Suchergebnisse - "Inflammation chemically induced"
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1
Autoren: et al.
Quelle: Inflammation
Schlagwörter: Inflammation, 0301 basic medicine, Mice, Inbred BALB C, 0303 health sciences, Research, Filaggrin Proteins, Dermatitis, Atopic/chemically induced [MeSH], 2,4-dinitrochlorobenzene, Dermatitis, Atopic/pathology [MeSH], Female [MeSH], Filaggrin Proteins [MeSH], Mice, Inbred BALB C [MeSH], Inflammation/chemically induced [MeSH], atopic dermatitis mouse model, Skin/drug effects [MeSH], sub-acute and chronic inflammation, Cytokines/metabolism [MeSH], Animals [MeSH], Skin/pathology [MeSH], Dermatitis, Atopic/metabolism [MeSH], Mice [MeSH], Dinitrochlorobenzene/toxicity [MeSH], Inflammation/metabolism [MeSH], Disease Models, Animal [MeSH], mild and moderate atopic dermatitis, Dexamethasone/pharmacology [MeSH], Skin/metabolism [MeSH], Dexamethasone, Dermatitis, Atopic, Mice, Disease Models, Animal, 03 medical and health sciences, Dinitrochlorobenzene, Animals, Cytokines, Female, Skin
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2
Autoren: et al.
Weitere Verfasser: et al.
Quelle: ISSN: 0197-0186.
Schlagwörter: MESH: Animals, MESH: Disease Models, MESH: Signal Transduction* / drug effects, MESH: Signal Transduction* / physiology, MESH: Hippocampus* / drug effects, MESH: Hippocampus* / metabolism, MESH: Inflammation / chemically induced, MESH: Inflammation / metabolism, MESH: Lipopolysaccharides* / toxicity, MESH: Male, MESH: Mice, Inbred C57BL, MESH: Mitochondria* / drug effects, MESH: Mitochondria* / metabolism, MESH: Mitochondrial Dynamics* / physiology, MESH: Mitochondrial Proteins* / metabolism, MESH: Neuroinflammatory Diseases* / chemically induced, MESH: Neuroinflammatory Diseases* / metabolism, MESH: STAT3 Transcription Factor* / metabolism, [SDV]Life Sciences [q-bio], [SDV.BC]Life Sciences [q-bio]/Cellular Biology, [SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology
Relation: info:eu-repo/semantics/altIdentifier/pmid/40209854; PUBMED: 40209854
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3
Autoren: et al.
Weitere Verfasser: et al.
Quelle: International journal of cardiology, vol. 382, pp. 76-82
Denegri, Andrea; Magnani, Giulia; Kraler, Simon; Bruno, Francesco; Klingenberg, Roland; Mach, Francois; Gencer, Baris; Räber, Lorenz; Rodondi, Nicolas; Rossi, Valentina A; Matter, Christian M; Nanchen, David; Obeid, Slayman; Lüscher, Thomas F (2023). History of peripheral artery disease and cardiovascular risk of real-word patients with acute coronary syndrome: Role of inflammation and comorbidities. International journal of cardiology, 382, pp. 76-82. Elsevier 10.1016/j.ijcard.2023.03.043 <http://dx.doi.org/10.1016/j.ijcard.2023.03.043>Schlagwörter: Inflammation, 610 Medicine & health, Hemorrhage, 2705 Cardiology and Cardiovascular Medicine, 3. Good health, Humans, Acute Coronary Syndrome/diagnosis, Acute Coronary Syndrome/epidemiology, Acute Coronary Syndrome/chemically induced, Cardiovascular Diseases/chemically induced, Platelet Aggregation Inhibitors/adverse effects, Prospective Studies, Risk Factors, Hemorrhage/diagnosis, Hemorrhage/epidemiology, Hemorrhage/chemically induced, Inflammation/diagnosis, Inflammation/epidemiology, Inflammation/chemically induced, Peripheral Arterial Disease/diagnosis, Peripheral Arterial Disease/epidemiology, Heart Disease Risk Factors, Acute coronary syndrome, Peripheral artery disease, Personalized therapy, Residual risk, Risk stratification, Peripheral Arterial Disease, 360 Social problems & social services, Cardiovascular Diseases, 10209 Clinic for Cardiology, Acute Coronary Syndrome, Platelet Aggregation Inhibitors
Dateibeschreibung: application/pdf; ZORA257558.pdf - application/pdf
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4
Autoren: et al.
