Search Results - "Inflammation/chemically induced"

Characterization of Different Inflammatory Skin Conditions in a Mouse Model of DNCB-Induced Atopic Dermatitis

Authors: Riedl, Rebecca Kühn, Annika Hupfer, Yvonne et al.

Source: Inflammation

Subject Terms: Inflammation, 0301 basic medicine, Mice, Inbred BALB C, 0303 health sciences, Research, Filaggrin Proteins, Dermatitis, Atopic/chemically induced [MeSH], 2,4-dinitrochlorobenzene, Dermatitis, Atopic/pathology [MeSH], Female [MeSH], Filaggrin Proteins [MeSH], Mice, Inbred BALB C [MeSH], Inflammation/chemically induced [MeSH], atopic dermatitis mouse model, Skin/drug effects [MeSH], sub-acute and chronic inflammation, Cytokines/metabolism [MeSH], Animals [MeSH], Skin/pathology [MeSH], Dermatitis, Atopic/metabolism [MeSH], Mice [MeSH], Dinitrochlorobenzene/toxicity [MeSH], Inflammation/metabolism [MeSH], Disease Models, Animal [MeSH], mild and moderate atopic dermatitis, Dexamethasone/pharmacology [MeSH], Skin/metabolism [MeSH], Dexamethasone, Dermatitis, Atopic, Mice, Disease Models, Animal, 03 medical and health sciences, Dinitrochlorobenzene, Animals, Cytokines, Female, Skin

Access URL: https://pubmed.ncbi.nlm.nih.gov/38150167
https://repository.publisso.de/resource/frl:6504113

Non-canonical STAT3 pathway induces alterations of mitochondrial dynamic proteins in the hippocampus of an LPS-induced murine neuroinflammation model

Authors: Millot, Périne Duquesne, Laurine San, Carine et al.

Contributors: Millot, Périne Duquesne, Laurine San, Carine et al.

Source: ISSN: 0197-0186.

Subject Terms: MESH: Animals, MESH: Disease Models, MESH: Signal Transduction* / drug effects, MESH: Signal Transduction* / physiology, MESH: Hippocampus* / drug effects, MESH: Hippocampus* / metabolism, MESH: Inflammation / chemically induced, MESH: Inflammation / metabolism, MESH: Lipopolysaccharides* / toxicity, MESH: Male, MESH: Mice, Inbred C57BL, MESH: Mitochondria* / drug effects, MESH: Mitochondria* / metabolism, MESH: Mitochondrial Dynamics* / physiology, MESH: Mitochondrial Proteins* / metabolism, MESH: Neuroinflammatory Diseases* / chemically induced, MESH: Neuroinflammatory Diseases* / metabolism, MESH: STAT3 Transcription Factor* / metabolism, [SDV]Life Sciences [q-bio], [SDV.BC]Life Sciences [q-bio]/Cellular Biology, [SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology

Relation: info:eu-repo/semantics/altIdentifier/pmid/40209854; PUBMED: 40209854

Availability: https://u-paris.hal.science/hal-05107434
https://u-paris.hal.science/hal-05107434v1/document
https://u-paris.hal.science/hal-05107434v1/file/1-s2.0-S019701862500052X-main.pdf
https://doi.org/10.1016/j.neuint.2025.105979

History of peripheral artery disease and cardiovascular risk of real-world patients with acute coronary syndrome: Role of inflammation and comorbidities

Authors: Denegri, Andrea Magnani, Giulia Kraler, Simon et al.

Contributors: Denegri, Andrea Magnani, Giulia Kraler, Simon et al.

