Suchergebnisse - "Gene Expression Regulation, Neoplastic/drug effects [MeSH]"
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1
Autoren: et al.
Quelle: Clin Exp Metastasis
Schlagwörter: 0301 basic medicine, Mice, Inbred BALB C, 0303 health sciences, Research, Gene Expression Profiling, Apoptosis, Gene Expression Regulation, Neoplastic, Mice, Disease Models, Animal, 03 medical and health sciences, Drug Resistance, Neoplasm, Cell Line, Tumor, Colonic Neoplasms, Animals, Humans, Fluorouracil, Cell Line, Tumor [MeSH], Mice, Inbred BALB C [MeSH], Prss11, Peritoneal Neoplasms/genetics [MeSH], Chemoresistance, Peritoneal Neoplasms/drug therapy [MeSH], Peritoneal Neoplasms/secondary [MeSH], Oligonucleotide Array Sequence Analysis [MeSH], Drug Resistance, Neoplasm/genetics [MeSH], Colonic Neoplasms/pathology [MeSH], Fluorouracil/pharmacology [MeSH], Disease Models, Animal [MeSH], BMP7, Fluorouracil/therapeutic use [MeSH], Cell survival, Humans [MeSH], Apoptosis/drug effects [MeSH], Colonic Neoplasms/genetics [MeSH], Animals [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Mice [MeSH], Colonic Neoplasms/drug therapy [MeSH], Chemotherapy, Gene Expression Profiling [MeSH], Peritoneal Neoplasms, Oligonucleotide Array Sequence Analysis
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2
Autoren: et al.
Quelle: Cell Oncol (Dordr)
Schlagwörter: Sulfonamides, Research, Apoptosis, Antineoplastic Agents, Hydroxamic Acids, 3. Good health, Histone Deacetylase Inhibitors, Gene Expression Regulation, Neoplastic, Organoids, Cell Line, Tumor [MeSH], Epigenetic, Organoids/drug effects [MeSH], Microsomes, Liver/drug effects [MeSH], Antineoplastic Agents/pharmacology [MeSH], Copper/chemistry [MeSH], Histone Deacetylase Inhibitor, Copper Complex, Hydroxamic Acids/pharmacology [MeSH], Organoids/metabolism [MeSH], Colonic Neoplasms/pathology [MeSH], Colonic Neoplasms/metabolism [MeSH], Microsomes, Liver/metabolism [MeSH], Histone Deacetylase Inhibitors/pharmacology [MeSH], Humans [MeSH], Apoptosis/drug effects [MeSH], Colonic Neoplasms/genetics [MeSH], Sulfonamides/pharmacology [MeSH], Colon Cancer, Gene Expression Regulation, Neoplastic/drug effects [MeSH], Colonic Neoplasms/drug therapy [MeSH], Hydroxamic Acids/therapeutic use [MeSH], Antineoplastic Agents/therapeutic use [MeSH], Cell Proliferation/drug effects [MeSH], Cell Line, Tumor, Colonic Neoplasms, Microsomes, Liver, Humans, Copper, Cell Proliferation
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3
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Radiat Oncol
Radiation Oncology, Vol 19, Iss 1, Pp 1-16 (2024)Schlagwörter: Male, 0301 basic medicine, DNA Repair, Medizin, R895-920, Heavy Ion Radiotherapy, Photon irradiation, Androgen receptor, TP53, Prostate cancer, Carbon ion irradiation, Medical physics. Medical radiology. Nuclear medicine, 03 medical and health sciences, Cell Line, Tumor, Humans, ddc:610, Cell Line, Tumor [MeSH], DNA Repair [MeSH], Heavy Ion Radiotherapy [MeSH], Humans [MeSH], Prostatic Neoplasms/metabolism [MeSH], Receptors, Androgen/metabolism [MeSH], Transcriptome/radiation effects [MeSH], Signal Transduction/radiation effects [MeSH], Prostatic Neoplasms/pathology [MeSH], Tumor Suppressor Protein p53/metabolism [MeSH], Prostatic Neoplasms/radiotherapy [MeSH], Receptors, Androgen/genetics [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Photons [MeSH], Carbon [MeSH], DNA Damage/radiation effects [MeSH], Male [MeSH], Gene Expression Regulation, Neoplastic/radiation effects [MeSH], Research, RC254-282, Photons, 0303 health sciences, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Prostatic Neoplasms, Medical sciences Medicine, Carbon, 3. Good health, Gene Expression Regulation, Neoplastic, Receptors, Androgen, Tumor Suppressor Protein p53, Transcriptome, DNA Damage, Signal Transduction
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4
Autoren: et al.