Quelle: J Neuroinflammation
Journal of Neuroinflammation, Vol 21, Iss 1, Pp 1-24 (2024)
Journal of neuroinflammation 21(1), 174 (2024). doi:10.1186/s12974-024-03164-xSchlagwörter: Lipopolysaccharides, Neuroinflammatory Diseases/metabolism [MeSH], Mice, Inbred C57BL [MeSH], Neuroinflammatory Diseases/genetics [MeSH], Inflammation/chemically induced [MeSH], Neuroinflammation, Microglia/drug effects [MeSH], Neuroinflammatory Diseases/chemically induced [MeSH], Lipopolysaccharides/pharmacology [MeSH], Neuroinflammatory Diseases/pathology [MeSH], Lipopolysaccharides/toxicity [MeSH], Microglia/metabolism [MeSH], Inflammation/genetics [MeSH], Lipopolysaccharide, Protein Deglycase DJ-1/deficiency [MeSH], Parkinson's disease, Humans [MeSH], Microglia, Animals [MeSH], Mice, Knockout [MeSH], Mice [MeSH], Microglia/pathology [MeSH], Microglia morphology, Inflammation/pathology [MeSH], Protein Deglycase DJ-1/genetics [MeSH], Research, Inflammation/metabolism [MeSH], Protein Deglycase DJ-1/metabolism [MeSH], PARK7/DJ-1, Protein Deglycase DJ-1, genetics [Protein Deglycase DJ-1], metabolism [Microglia], drug effects [Microglia], genetics [Inflammation], Mice, toxicity [Lipopolysaccharides], Animals, Humans, deficiency [Protein Deglycase DJ-1], ddc:610, chemically induced [Inflammation], RC346-429, pathology [Inflammation], Mice, Knockout, Inflammation, metabolism [Inflammation], PARK7 protein, mouse, pathology [Neuroinflammatory Diseases], genetics [Neuroinflammatory Diseases], pathology [Microglia], ddc, metabolism [Protein Deglycase DJ-1], pharmacology [Lipopolysaccharides], Mice, Inbred C57BL, metabolism [Neuroinflammatory Diseases], chemically induced [Neuroinflammatory Diseases], Neuroinflammatory Diseases, Neurology. Diseases of the nervous system
Dateibeschreibung: application/pdf
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5
Autoren: et al.
Quelle: Biochemical and Biophysical Research Communications
Schlagwörter: Dermatitis, Atopic* / chemically induced, Inflammation, 0301 basic medicine, 0303 health sciences, CD4 T cell +, Sensitization, Dermatitis, Atopic, 3. Good health, Disease Models, Animal, Mice, 03 medical and health sciences, Th2 Cells, IL-33, MC903, Animals, Cytokines, Challenge, Inflammation / chemically induced, Atopic dermatitis, Dermatitis, Atopic* / drug therapy, Skin
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6
Autoren: et al.