Source: International journal of cardiology, vol. 382, pp. 76-82
Denegri, Andrea; Magnani, Giulia; Kraler, Simon; Bruno, Francesco; Klingenberg, Roland; Mach, Francois; Gencer, Baris; Räber, Lorenz; Rodondi, Nicolas; Rossi, Valentina A; Matter, Christian M; Nanchen, David; Obeid, Slayman; Lüscher, Thomas F (2023). History of peripheral artery disease and cardiovascular risk of real-word patients with acute coronary syndrome: Role of inflammation and comorbidities. International journal of cardiology, 382, pp. 76-82. Elsevier 10.1016/j.ijcard.2023.03.043 <http://dx.doi.org/10.1016/j.ijcard.2023.03.043>

Subject Terms: Inflammation, 610 Medicine & health, Hemorrhage, 2705 Cardiology and Cardiovascular Medicine, 3. Good health, Humans, Acute Coronary Syndrome/diagnosis, Acute Coronary Syndrome/epidemiology, Acute Coronary Syndrome/chemically induced, Cardiovascular Diseases/chemically induced, Platelet Aggregation Inhibitors/adverse effects, Prospective Studies, Risk Factors, Hemorrhage/diagnosis, Hemorrhage/epidemiology, Hemorrhage/chemically induced, Inflammation/diagnosis, Inflammation/epidemiology, Inflammation/chemically induced, Peripheral Arterial Disease/diagnosis, Peripheral Arterial Disease/epidemiology, Heart Disease Risk Factors, Acute coronary syndrome, Peripheral artery disease, Personalized therapy, Residual risk, Risk stratification, Peripheral Arterial Disease, 360 Social problems & social services, Cardiovascular Diseases, 10209 Clinic for Cardiology, Acute Coronary Syndrome, Platelet Aggregation Inhibitors

File Description: application/pdf; ZORA257558.pdf - application/pdf

Access URL: https://pubmed.ncbi.nlm.nih.gov/36958395
http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_009B4EEC350F0
https://serval.unil.ch/notice/serval:BIB_009B4EEC350F
https://serval.unil.ch/resource/serval:BIB_009B4EEC350F.P001/REF.pdf
https://boris.unibe.ch/180603/

PARK7/DJ-1 deficiency impairs microglial activation in response to LPS-induced inflammation

Authors: Lind-Holm Mogensen, Frida Sousa, Carole Ameli, Corrado et al.

Source: J Neuroinflammation
Journal of Neuroinflammation, Vol 21, Iss 1, Pp 1-24 (2024)
Journal of neuroinflammation 21(1), 174 (2024). doi:10.1186/s12974-024-03164-x

Subject Terms: Lipopolysaccharides, Neuroinflammatory Diseases/metabolism [MeSH], Mice, Inbred C57BL [MeSH], Neuroinflammatory Diseases/genetics [MeSH], Inflammation/chemically induced [MeSH], Neuroinflammation, Microglia/drug effects [MeSH], Neuroinflammatory Diseases/chemically induced [MeSH], Lipopolysaccharides/pharmacology [MeSH], Neuroinflammatory Diseases/pathology [MeSH], Lipopolysaccharides/toxicity [MeSH], Microglia/metabolism [MeSH], Inflammation/genetics [MeSH], Lipopolysaccharide, Protein Deglycase DJ-1/deficiency [MeSH], Parkinson's disease, Humans [MeSH], Microglia, Animals [MeSH], Mice, Knockout [MeSH], Mice [MeSH], Microglia/pathology [MeSH], Microglia morphology, Inflammation/pathology [MeSH], Protein Deglycase DJ-1/genetics [MeSH], Research, Inflammation/metabolism [MeSH], Protein Deglycase DJ-1/metabolism [MeSH], PARK7/DJ-1, Protein Deglycase DJ-1, genetics [Protein Deglycase DJ-1], metabolism [Microglia], drug effects [Microglia], genetics [Inflammation], Mice, toxicity [Lipopolysaccharides], Animals, Humans, deficiency [Protein Deglycase DJ-1], ddc:610, chemically induced [Inflammation], RC346-429, pathology [Inflammation], Mice, Knockout, Inflammation, metabolism [Inflammation], PARK7 protein, mouse, pathology [Neuroinflammatory Diseases], genetics [Neuroinflammatory Diseases], pathology [Microglia], ddc, metabolism [Protein Deglycase DJ-1], pharmacology [Lipopolysaccharides], Mice, Inbred C57BL, metabolism [Neuroinflammatory Diseases], chemically induced [Neuroinflammatory Diseases], Neuroinflammatory Diseases, Neurology. Diseases of the nervous system

File Description: application/pdf

Access URL: https://pubmed.ncbi.nlm.nih.gov/39014482
https://doaj.org/article/087f0fcc0fd0451b80ec654106edcd26
https://repository.publisso.de/resource/frl:6498419
https://mediatum.ub.tum.de/1770480

CD4+ T cells play an essential role in chronic MC903-induced skin inflammation

Authors: Mi Hye Song Anupriya Gupta Varun Sasidharan Nair et al.

