Search Results - "Immunity, Innate genetics"

Validating genetic variants in innate immunity linked to infectious events in acute myeloid leukemia post-induction chemotherapy

Authors: Ulf Schnetzke Mike Fischer Christoph Röllig et al.

Source: Genes Immun

Subject Terms: Male, Adult, Adolescent, Immunity, Innate/genetics [MeSH], Aged [MeSH], Polymorphism, Single Nucleotide [MeSH], 631/250/254, Toll-Like Receptor 2/genetics [MeSH], Cohort Studies [MeSH], Male [MeSH], Leukemia, Myeloid, Acute/genetics [MeSH], Invasive Fungal Infections/genetics [MeSH], Cell Adhesion Molecules/genetics [MeSH], 38/23, Genetic Predisposition to Disease [MeSH], Receptors, Cell Surface/genetics [MeSH], Adolescent [MeSH], Female [MeSH], C-Reactive Protein [MeSH], Adult [MeSH], Humans [MeSH], Middle Aged [MeSH], 631/250/262/2106, Invasive Fungal Infections/drug therapy [MeSH], Serum Amyloid P-Component/genetics [MeSH], Article, Sepsis/drug therapy [MeSH], Induction Chemotherapy [MeSH], Lectins, C-Type/genetics [MeSH], Toll-Like Receptor 4/genetics [MeSH], Leukemia, Myeloid, Acute/drug therapy [MeSH], Sepsis/genetics [MeSH], article, Receptors, Cell Surface, Induction Chemotherapy, Middle Aged, Polymorphism, Single Nucleotide, Immunity, Innate, Toll-Like Receptor 2, Toll-Like Receptor 4, Cohort Studies, Leukemia, Myeloid, Acute, Serum Amyloid P-Component, Sepsis, Humans, Female, Lectins, C-Type, Cell Adhesion Molecules, Invasive Fungal Infections, Aged

Access URL: https://pubmed.ncbi.nlm.nih.gov/38982248
https://repository.publisso.de/resource/frl:6504915

Mutations in RNU4ATAC Are Associated With Chilblain‐Like Lesions and Enhanced Type I Interferon Signalling

Authors: Nic Robertson Aakash Joshi Francesca Ritchie et al.

Source: Eur J Immunol
Robertson, N, Joshi, A, Ritchie, F, Schim van der Loeff, I, Royan, D, Duker, A L, Rice, G I, Bober, M B, Mansour, S, Campbell, D I, Brennan, M, Brown, L, Jones, L, Williams, E, Jackson, A P & Crow, Y J 2025, 'Mutations in RNU4ATAC Are Associated With Chilblain-Like Lesions and Enhanced Type I Interferon Signalling', European journal of immunology, vol. 55, no. 5, e202451518. https://doi.org/10.1002/eji.202451518

Subject Terms: Male, RNA, Untranslated/genetics, Immunity, Notes and Insights, Untranslated/genetics, Innate/genetics, Interferon Type I/metabolism, Mutation, Humans, Signal Transduction/genetics, RNA, Female, Immunity, Innate/genetics, Chilblains/genetics

AI-driven immunology, quantum bioenergetics, bioelectromagnetic modulation, immunometabolism, epigenetic immunomodulation, precision immunotherapy, immune recalibration, quantum computing in biology, personalized medicine

Authors: Mohamed, Anwar

Subject Terms: Epigenomics, AI Immunology, Immune Cell Metabolism, RNA, Mitochondrial, CRISPR-Associated Proteins, Quantum Biology, Autoimmunity, Adaptive Immunity/immunology, Mitochondria/chemistry, Adaptive Immunity, Mitochondrial ADP, ATP Translocases/genetics, Epigenesis, Genetic, Redox Signaling, Inflammation/genetics, Immune Engineering, Systems Biology, Inflammation/metabolism, Longevity/genetics, Cytokine Storm, Histone Modification, Innate Immunity, Mitochondria, Quantum Immunology, Immune System Diseases, CRISPR, Autoimmunity/genetics, Mitochondria/pathology, Inflammation/therapy, Epigenetics, Inflammation Mediators, Epigenomics/education, Immune System/innervation, Mitochondrial ADP, ATP Translocases/immunology, Epigenetic Memory/immunology, Innate Immunity Recognition/immunology, Longevity, Innate Immunity Recognition/genetics, Bioenergetics, DNA, Mitochondrial, Immunity, Innate/immunology, Mitochondria/metabolism, Bioelectric Medicine, miRNA, Inflammation, Immunometabolism, Immune Reprogramming, Systems Biology/trends, Frequency Medicine, Innate Immunity Recognition, Immunity, Innate, ATP, Longevity/immunology, Adaptive Immunity/genetics, Immune system, Immune Aging, Immune System, Mitochondria/genetics, Immunity, Innate/genetics, Immune System/immunology, Mitochondrial ADP, ATP Translocases/metabolism, Epigenetic Memory, Epigenomics/trends, Inflammation/pathology

Single-cell RNA sequencing highlights the influence of innate and adaptive immune response mechanisms in psoriatic arthritis

Authors: Melanie R. Nielsen Marie Skougaard Clara Drachmann et al.