Quelle: Toxins (Basel)
Toxins, Vol 17, Iss 3, p 140 (2025)Schlagwörter: Male, Nociception, Synaptosomal-Associated Protein 25, Pain, Botulinum Toxins, Type A / therapeutic use, Carrageenan, Botulinum Toxins, Type A / pharmacology, Spinal Cord / drug effects, Article, Proto-Oncogene Proteins c-fos / metabolism, Rats, Sprague-Dawley, Ganglia, Spinal, Inflammation / drug therapy, Animals, neutralizing antitoxin, Botulinum Toxins, Type A, Rats, Wistar, Inflammation, Analgesics, carrageenan-induced bilateral inflammatory pain, c-Fos immunohistochemistry, Analgesics / therapeutic use, synaptosomal-associated protein 25, Synaptosomal-Associated Protein 25 / metabolism, trans-synaptic transport, Nociception / drug effects, botulinum toxin type A, Rats, Spinal Cord, Analgesics / pharmacology, Medicine, Inflammation / chemically induced, Spinal Cord / metabolism, Proto-Oncogene Proteins c-fos
Dateibeschreibung: application/pdf
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7
Autoren: et al.
Weitere Verfasser: et al.
Quelle: ISSN: 1661-6596.
Schlagwörter: DON, histology, immunity-related genes, mycotoxins, toxicity, MESH: Animal Feed, MESH: Animals, MESH: Spleen / pathology, MESH: Swine, MESH: Trichothecenes* / toxicity, MESH: Substances, MESH: European Union, MESH: Food Contamination / analysis, MESH: Inflammation* / chemically induced, MESH: Liver* / drug effects, MESH: Liver* / metabolism, MESH: Liver* / pathology, MESH: Spleen / drug effects, MESH: Spleen / metabolism, [SDV.TOX]Life Sciences [q-bio]/Toxicology
Relation: info:eu-repo/semantics/altIdentifier/pmid/39337277; PUBMED: 39337277
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8
Autoren: et al.
Quelle: Ecotoxicology and Environmental Safety, 272, 116111 (2024-03-01)
Schlagwörter: Chicken embryos, Immune challenge, In ovo feeding, Lipopolysaccharide, Zinc, NF-kappa B, Lipopolysaccharides, Toll-Like Receptor 4, zinc glycinate, Saline Solution, Glycine, Chick Embryo, Animals, Toll-Like Receptor 4/metabolism, Chickens/metabolism, Saline Solution/toxicity, Inflammation/chemically induced, Inflammation/veterinary, Homeostasis, Zinc/toxicity, Glycine/analogs & derivatives, NF-kappa B/genetics, NF-kappa B/metabolism, Lipopolysaccharides/toxicity, Health, Toxicology and Mutagenesis, Public Health, Environmental and Occupational Health, Pollution, General Medicine
Relation: https://api.elsevier.com/content/article/PII:S0147651324001866?httpAccept=text/xml; urn:issn:0147-6513; urn:issn:1090-2414; https://orbi.uliege.be/handle/2268/316290; info:hdl:2268/316290; info:pmid:38350216
Verfügbarkeit: https://orbi.uliege.be/handle/2268/316290
https://orbi.uliege.be/bitstream/2268/316290/1/published%20version_Zinc%20glycinate%20alleviates%20LPS-induced%20inflammation%20and%20intestinal%20barrier.pdf
https://doi.org/10.1016/j.ecoenv.2024.116111 -
9
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Neurochem Res
Schlagwörter: MESH: Inflammation, 6-Mercaptopurine, Lipopolysaccharides, 0301 basic medicine, MESH: Gene Expression, MESH: Rats, Interleukin-1beta/genetics [MeSH], Tumor Necrosis Factor-alpha/genetics [MeSH], Enteric Nervous System/cytology [MeSH], Inflammation/chemically induced [MeSH], Rats [MeSH], Lipopolysaccharides [MeSH], Animals [MeSH], Gene Expression/drug effects [MeSH], Inflammatory bowel diseases, Neurons/drug effects [MeSH], CXC motif ligand chemokines, Interleukin-1beta/pharmacology [MeSH], Mercaptopurine/pharmacology [MeSH], Mice [MeSH], Anti-Inflammatory Agents/pharmacology [MeSH], Original Paper, Cells, Cultured [MeSH], Tumor Necrosis Factor-alpha/metabolism [MeSH], Enteric glial cells, Interleukin-1beta/metabolism [MeSH], Enteric nervous system, Tumor Necrosis Factor-alpha/pharmacology [MeSH], [SDV]Life Sciences [q-bio], Interleukin-1beta, MESH: Neurons, Anti-Inflammatory Agents, Gene Expression, Enteric Nervous System, Mice, 03 medical and health sciences, MESH: Interleukin-1beta, Animals, MESH: Animals, MESH: Mice, Cells, Cultured, Inflammation, Neurons, MESH: Cells, 0303 health sciences, Cultured, MESH: Enteric Nervous System, Mercaptopurine, Tumor Necrosis Factor-alpha, Rats, 3. Good health, [SDV] Life Sciences [q-bio], MESH: Anti-Inflammatory Agents, MESH: Tumor Necrosis Factor-alpha, MESH: Mercaptopurine, MESH: Lipopolysaccharides