Source: Biochemical and Biophysical Research Communications

Subject Terms: Dermatitis, Atopic* / chemically induced, Inflammation, 0301 basic medicine, 0303 health sciences, CD4 T cell +, Sensitization, Dermatitis, Atopic, 3. Good health, Disease Models, Animal, Mice, 03 medical and health sciences, Th2 Cells, IL-33, MC903, Animals, Cytokines, Challenge, Inflammation / chemically induced, Atopic dermatitis, Dermatitis, Atopic* / drug therapy, Skin

Access URL: https://pubmed.ncbi.nlm.nih.gov/35500442
http://hdl.handle.net/10033/623191

Botulinum Toxin Type A Exerts Direct Trans-Synaptic Action at Bilateral Spinal Nociceptive Circuits

Authors: Dalia Nemanić Petra Šoštarić Patrik Meglić et al.

Source: Toxins (Basel)
Toxins, Vol 17, Iss 3, p 140 (2025)

Subject Terms: Male, Nociception, Synaptosomal-Associated Protein 25, Pain, Botulinum Toxins, Type A / therapeutic use, Carrageenan, Botulinum Toxins, Type A / pharmacology, Spinal Cord / drug effects, Article, Proto-Oncogene Proteins c-fos / metabolism, Rats, Sprague-Dawley, Ganglia, Spinal, Inflammation / drug therapy, Animals, neutralizing antitoxin, Botulinum Toxins, Type A, Rats, Wistar, Inflammation, Analgesics, carrageenan-induced bilateral inflammatory pain, c-Fos immunohistochemistry, Analgesics / therapeutic use, synaptosomal-associated protein 25, Synaptosomal-Associated Protein 25 / metabolism, trans-synaptic transport, Nociception / drug effects, botulinum toxin type A, Rats, Spinal Cord, Analgesics / pharmacology, Medicine, Inflammation / chemically induced, Spinal Cord / metabolism, Proto-Oncogene Proteins c-fos

File Description: application/pdf

Access URL: https://pubmed.ncbi.nlm.nih.gov/40137913
https://doaj.org/article/ed4af1ff95b442ab8596c1f16b9887e1
https://urn.nsk.hr/urn:nbn:hr:105:875315
https://doi.org/10.3390/toxins17030140

Deoxynivalenol Induces Local Inflammation and Lesions in Tissues at Doses Recommended by the EU

Authors: Pierron, Alix Balbo, Luciana Soler-Vasco, Laura et al.

Contributors: Pierron, Alix Balbo, Luciana Soler-Vasco, Laura et al.

Source: ISSN: 1661-6596.

Subject Terms: DON, histology, immunity-related genes, mycotoxins, toxicity, MESH: Animal Feed, MESH: Animals, MESH: Spleen / pathology, MESH: Swine, MESH: Trichothecenes* / toxicity, MESH: Substances, MESH: European Union, MESH: Food Contamination / analysis, MESH: Inflammation* / chemically induced, MESH: Liver* / drug effects, MESH: Liver* / metabolism, MESH: Liver* / pathology, MESH: Spleen / drug effects, MESH: Spleen / metabolism, [SDV.TOX]Life Sciences [q-bio]/Toxicology

Relation: info:eu-repo/semantics/altIdentifier/pmid/39337277; PUBMED: 39337277

Availability: https://hal.inrae.fr/hal-04720797
https://hal.inrae.fr/hal-04720797v1/document
https://hal.inrae.fr/hal-04720797v1/file/2024_Pierron_International%20Journal%20of%20Molecular%20Sciences.pdf
https://doi.org/10.3390/ijms25189790

Zinc glycinate alleviates LPS-induced inflammation and intestinal barrier disruption in chicken embryos by regulating zinc homeostasis and TLR4/NF-κB pathway.

Authors: Xiao, Chuanpi Comer, Luke Pan, Xue et al.

