Suchergebnisse - "Cell Movement/drug effects"
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1
Autoren: et al.
Quelle: Arch Toxicol
Archives of toxicology, vol. 99, no. 6, pp. 2463-2477Schlagwörter: Immunotoxicology, Langerhans Cells/drug effects, Humans, Cell Movement/drug effects, Thiazoles/toxicity, Dermatitis, Allergic Contact/etiology, Dermatitis, Allergic Contact/pathology, Skin/drug effects, Skin/cytology, Image Processing, Computer-Assisted/methods, Machine Learning, Software, Allergic contact dermatitis, DAB-staining CD1a immunohistochemistry, In vitro model, QuPath software, Skin histology, Skin sensitization
Dateibeschreibung: application/pdf
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2
Autoren: et al.
Quelle: Mol Ther
Schlosser, A, Pilecki, B, Allen, C, Benest, A V, Lynch, A P, Hua, J, Ved, N, Blackley, Z, Andersen, T L, Hennig, D, Graversen, J H, Möller, S, Skallerup, S, Ormhøj, M, Lange, C, Agostini, H T, Grauslund, J, Heegaard, S, Dacheva, I, Koss, M, Hu, W, Iglesias, B, Lawrence, M S, Beck, H C, Steffensen, L B, Laursen, N S, Andersen, G R, Holmskov, U, Bates, D O & Sorensen, G L 2025, 'Pharmacological blocking of microfibrillar-associated protein 4 reduces retinal neoangiogenesis and vascular leakage', Molecular Therapy, vol. 33, no. 3, pp. 1048-1072. https://doi.org/10.1016/j.ymthe.2025.01.038Schlagwörter: glycoprotein, integrin, extracellular matrix, Cell Movement/drug effects, neovascular age-related macular degeneration, Endothelial Cells/metabolism, Retinal Neovascularization, vascular leakage, MFAP4, Extracellular Matrix Proteins/metabolism, Capillary Permeability, Mice, Cell Movement, microfibrillar-associated protein 4, DME, Animals, Humans, Capillary Permeability/drug effects, x-ray crystallography, Retinal Neovascularization/drug therapy, Extracellular Matrix Proteins, Diabetic Retinopathy, Animal, Choroidal Neovascularization/drug therapy, Endothelial Cells, Choroidal Neovascularization, Disease Models, Animal, therapeutic antibody, Disease Models, nAMD, Diabetic Retinopathy/drug therapy, Original Article, RNA Splicing Factors, diabetic macular edema
Dateibeschreibung: pdf
Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/39863929
https://portal.findresearcher.sdu.dk/da/publications/517051ca-ba94-45f6-b470-5d3567790eed
https://doi.org/10.1016/j.ymthe.2025.01.038
https://pure.au.dk/portal/en/publications/d7778dee-8a01-4818-a4f5-14ab89f98c1c
http://www.scopus.com/inward/record.url?scp=85217266244&partnerID=8YFLogxK
https://doi.org/10.1016/j.ymthe.2025.01.038 -
3
Autoren: et al.
Quelle: Proceedings of the National Academy of Sciences. 122
Schlagwörter: CXCR4, Tumor, Leukemia, Carcinogenesis, receptor oligomerization, leukemia, CXCR4/metabolism, Cell Movement/drug effects, drug sensitization, Molecular Processes and Therapies [Topic 2], Lymphocytic, Cell Line, SDG 3 - Good Health and Well-being, Cardiovascular and Metabolic Diseases, B-Cell/metabolism, Receptors, Humans, Chronic, Protein Multimerization, Signal Transduction
Dateibeschreibung: application/pdf
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4
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Mol Cancer
Molecular Cancer, Vol 24, Iss 1, Pp 1-26 (2025)Schlagwörter: Male, Cancer therapy, Cell- och molekylärbiologi, Expression, Cell Movement/drug effects, Cell Proliferation/drug effects, Mice, 106023 Molekularbiologie, Platform, Cell Movement, Extracellular acidification, RC254-282, Set Enrichment Analysis, Tumor, Metabolic Reprogramming, 302055 Onkologie, Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Men, 106023 Molecular biology, Prostatic Neoplasms/metabolism, Mitochondria, Type 2 diabetes mellitus (T2DM), Health, SDG 3 – Gesundheit und Wohlergehen, Metabolic rewiring, Pioglitazone/pharmacology, Efficacy, Oxygen consumption rate, Outcomes, Cell Line, Hypoglycemic Agents/pharmacology, SDG 3 - Good Health and Well-being, Cell Line, Tumor, Mitochondria/metabolism, Humans, Animals, Hypoglycemic Agents, 302055 Oncology, Mass-Spectrometry, PPAR gamma/agonists, Cell Proliferation, PPAR agonists, Pioglitazone, Research, Prostatic Neoplasms, Energy metabolism, Xenograft Model Antitumor Assays, PPAR gamma, Cell and Molecular Biology
Dateibeschreibung: application/pdf; application/zip; text/xml
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5
Autoren: et al.