Quelle: J Cancer Res Clin Oncol
Schlagwörter: Adult, Aged, 80 and over, Male, 0301 basic medicine, Survivin, Imidazoles, Liposarcoma, Middle Aged, Prognosis, Aged, 80 and over [MeSH], Aged [MeSH], Survivin/antagonists, Naphthoquinones/administration, Tumor Cells, Cultured [MeSH], Etoposide/administration, Male [MeSH], Doxorubicin/administration, Targeted therapy, Liposarcoma/drug therapy [MeSH], Liposarcoma/classification [MeSH], Liposarcoma/pathology [MeSH], Female [MeSH], Follow-Up Studies [MeSH], Original Article – Cancer Research, Adult [MeSH], Humans [MeSH], Retrospective Studies [MeSH], Apoptosis, Middle Aged [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Survival Rate [MeSH], Antineoplastic Combined Chemotherapy Protocols/pharmacology [MeSH], Prognosis [MeSH], Liposarcoma/metabolism [MeSH], Inhibitor of apoptosis protein, Imidazoles/administration, Gene Expression Regulation, Neoplastic, Survival Rate, 03 medical and health sciences, 0302 clinical medicine, Doxorubicin, Antineoplastic Combined Chemotherapy Protocols, Tumor Cells, Cultured, Humans, Female, Aged, Etoposide, Follow-Up Studies, Naphthoquinones, Retrospective Studies
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5
Autoren: et al.
Quelle: J Cancer Res Clin Oncol
Schlagwörter: Adult, Male, Uveal Neoplasms, 0301 basic medicine, Skin Neoplasms, Autoimmunity, Endocrine System Diseases, Cohort Studies, 03 medical and health sciences, Melanoma, Cutaneous Malignant, 0302 clinical medicine, Uveal Melanoma, Germany, Biomarkers, Tumor, Humans, ddc:610, Neoplasm Metastasis, 10. No inequality, Immune Checkpoint Inhibitors, Melanoma, Inflammation, 3. Good health, Inflammation/blood [MeSH], Gene Expression Regulation, Neoplastic/immunology [MeSH], Inflammation/chemically induced [MeSH], Skin Neoplasms/blood [MeSH], Skin Neoplasms/drug therapy [MeSH], Cohort Studies [MeSH], Uveal Neoplasms/blood [MeSH], Autoimmunity/genetics [MeSH], Inflammation/genetics [MeSH], Male [MeSH], Immune Checkpoint Inhibitors/adverse effects [MeSH], Melanoma/pathology [MeSH], Case-Control Studies [MeSH], Autoimmunity/drug effects [MeSH], Uveal Neoplasms/pathology [MeSH], Diabetes Mellitus, Type 1/blood [MeSH], Melanoma/drug therapy [MeSH], Inflammation/immunology [MeSH], Female [MeSH], Original Article – Cancer Research, Melanoma/genetics [MeSH], Adult [MeSH], Diabetes Mellitus, Type 1/chemically induced [MeSH], Humans [MeSH], Neoplasm Metastasis [MeSH], Uveal Neoplasms/drug therapy [MeSH], Melanoma/blood [MeSH], Uveal Neoplasms/genetics [MeSH], Skin Neoplasms/pathology [MeSH], Immune checkpoint inhibitors, Immune Checkpoint Inhibitors/therapeutic use [MeSH], Cytokines, Gene Expression Regulation, Neoplastic/drug