Suchergebnisse - "Hyaluronan Receptors metabolism"

Targeting CD44 and other pleiotropic co-receptors as a means for broad inhibition of tumor growth and metastasis

Autoren: Lisa-Marie Mehner Leonel Munoz-Sagredo Steffen Joachim Sonnentag et al.

Quelle: Clin Exp Metastasis
Clinical and Experimental Metastasis

Schlagwörter: 0301 basic medicine, ddc:610, 0303 health sciences, Antineoplastic Agents, Review, Medicine & health, 3. Good health, 03 medical and health sciences, Hyaluronan Receptors, Neoplasms, Humans, Animals, Molecular Targeted Therapy, Neoplasm Metastasis, Cancer, Pleiotropicity, Humans [MeSH], Metastasis, Neoplasm Metastasis [MeSH], Hyaluronan Receptors/metabolism [MeSH], Antineoplastic Agents/pharmacology [MeSH], Animals [MeSH], Neoplasms/metabolism [MeSH], Neoplasms/pathology [MeSH], CD44, Antineoplastic Agents/therapeutic use [MeSH], Co-receptors, Signal Transduction [MeSH], Molecular Targeted Therapy/methods [MeSH], Signal Transduction

Dateibeschreibung: application/pdf

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38761292
https://publikationen.bibliothek.kit.edu/1000171434
https://publikationen.bibliothek.kit.edu/1000171434/153027455
https://doi.org/10.5445/IR/1000171434
https://repository.publisso.de/resource/frl:6524666

Correlation of microscopic tumor extension with tumor microenvironment in esophageal cancer patients

Autoren: Benjamin Terfa Igbo Christina Jentsch Annett Linge et al.

Quelle: Strahlenther Onkol

Schlagwörter: Male, 0301 basic medicine, Esophageal Neoplasms, Middle Aged, Hypoxia-Inducible Factor 1, alpha Subunit, Neoadjuvant Therapy, 03 medical and health sciences, Hyaluronan Receptors, Ki-67 Antigen, 0302 clinical medicine, Fiducial Markers, Focal Adhesion Kinase 1, Tumor Microenvironment, Biomarkers, Tumor, Humans, Original Article, Female, Female [MeSH], Hypoxia, Esophageal Neoplasms/therapy [MeSH], Aged [MeSH], Humans [MeSH], Hyaluronan Receptors/metabolism [MeSH], Multiparametric analysis, Fiducial Markers [MeSH], Middle Aged [MeSH], Esophageal Neoplasms/pathology [MeSH], Tumor stem cells, Ki-67 Antigen/analysis [MeSH], Neoadjuvant Therapy [MeSH], Clinical Target Volume, Hyaluronan Receptors/analysis [MeSH], Male [MeSH], Radiotherapy, Image-Guided [MeSH], Hypoxia-Inducible Factor 1, alpha Subunit/metabolism [MeSH], Proliferation, Immunohistochemical analysis, Biomarkers, Tumor/analysis [MeSH], Focal Adhesion Kinase 1/metabolism [MeSH], Tumor Microenvironment [MeSH], Aged, Radiotherapy, Image-Guided

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38727811
https://repository.publisso.de/resource/frl:6503627

Identification of Two Distinct Stem Cell Clusters, Lrig1-Derived and Wnt/CD44-Dependent, in Corneal Epithelium

Autoren: Barnes, Laurent Konstantinou, Evangelia Saurat, Jean-Hilaire et al.

Quelle: Int J Mol Sci
International journal of molecular sciences, vol. 26, no. 13

Schlagwörter: Stem Cells / metabolism, Nerve Tissue Proteins, 616.07, Ablation, Cornea, Epithelium, Corneal / metabolism, Wnt, Mice, Lrig1, Membrane Glycoproteins / metabolism, Animals, CD44, Wnt Signaling Pathway, Mice, Knockout, Stem cell, Communication, Hyaluronan Receptors / genetics, Epithelium, Corneal / cytology, Hyaluronan Receptors/metabolism, Hyaluronan Receptors/genetics, Epithelium, Corneal/metabolism, Epithelium, Corneal/cytology, Stem Cells/metabolism, Stem Cells/cytology, Membrane Glycoproteins/metabolism, Membrane Glycoproteins/genetics, Mice, Inbred C57BL, beta Catenin/metabolism, ablation, cornea, stem cell, tamoxifen, Tamoxifen, Hyaluronan Receptors / metabolism, Stem Cells / cytology, Membrane Glycoproteins / genetics, Beta Catenin / metabolism

Dateibeschreibung: application/pdf

Zugangs-URL: https://serval.unil.ch/resource/serval:BIB_7F9110C26982.P001/REF.pdf
http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_7F9110C269824
https://serval.unil.ch/notice/serval:BIB_7F9110C26982

CD44 signaling in Müller cells impacts photoreceptor function and survival in healthy and diseased retinas

Autoren: Ayten, Monika Straub, Tobias Kaplan, Lew et al.