Source: Front Immunol
Frontiers in Immunology, Vol 15 (2025)
Nielsen, M R, Skougaard, M, Drachmann, C, Stisen, Z R, Ditlev, S B, Jessen, L E & Kristensen, L E 2025, ' Single-cell RNA sequencing highlights the influence of innate and adaptive immune response mechanisms in psoriatic arthritis ', Frontiers in Immunology, vol. 15, 1490051 . https://doi.org/10.3389/fimmu.2024.1490051
Nielsen, M R, Skougaard, M, Drachmann, C, Stisen, Z R, Ditlev, S B, Jessen, L E & Kristensen, L E 2025, 'Single-cell RNA sequencing highlights the influence of innate and adaptive immune response mechanisms in psoriatic arthritis', Frontiers in Immunology, vol. 15, 1490051, pp. 1490051. https://doi.org/10.3389/fimmu.2024.1490051

Subject Terms: gene set enrichment analysis, Male, Adult, Immunology, Adaptive Immunity, Arthritis, Psoriatic/genetics, transcriptomics, Humans, Leukocytes, Mononuclear/immunology, Aged, psoriatic arthritis, Sequence Analysis, RNA, Gene Expression Profiling, Arthritis, Psoriatic, psoriasis, scRNAseq, bioinformatics, RC581-607, Middle Aged, Immunity, Innate, Adaptive Immunity/genetics, differential expression analysis, inflammation, Case-Control Studies, Leukocytes, Mononuclear, Female, Immunity, Innate/genetics, Immunologic diseases. Allergy, Single-Cell Analysis, Transcriptome

File Description: application/pdf

Access URL: https://pubmed.ncbi.nlm.nih.gov/40084238
https://doaj.org/article/d18410b4645f456b9de830ee4d2619dc
https://curis.ku.dk/ws/files/432939722/fimmu_3_1490051.pdf
http://www.scopus.com/inward/record.url?scp=86000591793&partnerID=8YFLogxK
https://doi.org/10.3389/fimmu.2024.1490051
https://pure.au.dk/portal/en/publications/ae0ae16f-ab41-438d-926f-050c66b393a9

Innate immune sensing of danger signals: novel mechanism of heme complex-mediated lytic cell death

Authors: Chintan Chhatbar Michael Schulz Roman Sankowski

Source: Signal Transduct Target Ther
Signal Transduction and Targeted Therapy, Vol 9, Iss 1, Pp 1-3 (2024)

Subject Terms: Cell Death, QH301-705.5, Medicine, Humans, Animals, research-highlight, Immunity, Innate/genetics [MeSH], Research Highlight, Heme/genetics [MeSH], Immunity, Innate/immunology [MeSH], Humans [MeSH], Cell Death/immunology [MeSH], 631/250/262, Animals [MeSH], 631/250/2504, Cell Death/genetics [MeSH], Heme/immunology [MeSH], Heme, Biology (General), Immunity, Innate

File Description: pdf

Access URL: https://pubmed.ncbi.nlm.nih.gov/39349952
https://doaj.org/article/513232146ccf4c3da5161199a9d011da
https://repository.publisso.de/resource/frl:6503375

Different Innate Immune Responses in BALB/c and C57BL/6 Strains following Corneal Transplantation

Authors: Tim Bleul Xinyu Zhuang Antonia Hildebrand et al.

Source: Journal of Innate Immunity, Pp 1-11 (2020)

Subject Terms: Graft Rejection, 0301 basic medicine, balb/c, keratoplasty, macrophage, c57bl/6, Corneal Transplantation, Mice, 03 medical and health sciences, Anterior Eye Segment, Cell Movement, Hornhauttransplantation, Animals, Transplantation, Homologous, Genetic Predisposition to Disease, Internal medicine, Immunity, Innate/genetics [MeSH], Mice, Inbred BALB C [MeSH], Mice, Inbred C57BL [MeSH], Cell Movement [MeSH], Tomography, Optical Coherence [MeSH], Macrophages/immunology [MeSH], Keratoplasty, ADP-ribosyl Cyclase 1/metabolism [MeSH], Graft Rejection/immunology [MeSH], BALB/c, Corneal Transplantation [MeSH], Research Article, Corneal graft rejection, Anterior Eye Segment/diagnostic imaging [MeSH], Transplantation, Homologous [MeSH], Genetic Predisposition to Disease [MeSH], Anterior Eye Segment/immunology [MeSH], C57BL/6, Macrophage, Antigens, Differentiation/metabolism [MeSH], Animals [MeSH], Mice [MeSH], Genetic Background [MeSH], Graft Rejection/genetics [MeSH], Transplantation, Isogeneic [MeSH], Macrophage Activation [MeSH], Mice, Inbred BALB C, 0303 health sciences, Macrophages, corneal graft rejection, Macrophage Activation, RC31-1245, ADP-ribosyl Cyclase 1, Antigens, Differentiation, Immunity, Innate, 3. Good health, Mice, Inbred C57BL, Transplantation, Isogeneic, Makrophage, Medicine, Genetic Background, Tomography, Optical Coherence

File Description: pdf

Access URL: https://www.karger.com/Article/Pdf/509716
https://pubmed.ncbi.nlm.nih.gov/32906119
https://doaj.org/article/dab68f9856af45bdb5df3679f2a02093
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7879253
https://pubmed.ncbi.nlm.nih.gov/32906119/
https://www.scilit.net/article/0d4c0c227deff35dad5186e7e19a8676
https://repository.publisso.de/resource/frl:6479144

The transcription factor NRF2 enhances melanoma malignancy by blocking differentiation and inducing COX2 expression

Authors: Christina Jessen Julia K. C. Kreß Apoorva Baluapuri et al.