effects [MeSH], Autoimmunity/immunology [MeSH], Skin Neoplasms/genetics [MeSH], Endocrine System Diseases/blood [MeSH], Biomarkers, Tumor/blood [MeSH], Biomarkers, Tumor/genetics [MeSH], Germany [MeSH], Endocrine System Diseases/immunology [MeSH], Diabetes Mellitus, Type 1/immunology [MeSH], Immune-related adverse events, Endocrine System Diseases/chemically induced [MeSH], Immune Checkpoint Inhibitors/pharmacology [MeSH], Gene Expression Regulation, Neoplastic, Diabetes Mellitus, Type 1, Case-Control Studies, Female
Dateibeschreibung: application/pdf
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00432-021-03550-5.pdf
https://pubmed.ncbi.nlm.nih.gov/33837821
https://link.springer.com/content/pdf/10.1007/s00432-021-03550-5.pdf
https://www.ncbi.nlm.nih.gov/pubmed/33837821
https://europepmc.org/article/PMC/PMC8076116
https://link.springer.com/article/10.1007/s00432-021-03550-5
https://pubmed.ncbi.nlm.nih.gov/33837821/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8076116
https://opus4.kobv.de/opus4-fau/frontdoor/index/index/docId/22122
https://repository.publisso.de/resource/frl:6449924 -
6
Autoren: et al.
Quelle: Br J Cancer
Schlagwörter: 0301 basic medicine, 0303 health sciences, Cell Death, Plasma Gases, Article, 3. Good health, Gene Expression Regulation, Neoplastic, 03 medical and health sciences, Drug Resistance, Neoplasm, Neoplasms, Metabolome, Humans, Metabolome/drug effects [MeSH], Energy Metabolism/physiology [MeSH], Humans [MeSH], Reactive Oxygen Species/metabolism [MeSH], Neoplasms/genetics [MeSH], Metabolic Networks and Pathways/drug effects [MeSH], Neoplasms/metabolism [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Plasma Gases/pharmacology [MeSH], Cell Death/drug effects [MeSH], Drug Resistance, Neoplasm/physiology [MeSH], Neoplasms/pathology [MeSH], Neoplasms/therapy [MeSH], Cells, Cultured [MeSH], Physics, Plasma Gases/therapeutic use [MeSH], Argon/pharmacology [MeSH], HeLa Cells [MeSH], Cancer metabolism, Amino Acids/metabolism [MeSH], Argon/therapeutic use [MeSH], Metabolic Networks and Pathways/genetics [MeSH], Amino Acids, Argon, Energy Metabolism, Reactive Oxygen Species, Cells, Cultured, Metabolic Networks and Pathways, HeLa Cells
Zugangs-URL: https://www.nature.com/articles/s41416-021-01335-8.pdf
https://pubmed.ncbi.nlm.nih.gov/33767419
https://www.ncbi.nlm.nih.gov/pubmed/33767419
https://www.nature.com/articles/s41416-021-01335-8.pdf
https://pubmed.ncbi.nlm.nih.gov/33767419/
https://europepmc.org/article/MED/33767419
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8144554
https://www.nature.com/articles/s41416-021-01335-8
https://repository.publisso.de/resource/frl:6442874 -
7
Autoren: et al.