Quelle: Pflugers Arch
Schlagwörter: 0301 basic medicine, 0303 health sciences, Signaling and Cell Physiology, Neutrophils, Tumor Necrosis Factor-alpha, NADPH Oxidases, Complement C5a, Complement C5a/metabolism [MeSH], Complement C5a/pharmacology [MeSH], Mice, Inbred C57BL [MeSH], Reactive Oxygen Species/metabolism [MeSH], Neutrophils/metabolism [MeSH], Membrane Potentials/physiology [MeSH], Neutrophils/physiology [MeSH], Actin Cytoskeleton/metabolism [MeSH], NADPH Oxidase 2/metabolism [MeSH], Neutrophils/drug effects [MeSH], Cell Movement/drug effects [MeSH], Animals [MeSH], ROS, Neutrophil Activation [MeSH], Mice [MeSH], Chemotaxis, Membrane potential dynamics, Tumor Necrosis Factor-alpha/metabolism [MeSH], NADPH Oxidases/metabolism [MeSH], Tumor Necrosis Factor-alpha/pharmacology [MeSH], Neutrophil Activation, Membrane Potentials, Mice, Inbred C57BL, Mice, Actin Cytoskeleton, 03 medical and health sciences, Cell Movement, NADPH Oxidase 2, Animals, Reactive Oxygen Species
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6
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Scientific Reports
Schlagwörter: Lactoferrin/pharmacology, Humans, Cell Cycle Checkpoints/drug effects, Carcinoma, Non-Small-Cell Lung/genetics, Cattle, Transcriptome/drug effects, Lung Neoplasms/genetics, Animals, Cell Line, Tumor, Apoptosis/drug effects, Gene Expression Regulation, Neoplastic/drug effects, A549 Cells, Cell Movement/drug effects, Gene Expression Profiling, Mitochondria/drug effects, Cell Proliferation/drug effects
Relation: https://eprints.qut.edu.au/259236/
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7
Autoren: et al.
Quelle: Garra, S, Mejlstrup Hymøller, C, Di Molfetta, D, Zagaria, N, Gena, P, Cardone, R A, Rützler, M, Birkelund, S & Calamita, G 2025, 'Selective Blockade of Two Aquaporin Channels, AQP3 and AQP9, Impairs Human Leukocyte Migration', Cells, vol. 14, no. 12, 880. https://doi.org/10.3390/cells14120880
Schlagwörter: Aquaporin 3/antagonists & inhibitors, Aquaporins/antagonists & inhibitors, Cell Movement/drug effects, Humans, Klebsiella pneumoniae/drug effects, Leukocytes, Mononuclear/metabolism, Leukocytes/metabolism, Lipopolysaccharides/pharmacology, Neutrophils/metabolism, Phagocytosis/drug effects, phagocytosis, LPS, cell motility, innate immunity, white blood cells, host–bacteria relationship, inflammation, aquaporin inhibitors
Dateibeschreibung: application/pdf
Relation: info:eu-repo/semantics/altIdentifier/pmid/40558507; info:eu-repo/semantics/altIdentifier/pissn/2073-4409
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8
Autoren: et al.
Quelle: González-Novo, R, Armesto, M, González-Murillo, Á, Dreger, M, Hurlstone, A F L, Benito, A, Samaniego, R, Ramírez, M & Redondo-Muñoz, J 2025, 'Dual effect of targeting LSD1 on the invasiveness and the mechanical response of acute lymphoblastic leukemia cells', Biomedicine & pharmacotherapy = Biomédecine & pharmacothérapie, vol. 183, 117830. https://doi.org/10.1016/j.biopha.2025.117830
Schlagwörter: Histone Demethylases/metabolism, Humans, Neoplasm Invasiveness, Precursor Cell Lymphoblastic Leukemia-Lymphoma/pathology, Animals, Cell Line, Tumor, Cell Movement/drug effects, Apoptosis/drug effects, Mice
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9
Autoren: et al.