Quelle: J Neuroinflammation
Journal of Neuroinflammation, Vol 21, Iss 1, Pp 1-16 (2024)

Schlagwörter: Müller cells, Mice, Knockout, 0301 basic medicine, 0303 health sciences, Cell Survival, Research, SLC1A2, Ependymoglial Cells, Mice, Transgenic, Retina, Retinitis pigmentosa, Mice, Inbred C57BL, Mice, 03 medical and health sciences, Hyaluronan Receptors, Animals, Mice, Inbred C57BL [MeSH], Retinitis Pigmentosa/genetics [MeSH], Glutamate, Gliosis, Mice, Transgenic [MeSH], Signal Transduction/physiology [MeSH], CD44, Retina/pathology [MeSH], Neuroinflammation in the Retina, Retinitis Pigmentosa/pathology [MeSH], Photoreceptor Cells, Vertebrate/metabolism [MeSH], Ependymoglial Cells/metabolism [MeSH], Retinitis Pigmentosa/metabolism [MeSH], Inflammation, Hyaluronan Receptors/genetics [MeSH], Hyaluronan Receptors/metabolism [MeSH], Animals [MeSH], Mice, Knockout [MeSH], Mice [MeSH], Retina/metabolism [MeSH], Cell Survival/physiology [MeSH], Neurology. Diseases of the nervous system, RC346-429, Retinitis Pigmentosa, Signal Transduction, Photoreceptor Cells, Vertebrate

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/39095775
https://doaj.org/article/c354d0f8e4954bfb929ca8e81e3fbbff
https://repository.publisso.de/resource/frl:6507532

Small cell lung cancer stem cells display mesenchymal properties and exploit immune checkpoint pathways in activated cytotoxic T lymphocytes

Autoren: Feyza Gul Ozbay Aysegul Uner Dorina Esendagli et al.

Quelle: Cancer Immunol Immunother

Schlagwörter: Cancer Research, Lung Neoplasms, Apoptosis, Mesenchymal Stem Cells, CD8-Positive T-Lymphocytes, Programmed Cell Death 1 Ligand 2 Protein, Small Cell Lung Carcinoma, B7-H1 Antigen, 3. Good health, 03 medical and health sciences, Hyaluronan Receptors, 0302 clinical medicine, Neoplastic Stem Cells, Tumor Cells, Cultured, Humans, Neoplastic Stem Cells/pathology [MeSH], Cancer stem cell, Neoplastic Stem Cells/metabolism [MeSH], Apoptosis [MeSH], B7-H1 Antigen/metabolism [MeSH], Original Article, Tumor Cells, Cultured [MeSH], TIM-3, Lung Neoplasms/immunology [MeSH], Small Cell Lung Carcinoma/pathology [MeSH], CD8-Positive T-Lymphocytes/immunology [MeSH], LAG3, Mesenchymal Stem Cells/immunology [MeSH], Cell Proliferation [MeSH], Mesenchymal Stem Cells/pathology [MeSH], T-Lymphocytes, Cytotoxic/immunology [MeSH], Neoplastic Stem Cells/immunology [MeSH], Humans [MeSH], Small Cell Lung Carcinoma/metabolism [MeSH], Hyaluronan Receptors/metabolism [MeSH], Programmed Cell Death 1 Ligand 2 Protein/metabolism [MeSH], PD-1, Mesenchymal Stem Cells/metabolism [MeSH], Immunotherapy, Lung Neoplasms/pathology [MeSH], Lung cancer, Lung Neoplasms/metabolism [MeSH], Small Cell Lung Carcinoma/immunology [MeSH], Cell Proliferation, T-Lymphocytes, Cytotoxic

Dateibeschreibung: application/pdf

Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00262-021-02998-1.pdf
https://pubmed.ncbi.nlm.nih.gov/34228218
https://link.springer.com/content/pdf/10.1007/s00262-021-02998-1.pdf
https://pubmed.ncbi.nlm.nih.gov/34228218/
https://europepmc.org/article/MED/34228218
https://www.ncbi.nlm.nih.gov/pubmed/34228218
https://link.springer.com/article/10.1007/s00262-021-02998-1
https://edoc.mdc-berlin.de/20413/
https://edoc.mdc-berlin.de/id/eprint/20413/2/20413suppl.pdf
https://repository.publisso.de/resource/frl:6451417
https://aperta.ulakbim.gov.tr/record/239058

Differential expression of stem cell markers in proliferating cells in glioma

Autoren: Marten Rehfeld Jakob Matschke Christian Hagel et al.