Source: Oncogene

Subject Terms: Male, 0301 basic medicine, Lung Neoplasms, Datasets as Topic, Adenocarcinoma of Lung, Cell Line, Tumor [MeSH], Immunity, Innate/genetics [MeSH], Gene Expression Regulation, Neoplastic/immunology [MeSH], Melanoma, Lung Neoplasms/genetics [MeSH], Tumor Escape/genetics [MeSH], NF-E2-Related Factor 2/metabolism [MeSH], NF-E2-Related Factor 2/genetics [MeSH], Activating Transcription Factor 4/metabolism [MeSH], Gene Knockout Techniques [MeSH], Male [MeSH], Adenocarcinoma of Lung/genetics [MeSH], Melanoma/pathology [MeSH], Lung Neoplasms/immunology [MeSH], Microphthalmia-Associated Transcription Factor/metabolism [MeSH], Skin Neoplasms/immunology [MeSH], Disease Models, Animal [MeSH], Female [MeSH], Oncogenes, Melanoma/genetics [MeSH], Datasets as Topic [MeSH], Humans [MeSH], Gene Knockdown Techniques [MeSH], Cell Differentiation/genetics [MeSH], Adenocarcinoma of Lung/immunology [MeSH], Skin Neoplasms/pathology [MeSH], Animals [MeSH], Mice [MeSH], Article, Skin Neoplasms/genetics [MeSH], Melanoma/immunology [MeSH], Lung Neoplasms/pathology [MeSH], Dinoprostone/metabolism [MeSH], Cyclooxygenase 2/metabolism [MeSH], Adenocarcinoma of Lung/pathology [MeSH], Dinoprostone, Gene Knockout Techniques, Mice, 03 medical and health sciences, Cell Line, Tumor, Animals, Humans, ddc:610, Microphthalmia-Associated Transcription Factor, 0303 health sciences, Cell Differentiation, Activating Transcription Factor 4, Immunity, Innate, 3. Good health, Gene Expression Regulation, Neoplastic, Disease Models, Animal, Cyclooxygenase 2, Gene Knockdown Techniques, Female, ddc:570

File Description: application/pdf

Access URL: https://www.nature.com/articles/s41388-020-01477-8.pdf
https://pubmed.ncbi.nlm.nih.gov/32978520
https://ora.ox.ac.uk/objects/uuid:86dc57a2-0392-477d-b201-b1d92016a4a8
https://doi.org/10.1038/s41388-020-01477-8
https://repository.publisso.de/resource/frl:6471342
https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-235064
https://opus.bibliothek.uni-wuerzburg.de/files/23506/s41388-020-01477-8.pdf
https://opus.bibliothek.uni-wuerzburg.de/frontdoor/index/index/docId/23506
https://doi.org/10.1038/s41388-020-01477-8

The temporal pattern of immune and inflammatory proteins prior to a recurrent coronary event in post-acute coronary syndrome patients

Authors: Maxime M. Vroegindewey Rohit M. Oemrawsingh Isabella Kardys et al.

Contributors: Maxime M. Vroegindewey Rohit M. Oemrawsingh Isabella Kardys et al.

Source: Biomarkers, 24, 2, pp. 199-205

Subject Terms: Inflammation/blood, EXPRESSION, Male, Proteomics, 0301 basic medicine, Health, Toxicology and Mutagenesis, Chemokine CXCL1, Clinical Biochemistry, Radboud University Medical Center, TRAIL, temporal pattern, Biochemistry, GRO-ALPHA, 03 medical and health sciences, Radboudumc 16: Vascular damage RIHS: Radboud Institute for Health Sciences, proteomics, Signaling Lymphocytic Activation Molecule Family, Receptors, TNF-Related Apoptosis-Inducing Ligand/genetics, Journal Article, Humans, Acute Coronary Syndrome, Aged, Inflammation, 0303 health sciences, Signaling Lymphocytic Activation Molecule Family/genetics, biomarkers, Middle Aged, proteins, Immunity, Innate, Chemokine CXCL1/genetics, 3. Good health, RECEPTORS, Receptors, TNF-Related Apoptosis-Inducing Ligand, ATHEROSCLEROSIS, immune and inflammatory system, Acute Coronary Syndrome/blood, Female, Immunity, Innate/genetics, Acute coronary syndrome, Biomarkers/blood, Biomarkers, EMC COEUR-09