Quelle: J Cancer Res Clin Oncol
Schlagwörter: Glutamate Carboxypeptidase II, Male, Secretory Pathway, Edetic Acid/analogs, Cell Line, Tumor [MeSH], Glutamate Carboxypeptidase II/metabolism [MeSH], Prostate-Specific Antigen/drug effects [MeSH], Prostatic Neoplasms/metabolism [MeSH], Prostatic Neoplasms/drug therapy [MeSH], Prostatic Neoplasms/pathology [MeSH], Androgen Antagonists/therapeutic use [MeSH], Adenocarcinoma/metabolism [MeSH], Male [MeSH], Adenocarcinoma/drug therapy [MeSH], Adenocarcinoma/pathology [MeSH], Edetic Acid/pharmacokinetics [MeSH], Antigens, Surface/genetics [MeSH], Positron Emission Tomography Computed Tomography/methods [MeSH], [, Glutamate Carboxypeptidase II/genetics [MeSH], Prostate cancer, Original Article – Cancer Research, PC-3 Cells [MeSH], Humans [MeSH], VPC-13566, Androstenes/therapeutic use [MeSH], Secretory Pathway/drug effects [MeSH], Prostate-specific membrane antigen, Antigens, Surface/metabolism [MeSH], Androgen Antagonists/pharmacology [MeSH], Androgen antagonist, Gene Expression Regulation, Neoplastic/drug effects [MeSH], Androstenes/pharmacology [MeSH], Oligopeptides/pharmacokinetics [MeSH], Adenocarcinoma/diagnosis [MeSH], Prostate-Specific Antigen/metabolism [MeSH], Prostatic Neoplasms/diagnosis [MeSH], Abiraterone, Prostatic Neoplasms, Androgen Antagonists, Gallium Radioisotopes, Adenocarcinoma, Prostate-Specific Antigen, 3. Good health, Gene Expression Regulation, Neoplastic, 03 medical and health sciences, 0302 clinical medicine, Cell Line, Tumor, Positron Emission Tomography Computed Tomography, Antigens, Surface, PC-3 Cells, Humans, Androstenes, Oligopeptides, Edetic Acid, Gallium Isotopes
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00432-021-03583-w.pdf
https://pubmed.ncbi.nlm.nih.gov/33760944
https://europepmc.org/article/PMC/PMC8076114
https://www.ncbi.nlm.nih.gov/pubmed/33760944
https://link.springer.com/content/pdf/10.1007/s00432-021-03583-w.pdf
https://link.springer.com/article/10.1007/s00432-021-03583-w
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8076114
https://pubmed.ncbi.nlm.nih.gov/33760944/
https://repository.publisso.de/resource/frl:6449904 -
8
Autoren:
Quelle: Leukemia
Schlagwörter: 0301 basic medicine, Tumour immunology, B-Lymphocytes/metabolism [MeSH], Oncogenesis, Tumor Microenvironment/immunology [MeSH], Disease Management [MeSH], Immune Evasion [MeSH], Hodgkin Disease/diagnosis [MeSH], Hodgkin Disease/therapy [MeSH], Hodgkin Disease/metabolism [MeSH], Hodgkin Disease/etiology [MeSH], Signal Transduction/drug effects [MeSH], Biomarkers, Tumor [MeSH], Transcription Factors/metabolism [MeSH], Genetic Variation [MeSH], Genetic Predisposition to Disease [MeSH], Cell Transformation, Neoplastic/metabolism [MeSH], B-Lymphocytes/immunology [MeSH], Humans [MeSH], Review Article, Cell Transformation, Neoplastic/genetics [MeSH], Cell Transformation, Neoplastic/immunology [MeSH], Animals [MeSH], Disease Susceptibility [MeSH], B-Lymphocytes/pathology [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Cancer genetics, Tumor Microenvironment/genetics [MeSH], B-Lymphocytes, 0303 health sciences, Disease Management, Genetic Variation, Hodgkin Disease, 3. Good health, Gene Expression Regulation, Neoplastic, 03 medical and health sciences, Cell Transformation, Neoplastic, Biomarkers, Tumor, Tumor Microenvironment, Animals, Humans, Genetic Predisposition to Disease, Disease Susceptibility, Immune Evasion, Signal Transduction, Transcription Factors
Zugangs-URL: https://www.nature.com/articles/s41375-021-01204-6.pdf
https://pubmed.ncbi.nlm.nih.gov/33686198
https://www.ncbi.nlm.nih.gov/pubmed/33686198
https://www.scilit.net/article/b6c87d23a2115a582621782445367dd8?action=show-references
https://pubmed.ncbi.nlm.nih.gov/33686198/
https://www.nature.com/articles/s41375-021-01204-6.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8024192
https://europepmc.org/article/PMC/PMC8024192
https://repository.publisso.de/resource/frl:6443842 -
9
Pharmaceutical immunoglobulin G impairs anti-carcinoma activity of oxaliplatin in colon cancer cells
Autoren: et al.