Source: Ecotoxicology and Environmental Safety, 272, 116111 (2024-03-01)

Subject Terms: Chicken embryos, Immune challenge, In ovo feeding, Lipopolysaccharide, Zinc, NF-kappa B, Lipopolysaccharides, Toll-Like Receptor 4, zinc glycinate, Saline Solution, Glycine, Chick Embryo, Animals, Toll-Like Receptor 4/metabolism, Chickens/metabolism, Saline Solution/toxicity, Inflammation/chemically induced, Inflammation/veterinary, Homeostasis, Zinc/toxicity, Glycine/analogs & derivatives, NF-kappa B/genetics, NF-kappa B/metabolism, Lipopolysaccharides/toxicity, Health, Toxicology and Mutagenesis, Public Health, Environmental and Occupational Health, Pollution, General Medicine

Relation: https://api.elsevier.com/content/article/PII:S0147651324001866?httpAccept=text/xml; urn:issn:0147-6513; urn:issn:1090-2414; https://orbi.uliege.be/handle/2268/316290; info:hdl:2268/316290; info:pmid:38350216

Availability: https://orbi.uliege.be/handle/2268/316290
https://orbi.uliege.be/bitstream/2268/316290/1/published%20version_Zinc%20glycinate%20alleviates%20LPS-induced%20inflammation%20and%20intestinal%20barrier.pdf
https://doi.org/10.1016/j.ecoenv.2024.116111

Limited Impact of 6-Mercaptopurine on Inflammation-Induced Chemokines Expression Profile in Primary Cultures of Enteric Nervous System

Authors: Kneusels, Jan Kaehler, Meike Cascorbi, Ingolf et al.

Contributors: Kneusels, Jan Kaehler, Meike Cascorbi, Ingolf et al.

Source: Neurochem Res

Subject Terms: MESH: Inflammation, 6-Mercaptopurine, Lipopolysaccharides, 0301 basic medicine, MESH: Gene Expression, MESH: Rats, Interleukin-1beta/genetics [MeSH], Tumor Necrosis Factor-alpha/genetics [MeSH], Enteric Nervous System/cytology [MeSH], Inflammation/chemically induced [MeSH], Rats [MeSH], Lipopolysaccharides [MeSH], Animals [MeSH], Gene Expression/drug effects [MeSH], Inflammatory bowel diseases, Neurons/drug effects [MeSH], CXC motif ligand chemokines, Interleukin-1beta/pharmacology [MeSH], Mercaptopurine/pharmacology [MeSH], Mice [MeSH], Anti-Inflammatory Agents/pharmacology [MeSH], Original Paper, Cells, Cultured [MeSH], Tumor Necrosis Factor-alpha/metabolism [MeSH], Enteric glial cells, Interleukin-1beta/metabolism [MeSH], Enteric nervous system, Tumor Necrosis Factor-alpha/pharmacology [MeSH], [SDV]Life Sciences [q-bio], Interleukin-1beta, MESH: Neurons, Anti-Inflammatory Agents, Gene Expression, Enteric Nervous System, Mice, 03 medical and health sciences, MESH: Interleukin-1beta, Animals, MESH: Animals, MESH: Mice, Cells, Cultured, Inflammation, Neurons, MESH: Cells, 0303 health sciences, Cultured, MESH: Enteric Nervous System, Mercaptopurine, Tumor Necrosis Factor-alpha, Rats, 3. Good health, [SDV] Life Sciences [q-bio], MESH: Anti-Inflammatory Agents, MESH: Tumor Necrosis Factor-alpha, MESH: Mercaptopurine, MESH: Lipopolysaccharides

File Description: application/pdf

Access URL: https://link.springer.com/content/pdf/10.1007/s11064-021-03324-y.pdf
https://pubmed.ncbi.nlm.nih.gov/33864170
https://www.ncbi.nlm.nih.gov/pubmed/33864170
https://link.springer.com/article/10.1007/s11064-021-03324-y
https://europepmc.org/article/PMC/PMC8187225
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8187225
https://link.springer.com/content/pdf/10.1007/s11064-021-03324-y.pdf
https://hal.science/hal-04178448v1
https://hal.science/hal-04178448v1/document
https://doi.org/10.1007/s11064-021-03324-y
https://repository.publisso.de/resource/frl:6445958

Inflammatory markers in autoimmunity induced by checkpoint inhibitors

Authors: Beate Husain Michael Constantin Kirchberger Michael Erdmann et al.