Quelle: J Cancer Res Clin Oncol

Schlagwörter: 0301 basic medicine, 0303 health sciences, Brain Neoplasms, Lewis X Antigen, RNA-Binding Proteins, Nerve Tissue Proteins, Glioma, 3. Good health, Gene Expression Regulation, Neoplastic, Nestin, 03 medical and health sciences, Hyaluronan Receptors, Gene Expression Regulation, Neoplastic [MeSH], Lewis X Antigen/metabolism [MeSH], Cell Proliferation [MeSH], Glioma/metabolism [MeSH], Original Article – Cancer Research, Neoplastic Stem Cells/pathology [MeSH], RNA-Binding Proteins/metabolism [MeSH], Humans [MeSH], Nerve Tissue Proteins/metabolism [MeSH], Nestin/metabolism [MeSH], AC133 Antigen/metabolism [MeSH], Hyaluronan Receptors/metabolism [MeSH], Stem cells, Expression profile, Neoplastic Stem Cells/metabolism [MeSH], Glioblastoma, Glioma/pathology [MeSH], Biomarkers, Tumor/metabolism [MeSH], Proliferating cells, Ki67, Brain Neoplasms/metabolism [MeSH], Brain Neoplasms/pathology [MeSH], Biomarkers, Tumor, Neoplastic Stem Cells, Humans, AC133 Antigen, Cell Proliferation

Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00432-021-03704-5.pdf
https://pubmed.ncbi.nlm.nih.gov/34170383
https://link.springer.com/content/pdf/10.1007/s00432-021-03704-5.pdf
https://europepmc.org/article/MED/34170383
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8397690
https://link.springer.com/article/10.1007/s00432-021-03704-5
https://pubmed.ncbi.nlm.nih.gov/34170383/
https://repository.publisso.de/resource/frl:6449844

CD44v3-Positive Intermediate Progenitor Cells Contribute to Airway Goblet Cell Hyperplasia

Autoren: Joo Heon Yoon Ji-Suk Ahn Augustine M.K. Choi et al.

Weitere Verfasser: Joo Heon Yoon Ji-Suk Ahn Augustine M.K. Choi et al.

Quelle: American Journal of Respiratory Cell and Molecular Biology. 64:247-259

Schlagwörter: Hyperplasia / metabolism, Respiratory System, Hypersensitivity / pathology, Stem Cells / pathology, Inflammation / pathology, 0302 clinical medicine, Biomarkers / metabolism, Respiratory System / metabolism, CD44, Exons / genetics, Cells, Cultured, Respiratory System / pathology, Cultured, Stem Cells, 05 social sciences, Cell Differentiation, Hyperplasia / pathology, Exons, Up-Regulation, 3. Good health, Hyaluronan Receptors, Stem Cells / metabolism, Goblet Cells, Goblet Cells / metabolism, Hypersensitivity / metabolism, Epithelial Cells / metabolism, Mucins / metabolism, Cells, Inflammation / metabolism, Epithelial Cells / pathology, 03 medical and health sciences, allergic airway disease, 0502 economics and business, Hypersensitivity, Humans, RNA, Messenger, Goblet Cells / pathology, Nasal Mucosa / pathology, Hyaluronan Receptors / metabolism, Nasal Mucosa / metabolism, Cell Proliferation, Inflammation, Messenger / genetics, Hyperplasia, intermediate progenitor cells, Mucins, human nasal epithelial cells, Epithelial Cells, Cell Proliferation / physiology, Nasal Mucosa, Up-Regulation / physiology, RNA, Cell Differentiation / physiology, interleukin-4, Biomarkers

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/33264080

Fat1 deletion promotes hybrid EMT state, tumour stemness and metastasis

Autoren: Frédérique De Cock Françoise Helmbacher Pedro Jaén et al.

Weitere Verfasser: Frédérique De Cock Françoise Helmbacher Pedro Jaén et al.

Quelle: Zaguán. Repositorio Digital de la Universidad de Zaragoza
instname
RISalud-ANDALUCIA. Repositorio Institucional de Salud de Andalucía
Universidad de Zaragoza
Zaguán: Repositorio Digital de la Universidad de Zaragoza
Nature (London)
Nature

Schlagwörter: Proteomics, 0301 basic medicine, Skin Neoplasms, Lung Neoplasms, [SDV]Life Sciences [q-bio], SOX2, Mesoderm, Mice, Neoplasms, Hyaluronan Receptors -- metabolism, RECURRENT, CD44, Neoplasm Metastasis, Transcription Factors -- metabolism, Phosphoproteins -- analysis -- metabolism, 0303 health sciences, src-Family Kinases -- metabolism, Adaptor Proteins, Neoplasms -- drug therapy -- genetics -- pathology, OPEN-LABEL, Cadherins, Lung Neoplasms -- genetics -- pathology, CANCER, 3. Good health, Signal Transducing -- metabolism, [SDV] Life Sciences [q-bio], Multidisciplinary Sciences, Gene Expression Regulation, Neoplastic, src-Family Kinases, Hyaluronan Receptors, Phenotype, Carcinoma, Squamous Cell, Disease Progression, Neoplastic Stem Cells, Science & Technology - Other Topics, SQUAMOUS-CELL CARCINOMA, Signal Transduction, EXPRESSION, Cadherins -- deficiency -- genetics -- metabolism, Enhancer of Zeste Homolog 2 Protein -- metabolism, GENES, Epithelial-Mesenchymal Transition, General Science & Technology, BIOLOGY, [SDV.CAN]Life Sciences [q-bio]/Cancer, Epithelial Cells -- metabolism -- pathology, Skin Neoplasms -- genetics -- pathology, Zinc Finger E-box-Binding Homeobox 1 -- metabolism, 03 medical and health sciences, Neoplastic Stem Cells -- metabolism -- pathology, [SDV.CAN] Life Sciences [q-bio]/Cancer, Animals, Humans, Neoplasm Metastasis -- drug therapy -- genetics, Enhancer of Zeste Homolog 2 Protein, Mesoderm -- metabolism -- pathology, Adaptor Proteins, Signal Transducing, Neoplastic, Science & Technology, Epithelial-Mesenchymal Transition -- drug effects -- genetics, SOXB1 Transcription Factors, Carcinoma, Médecine pathologie humaine, Biologie moléculaire, Zinc Finger E-box-Binding Homeobox 1, YAP-Signaling Proteins, Epithelial Cells, Phosphoproteins, Sciences biomédicales, Cancérologie, SOXB1 Transcription Factors -- metabolism, Gene Expression Regulation, Biologie cellulaire, Squamous Cell -- genetics -- pathology, RESISTANCE, Gene Deletion, Transcription Factors