Quelle: Br J Cancer
Schlagwörter: Male, Cell Survival, Immunoglobulins, Intravenous, Middle Aged, Xenograft Model Antitumor Assays, Article, 3. Good health, Gene Expression Regulation, Neoplastic, Oxaliplatin, 03 medical and health sciences, 0302 clinical medicine, Cell Line, Tumor, Colonic Neoplasms, Cell Line, Tumor [MeSH], Aged [MeSH], Humans [MeSH], Molecular medicine, Cell Survival/drug effects [MeSH], Middle Aged [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Preclinical research, Oxaliplatin/pharmacology [MeSH], Male [MeSH], Colonic Neoplasms/drug therapy [MeSH], Immunoglobulins, Intravenous/pharmacology [MeSH], Xenograft Model Antitumor Assays [MeSH], Drug Interactions [MeSH], Colonic Neoplasms/metabolism [MeSH], Immunoglobulins, Intravenous/administration, Cell Proliferation/drug effects [MeSH], Extracellular Signal-Regulated MAP Kinases/drug effects [MeSH], Colon cancer, Humans, Drug Interactions, Extracellular Signal-Regulated MAP Kinases, Aged, Cell Proliferation
Zugangs-URL: https://www.nature.com/articles/s41416-021-01272-6.pdf
https://pubmed.ncbi.nlm.nih.gov/33558709
https://europepmc.org/article/PMC/PMC8039037
https://www.nature.com/articles/s41416-021-01272-6.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8039037
http://www-nature-com-443.webvpn.bjmu.tsg211.com/articles/s41416-021-01272-6
https://pubmed.ncbi.nlm.nih.gov/33558709/
https://www.ncbi.nlm.nih.gov/pubmed/33558709
https://repository.publisso.de/resource/frl:6442896 -
10
Autoren: et al.
Quelle: Mol Neurobiol
Schlagwörter: Male, 0301 basic medicine, 0303 health sciences, Curcumin, Brain Neoplasms, Antineoplastic Agents, Apoptosis, Article, 6. Clean water, Gene Expression Regulation, Neoplastic, 03 medical and health sciences, Glioma, Cell Line, Tumor [MeSH], Brain Neoplasms/genetics [MeSH], Humans [MeSH], Reactive Oxygen Species/metabolism [MeSH], Cell death, Apoptosis/drug effects [MeSH], Antineoplastic Agents/pharmacology [MeSH], Glioblastoma/pathology [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Cellular engineering, Male [MeSH], Brain tumor, Cytokine, Brain Neoplasms/drug therapy [MeSH], Curcumin/pharmacology [MeSH], Cell Line, Tumor, Humans, Glioblastoma, Reactive Oxygen Species
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s12035-020-01922-5.pdf
https://pubmed.ncbi.nlm.nih.gov/32430842
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7340659
https://link.springer.com/content/pdf/10.1007/s12035-020-01922-5.pdf
https://pubmed.ncbi.nlm.nih.gov/32430842/
https://europepmc.org/article/PMC/PMC7340659
https://link.springer.com/article/10.1007/s12035-020-01922-5
https://repository.publisso.de/resource/frl:6471718 -
11
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Br J Cancer