Source: J Cancer Res Clin Oncol

Subject Terms: Adult, Male, Uveal Neoplasms, 0301 basic medicine, Skin Neoplasms, Autoimmunity, Endocrine System Diseases, Cohort Studies, 03 medical and health sciences, Melanoma, Cutaneous Malignant, 0302 clinical medicine, Uveal Melanoma, Germany, Biomarkers, Tumor, Humans, ddc:610, Neoplasm Metastasis, 10. No inequality, Immune Checkpoint Inhibitors, Melanoma, Inflammation, 3. Good health, Inflammation/blood [MeSH], Gene Expression Regulation, Neoplastic/immunology [MeSH], Inflammation/chemically induced [MeSH], Skin Neoplasms/blood [MeSH], Skin Neoplasms/drug therapy [MeSH], Cohort Studies [MeSH], Uveal Neoplasms/blood [MeSH], Autoimmunity/genetics [MeSH], Inflammation/genetics [MeSH], Male [MeSH], Immune Checkpoint Inhibitors/adverse effects [MeSH], Melanoma/pathology [MeSH], Case-Control Studies [MeSH], Autoimmunity/drug effects [MeSH], Uveal Neoplasms/pathology [MeSH], Diabetes Mellitus, Type 1/blood [MeSH], Melanoma/drug therapy [MeSH], Inflammation/immunology [MeSH], Female [MeSH], Original Article – Cancer Research, Melanoma/genetics [MeSH], Adult [MeSH], Diabetes Mellitus, Type 1/chemically induced [MeSH], Humans [MeSH], Neoplasm Metastasis [MeSH], Uveal Neoplasms/drug therapy [MeSH], Melanoma/blood [MeSH], Uveal Neoplasms/genetics [MeSH], Skin Neoplasms/pathology [MeSH], Immune checkpoint inhibitors, Immune Checkpoint Inhibitors/therapeutic use [MeSH], Cytokines, Gene Expression Regulation, Neoplastic/drug effects [MeSH], Autoimmunity/immunology [MeSH], Skin Neoplasms/genetics [MeSH], Endocrine System Diseases/blood [MeSH], Biomarkers, Tumor/blood [MeSH], Biomarkers, Tumor/genetics [MeSH], Germany [MeSH], Endocrine System Diseases/immunology [MeSH], Diabetes Mellitus, Type 1/immunology [MeSH], Immune-related adverse events, Endocrine System Diseases/chemically induced [MeSH], Immune Checkpoint Inhibitors/pharmacology [MeSH], Gene Expression Regulation, Neoplastic, Diabetes Mellitus, Type 1, Case-Control Studies, Female

File Description: application/pdf

Access URL: https://link.springer.com/content/pdf/10.1007/s00432-021-03550-5.pdf
https://pubmed.ncbi.nlm.nih.gov/33837821
https://link.springer.com/content/pdf/10.1007/s00432-021-03550-5.pdf
https://www.ncbi.nlm.nih.gov/pubmed/33837821
https://europepmc.org/article/PMC/PMC8076116
https://link.springer.com/article/10.1007/s00432-021-03550-5
https://pubmed.ncbi.nlm.nih.gov/33837821/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8076116
https://opus4.kobv.de/opus4-fau/frontdoor/index/index/docId/22122
https://repository.publisso.de/resource/frl:6449924

Retinal tissue develops an inflammatory reaction to tobacco smoke and electronic cigarette vapor in mice

Authors: Wang, Feng Hadzic, Stefan Roxlau, Elsa T. et al.