Quelle: Sci Adv
Maldonado, H, Dreger, M, Bedgood, L D, Kyriakou, T, Wolanska, K I, Rigby, M E, Marotta, V E, Webster, J M, Wang, J, Rusilowicz-Jones, E V, Marshall, J F, Coulson, J M, Macpherson, I R, Hurlstone, A & Morgan, M R 2024, 'A trafficking regulatory subnetwork governs α Vβ 6 integrin-HER2 cross-talk to control breast cancer invasion and drug resistance.', Science Advances, vol. 10, no. 49, pp. eadk9944. https://doi.org/10.1126/sciadv.adk9944Schlagwörter: 0301 basic medicine, Integrins, Receptor, ErbB-2, Receptor, ErbB-2/metabolism, Drug Resistance, Cell Movement/drug effects, Breast Neoplasms, Integrins/metabolism, Trastuzumab/pharmacology, Cell Line, ACTIVATION, Mice, 03 medical and health sciences, RAB GTPASES, Cell Movement, Antigens, Neoplasm, Signal Transduction/drug effects, Cell Line, Tumor, Humans, Animals, rab GTP-Binding Proteins/metabolism, Neoplasm Invasiveness, Antigens, ERBB2, rab5 GTP-Binding Proteins, 0303 health sciences, Tumor, PROGNOSTIC INDICATOR, rab7 GTP-Binding Proteins, TGF-BETA, Trastuzumab, PANCREATIC-CANCER, TRASTUZUMAB RESISTANCE, ALPHA-V-BETA-6 INTEGRIN, Protein Transport, rab5 GTP-Binding Proteins/metabolism, Breast Neoplasms/metabolism, Drug Resistance, Neoplasm, rab GTP-Binding Proteins, GROWTH, Neoplasm, ErbB-2/metabolism, Female, SQUAMOUS-CELL CARCINOMA, Biomedicine and Life Sciences, rab7 GTP-Binding Proteins/metabolism, Receptor, Signal Transduction
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10
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Sci Rep
Scientific Reports, Vol 14, Iss 1, Pp 1-10 (2024)Schlagwörter: Science, Tartrazine/toxicity, Neovascularization, Physiologic, Cell Movement/drug effects, Human Umbilical Vein Endothelial Cells/drug effects, Endothelial Cells/metabolism, Article, Cell Proliferation/drug effects, Larva/drug effects, Food science, Animals, Genetically Modified, Blood vessels, Zebrafish, HUVEC, Cell Movement, Sciences des denrées alimentaires, Rho GTPases, Tartrazine/pharmacology, Human Umbilical Vein Endothelial Cells, Animals, Humans, Zebrafish, Tartrazine, Endothelial Cells/drug effects, Cell Proliferation, BMP pathways, Food Coloring Agents, Endothelial Cells, Neovascularization, Physiologic/drug effects, Life sciences, Larva, Food Coloring Agents/pharmacology, Sciences du vivant, Medicine, Zebrafish/embryology
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11
Autoren: et al.
Quelle: J Cell Mol Med
Liu, H, Li, H, Bai, X, Zhao, Y, Cai, Y, Pan, H, Guo, L, Liu, K, Liu, Q, Huang, X, Zampetaki, A, Margariti, A, Zeng, L & Cai, T 2024, 'Histone deacetylase 7-derived 7-amino acid peptide increases skin wound healing via regulating epidermal fibroblast proliferation and migration', Journal of Cellular and Molecular Medicine, vol. 28, no. 22, e70209. https://doi.org/10.1111/jcmm.70209Schlagwörter: Male, 0301 basic medicine, Cell Movement/drug effects, Histone Deacetylases, Cell Proliferation/drug effects, Rats, Sprague-Dawley, 03 medical and health sciences, Cell Movement, Signal Transduction/drug effects, Animals, Humans, Phosphorylation, Wound Healing/drug effects, Cell Proliferation, Skin, Fibroblasts/metabolism, Peptides/pharmacology, Wound Healing, 0303 health sciences, Phosphorylation/drug effects, Cyclin-Dependent Kinase 6, Fibroblasts, Cyclin-Dependent Kinase 6/metabolism, Rats, Skin/drug effects, Original Article, Sprague-Dawley, Histone Deacetylases/metabolism, Epidermis, Peptides, Epidermis/metabolism, Signal Transduction
Dateibeschreibung: application/pdf
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12
Autoren: et al.