Dateibeschreibung: application/pdf; 1 full-text file(s): application/pdf

Zugangs-URL: https://lirias.kuleuven.be/bitstream/123456789/665166/3/374935_0_art_file_3429067_q9yvjm_convrt.pdf
https://pubmed.ncbi.nlm.nih.gov/33328637
http://zaguan.unizar.es/record/102099
https://hdl.handle.net/10668/26673
https://hal.archives-ouvertes.fr/hal-03081403
https://pubmed.ncbi.nlm.nih.gov/33328637/
https://www.ncbi.nlm.nih.gov/pubmed/33328637
https://biblio.ugent.be/publication/8696394
http://pubmed.ncbi.nlm.nih.gov/33328637/
https://www.nature.com/articles/s41586-020-03046-1
https://hal.science/hal-03081403v1/document
https://doi.org/10.1038/s41586-020-03046-1
https://hal.science/hal-03081403v1
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/316454

Chemical modification of hyaluronan oligosaccharides differentially modulates hyaluronan–hyaladherin interactions

Autoren: Rebecca J. Dodd Charles D. Blundell Benedict M. Sattelle et al.

Quelle: J Biol Chem
Dodd, R J, Blundell, C D, Sattelle, B M, Enghild, J J, Milner, C M & Day, A J 2024, 'Chemical modification of hyaluronan oligosaccharides differentially modulates hyaluronan-hyaladherin interactions', The Journal of biological chemistry, vol. 300, no. 9, pp. 107668. https://doi.org/10.1016/j.jbc.2024.107668
Dodd, R J, Blundell, C D, Sattelle, B M, Enghild, J J, Milner, C M & Day, A J 2024, 'Chemical modification of hyaluronan oligosaccharides differentially modulates hyaluronan–hyaladherin interactions', The Journal of Biological Chemistry, vol. 300, no. 9, 107668. https://doi.org/10.1016/j.jbc.2024.107668

Schlagwörter: extracellular matrix, Oligosaccharides, Oligosaccharides/chemistry, hyaluronan, thermodynamics, Hyaluronan Receptors, Hyaluronic Acid/chemistry, structural biology, Humans, CD44, Hyaluronan Receptors/metabolism, Hyaluronic Acid, chemical modification, hyaluronan–protein interactions, TSG-6, Cell Adhesion Molecules, Cell Adhesion Molecules/metabolism, Research Article, Protein Binding

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/39128716
https://pure.au.dk/portal/en/publications/505b6ae3-2a92-4632-8d1d-9c459c195110
https://doi.org/10.1016/j.jbc.2024.107668
http://www.scopus.com/inward/record.url?scp=85204565091&partnerID=8YFLogxK

Fat1 deletion promotes hybrid EMT state, tumour stemness and metastasis

Autoren: Pastushenko, Ievgenia Mauri, Federico Song, Yura et al.

Quelle: Nature (London), 589 (7842

Schlagwörter: Sciences biomédicales, Adaptor Proteins, Signal Transducing -- metabolism, Animals, Cadherins -- deficiency -- genetics -- metabolism, Carcinoma, Squamous Cell -- genetics -- pathology, Disease Progression, Enhancer of Zeste Homolog 2 Protein -- metabolism, Epithelial Cells -- metabolism -- pathology, Epithelial-Mesenchymal Transition -- drug effects -- genetics, Gene Deletion, Gene Expression Regulation, Neoplastic, Humans, Hyaluronan Receptors -- metabolism, Lung Neoplasms -- genetics -- pathology, Mesoderm -- metabolism -- pathology, Mice, Neoplasm Metastasis -- drug therapy -- genetics, Neoplasms -- drug therapy -- genetics -- pathology, Neoplastic Stem Cells -- metabolism -- pathology, Phenotype, Phosphoproteins -- analysis -- metabolism, Proteomics, SOXB1 Transcription Factors -- metabolism, Signal Transduction, Skin Neoplasms -- genetics -- pathology, Transcription Factors -- metabolism, YAP-Signaling Proteins

Dateibeschreibung: 1 full-text file(s): application/pdf

Relation: uri/info:pii/3046; uri/info:pmid/33328637; uri/info:scp/85097603958; uri/info:pmcid/PMC7612440; https://dipot.ulb.ac.be/dspace/bitstream/2013/340069/3/PastushenkoNature2020.pdf

Verfügbarkeit: http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/340069
https://dipot.ulb.ac.be/dspace/bitstream/2013/340069/3/PastushenkoNature2020.pdf

Inhibitor of DNA binding 2 is a novel therapeutic target for stemness of head and neck squamous cell carcinoma

Autoren: Bon Seok Koo Jin-Man Kim Young Soo Rho et al.