Schlagwörter: Adult, Male, 0301 basic medicine, Cell Survival, Pyridines, Aminopyridines, Article, Piperazines, 03 medical and health sciences, Cell Line, Tumor, Humans, Child, Cell Proliferation, 0303 health sciences, Sequence Analysis, RNA, Gene Expression Profiling, Cyclin-Dependent Kinase 4, Cyclin-Dependent Kinase 6, Neoplasms, Germ Cell and Embryonal, 3. Good health, Gene Expression Regulation, Neoplastic, Cell Line, Tumor [MeSH], Checkpoints, Purines/pharmacology [MeSH], Molecular medicine, Cyclin-Dependent Kinase 4/metabolism [MeSH], Piperazines/pharmacology [MeSH], Cyclin-Dependent Kinase 4/antagonists, Translational research, Aminopyridines/pharmacology [MeSH], Neoplasms, Germ Cell and Embryonal/drug therapy [MeSH], Germ cell tumours, Male [MeSH], Cyclin-Dependent Kinase 6/antagonists, Child [MeSH], Up-Regulation/drug effects [MeSH], Cyclin-Dependent Kinase 4/genetics [MeSH], Female [MeSH], Neoplasms, Germ Cell and Embryonal/metabolism [MeSH], Pyridines/pharmacology [MeSH], Drug Resistance, Neoplasm/drug effects [MeSH], Adult [MeSH], Humans [MeSH], Neoplasms, Germ Cell and Embryonal/genetics [MeSH], Sequence Analysis, RNA [MeSH], Cell Survival/drug effects [MeSH], Cyclin-Dependent Kinase 6/metabolism [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Cisplatin/pharmacology [MeSH], Drug screening, Cyclin-Dependent Kinase 6/genetics [MeSH], Gene Expression Profiling [MeSH], Cell Proliferation/drug effects [MeSH], Drug Resistance, Neoplasm, Purines, Female, Cisplatin
Zugangs-URL: https://www.nature.com/articles/s41416-020-0891-x.pdf
https://pubmed.ncbi.nlm.nih.gov/32418994
https://www.nature.com/articles/s41416-020-0891-x/
https://pubmed.ncbi.nlm.nih.gov/32418994/
https://www.nature.com/articles/s41416-020-0891-x.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7403155
http://www-nature-com-443.webvpn.bjmu.tsg211.com/articles/s41416-020-0891-x
https://resolver.sub.uni-goettingen.de/purl?gro-2/73730
https://repository.publisso.de/resource/frl:6471430 -
12
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Leukemia
Digital.CSIC. Repositorio Institucional del CSIC
instname
RISalud-ANDALUCIA. Repositorio Institucional de Salud de AndalucíaSchlagwörter: Inhibidores selectivos de HDAC6, Inhibidores de histona deacetilasa, Expresión de CD38, Myeloma, Antineoplastic Agents, Histone Deacetylase 6, Hydroxamic Acids, Models, Biological, Article, Immunophenotyping, Histone Deacetylase Inhibitors/therapeutic use [MeSH], Cell Line, Tumor [MeSH], Antibodies, Monoclonal/pharmacology [MeSH], Drug Synergism [MeSH], T-Lymphocyte Subsets/immunology [MeSH], Antineoplastic Agents/pharmacology [MeSH], Models, Biological [MeSH], Multiple Myeloma/genetics [MeSH], T-Lymphocyte Subsets/metabolism [MeSH], ADP-ribosyl Cyclase 1/metabolism [MeSH], Antibody-Dependent Cell Cytotoxicity/drug effects [MeSH], Immunophenotyping [MeSH], ADP-ribosyl Cyclase 1/genetics [MeSH], Histone