File Description: image/pdf

Access URL: https://www.tandfonline.com/doi/pdf/10.1080/1354750X.2018.1539768?needAccess=true
https://pubmed.ncbi.nlm.nih.gov/30514120
https://hdl.handle.net/https://repository.ubn.ru.nl/handle/2066/203488
https://doi.org/10.1080/1354750X.2018.1539768
https://hdl.handle.net/11370/200b825b-a27b-40d9-90ce-add0fd3bfd06
https://research.rug.nl/en/publications/200b825b-a27b-40d9-90ce-add0fd3bfd06
https://doi.org/10.1080/1354750X.2018.1539768
https://repub.eur.nl/pub/117040
https://www.narcis.nl/publication/RecordID/oai%3Arepub.eur.nl%3A113070
https://www.tandfonline.com/doi/full/10.1080/1354750X.2018.1539768
https://www.ncbi.nlm.nih.gov/pubmed/30514120
https://repub.eur.nl/pub/117040/Repub_117040_O-A.pdf
https://repository.ubn.ru.nl/handle/2066/203488
https://dspace.library.uu.nl/handle/1874/394278
https://repository.ubn.ru.nl//bitstream/handle/2066/203488/203488.pdf
https://hdl.handle.net/2066/203488
https://discovery-pp.ucl.ac.uk/id/eprint/10064183/

CD127+ CD94+ innate lymphoid cells expressing granulysin and perforin are expanded in patients with Crohn’s disease

Authors: W. A. Bemelman M Becker Chantal M. A. Kradolfer et al.

Contributors: W. A. Bemelman M Becker Chantal M. A. Kradolfer et al.

Source: Nat Commun
Nature Communications, Vol 12, Iss 1, Pp 1-11 (2021)

Subject Terms: Antigens, Differentiation, T-Lymphocyte, 0301 basic medicine, Cells, Science, Innate/genetics, Lymphocytes/immunology, Enzyme-Linked Immunosorbent Assay, Real-Time Polymerase Chain Reaction, Antigens, Differentiation, T-Lymphocyte/genetics, Article, Interleukin-7 Receptor alpha Subunit, 03 medical and health sciences, 0302 clinical medicine, Crohn Disease, Humans, Lymphocytes, Antigens, NK Cell Lectin-Like Receptor Subfamily D/metabolism, Cells, Cultured, Inflammation, Crohn Disease/genetics, Cultured, Perforin, Immunity, Perforin/genetics, Inflammation/immunology, Flow Cytometry, Immunity, Innate, 3. Good health, T-Lymphocyte/genetics, Differentiation, Interleukin-7 Receptor alpha Subunit/metabolism, Immunity, Innate/genetics, NK Cell Lectin-Like Receptor Subfamily D

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Access URL: https://www.nature.com/articles/s41467-021-26187-x.pdf
https://pubmed.ncbi.nlm.nih.gov/34615883
https://doaj.org/article/1fc6af1d913c45e8876e1267a084185f
https://research-portal.uu.nl/en/publications/36cd44ad-d4ce-4b0e-a4a7-c5ca87943d1a
https://doi.org/10.1038/s41467-021-26187-x
https://researchinformation.amsterdamumc.org/en/publications/cd127-cd94-innate-lymphoid-cells-expressing-granulysin-and-perfor
https://pubmed.ncbi.nlm.nih.gov/34615883/
http://www-nature-com-443.webvpn.bjmu.tsg211.com/articles/s41467-021-26187-x
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8494908
https://repub.eur.nl/pub/136943
https://www.nature.com/articles/s41467-021-26187-x.pdf
https://dspace.library.uu.nl/handle/1874/413632

Automated machine learning optimizes and accelerates predictive modeling from COVID-19 high throughput datasets

Authors: Vincenzo Lagani Ioannis Tsamardinos Naomi Thomson et al.

Source: Sci Rep
Scientific Reports, Vol 11, Iss 1, Pp 1-13 (2021)
Papoutsoglou, G, Karaglani, M, Lagani, V, Thomson, N, Røe, O D, Tsamardinos, I & Chatzaki, E 2021, 'Automated machine learning optimizes and accelerates predictive modeling from COVID-19 high throughput datasets', Scientific Reports, vol. 11, no. 1, 15107. https://doi.org/10.1038/s41598-021-94501-0

Subject Terms: Proteomics, 0301 basic medicine, COVID-19/diagnosis, Databases, Factual, Science, Severity of Illness Index, Article, Machine Learning, Interferon-gamma, 03 medical and health sciences, SARS-CoV-2/genetics, Databases, Genetic, Humans, Metabolomics, Computer Simulation, Databases, Protein, 0303 health sciences, SARS-CoV-2, Gene Expression Profiling, COVID-19, Prognosis, Immunity, Innate, 3. Good health, ROC Curve, Signal Transduction/genetics, Medicine, Immunity, Innate/genetics, Biomarkers/blood, Interferon-gamma/blood, Biomarkers, Software, Signal Transduction

File Description: application/pdf

Access URL: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8302755
https://pubmed.ncbi.nlm.nih.gov/34302024
https://doaj.org/article/760ad289441a49f7b30b75092e2b03be
https://www.nature.com/articles/s41598-021-94501-0
https://vbn.aau.dk/da/publications/automated-machine-learning-optimizes-and-accelerates-predictive-m
https://www.nature.com/articles/s41598-021-94501-0.pdf
https://pubmed.ncbi.nlm.nih.gov/34302024/
https://search.bvsalud.org/global-literature-on-novel-coronavirus-2019-ncov/resource/pt/covidwho-1322506
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8302755/

Vpx enhances innate immune responses independently of SAMHD1 during HIV-1 infection

Authors: Oya Cingöz Nicolas D. Arnow Mireia Puig Torrents et al.