Dateibeschreibung: application/pdf
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s11064-021-03324-y.pdf
https://pubmed.ncbi.nlm.nih.gov/33864170
https://www.ncbi.nlm.nih.gov/pubmed/33864170
https://link.springer.com/article/10.1007/s11064-021-03324-y
https://europepmc.org/article/PMC/PMC8187225
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8187225
https://link.springer.com/content/pdf/10.1007/s11064-021-03324-y.pdf
https://hal.science/hal-04178448v1
https://hal.science/hal-04178448v1/document
https://doi.org/10.1007/s11064-021-03324-y
https://repository.publisso.de/resource/frl:6445958 -
10
Autoren: et al.
Quelle: J Cancer Res Clin Oncol
Schlagwörter: Adult, Male, Uveal Neoplasms, 0301 basic medicine, Skin Neoplasms, Autoimmunity, Endocrine System Diseases, Cohort Studies, 03 medical and health sciences, Melanoma, Cutaneous Malignant, 0302 clinical medicine, Uveal Melanoma, Germany, Biomarkers, Tumor, Humans, ddc:610, Neoplasm Metastasis, 10. No inequality, Immune Checkpoint Inhibitors, Melanoma, Inflammation, 3. Good health, Inflammation/blood [MeSH], Gene Expression Regulation, Neoplastic/immunology [MeSH], Inflammation/chemically induced [MeSH], Skin Neoplasms/blood [MeSH], Skin Neoplasms/drug therapy [MeSH], Cohort Studies [MeSH], Uveal Neoplasms/blood [MeSH], Autoimmunity/genetics [MeSH], Inflammation/genetics [MeSH], Male [MeSH], Immune Checkpoint Inhibitors/adverse effects [MeSH], Melanoma/pathology [MeSH], Case-Control Studies [MeSH], Autoimmunity/drug effects [MeSH], Uveal Neoplasms/pathology [MeSH], Diabetes Mellitus, Type 1/blood [MeSH], Melanoma/drug therapy [MeSH], Inflammation/immunology [MeSH], Female [MeSH], Original Article – Cancer Research, Melanoma/genetics [MeSH], Adult [MeSH], Diabetes Mellitus, Type 1/chemically induced [MeSH], Humans [MeSH], Neoplasm Metastasis [MeSH], Uveal Neoplasms/drug therapy [MeSH], Melanoma/blood [MeSH], Uveal Neoplasms/genetics [MeSH], Skin Neoplasms/pathology [MeSH], Immune checkpoint inhibitors, Immune Checkpoint Inhibitors/therapeutic use [MeSH], Cytokines, Gene Expression Regulation, Neoplastic/drug effects [MeSH], Autoimmunity/immunology [MeSH], Skin Neoplasms/genetics [MeSH], Endocrine System Diseases/blood [MeSH], Biomarkers, Tumor/blood [MeSH], Biomarkers, Tumor/genetics [MeSH], Germany [MeSH], Endocrine System Diseases/immunology [MeSH], Diabetes Mellitus, Type 1/immunology [MeSH], Immune-related adverse events, Endocrine System Diseases/chemically induced [MeSH], Immune Checkpoint Inhibitors/pharmacology [MeSH], Gene Expression Regulation, Neoplastic, Diabetes Mellitus, Type 1, Case-Control Studies, Female
Dateibeschreibung: application/pdf
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00432-021-03550-5.pdf
https://pubmed.ncbi.nlm.nih.gov/33837821
https://link.springer.com/content/pdf/10.1007/s00432-021-03550-5.pdf
https://www.ncbi.nlm.nih.gov/pubmed/33837821
https://europepmc.org/article/PMC/PMC8076116
https://link.springer.com/article/10.1007/s00432-021-03550-5
https://pubmed.ncbi.nlm.nih.gov/33837821/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8076116
https://opus4.kobv.de/opus4-fau/frontdoor/index/index/docId/22122
https://repository.publisso.de/resource/frl:6449924 -
11
Autoren: et al.