Deacetylase 6/antagonists, Hydroxamic Acids/pharmacology [MeSH], Combination drug therapy, Membrane Glycoproteins/metabolism [MeSH], Multiple Myeloma/metabolism [MeSH], Multiple Myeloma/drug therapy [MeSH], Antibody-Dependent Cell Cytotoxicity/immunology [MeSH], Pyrimidines/pharmacology [MeSH], Histone Deacetylase Inhibitors/pharmacology [MeSH], Humans [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Membrane Glycoproteins/genetics [MeSH], Immunotherapy, T-Lymphocyte Subsets, Cell Line, Tumor, Humans, Terapia con anticuerpos anti‑CD38, Membrane Glycoproteins, Terapia combinada, Mieloma múltiple, Antibody-Dependent Cell Cytotoxicity, Antibodies, Monoclonal, Drug Synergism, ADP-ribosyl Cyclase 1, 3. Good health, Gene Expression Regulation, Neoplastic, Histone Deacetylase Inhibitors, Pyrimidines, Multiple Myeloma
Dateibeschreibung: application/pdf; application/zip
Zugangs-URL: https://www.nature.com/articles/s41375-020-0840-y.pdf
https://pubmed.ncbi.nlm.nih.gov/32350373
http://hdl.handle.net/10261/236784
http://hdl.handle.net/10668/15467
https://www.nature.com/articles/s41375-020-0840-y.pdf
https://www.scilit.net/article/f759f2444e62c2284708327891600b79?action=show-references
https://www.nature.com/articles/s41375-020-0840-y
https://pubmed.ncbi.nlm.nih.gov/32350373/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8318885
https://digital.csic.es/handle/10261/236784
https://repository.publisso.de/resource/frl:6471038 -
13
Autoren: et al.
Quelle: Oncogene
Schlagwörter: 0301 basic medicine, 0303 health sciences, Brain Neoplasms, Chemokine CX3CL1, Primary Cell Culture, Chemokine CXCL16, Real-Time Polymerase Chain Reaction, Cell Line, Tumor [MeSH], Humans [MeSH], Chemokine CXCL12 [MeSH], Chemokine CX3CL1 [MeSH], CNS cancer, Antineoplastic Agents, Alkylating/pharmacology [MeSH], Glioblastoma/pathology [MeSH], Chemokine CXCL16 [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Article, CRISPR-Cas Systems [MeSH], Transcriptome [MeSH], Chemotherapy, Primary Cell Culture [MeSH], Cell Proliferation/drug effects [MeSH], Temozolomide/pharmacology [MeSH], Brain Neoplasms/pathology [MeSH], Real-Time Polymerase Chain Reaction [MeSH], Chemokine CXCL12, Gene Expression Regulation, Neoplastic, 03 medical and health sciences, Cell Line, Tumor, Temozolomide, Humans, CRISPR-Cas Systems, Glioblastoma, Transcriptome, Antineoplastic Agents, Alkylating, Cell Proliferation
Zugangs-URL: https://www.nature.com/articles/s41388-020-1302-8.pdf
https://pubmed.ncbi.nlm.nih.gov/32346064
https://www.ncbi.nlm.nih.gov/pubmed/32346064
https://pubmed.ncbi.nlm.nih.gov/32346064/
https://www.nature.com/articles/s41388-020-1302-8.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7253351
https://www.nature.com/articles/s41388-020-1302-8
https://repository.publisso.de/resource/frl:6471374 -
14
Autoren: et al.