Source: Retrovirology
Retrovirology, Vol 18, Iss 1, Pp 1-15 (2021)

Subject Terms: 0301 basic medicine, Macrophage, THP-1 Cells, MDM, HIV Infections, Virus Replication, Cell Line, ISG, SAM Domain and HD Domain-Containing Protein 1, 03 medical and health sciences, ISRE, Humans, Viral Regulatory and Accessory Proteins, ddc:610, Innate immunity, 0303 health sciences, Research, Immunity, Innate/genetics [MeSH], Interferon, Cell Line [MeSH], SAM Domain and HD Domain-Containing Protein 1/metabolism [MeSH], THP-1 Cells [MeSH], Virus Replication [MeSH], Host-Pathogen Interactions/immunology [MeSH], HIV-2/genetics [MeSH], HIV-2/physiology [MeSH], Viral Regulatory and Accessory Proteins/genetics [MeSH], HIV Infections/immunology [MeSH], Humans [MeSH], HIV Infections/genetics [MeSH], Infection, SAM Domain and HD Domain-Containing Protein 1/immunology [MeSH], THP-1, Leukocytes, Mononuclear/virology [MeSH], SAM Domain and HD Domain-Containing Protein 1/genetics [MeSH], Proteolysis [MeSH], Host-Pathogen Interactions/genetics [MeSH], Viral Regulatory and Accessory Proteins/immunology [MeSH], HEK293 Cells [MeSH], RC581-607, Immunity, Innate, 3. Good health, HEK293 Cells, HIV-2, Host-Pathogen Interactions, Proteolysis, Leukocytes, Mononuclear, Immunologic diseases. Allergy, 610 Medizin und Gesundheit

File Description: application/pdf

Access URL: https://pubmed.ncbi.nlm.nih.gov/33563288
https://doaj.org/article/5710cfeed3474dbcba859ca5edfb2a2e
https://link.springer.com/content/pdf/10.1186/s12977-021-00548-2.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7871410
https://europepmc.org/article/MED/33563288
https://retrovirology.biomedcentral.com/articles/10.1186/s12977-021-00548-2
https://edoc.rki.de/handle/176904/7837
https://pubmed.ncbi.nlm.nih.gov/33563288/
http://edoc.rki.de/176904/7837
https://repository.publisso.de/resource/frl:6465812

Genome-to-genome analysis highlights the effect of the human innate and adaptive immune systems on the hepatitis C virus

Authors: M Azim Ansari Vincent Pedergnana Camilla L C Ip et al.

Contributors: M Azim Ansari Vincent Pedergnana Camilla L C Ip et al.

Source: Nat Genet
Nature genetics, vol. 49, no. 5, pp. 666-673

Subject Terms: 0301 basic medicine, IL28B, Hepacivirus, Adaptive Immunity, Viral Nonstructural Proteins, HLA Antigens, INFECTION, Innate, WIDE ASSOCIATION, HCV CLEARANCE, Viral, Chronic, Genetics & Heredity, Principal Component Analysis, 0303 health sciences, Genome, [SDV.MHEP] Life Sciences [q-bio]/Human health and pathology, GENETIC-VARIATION, 11 Medical And Health Sciences, Viral Load, Hepatitis C, 3. Good health, Host-Pathogen Interactions, Life Sciences & Biomedicine, Human, INTERFERON, Genotype, name=Genetics, Genome, Viral, Adaptive Immunity/genetics, Alleles, Genetic Variation, Genome, Human/genetics, Genome, Viral/genetics, HLA Antigens/genetics, Hepacivirus/genetics, Hepacivirus/physiology, Hepatitis C, Chronic/genetics, Hepatitis C, Chronic/virology, Host-Pathogen Interactions/genetics, Humans, Immunity, Innate/genetics, Interleukins/genetics, Logistic Models, Viral Load/genetics, Viral Nonstructural Proteins/genetics, Article, 03 medical and health sciences, DIRECT-ACTING ANTIVIRALS, Science & Technology, Genome, Human, Interleukins, Immunity, DISEASE ASSOCIATION, 06 Biological Sciences, Hepatitis C, Chronic, Immunity, Innate, GENOTYPES 1, RESISTANCE, STOP-HCV Consortium, Developmental Biology