Quelle: Arch Dermatol Res
Schlagwörter: Keratinocytes, 0301 basic medicine, ddc:610, Original Paper, Skin Neoplasms, Pyridines [MeSH], Cell Line, Tumor [MeSH], Beta-tubulin-polymerization, Antineoplastic Agents/pharmacology [MeSH], Keratinocytes/drug effects [MeSH], Skin Neoplasms/drug therapy [MeSH], Cell Movement/drug effects [MeSH], Morpholines [MeSH], Keratinocytes/metabolism [MeSH], Tirbanibulin, Keratosis, Actinic/drug therapy [MeSH], Signal Transduction/drug effects [MeSH], Skin Neoplasms/metabolism [MeSH], Carcinoma, Squamous Cell/pathology [MeSH], Keratosis, Actinic/pathology [MeSH], Polymerization/drug effects [MeSH], Actinic keratosis (AK), Humans [MeSH], Apoptosis/drug effects [MeSH], Tubulin/metabolism [MeSH], Skin Neoplasms/pathology [MeSH], Carcinoma, Squamous Cell/drug therapy [MeSH], Cutaneous squamous cell carcinoma (cSCC), Acetamides [MeSH], Keratosis, Actinic/metabolism [MeSH], Cell Proliferation/drug effects [MeSH], Carcinoma, Squamous Cell/metabolism [MeSH], Pyridines, Morpholines, Apoptosis, Antineoplastic Agents, Polymerization, Keratosis, Actinic, 03 medical and health sciences, 0302 clinical medicine, Tubulin, Cell Movement, Cell Line, Tumor, Acetamides, Carcinoma, Squamous Cell, Humans, Cell Proliferation, Signal Transduction
Dateibeschreibung: application/pdf
Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/38847867
https://nbn-resolving.org/urn:nbn:de:hbz:294-114483
https://hss-opus.ub.ruhr-uni-bochum.de/opus4/frontdoor/index/index/docId/11448
https://hss-opus.ub.ruhr-uni-bochum.de/opus4/files/11448/DeTempleViola07062024.pdf
https://repository.publisso.de/resource/frl:6500757 -
13
Autoren: et al.
Quelle: Pharm Res
Marwah, M K, Shokr, H, Sanchez-Aranguren, L, Badhan, R K S, Wang, K & Ahmad, S 2022, ' Transdermal Delivery of a Hydrogen Sulphide Donor, ADT-OH Using Aqueous Gel Formulations for the Treatment of Impaired Vascular Function : an Ex Vivo Study ', Pharmaceutical Research, vol. 39, no. 2, pp. 341-352 . https://doi.org/10.1007/s11095-021-03164-zSchlagwörter: Physiologic/drug effects, 0301 basic medicine, Cells, Drug Compounding, Skin Absorption, Neovascularization, Physiologic, Cell Movement/drug effects, Human Umbilical Vein Endothelial Cells/drug effects, Thiones/administration & dosage, Administration, Cutaneous, Energy Metabolism/drug effects, Oxygen Consumption/drug effects, Mice, 03 medical and health sciences, Oxygen Consumption, Cell Movement, Human Umbilical Vein Endothelial Cells, Animals, Humans, Hydrogen Sulfide, Neovascularization, Cells, Cultured, 0303 health sciences, Cultured, Thiones, Neovascularization, Physiologic/drug effects, Mitochondria/drug effects, Mitochondria, Cutaneous, Administration, Hydrogen Sulfide/administration & dosage, Female, Energy Metabolism, Gels, Research Paper
Dateibeschreibung: application/pdf
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14
Autoren: et al.
Weitere Verfasser: et al.