Quelle: J Mol Med (Berl)
Schlagwörter: Inflammation, Male, 0301 basic medicine, Nicotine, ddc:610, 0303 health sciences, Smoking, Retinal Pigment Epithelium, Tobacco Products, Electronic Nicotine Delivery Systems, Retina, 3. Good health, Mice, Inbred C57BL, Smoke/adverse effects [MeSH], E-Cigarette Vapor/adverse effects [MeSH], Nicotine/adverse effects [MeSH], Mice, Inbred C57BL [MeSH], Inflammation/chemically induced [MeSH], Retina/drug effects [MeSH], Retinal Pigment Epithelium/pathology [MeSH], Tobacco/adverse effects [MeSH], Electronic Nicotine Delivery Systems [MeSH], Animals [MeSH], E-cigarette vapor, Smoking/adverse effects [MeSH], C-cigarette smoke, Original Article, Models, Animal [MeSH], Mice [MeSH], Male [MeSH], Inflammation/pathology [MeSH], Inflammatory reaction, Retinal Pigment Epithelium/drug effects [MeSH], Retina/pathology [MeSH], Angiogenesis, Tobacco Smoke Pollution/adverse effects [MeSH], AMD, Mice, 03 medical and health sciences, E-Cigarette Vapor, Smoke, Models, Animal, Animals, Tobacco Smoke Pollution
Dateibeschreibung: application/pdf
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00109-021-02108-9.pdf
https://europepmc.org/articles/pmc8455497?pdf=render
https://pubmed.ncbi.nlm.nih.gov/34264377
https://www.researchsquare.com/article/rs-233889/v1.pdf?c=1631890946000
https://link.springer.com/article/10.1007/s00109-021-02108-9
https://www.researchsquare.com/article/rs-233889/v1
https://pubmed.ncbi.nlm.nih.gov/34264377/
https://link.springer.com/content/pdf/10.1007/s00109-021-02108-9.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8455497/
https://repository.publisso.de/resource/frl:6448279 -
12
Autoren: et al.