Source: J Mol Med (Berl)

Subject Terms: Inflammation, Male, 0301 basic medicine, Nicotine, ddc:610, 0303 health sciences, Smoking, Retinal Pigment Epithelium, Tobacco Products, Electronic Nicotine Delivery Systems, Retina, 3. Good health, Mice, Inbred C57BL, Smoke/adverse effects [MeSH], E-Cigarette Vapor/adverse effects [MeSH], Nicotine/adverse effects [MeSH], Mice, Inbred C57BL [MeSH], Inflammation/chemically induced [MeSH], Retina/drug effects [MeSH], Retinal Pigment Epithelium/pathology [MeSH], Tobacco/adverse effects [MeSH], Electronic Nicotine Delivery Systems [MeSH], Animals [MeSH], E-cigarette vapor, Smoking/adverse effects [MeSH], C-cigarette smoke, Original Article, Models, Animal [MeSH], Mice [MeSH], Male [MeSH], Inflammation/pathology [MeSH], Inflammatory reaction, Retinal Pigment Epithelium/drug effects [MeSH], Retina/pathology [MeSH], Angiogenesis, Tobacco Smoke Pollution/adverse effects [MeSH], AMD, Mice, 03 medical and health sciences, E-Cigarette Vapor, Smoke, Models, Animal, Animals, Tobacco Smoke Pollution

File Description: application/pdf

Access URL: https://link.springer.com/content/pdf/10.1007/s00109-021-02108-9.pdf
https://europepmc.org/articles/pmc8455497?pdf=render
https://pubmed.ncbi.nlm.nih.gov/34264377
https://www.researchsquare.com/article/rs-233889/v1.pdf?c=1631890946000
https://link.springer.com/article/10.1007/s00109-021-02108-9
https://www.researchsquare.com/article/rs-233889/v1
https://pubmed.ncbi.nlm.nih.gov/34264377/
https://link.springer.com/content/pdf/10.1007/s00109-021-02108-9.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8455497/
https://repository.publisso.de/resource/frl:6448279

LPS-induced expression and release of monocyte tissue factor in patients with haemophilia

Authors: Holstein, Katharina Matysiak, Anna Witt, Leonora et al.

Source: Ann Hematol

Subject Terms: Adult, Inflammation, Lipopolysaccharides, Male, 0301 basic medicine, Secretory Pathway, Middle Aged, Hemophilia A, Severity of Illness Index, Monocytes, Thromboplastin, 3. Good health, Young Adult, 03 medical and health sciences, 0302 clinical medicine, Case-Control Studies, Humans, Original Article, Female, Female [MeSH], Adult [MeSH], Hemophilia A/pathology [MeSH], Inflammation/chemically induced [MeSH], Thromboplastin/metabolism [MeSH], Humans [MeSH], Severity of Illness Index [MeSH], Lipopolysaccharides/pharmacology [MeSH], Secretory Pathway/drug effects [MeSH], Middle Aged [MeSH], Hemophilia A/metabolism [MeSH], Monocytes/metabolism [MeSH], Monocytes/drug effects [MeSH], Microvesicles, Tissue factor, Male [MeSH], Haemophilia, Hepatitis, Inflammation/pathology [MeSH], Young Adult [MeSH], Inflammation/metabolism [MeSH], Case-Control Studies [MeSH]

Access URL: https://link.springer.com/content/pdf/10.1007/s00277-020-04075-6.pdf
https://pubmed.ncbi.nlm.nih.gov/32430703
https://link.springer.com/content/pdf/10.1007/s00277-020-04075-6.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7316670
https://pubmed.ncbi.nlm.nih.gov/32430703/
https://link.springer.com/article/10.1007/s00277-020-04075-6
https://europepmc.org/articles/PMC7316670/
https://repository.publisso.de/resource/frl:6466992

Establishing relationships between particle-induced in vitro and in vivo inflammation endpoints to better extrapolate between in vitro markers and in vivo fibrosis

Authors: McLean, P Mueller, W Gosens, I et al.

Contributors: McLean, P Mueller, W Gosens, I et al.