Weitere Verfasser: Bon Seok Koo Jin-Man Kim Young Soo Rho et al.

Quelle: Br J Cancer

Schlagwörter: 0301 basic medicine, Cisplatin/pharmacology, Nude, Drug Resistance, Gene Expression, Mice, Cell Self Renewal/genetics, Neoplasm Transplantation/pathology, Cell Self Renewal, Inbred BALB C, Inhibitor of Differentiation Protein 2, Mice, Inbred BALB C, 0303 health sciences, Tumor, Head and Neck Neoplasms/genetics, Inhibitor of Differentiation Protein 2/metabolism, Head and Neck Neoplasms/metabolism, Inhibitor of Differentiation Protein 2/genetics, 3. Good health, Survival Rate, Phenotype, Hyaluronan Receptors, Head and Neck Neoplasms, Carcinoma, Squamous Cell, Female, Antineoplastic Agents/pharmacology, Cell Survival/drug effects, Squamous Cell/drug therapy, Neoplastic Stem Cells/metabolism, Cell Survival, Mice, Nude, Antineoplastic Agents, Cell Line, 03 medical and health sciences, Cell Line, Tumor, Animals, Humans, Gene Silencing, Hyaluronan Receptors/metabolism, Squamous Cell/genetics, Cell Proliferation, Neoplasm/genetics, Carcinoma, Head and Neck Neoplasms/drug therapy, Squamous Cell/metabolism, Drug Resistance, Neoplasm, Cellular, Spheroids, Cisplatin, Translational Therapeutics, Neoplasm Transplantation

Dateibeschreibung: application/pdf

Zugangs-URL: https://www.nature.com/articles/bjc2017373.pdf
https://pubmed.ncbi.nlm.nih.gov/29096401
https://ir.ymlib.yonsei.ac.kr/bitstream/22282913/166159/1/T999901449.pdf
https://yonsei.pure.elsevier.com/en/publications/inhibitor-of-dna-binding-2-is-a-novel-therapeutic-target-for-stem
https://www.nature.com/articles/bjc2017373
https://www.nature.com/articles/bjc2017373.pdf
https://pubmed.ncbi.nlm.nih.gov/29096401/
https://ir.ymlib.yonsei.ac.kr/handle/22282913/166159

In-vitro chondrogenic potential of synovial stem cells and chondrocytes allocated for autologous chondrocyte implantation — a comparison: Synovial stem cells as an alternative cell source for autologous chondrocyte implantation

Autoren: Anke Bernstein Hagen Schmal Norbert P. Südkamp et al.

Quelle: Kubosch, E J, Heidt, E, Niemeyer, P, Bernstein, A, Südkamp, N P & Schmal, H 2017, ' In-vitro chondrogenic potential of synovial stem cells and chondrocytes allocated for autologous chondrocyte implantation-a comparison : Synovial stem cells as an alternative cell source for autologous chondrocyte implantation ', International Orthopaedics, vol. 41, no. 5, pp. 991–998 . https://doi.org/10.1007/s00264-017-3400-y

Schlagwörter: Adult, Male, 0301 basic medicine, Transplantation, Autologous/methods, Autologous/methods, Cells, 03 medical and health sciences, Cartilage repair, Collagen Type II/metabolism, Chondrocytes, Osteoarthritis, Knee/metabolism, Osteogenesis, Osteoarthritis, 80 and over, Cell Differentiation/physiology, Humans, Orthopedic Procedures, Aggrecans, Hyaluronan Receptors/metabolism, Collagen Type II, Cells, Cultured, Aged, Aged, 80 and over, Transplantation, 0303 health sciences, Cultured, Stem Cells/metabolism, Cartilage/metabolism, Mesenchymal Stem Cells/cytology, Chondrocytes/metabolism, Cell Differentiation, Mesenchymal Stem Cells, Middle Aged, Osteoarthritis, Knee, Flow Cytometry, Knee/metabolism, Cartilage, Hyaluronan Receptors, Autologous chondrocyte implantation, Collagen type II, Cell quality, Aggrecans/metabolism, Female, Chondrogenesis, Synoviocytes/metabolism

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/28124102
https://core.ac.uk/display/80329945
https://europepmc.org/article/MED/28124102
https://link.springer.com/article/10.1007/s00264-017-3400-y
https://rd.springer.com/article/10.1007/s00264-017-3400-y
https://www.ncbi.nlm.nih.gov/pubmed/28124102
https://pubmed.ncbi.nlm.nih.gov/28124102/
https://portal.findresearcher.sdu.dk/da/publications/413eafab-a65a-4809-b77c-ae257504f670
https://portal.findresearcher.sdu.dk/da/publications/413eafab-a65a-4809-b77c-ae257504f670
https://doi.org/10.1007/s00264-017-3400-y

Impact of Truncated O-glycans in Gastric-Cancer-Associated CD44v9 Detection

Autoren: Moreira, IB Pinto, F Gomes, C et al.