File Description: application/pdf; 666 - +

Access URL: https://www.nature.com/articles/ng.3835.pdf
https://pubmed.ncbi.nlm.nih.gov/28394351
https://hal.science/hal-03691457v1
https://doi.org/10.1038/ng.3835
https://eprints.gla.ac.uk/138360/7/138360.pdf
https://dl.uswr.ac.ir/handle/Hannan/31989
https://serval.unil.ch/notice/serval:BIB_179F1128ACCA
https://eprints.soton.ac.uk/413488/
http://eprints.gla.ac.uk/138360/
https://www.cabdirect.org/cabdirect/abstract/20173180744
http://europepmc.org/abstract/MED/28394351
http://hdl.handle.net/10044/1/44869
https://ora.ox.ac.uk/objects/uuid:f82608ed-7142-4e4c-9d07-789b16d72c09
https://doi.org/10.1038/ng.3835
http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_179F1128ACCA9
http://europepmc.org/abstract/med/28394351
https://serval.unil.ch/resource/serval:BIB_179F1128ACCA.P001/REF.pdf
https://serval.unil.ch/notice/serval:BIB_179F1128ACCA

Innate immunity and the evolution of resistance to an emerging infectious disease in a wild bird

Authors: Bonneaud, Camille Balenger, Susan L. Zhang, Jiangwen et al.

Source: Molecular Ecology. 21:2628-2639

Subject Terms: 0301 basic medicine, Immunity, Innate - Genetics, Molecular Sequence Data, Wild, Mycoplasma gallisepticum, Animals, Wild, 03 medical and health sciences, time-series microarray, Finches - Genetics - Immunology - Microbiology, cell-mediated immunity, Animals, Mycoplasma Infections, Bird Diseases - Microbiology, Disease Resistance, Oligonucleotide Array Sequence Analysis, 0303 health sciences, immunosuppression, Mycoplasma Gallisepticum - Pathogenicity, Bird Diseases, Immunity, Mycoplasma Infections - Immunology - Veterinary, Biological Evolution, Immunity, Innate, United States, 3. Good health, microevolution, Disease Resistance - Genetics - Immunology, Gene Expression Regulation, Innate - Genetics, Finches

File Description: application/pdf

Access URL: https://dash.harvard.edu/bitstream/1/11688779/1/edwards-innate-immunity.pdf
https://pubmed.ncbi.nlm.nih.gov/22512302
https://experts.umn.edu/en/publications/innate-immunity-and-the-evolution-of-resistance-to-an-emerging-in
https://dash.harvard.edu/bitstream/handle/1/11688779/edwards-innate-immunity.pdf?sequence=1
https://dash.harvard.edu/handle/1/11688779
https://dash.harvard.edu/bitstream/1/11688779/1/edwards-innate-immunity.pdf
https://onlinelibrary.wiley.com/doi/10.1111/j.1365-294X.2012.05551.x
https://www.auburn.edu/academic/cosam/faculty/biology/hill/lab/documents/197.pdf
http://hdl.handle.net/10722/180753

Leprosy and the natural selection for psoriasis

Authors: Bassukas, I. D. Gaitanis, G. Hundeiker, M.

Source: Medical Hypotheses. 78:183-190

Subject Terms: 0301 basic medicine, European Continental Ancestry Group/*genetics, Receptor, ErbB-2, Selection, Genetic, Models, Immunological, White People, 03 medical and health sciences, 0302 clinical medicine, Leprosy, Leprosy/epidemiology/genetics/*immunology, Prevalence, Immunity, Innate/genetics/*immunology, Humans, Psoriasis, Genetic Predisposition to Disease, Toll-Like Receptors/immunology, Mitogen-Activated Protein Kinase 3, Psoriasis/epidemiology/genetics/*immunology, Toll-Like Receptors, Genetic Predisposition to Disease/*genetics, Mitogen-Activated Protein Kinase 3/metabolism, Receptor, erbB-2/metabolism, History, Medieval, Immunity, Innate, 3. Good health

Access URL: https://pubmed.ncbi.nlm.nih.gov/22079652
https://www.ncbi.nlm.nih.gov/pubmed/22079652
http://europepmc.org/abstract/MED/22079652
https://www.sciencedirect.com/science/article/abs/pii/S0306987711005366
https://pubmed.ncbi.nlm.nih.gov/22079652/
http://www.sciencedirect.com/science/article/pii/S0306987711005366
http://olympias.lib.uoi.gr/jspui/handle/123456789/20445

Host Immune and Apoptotic Responses to Avian Influenza Virus H9N2 in Human Tracheobronchial Epithelial Cells

Authors: Harper, R Yang, Z Gao, W et al.