Quelle: Br J Cancer
Schlagwörter: Glycine Hydroxymethyltransferase, 0301 basic medicine, Cell Survival, NF-E2-Related Factor 2, Antineoplastic Agents, Cell Line, Tumor [MeSH], Homeostasis/drug effects [MeSH], Phosphoglycerate Dehydrogenase/genetics [MeSH], Humans [MeSH], Reactive Oxygen Species/metabolism [MeSH], Tumor Microenvironment/drug effects [MeSH], Molecular medicine, CNS cancer, Cell Survival/drug effects [MeSH], Antineoplastic Agents/pharmacology [MeSH], Phosphoglycerate Dehydrogenase/antagonists, Glioblastoma/metabolism [MeSH], Glycine Hydroxymethyltransferase/genetics [MeSH], Glioblastoma/pathology [MeSH], NF-E2-Related Factor 2/genetics [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Article, Serine/metabolism [MeSH], Oxidation-Reduction/drug effects [MeSH], Mechanisms of disease, Glioblastoma/genetics [MeSH], Cell Proliferation/drug effects [MeSH], 3. Good health, Gene Expression Regulation, Neoplastic, 03 medical and health sciences, Cell Line, Tumor, Serine, Tumor Microenvironment, Homeostasis, Humans, Glioblastoma, Reactive Oxygen Species, Oxidation-Reduction, Phosphoglycerate Dehydrogenase, Cell Proliferation
Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/32203214
https://www.nature.com/articles/s41416-020-0794-x
https://pubmed.ncbi.nlm.nih.gov/32203214/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7188854
https://www.nature.com/articles/s41416-020-0794-x.pdf
https://europepmc.org/articles/PMC7188854/figure/Fig2/
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7188854
https://repository.publisso.de/resource/frl:6471422 -
15
Autoren: et al.
Weitere Verfasser: et al.
Quelle: BMC Cancer
BMC Cancer, Vol 20, Iss 1, Pp 1-19 (2020)
BMC cancer
England
BMC Cancer 20:1039 (2020)Schlagwörter: Cetuximab, Apoptosis, Afatinib, Cell Movement, Stomach Neoplasms, Gastric Cancer, Gene Expression, Motility, Phosphoprotein, Trastuzumab, Antineoplastic Combined Chemotherapy Protocols, Research Article, Cell and molecular biology, Gastric cancer, Gene expression, Biomarkers, Tumor, Tumor Cells, Cultured, Humans, Cetuximab/administration, Phosphoproteins/metabolism [MeSH], Stomach Neoplasms/pathology [MeSH], Trastuzumab/administration, Afatinib/administration, Cell Movement [MeSH], Phosphoproteins/genetics [MeSH], Apoptosis [MeSH], Tumor Cells, Cultured [MeSH], Stomach Neoplasms/drug therapy [MeSH], Phenotype [MeSH], Stomach Neoplasms/genetics [MeSH], Cell Proliferation [MeSH], Humans [MeSH], Stomach Neoplasms/metabolism [MeSH], Gene Expression Regulation, Neoplastic/drug effects [MeSH], Antineoplastic Combined Chemotherapy Protocols/pharmacology [MeSH], Biomarkers, Tumor/metabolism [MeSH], Biomarkers, Tumor/genetics [MeSH], Gene Expression Profiling [MeSH], Cell Cycle [MeSH], RC254-282, Cell Proliferation, Gene Expression Profiling, Cell Cycle, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Phosphoproteins, ddc, 3. Good health, Gene Expression Regulation, Neoplastic, Phenotype
Dateibeschreibung: application/pdf
Zugangs-URL: https://bmccancer.biomedcentral.com/track/pdf/10.1186/s12885-020-07540-7
https://pubmed.ncbi.nlm.nih.gov/33115415
https://doaj.org/article/7f479c0cef6b4a9a9508b50dc6678e56
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7594334/
https://europepmc.org/article/MED/33115415
https://mediatum.ub.tum.de/1617354
https://link.springer.com/content/pdf/10.1186/s12885-020-07540-7.pdf
https://bmccancer.biomedcentral.com/articles/10.1186/s12885-020-07642-2
https://pubmed.ncbi.nlm.nih.gov/33115415/
http://hdl.handle.net/10033/622551
https://push-zb.helmholtz-muenchen.de/frontdoor.php?source_opus=60438
https://repository.publisso.de/resource/frl:6464617
https://mediatum.ub.tum.de/1597566
https://mediatum.ub.tum.de/doc/1704471/document.pdf
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