Weitere Verfasser: Moreira, IB Pinto, F Gomes, C et al.

Schlagwörter: CD44, Gastric cancer, Glycosylation, Truncated O-glycans, Antibodies, Monoclonal / metabolism, Cell Line, Tumor, Gene Expression Regulation, Neoplastic, Humans, Hyaluronan Receptors / genetics, Hyaluronan Receptors / metabolism, Models, Biological, Polysaccharides / metabolism, Stomach Neoplasms / genetics, Stomach Neoplasms / metabolism

Dateibeschreibung: application/pdf

Relation: info:eu-repo/grantAgreement/FCT/9471 - RIDTI/PTDC%2FBBB-EBI%2F0567%2F2014/PT; info:eu-repo/grantAgreement/FCT/6817 - DCRRNI ID/UID%2FBIM%2F04293%2F2013/PT; info:eu-repo/grantAgreement/FCT/9471 - RIDTI/PTDC%2FMED-QUI%2F29780%2F2017/PT; info:eu-repo/grantAgreement/FCT/POR_NORTE/SFRH%2FBPD%2F115730%2F2016/PT; Cells, vol.9(2):264; https://www.mdpi.com/2073-4409/9/2/264; https://hdl.handle.net/10216/145268

Verfügbarkeit: https://hdl.handle.net/10216/145268
https://doi.org/10.3390/cells9020264

Tumor-associated mesenchymal stem-like cells provide extracellular signaling cue for invasiveness of glioblastoma cells

Autoren: Min Jung Kim Eun Jung Lim In Gyu Kim et al.

Weitere Verfasser: Min Jung Kim Eun Jung Lim In Gyu Kim et al.

Quelle: Oncotarget

Schlagwörter: Male, 0301 basic medicine, C5a, mesenchymal stem-like cells, Nude, Mice, Nude, Ligands, hyaluronic acid synthase-2, Extracellular Matrix Proteins/metabolism, Glioblastoma/metabolism, Mice, 03 medical and health sciences, hyaluronic acid, extracellular matrix remodeling, Brain Neoplasms/metabolism, Animals, Humans, Neoplasm Invasiveness, Mesenchymal Stromal Cells/metabolism, Hyaluronan Receptors/metabolism, Hyaluronic Acid, Aged, Extracellular Matrix Proteins, 0303 health sciences, Hyaluronic Acid/pharmacology, Brain Neoplasms, Mesenchymal Stem Cells, 3. Good health, Glioblastoma/pathology, Mesenchymal Stromal Cells/pathology, Hyaluronan Receptors, Brain Neoplasms/pathology, Heterografts, Female, Glioblastoma, Research Paper, Signal Transduction

Dateibeschreibung: application/pdf

Zugangs-URL: http://www.oncotarget.com/index.php?journal=oncotarget&page=article&op=download&path%5B%5D=13638&path%5B%5D=43319
https://pubmed.ncbi.nlm.nih.gov/27903965
http://www.oncotarget.com/fulltext/13638
https://ir.ymlib.yonsei.ac.kr/bitstream/22282913/154084/1/T201700104.pdf
https://hanyang.elsevierpure.com/en/publications/tumor-associated-mesenchymal-stem-like-cells-provide-extracellula
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5352067
https://ir.ymlib.yonsei.ac.kr/handle/22282913/154084
https://www.ncbi.nlm.nih.gov/pubmed/27903965

Nanovesicle-mediated systemic delivery of microRNA-34a for CD44 overexpressing gastric cancer stem cell therapy

Autoren: Yong Min Huh Hwunjae Lee Seungmin Han et al.

Weitere Verfasser: Yong Min Huh Hwunjae Lee Seungmin Han et al.

Quelle: Biomaterials. 105:12-24

Schlagwörter: Male, 0301 basic medicine, Neoplastic Stem Cells/metabolism, Nanoconjugates, Nanocapsules/chemistry, Stomach Neoplasms/therapy, Cell Line, Nanovesicles, Mice, 03 medical and health sciences, Nanocapsules, Stomach Neoplasms, Cell Line, Tumor, Animals, Humans, Molecular Targeted Therapy, Inbred BALB C, pH-responsive, Mice, Inbred BALB C, 0303 health sciences, Tumor, Neoplastic Stem Cells/pathology, Cancer stem cells, miR-34a delivery, Neoplastic Stem Cells/drug effects, CD44 suppression, MicroRNAs/administration & dosage, Nanocapsules/administration & dosage, Nanoconjugates/administration & dosage, 3. Good health, MicroRNAs, Hyaluronan Receptors, Treatment Outcome, Hyaluronan Receptors/metabolism, Nanoconjugates/chemistry, Neoplastic Stem Cells, Molecular Targeted Therapy/methods, Stomach Neoplasms/metabolism