Quelle: Ann Hematol
Schlagwörter: Adult, Inflammation, Lipopolysaccharides, Male, 0301 basic medicine, Secretory Pathway, Middle Aged, Hemophilia A, Severity of Illness Index, Monocytes, Thromboplastin, 3. Good health, Young Adult, 03 medical and health sciences, 0302 clinical medicine, Case-Control Studies, Humans, Original Article, Female, Female [MeSH], Adult [MeSH], Hemophilia A/pathology [MeSH], Inflammation/chemically induced [MeSH], Thromboplastin/metabolism [MeSH], Humans [MeSH], Severity of Illness Index [MeSH], Lipopolysaccharides/pharmacology [MeSH], Secretory Pathway/drug effects [MeSH], Middle Aged [MeSH], Hemophilia A/metabolism [MeSH], Monocytes/metabolism [MeSH], Monocytes/drug effects [MeSH], Microvesicles, Tissue factor, Male [MeSH], Haemophilia, Hepatitis, Inflammation/pathology [MeSH], Young Adult [MeSH], Inflammation/metabolism [MeSH], Case-Control Studies [MeSH]
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00277-020-04075-6.pdf
https://pubmed.ncbi.nlm.nih.gov/32430703
https://link.springer.com/content/pdf/10.1007/s00277-020-04075-6.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7316670
https://pubmed.ncbi.nlm.nih.gov/32430703/
https://link.springer.com/article/10.1007/s00277-020-04075-6
https://europepmc.org/articles/PMC7316670/
https://repository.publisso.de/resource/frl:6466992 -
13
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Part Fibre Toxicol
Particle and Fibre Toxicology, Vol 20, Iss 1, Pp 1-19 (2023)Schlagwörter: Inflammation, 0301 basic medicine, 0303 health sciences, Inflammation/chemically induced, Adverse Outcome Pathways, Health, Toxicology and Mutagenesis, Pneumonia/chemically induced, Research, Pulmonary Fibrosis, Pulmonary Fibrosis/chemically induced, Pneumonia, HD7260-7780.8, Toxicology, Silicon Dioxide, Risk Assessment, 03 medical and health sciences, Silicon Dioxide/chemistry, RA1190-1270, Toxicology. Poisons, Humans, Industrial hygiene. Industrial welfare, Risk Assessment/methods
Dateibeschreibung: application/pdf
Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/36759844
https://doaj.org/article/5f0878caca054e3e8d5f98b2ff24b2f6
https://research-portal.uu.nl/en/publications/d2118ce2-00af-4989-9d69-da4cef7e3da5
https://doi.org/10.1186/s12989-023-00516-y
https://dspace.library.uu.nl/handle/1874/428747
https://hdl.handle.net/10029/626439
https://rivm.openrepository.com/handle/10029/654508 -
14
Autoren: et al.
Schlagwörter: Animals, Anti-Inflammatory Agents/pharmacology, Anti-Inflammatory Agents/therapeutic use, Aorta/drug effects, Aorta/metabolism, Cannabinoids/pharmacology, Cannabinoids/therapeutic use, Cells, Cultured, Chemokine CCL2/metabolism, Coronary Vessels/drug effects, Coronary Vessels/metabolism, Disease Models, Animal, Dose-Response Relationship, Drug, Endothelial Cells/drug effects, Endothelial Cells/metabolism, Humans, Inflammation/chemically induced, Inflammation/metabolism, Intercellular Adhesion Molecule-1/metabolism, Leukocyte Rolling/drug effects, Lipopolysaccharides, Male, Mice, Inbred C57BL, Monocytes/drug effects, Monocytes/metabolism, NF-kappa B/metabolism
Dateibeschreibung: application/pdf
Relation: American Journal of Physiology - Heart and Circulatory Physiology; https://iris.unil.ch/handle/iris/64366; serval:BIB_1486D98A14E4; 000250058400024
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15
Autoren: et al.
Schlagwörter: Animals, Congenic, Arenaviridae Infections/immunology, Cercopithecus aethiops, Flow Cytometry, Gene Expression Regulation/immunology, Histocompatibility Antigens Class I/genetics, Histocompatibility Antigens Class I/immunology, Humans, Inflammation/chemically induced, Inflammation/immunology, Interferon Inducers/toxicity, Intracellular Signaling Peptides and Proteins/genetics, Intracellular Signaling Peptides and Proteins/immunology, Killer Cells, Natural/immunology, Lymphocytic choriomeningitis virus, Mice, Inbred C57BL, Knockout, Transgenic, Poly I-C/toxicity, Reverse Transcriptase Polymerase Chain Reaction, Self Tolerance/immunology, Spleen/cytology, Spleen/immunology, T-Lymphocytes/drug effects, T-Lymphocytes/immunology, Vero Cells
Dateibeschreibung: application/pdf
Relation: Nature Communications; https://iris.unil.ch/handle/iris/219167; serval:BIB_8FDEF0BFED69; 000371014300001
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16
Autoren: et al.