Source: American Journal of Respiratory Cell and Molecular Biology. 44:24-33

Subject Terms: 0301 basic medicine, CASP8 and FADD-Like Apoptosis Regulating Protein, Host responses, Avian influenza virus, Apoptosis, Bronchi, Apoptosis - genetics, Birds, DEAD-box RNA Helicases, 03 medical and health sciences, Cytopathogenic Effect, Viral, Bronchial epithelial cells, Influenza, Human, Influenza A Virus, Influenza A Virus, H9N2 Subtype, Animals, Humans, Bronchi - immunology - pathology - virology, Cells, Cultured, 0303 health sciences, Epithelial Cells - immunology - pathology - virology, Influenza A Virus, H9N2 Subtype - growth and development - pathogenicity, Immunity, Epithelial Cells, H9N2, Immunity, Innate, 3. Good health, Enzyme Activation, Innate - genetics, Gene Expression Regulation, Caspases, Influenza in Birds, Immunity, Innate - genetics, Cytokines, Chemokines, Inflammation Mediators, H9N2 Subtype - growth and development - pathogenicity, BH3 Interacting Domain Death Agonist Protein

Access URL: https://europepmc.org/articles/pmc3159084?pdf=render
https://pubmed.ncbi.nlm.nih.gov/20118223
http://hdl.handle.net/10722/125148

Mitochondrial DNA haplogroups influence the therapeutic response to riboflavin in migraineurs

Authors: DI LORENZO C PIERELLI, Francesco COPPOLA, GIANLUCA et al.

Source: Neurology. 72:1588-1594

Subject Terms: Adult, Male, Mitochondrial Diseases, Mutation/genetics, Genotype, Migraine Disorders, Riboflavin, Haplotypes/genetics, Mitochondrial Diseases/complications/genetics/metabolism, DNA Mutational Analysis, Migraine with Aura, Migraine with Aura/drug therapy/genetics/metabolism, Sciences de la santé humaine, DNA, Mitochondrial, Young Adult, 03 medical and health sciences, 0302 clinical medicine, Neurologie, Riboflavin/administration & dosage/adverse effects, Humans, Genetic Predisposition to Disease, Genetic Testing, Human health sciences, Migraine Disorders/drug therapy/genetics/metabolism, Genetic Screening, Genetic Predisposition to Disease/genetics, Brain, Middle Aged, Immunity, Innate, 3. Good health, Treatment Outcome, Neurology, Haplotypes, Mutation, Female, DNA, Mitochondrial/analysis/genetics, Immunity, Innate/genetics, Brain/drug effects/metabolism/physiopathology

Access URL: https://pubmed.ncbi.nlm.nih.gov/19414726
http://n.neurology.org/content/72/18/1588
https://n.neurology.org/content/72/18/1588
https://n.neurology.org/content/72/18/1588.full.pdf
https://orbi.uliege.be/handle/2268/23517
https://www.ncbi.nlm.nih.gov/pubmed/19414726
https://www.cabdirect.org/abstracts/20093152082.html
https://hdl.handle.net/2268/23517
https://doi.org/10.1212/WNL.0b013e3181a41269
https://hdl.handle.net/11573/361720
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=000265782600010&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=0c7ff228ccbaaa74236f48834a34396a
https://doi.org/10.1212/WNL.0b013e3181a41269

Determination of sex and scrapie resistance genotype in preimplantation ovine embryos

Authors: Guignot, Florence Baril, Gérard Dupont, Francis et al.

Contributors: Guignot, Florence Baril, Gérard Dupont, Francis et al.

Source: Molecular Reproduction and Development 2 (76), 183-190. (2009)

Subject Terms: 0301 basic medicine, Sex Determination Analysis, [SDV.BID.SPT]Life Sciences [q-bio]/Biodiversity/Systematics, [SDV.BA] Life Sciences [q-bio]/Animal biology, Biopsy, reproduction animale, biopsy, dpi, PrP genotyping, sex diagnosis, vitrification, transfer, [SDV.GEN] Life Sciences [q-bio]/Genetics, blastocyste, 0302 clinical medicine, ovin, Biologie de la reproduction, Reproductive Biology, détermination du sexe, prp genotyping, [SDV.BA]Life Sciences [q-bio]/Animal biology, [SDV.BID.EVO]Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE], Phylogenetics and taxonomy, Life sciences, Sciences du vivant, génotype, Genotype, Scrapie/genetics/immunology, Prions, transfert d'embryon, génotype résistant, biopsie, prion, Veterinary medicine & animal health, 03 medical and health sciences, [SDV.BID.SPT] Life Sciences [q-bio]/Biodiversity/Systematics, Phylogenetics and taxonomy, Prions/genetics, [SDV.BID.EVO] Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE], Animals, Embryo Transfer/methods, DNA Primers, Cryopreservation, [SDV.GEN]Life Sciences [q-bio]/Genetics, DNA Primers/genetics, Sheep, scrapie, Embryo Transfer, Embryo, Mammalian, Survival Analysis, Embryo, Mammalian/embryology, Médecine vétérinaire & santé animale, Immunity, Innate, embryon, Immunity, Innate/genetics, Sex Determination (Analysis), Scrapie

File Description: application/pdf

Access URL: https://hal.archives-ouvertes.fr/hal-02360345/file/Guignot_MRD_2009_Postprint_1.pdf
https://pubmed.ncbi.nlm.nih.gov/18543282
https://www.bib.irb.hr/420289
https://www.cabdirect.org/cabdirect/abstract/20093036795
https://www.ncbi.nlm.nih.gov/pubmed/18543282
https://hal.archives-ouvertes.fr/hal-02360345/document
https://orbi.uliege.be/handle/2268/21373
https://pubmed.ncbi.nlm.nih.gov/18543282/
https://core.ac.uk/display/13349860
http://prodinra.inra.fr/record/30013

BXSB/long-livedIs a Recombinant Inbred Strain Containing Powerful Disease Suppressor Loci

Authors: Haywood, M. E. Gabriel, Luisa Rose, S. J. et al.