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/27497057
https://pubmed.ncbi.nlm.nih.gov/27497057/
https://www.sciencedirect.com/science/article/abs/pii/S0142961216303738
http://www.sciencedirect.com/science/article/pii/S0142961216303738
https://ir.ymlib.yonsei.ac.kr/handle/22282913/151922
https://yonsei.pure.elsevier.com/en/publications/nanovesicle-mediated-systemic-delivery-of-microrna-34a-for-cd44-o
https://www.ncbi.nlm.nih.gov/pubmed/27497057

Tumour-vasculature development via endothelial-to-mesenchymal transition after radiotherapy controls CD44v6+ cancer cell and macrophage polarization

Weitere Verfasser: Seo-Hyun Choi A-Ram Kim Jae-Kyung Nam et al.

Schlagwörter: Animals, Cell Line, Tumor, Cell Transdifferentiation/drug effects, Cell Transdifferentiation/genetics, Endothelial Cells/metabolism, Epithelial-Mesenchymal Transition/genetics, Epithelial-Mesenchymal Transition/physiology, Female, Heterocyclic Compounds/pharmacology, Humans, Hyaluronan Receptors/metabolism, Immunohistochemistry, Macrophages/metabolism, Male, Mice, Inbred BALB C, Inbred C57BL, Neoplastic Stem Cells/metabolism, Receptor, Transforming Growth Factor-beta Type II/genetics, Transforming Growth Factor-beta Type II/metabolism, Receptors, CXCR4/genetics, CXCR4/metabolism, Signal Transduction/genetics, Signal Transduction/physiology

Relation: NATURE COMMUNICATIONS; J02293; https://ir.ymlib.yonsei.ac.kr/handle/22282913/166809; T201804744; NATURE COMMUNICATIONS, Vol.9(1) : 5108, 2018; 58186

Verfügbarkeit: https://ir.ymlib.yonsei.ac.kr/handle/22282913/166809
https://doi.org/10.1038/s41467-018-07470-w

Proteomic analysis of CD44(+) and CD44(−) gastric cancer cells

Autoren: Dayeon Yu Hyun Soo Shin Go Choi et al.

Weitere Verfasser: Dayeon Yu Hyun Soo Shin Go Choi et al.

Quelle: Molecular and Cellular Biochemistry. 396:213-220

Schlagwörter: Electrophoresis, Proteomics, 0301 basic medicine, Cell Separation, HSC70 Heat-Shock Proteins/metabolism, 03 medical and health sciences, Neoplastic Stem Cells/metabolism, Stomach Neoplasms, Tumor/metabolism, Biomarkers, Tumor, Humans, Electrophoresis, Gel, Two-Dimensional, CD44, 2. Zero hunger, Gel, 0303 health sciences, Cytoskeletal Proteins/metabolism, Cancer stem cells, Gene Expression Profiling, Stomach Neoplasms/pathology, HSC70 Heat-Shock Proteins, Proteins, Reproducibility of Results, 2-DE, Up-Regulation, 3. Good health, Cytoskeletal Proteins, Hyaluronan Receptors, Hyaluronan Receptors/metabolism, Phosphopyruvate Hydratase, Two-Dimensional, Proteins/genetics, Neoplastic Stem Cells, Proteins/metabolism, Proteins/analysis, Proteomics/methods, Phosphopyruvate Hydratase/metabolism, Gastric cancer, Biomarkers, Stomach Neoplasms/metabolism

Dateibeschreibung: 213~220

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/25081334
https://mdanderson.elsevierpure.com/en/publications/proteomic-analysis-of-cd44-and-cd44-gastric-cancer-cells
https://www.ncbi.nlm.nih.gov/pubmed/25081334
https://link.springer.com/article/10.1007/s11010-014-2156-6
https://yonsei.pure.elsevier.com/en/publications/proteomic-analysis-of-cd44-and-cd44-gastric-cancer-cells
https://europepmc.org/abstract/MED/25081334
https://link.springer.com/article/10.1007/s11010-014-2156-6/fulltext.html

Candidate Gene Polymorphisms in Patients with Acetaminophen-Induced Acute Liver Failure

Autoren: Court, Michael H Peter, Inga Hazarika, Suwagmani et al.