Source: Part Fibre Toxicol
Particle and Fibre Toxicology, Vol 20, Iss 1, Pp 1-19 (2023)

Subject Terms: Inflammation, 0301 basic medicine, 0303 health sciences, Inflammation/chemically induced, Adverse Outcome Pathways, Health, Toxicology and Mutagenesis, Pneumonia/chemically induced, Research, Pulmonary Fibrosis, Pulmonary Fibrosis/chemically induced, Pneumonia, HD7260-7780.8, Toxicology, Silicon Dioxide, Risk Assessment, 03 medical and health sciences, Silicon Dioxide/chemistry, RA1190-1270, Toxicology. Poisons, Humans, Industrial hygiene. Industrial welfare, Risk Assessment/methods

File Description: application/pdf

Access URL: https://pubmed.ncbi.nlm.nih.gov/36759844
https://doaj.org/article/5f0878caca054e3e8d5f98b2ff24b2f6
https://research-portal.uu.nl/en/publications/d2118ce2-00af-4989-9d69-da4cef7e3da5
https://doi.org/10.1186/s12989-023-00516-y
https://dspace.library.uu.nl/handle/1874/428747
https://hdl.handle.net/10029/626439
https://rivm.openrepository.com/handle/10029/654508

CB2-receptor stimulation attenuates TNF-alpha-induced human endothelial cell activation, transendothelial migration of monocytes, and monocyte-endothelial adhesion.

Authors: Rajesh, M. Mukhopadhyay, P. Bátkai, S. et al.

Subject Terms: Animals, Anti-Inflammatory Agents/pharmacology, Anti-Inflammatory Agents/therapeutic use, Aorta/drug effects, Aorta/metabolism, Cannabinoids/pharmacology, Cannabinoids/therapeutic use, Cells, Cultured, Chemokine CCL2/metabolism, Coronary Vessels/drug effects, Coronary Vessels/metabolism, Disease Models, Animal, Dose-Response Relationship, Drug, Endothelial Cells/drug effects, Endothelial Cells/metabolism, Humans, Inflammation/chemically induced, Inflammation/metabolism, Intercellular Adhesion Molecule-1/metabolism, Leukocyte Rolling/drug effects, Lipopolysaccharides, Male, Mice, Inbred C57BL, Monocytes/drug effects, Monocytes/metabolism, NF-kappa B/metabolism

File Description: application/pdf

Relation: American Journal of Physiology - Heart and Circulatory Physiology; https://iris.unil.ch/handle/iris/64366; serval:BIB_1486D98A14E4; 000250058400024

Availability: https://iris.unil.ch/handle/iris/64366
https://doi.org/10.1152/ajpheart.00688.2007

NLRC5 shields T lymphocytes from NK-cell-mediated elimination under inflammatory conditions.

Authors: Ludigs, K. Jandus, C. Utzschneider, D.T. et al.

Subject Terms: Animals, Congenic, Arenaviridae Infections/immunology, Cercopithecus aethiops, Flow Cytometry, Gene Expression Regulation/immunology, Histocompatibility Antigens Class I/genetics, Histocompatibility Antigens Class I/immunology, Humans, Inflammation/chemically induced, Inflammation/immunology, Interferon Inducers/toxicity, Intracellular Signaling Peptides and Proteins/genetics, Intracellular Signaling Peptides and Proteins/immunology, Killer Cells, Natural/immunology, Lymphocytic choriomeningitis virus, Mice, Inbred C57BL, Knockout, Transgenic, Poly I-C/toxicity, Reverse Transcriptase Polymerase Chain Reaction, Self Tolerance/immunology, Spleen/cytology, Spleen/immunology, T-Lymphocytes/drug effects, T-Lymphocytes/immunology, Vero Cells

File Description: application/pdf

Relation: Nature Communications; https://iris.unil.ch/handle/iris/219167; serval:BIB_8FDEF0BFED69; 000371014300001

Availability: https://iris.unil.ch/handle/iris/219167
https://doi.org/10.1038/ncomms10554

Targeting iron-mediated retinal degeneration by local delivery of transferrin.

Authors: Picard, E. Le Rouzic, Q. Oudar, A. et al.

Subject Terms: Animals, Cells, Cultured, Disease Models, Animal, Homeostasis/drug effects, Immunoenzyme Techniques, Inflammation/chemically induced, Inflammation/prevention & control, Iron/toxicity, Male, Mice, Oxidative Stress/drug effects, RNA, Messenger/genetics, Rats, Wistar, Real-Time Polymerase Chain Reaction, Retinal Degeneration/chemically induced, Retinal Degeneration/metabolism, Reverse Transcriptase Polymerase Chain Reaction, Transferrin/pharmacology

File Description: application/pdf

Relation: Free Radical Biology and Medicine; https://iris.unil.ch/handle/iris/204127; serval:BIB_D77ED06BE30E; 000366355800104

Availability: https://iris.unil.ch/handle/iris/204127
https://doi.org/10.1016/j.freeradbiomed.2015.08.018

Neurotoxicant-induced inflammatory response in three-dimensional brain cell cultures.

