmacology [MeSH], Isolated Heart Preparation [MeSH], Animals, Myocytes, Cardiac, Rats, Wistar, 0303 health sciences, Mitochondrial Permeability Transition Pore, Receptor, Melatonin, MT2, Cardiovascular Agents, Isolated Heart Preparation, 3. Good health, Disease Models, Animal, Indenes, Reactive Oxygen Species, Signal Transduction

Prístupová URL adresa: https://link.springer.com/content/pdf/10.1007/s10557-020-06972-4.pdf
https://pubmed.ncbi.nlm.nih.gov/32236860
https://www.scilit.net/article/5cf36ecad0de0bd79b64eab2d29c7fd2
https://www.narcis.nl/publication/RecordID/oai%3Apure.amc.nl%3Apublications%2Fb5eab9c6-3977-45a1-b100-bfb45916dddb
https://link.springer.com/content/pdf/10.1007/s10557-020-06972-4.pdf
https://link.springer.com/article/10.1007/s10557-020-06972-4
https://pubmed.ncbi.nlm.nih.gov/32236860/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7242242
https://repository.publisso.de/resource/frl:6470798

Cardioprotection by post-conditioning with exogenous triiodothyronine in isolated perfused rat hearts and isolated adult rat cardiomyocytes

Autori: Helmut Raphael Lieder Felix Braczko Nilgün Gedik a ďalší

Zdroj: Basic Res Cardiol

Predmety: Male, Mitogen-Activated Protein Kinase 1, 0301 basic medicine, Mitogen-Activated Protein Kinase 3, Medizin, Myocardial Infarction, Myocardial Reperfusion Injury/physiopathology [MeSH], Phosphorylation [MeSH], Original Contribution, Triiodothyronine, Myocardial Infarction/pathology [MeSH], Post-conditioning, Mitogen-Activated Protein Kinase 3/metabolism [MeSH], Myocardial Infarction/metabolism [MeSH], Mitochondria, Heart/metabolism [MeSH], Male [MeSH], Myocardial Infarction/physiopathology [MeSH], Myocytes, Cardiac/metabolism [MeSH], Ischemia/reperfusion, Myocytes, Cardiac/drug effects [MeSH], Triiodothyronine/pharmacology [MeSH], Disease Models, Animal [MeSH], Ischemic conditioning, Cell Hypoxia [MeSH], Myocardial Reperfusion Injury/pathology [MeSH], Mitochondria, Heart/pathology [MeSH], Myocardial Infarction/prevention, Myocardial Reperfusion Injury/metabolism [MeSH], Proto-Oncogene Proteins c-akt [MeSH], Cardioprotection, Mitochondria, Heart/drug effects [MeSH], Animals [MeSH], Myocytes, Cardiac/pathology [MeSH], Myocardial Reperfusion Injury/prevention, Rats, Inbred Lew [MeSH], Ventricular Function, Left/drug effects [MeSH], Signal Transduction [MeSH], Mitogen-Activated Protein Kinase 1/metabolism [MeSH], Isolated Heart Preparation [MeSH], Isolated Heart Preparation, Myocardial Reperfusion Injury, Cell Hypoxia, Mitochondria, Heart, Ventricular Function, Left, Rats, Disease Models, Animal, 03 medical and health sciences, 0302 clinical medicine, Rats, Inbred Lew, Animals, Myocytes, Cardiac, Phosphorylation, Proto-Oncogene Proteins c-akt, Signal Transduction

Prístupová URL adresa: https://link.springer.com/content/pdf/10.1007/s00395-021-00868-6.pdf
https://pubmed.ncbi.nlm.nih.gov/33876304
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8055637
https://pubmed.ncbi.nlm.nih.gov/33876304/
https://link.springer.com/content/pdf/10.1007/s00395-021-00868-6.pdf
https://link.springer.com/article/10.1007/s00395-021-00868-6
https://europepmc.org/article/PMC/PMC8055637
https://repository.publisso.de/resource/frl:6450756

Neuronal guidance proteins in cardiovascular inflammation

Autori: Marius Keller Valbona Mirakaj Michael Koeppen a ďalší

Zdroj: Basic Res Cardiol

Predmety: Inflammation, 2. Zero hunger, 0301 basic medicine, 0303 health sciences, Myocardial Reperfusion Injury/pathology [MeSH], Cardiovascular inflammation, Intercellular Signaling Peptides and Proteins/metabolism [MeSH], Thrombosis/pathology [MeSH], Thrombosis/metabolism [MeSH], Myocardial Reperfusion Injury/metabolism [MeSH], Humans [MeSH], Nerve Tissue Proteins/metabolism [MeSH], Atherosclerosis, Inflammation Mediators/metabolism [MeSH], Animals [MeSH], Neuronal guidance proteins, Atherosclerosis/pathology [MeSH], Myocardium/pathology [MeSH], Inflammation/pathology [MeSH], Plaque, Atherosclerotic [MeSH], Inflammation/metabolism [MeSH], Signal Transduction [MeSH], Review, Atherosclerosis/metabolism [MeSH], Myocardial infarction, Axon Guidance [MeSH], Myocardium/metabolism [MeSH], Myocardium, Myocardial Reperfusion Injury, Nerve Tissue Proteins, Thrombosis, Plaque, Atherosclerotic, Axon Guidance, 3. Good health, 03 medical and health sciences, Animals, Humans, Intercellular Signaling Peptides and Proteins, Inflammation Mediators, Signal Transduction

Prístupová URL adresa: https://link.springer.com/content/pdf/10.1007/s00395-021-00847-x.pdf
https://pubmed.ncbi.nlm.nih.gov/33511463
https://publikationen.uni-tuebingen.de/xmlui/handle/10900/118953
https://europepmc.org/article/PMC/PMC7843241
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7843241
https://link.springer.com/article/10.1007/s00395-021-00847-x
https://bibliographie.uni-tuebingen.de/xmlui/handle/10900/118953
https://link.springer.com/article/10.1007/s00395-021-00847-x?error=cookies_not_supported&error=cookies_not_supported&error=cookies_not_supported&error=cookies_not_supported&code=e72f2ff0-809b-44f2-ba22-775677d8a07e&code=93a94b4d-a4e8-4975-8a63-3283a42dca69&code=8fcb2ab1-bae3-4876-8915-1eb907267f79&code=2bb143b6-f6d5-4bfe-b382-5e2bb3db3c51
https://repository.publisso.de/resource/frl:6450772