Schlagwörter: Animals, Cells, Cultured, Disease Models, Animal, Homeostasis/drug effects, Immunoenzyme Techniques, Inflammation/chemically induced, Inflammation/prevention & control, Iron/toxicity, Male, Mice, Oxidative Stress/drug effects, RNA, Messenger/genetics, Rats, Wistar, Real-Time Polymerase Chain Reaction, Retinal Degeneration/chemically induced, Retinal Degeneration/metabolism, Reverse Transcriptase Polymerase Chain Reaction, Transferrin/pharmacology
Dateibeschreibung: application/pdf
Relation: Free Radical Biology and Medicine; https://iris.unil.ch/handle/iris/204127; serval:BIB_D77ED06BE30E; 000366355800104
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17
Autoren:
Schlagwörter: Brain/growth & development, Brain/pathology, Cells, Cultured, Cytological Techniques, Glial Fibrillary Acidic Protein/metabolism, Humans, Inflammation/chemically induced, Inflammation/pathology, Neuroglia/pathology, Neurons/pathology, Neurotoxicity Syndromes/pathology
Relation: Human and Experimental Toxicology; https://iris.unil.ch/handle/iris/174928; serval:BIB_6CBE69625C7B; 000246413500009
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18
Autoren: et al.
Schlagwörter: 11-beta-Hydroxysteroid Dehydrogenase Type 1/biosynthesis, 11-beta-Hydroxysteroid Dehydrogenase Type 1/drug effects, Animals, Cell Line, Cells, Cultured, Cytokines/biosynthesis, Glucocorticoids/metabolism, In Vitro Techniques, Inflammation/chemically induced, Inflammation/metabolism, Insulin Secretion/drug effects, Metabolic Syndrome/metabolism, Mice, Inbred Strains, Nerve Degeneration/chemically induced, Nerve Degeneration/metabolism, Neuroglia/drug effects, Neurons/drug effects, Rats, Sprague-Dawley, Receptors, Glucocorticoid/drug effects, Reproducibility of Results, Trimethyltin Compounds/toxicity, 11β-Hydroxysteroid dehydrogenase (11β-hsd1), Astrocyte, Glucocorticoid, Insulin, Interleukin-6 (IL-6)
Relation: Archives of Toxicology; https://iris.unil.ch/handle/iris/162606; serval:BIB_D7E0994E9422; 000475702100014
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19
Autoren: et al.
Schlagwörter: Animals *Behavior, Animal Freund's Adjuvant Ganglia, Spinal/drug effects/*metabolism Hyperalgesia/etiology/*metabolism Inflammation/chemically induced/complications/*metabolism Male Neuralgia/etiology/*metabolism Neurons/drug effects/*metabolism Rats Rats, Sprague-Dawley TRPV Cation Channels/*metabolism Up-Regulation/drug effects
Relation: PAIN; https://iris.unil.ch/handle/iris/141089; serval:BIB_B48115DE18F6; 000234355900026
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20
Autoren: et al.
Schlagwörter: Animals, Colitis/chemically induced, Colitis/genetics, Colon/metabolism, Cytoskeletal Proteins/metabolism, Dextran Sulfate, Disease Models, Animal, Genetic Predisposition to Disease, Inflammation/chemically induced, Inflammation/genetics, Intestinal Mucosa/metabolism, Rats, Serine Endopeptidases/metabolism
Dateibeschreibung: application/pdf
Relation: Inflammatory Bowel Diseases; https://iris.unil.ch/handle/iris/215658; serval:BIB_A9B4457513BE; 000393893400008
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