Quelle: Drug Metabolism and Disposition. 42:28-32

Schlagwörter: 0301 basic medicine, Genotype, Genetic - genetics, Hyaluronan Receptors - genetics, Chemical and Drug Induced Liver Injury - metabolism, Chemical and Drug Induced Liver Injury - genetics, Acute - metabolism, Acute - genetics, 03 medical and health sciences, Hyaluronan Receptors - metabolism, Risk Factors, Cytochrome P-450 CYP3A - genetics, Cytochrome P-450 CYP3A, Humans, Polymorphism, Alleles, Acetaminophen, 0303 health sciences, Polymorphism, Genetic, 4. Education, Acetaminophen - adverse effects, Liver Failure, Acute, 3. Good health, Biomarkers - metabolism, Hyaluronan Receptors, Acute - chemically induced, Cytochrome P-450 CYP3A - metabolism, Chemical and Drug Induced Liver Injury, Liver Failure, Biomarkers

Zugangs-URL: https://europepmc.org/articles/pmc3876784?pdf=render
https://pubmed.ncbi.nlm.nih.gov/24104197
https://dmd.aspetjournals.org/content/42/1/28
https://www.ncbi.nlm.nih.gov/pubmed/24104197
https://dmd.aspetjournals.org/content/dmd/early/2013/10/08/dmd.113.053546.full.pdf
http://dmd.aspetjournals.org/content/early/2013/10/08/dmd.113.053546
https://utsouthwestern.pure.elsevier.com/en/publications/candidate-gene-polymorphisms-in-patients-with-acetaminophen-induc
http://dmd.aspetjournals.org/lookup/doi/10.1124/dmd.113.053546

Hyaluronic acid receptor-targetable imidazolized nanovectors for induction of gastric cancer cell death by RNA interference

Autoren: Taeyun Kwon Eun Kyung Lim Seungjoo Haam et al.

Weitere Verfasser: Taeyun Kwon Eun Kyung Lim Seungjoo Haam et al.

Quelle: Biomaterials. 34:4327-4338

Schlagwörter: 0301 basic medicine, Magnetic Resonance Spectroscopy, Small Interfering/metabolism, Hyaluronic acid, Genetic Vectors, Static Electricity, Intracellular Space, Buffering, Electrophoretic Mobility Shift Assay, Transfection, Genetic Vectors/metabolism, Cell Line, Stomach Neoplasms/pathology, 03 medical and health sciences, RNA interference, RNA Interference, Cell Death/drug effects, Stomach Neoplasms, Cell Line, Tumor, DNA/metabolism, Humans, Polylysine, Gene delivery, Particle Size, RNA, Small Interfering, Imidazole, Cell Proliferation, Tumor, Cell Death, Intracellular Space/metabolism, Nanoparticles/chemistry, Imidazoles, DNA, Plasmids/metabolism, Hydrogen-Ion Concentration, Imidazoles/chemistry, 3. Good health, Hyaluronan Receptors, Hyaluronan Receptors/metabolism, Polylysine/chemistry, RNA, Nanoparticles, bcl-X Protein/metabolism, Plasmids

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/23478035
https://www.sciencedirect.com/science/article/abs/pii/S0142961213001750
https://pubmed.ncbi.nlm.nih.gov/23478035/
https://mdanderson.elsevierpure.com/en/publications/hyaluronic-acid-receptor-targetable-imidazolized-nanovectors-for-
https://yonsei.pure.elsevier.com/en/publications/hyaluronic-acid-receptor-targetable-imidazolized-nanovectors-for-
https://mdanderson.influuent.utsystem.edu/en/publications/hyaluronic-acid-receptor-targetable-imidazolized-nanovectors-for-

Hyaluronan-modified magnetic nanoclusters for detection of CD44-overexpressing breast cancer by MR imaging

Autoren: Kwangyeol Lee Hyun Ouk Kim Seungjoo Haam et al.

Weitere Verfasser: Kwangyeol Lee Hyun Ouk Kim Seungjoo Haam et al.

Quelle: Biomaterials. 32:7941-7950

Schlagwörter: Breast Neoplasms/pathology, Magnetic Resonance Spectroscopy, Cell Survival, Nude, Static Electricity, Nanoparticles/ultrastructure, Mice, Nude, Breast Neoplasms, 02 engineering and technology, Breast Neoplasms/diagnosis, 01 natural sciences, Cell Line, Magnets/chemistry, Mice, Magnetic resonance imaging, CD44Breast cancer, Cell Line, Tumor, Magnetic nanoclusters, Spectroscopy, Fourier Transform Infrared, Animals, Humans, Tissue Distribution/drug effects, Hyaluronic Acid, Particle Size, Spectroscopy, Hyaluronan, Micelles, Tumor, Hyaluronic Acid/pharmacology, Magnetic Phenomena, Photoelectron Spectroscopy, Nanoparticles/chemistry, Temperature, Hyaluronic Acid/chemistry, Magnetic Resonance Imaging, 0104 chemical sciences, 3. Good health, Hyaluronan Receptors, Hyaluronan Receptors/metabolism, Fourier Transform Infrared, Magnets, Nanoparticles, Female, 0210 nano-technology

Dateibeschreibung: 7941~7950

Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/21777976
https://koreauniv.pure.elsevier.com/en/publications/hyaluronan-modified-magnetic-nanoclusters-for-detection-of-cd44-o
https://pubag.nal.usda.gov/catalog/903593
https://www.sciencedirect.com/science/article/pii/S0142961211007708
https://yonsei.pure.elsevier.com/en/publications/hyaluronan-modified-magnetic-nanoclusters-for-detection-of-cd44-o
http://www.sciencedirect.com/science/article/pii/S0142961211007708
https://www.ncbi.nlm.nih.gov/pubmed/21777976