Source: Journal of Immunology, Vol. 179, No 4 (2007) pp. 2428-2434

Subject Terms: Recombination, Genetic, 0301 basic medicine, 0303 health sciences, Quantitative Trait Loci, Mice, Inbred Strains, ddc:616.07, 616.07, Breeding, Immunity, Innate/*genetics, Polymorphism, Single Nucleotide, Chromosomes, Immunity, Innate, 3. Good health, Recombination, Genetic/immunology, Mice, 03 medical and health sciences, Lupus Erythematosus, Systemic/genetics/immunology, Species Specificity, Chromosomes/*genetics/immunology, Animals, Lupus Erythematosus, Systemic, Genetic Predisposition to Disease, Quantitative Trait Loci/*genetics/immunology

File Description: application/pdf

Access URL: http://www.jimmunol.org/content/jimmunol/179/4/2428.full.pdf
https://pubmed.ncbi.nlm.nih.gov/17675504
https://archive-ouverte.unige.ch/unige:11294
https://researchportal.bath.ac.uk/en/publications/bxsblong-lived-is-a-recombinant-inbred-strain-containing-powerful-2
http://opus.bath.ac.uk/19266/
https://www.ncbi.nlm.nih.gov/pubmed/17675504
https://www.jimmunol.org/lookup/doi/10.4049/jimmunol.179.4.2428
https://europepmc.org/article/MED/17675504
https://archive-ouverte.unige.ch/unige:11294

Evolutionary Dynamics of Immune-Related Genes and Pathways in Disease-Vector Mosquitoes

Authors: Waterhouse, Robert Kriventseva, Evgenia Meister, Stephen et al.

Contributors: Waterhouse, Robert Kriventseva, Evgenia Meister, Stephen et al.

Source: Science, Vol. 316, No 5832 (2007) pp. 1738-1743

Subject Terms: 576.5, 0301 basic medicine, Carrier Proteins/genetics/physiology, Insect Vectors/ genetics/immunology, Epidemiology, Evolution, Medical Immunology, Genes, Insect, Biochemistry, Animal Diseases, Evolution, Molecular, 03 medical and health sciences, Species Specificity, Aedes, Anopheles, Genetics, Animals, ddc:576.5, Anopheles/ genetics/immunology, Melanins, Antimicrobial Cationic Peptides/physiology, 0303 health sciences, Immunity, Innate/ genetics, Melanins/metabolism, Immunity, Innate, Aedes/ genetics/immunology, Insect Vectors, Malaria, 3. Good health, Drosophila melanogaster, Multigene Family, Insect Proteins/genetics/physiology, Drosophila melanogaster/genetics/immunology, Malaria/transmission, Insect Proteins, Carrier Proteins, Entomology, Antimicrobial Cationic Peptides, Signal Transduction

File Description: application/pdf

Access URL: https://dr.lib.iastate.edu/bitstreams/8445d617-7239-40bf-af42-2ec0ed91e719/download
https://pubmed.ncbi.nlm.nih.gov/17588928
https://jhu.pure.elsevier.com/en/publications/evolutionary-dynamics-of-immune-related-genes-and-pathways-in-dis-5
https://www3.nd.edu/~dseverso/Pubs/Waterhouse_Science_2007.pdf
https://science.sciencemag.org/content/316/5832/1738.full
https://lib.dr.iastate.edu/cgi/viewcontent.cgi?article=1032&context=ent_pubs
http://europepmc.org/articles/PMC2042107
https://core.ac.uk/display/38935294
https://archive-ouverte.unige.ch/unige:9273

Enhancing disease resistances of Super Hybrid Rice with four antifungal genes

Authors: GuoYing Xiao Baojian Li LongPing Yuan et al.

Source: Science in China Series C: Life Sciences. 50:31-39

Subject Terms: 2. Zero hunger, 0106 biological sciences, 0301 basic medicine, Immunity, Innate - Genetics, Immunity, Fungi - Genetics, Fungi, Genetically Modified, Oryza, Plants, Plants, Genetically Modified, 01 natural sciences, Immunity, Innate, 03 medical and health sciences, Plant Diseases - Genetics - Microbiology, Innate - Genetics, Oryza Sativa - Genetics - Microbiology, Plant Diseases

Access URL: https://pubmed.ncbi.nlm.nih.gov/17393080
https://link.springer.com/article/10.1007%2Fs11427-007-0001-9
https://core.ac.uk/display/38015299
https://en.cnki.com.cn/Article_en/CJFDTOTAL-JCXG200701005.htm
https://europepmc.org/article/MED/17393080
https://www.ncbi.nlm.nih.gov/pubmed/17393080
http://hdl.handle.net/10722/179797