Authors: Monnet-Tschudi, F. Zurich, M.G. Honegger, P.

Subject Terms: Brain/growth & development, Brain/pathology, Cells, Cultured, Cytological Techniques, Glial Fibrillary Acidic Protein/metabolism, Humans, Inflammation/chemically induced, Inflammation/pathology, Neuroglia/pathology, Neurons/pathology, Neurotoxicity Syndromes/pathology

Relation: Human and Experimental Toxicology; https://iris.unil.ch/handle/iris/174928; serval:BIB_6CBE69625C7B; 000246413500009

Availability: https://iris.unil.ch/handle/iris/174928
https://doi.org/10.1177/0960327107074589

In vitro models to study insulin and glucocorticoids modulation of trimethyltin (TMT)-induced neuroinflammation and neurodegeneration, and in vivo validation in db/db mice.

Authors: Sandström, J. Kratschmar, D.V. Broyer, A. et al.

Subject Terms: 11-beta-Hydroxysteroid Dehydrogenase Type 1/biosynthesis, 11-beta-Hydroxysteroid Dehydrogenase Type 1/drug effects, Animals, Cell Line, Cells, Cultured, Cytokines/biosynthesis, Glucocorticoids/metabolism, In Vitro Techniques, Inflammation/chemically induced, Inflammation/metabolism, Insulin Secretion/drug effects, Metabolic Syndrome/metabolism, Mice, Inbred Strains, Nerve Degeneration/chemically induced, Nerve Degeneration/metabolism, Neuroglia/drug effects, Neurons/drug effects, Rats, Sprague-Dawley, Receptors, Glucocorticoid/drug effects, Reproducibility of Results, Trimethyltin Compounds/toxicity, 11β-Hydroxysteroid dehydrogenase (11β-hsd1), Astrocyte, Glucocorticoid, Insulin, Interleukin-6 (IL-6)

Relation: Archives of Toxicology; https://iris.unil.ch/handle/iris/162606; serval:BIB_D7E0994E9422; 000475702100014

Availability: https://iris.unil.ch/handle/iris/162606
https://doi.org/10.1007/s00204-019-02455-0

Peripheral inflammation induces up-regulation of TRPV2 expression in rat DRG

Authors: Shimosato, G. Amaya, F. Ueda, M. et al.

Subject Terms: Animals *Behavior, Animal Freund's Adjuvant Ganglia, Spinal/drug effects/*metabolism Hyperalgesia/etiology/*metabolism Inflammation/chemically induced/complications/*metabolism Male Neuralgia/etiology/*metabolism Neurons/drug effects/*metabolism Rats Rats, Sprague-Dawley TRPV Cation Channels/*metabolism Up-Regulation/drug effects

Relation: PAIN; https://iris.unil.ch/handle/iris/141089; serval:BIB_B48115DE18F6; 000234355900026

Availability: https://iris.unil.ch/handle/iris/141089
https://doi.org/10.1016/j.pain.2005.10.002

Altered Prostasin (CAP1/Prss8) Expression Favors Inflammation and Tissue Remodeling in DSS-induced Colitis.

Authors: Keppner, A. Malsure, S. Nobile, A. et al.

Subject Terms: Animals, Colitis/chemically induced, Colitis/genetics, Colon/metabolism, Cytoskeletal Proteins/metabolism, Dextran Sulfate, Disease Models, Animal, Genetic Predisposition to Disease, Inflammation/chemically induced, Inflammation/genetics, Intestinal Mucosa/metabolism, Rats, Serine Endopeptidases/metabolism

File Description: application/pdf

Relation: Inflammatory Bowel Diseases; https://iris.unil.ch/handle/iris/215658; serval:BIB_A9B4457513BE; 000393893400008

Availability: https://iris.unil.ch/handle/iris/215658
https://doi.org/10.1097/MIB.0000000000000940