Quelle: Biochemical and Biophysical Research Communications. 581:74-80
Schlagwörter: 0301 basic medicine, Osteoblasts / pathology, Culture, Proliferation, Osteoblasts / drug effects, Cell Nucleus / drug effects, MAP Kinase Signaling System / drug effects, Invasion, Cell Movement, beta Catenin / metabolism, Bone Marrow Cells / drug effects, Phosphorylation, Wnt Signaling Pathway, beta Catenin, Bone Marrow Cells / cytology, Mitogen-Activated Protein Kinase 1, Cell Movement / genetics, Diffusion Chambers, Osteosarcoma, 0303 health sciences, Tumor, Mitogen-Activated Protein Kinase 3, Protein Transport / drug effects, Cell Movement / drug effects, Osteoblasts / metabolism, Mitogen-Activated Protein Kinase 3 / genetics, 3. Good health, Gene Expression Regulation, Neoplastic, Protein Transport, Mitogen-Activated Protein Kinase 1 / genetics, Diffusion Chambers, Culture, Cell Proliferation / genetics, beta Catenin / genetics, Mitogen-Activated Protein Kinase 1 / antagonists & inhibitors, MAP Kinase Signaling System, Primary Cell Culture, Epidermal Growth Factor / pharmacology, Bone Marrow Cells, Cell Line, 03 medical and health sciences, Cell Line, Tumor, Glycogen Synthase Kinase 3 beta / genetics, Humans, Cell Proliferation, Cell Nucleus, Neoplastic, Phosphorylation / drug effects, Glycogen Synthase Kinase 3 beta, Osteoblasts, Mitogen-Activated Protein Kinase 3 / antagonists & inhibitors, Epidermal Growth Factor, Cell Nucleus / metabolism, Epidermal growth factor, Bone Marrow Cells / metabolism, Glycogen Synthase Kinase 3 beta / metabolism, Lithium Chloride / pharmacology, Lithium chloride, Gene Expression Regulation, Mitogen-Activated Protein Kinase 1 / metabolism, Wnt Signaling Pathway / drug effects, Cell Proliferation / drug effects, Lithium Chloride, Mitogen-Activated Protein Kinase 3 / metabolism
Zugangs-URL: https://pubmed.ncbi.nlm.nih.gov/34656851
https://pubmed.ncbi.nlm.nih.gov/34656851/
https://www.sciencedirect.com/science/article/pii/S0006291X21014327
https://www.ncbi.nlm.nih.gov/pubmed/34656851
https://yonsei.pure.elsevier.com/en/publications/lithium-chloride-inhibits-the-migration-and-invasion-of-osteosarc -
15
Autoren: et al.
Quelle: Neurotox Res
Schlagwörter: Neurons, 0301 basic medicine, Insecticides, 0303 health sciences, Dose-Response Relationship, Drug, Neuronal Outgrowth, Cell Differentiation, Cell Line, 3. Good health, 03 medical and health sciences, Dose-Response Relationship, Drug [MeSH], Cell Differentiation/physiology [MeSH], Cell Movement/physiology [MeSH], Humans [MeSH], Neurons/metabolism [MeSH], Cell Differentiation/drug effects [MeSH], Cell Line [MeSH], Neuronal Outgrowth/drug effects [MeSH], Cell Movement/drug effects [MeSH], NT2, Neurite outgrowth, Pyrazoles/toxicity [MeSH], Neurons/drug effects [MeSH], Rotenone/toxicity [MeSH], Neurotoxicity Syndromes/pathology [MeSH], Original Article, Insecticides/toxicity [MeSH], Migration, Neuronal Outgrowth/physiology [MeSH], NTera-2, Neurons/pathology [MeSH], Neurotoxicity Syndromes/metabolism [MeSH], DNT, Differentiation, Cell Movement, Rotenone, Humans, Pyrazoles, Neurotoxicity Syndromes
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s12640-021-00364-8.pdf
https://pubmed.ncbi.nlm.nih.gov/33871813
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8275550
https://link.springer.com/article/10.1007/s12640-021-00364-8
https://europepmc.org/article/MED/33871813
https://link.springer.com/content/pdf/10.1007/s12640-021-00364-8.pdf
https://www.ncbi.nlm.nih.gov/pubmed/33871813
https://pubmed.ncbi.nlm.nih.gov/33871813/
https://repository.publisso.de/resource/frl:6444214 -
16
Autoren: et al.
Quelle: Arch Toxicol
Schlagwörter: 0301 basic medicine, Alkylating Agents, Wound Healing, Apoptosis, Mesenchymal Stem Cells, Hydrogen Peroxide, Biologics, 3. Good health, 03 medical and health sciences, Cell Movement, 13. Climate action, Chemokines/metabolism [MeSH], Chemical Warfare Agents/toxicity [MeSH], Senescence, Skin/injuries [MeSH], Skin/drug effects [MeSH], Wound Healing/drug effects [MeSH], Cell Movement/drug effects [MeSH], Cytokines/metabolism [MeSH], Alkylating Agents/toxicity [MeSH], Mesenchymal Stem Cells/drug effects [MeSH], Sulfur mustard, Chemical warfare agents, Mesenchymal Stem Cells/cytology [MeSH], Hydrogen Peroxide/toxicity [MeSH], Mustard Gas/toxicity [MeSH], Cellular Senescence/drug effects [MeSH], Wound healing disorder, Humans [MeSH], Apoptosis/drug effects [MeSH], Mesenchymal Stem Cells/metabolism [MeSH], Biomarkers/metabolism [MeSH], Cells, Cultured [MeSH], Cytokines/genetics [MeSH], Cell Proliferation/drug effects [MeSH], Mesenchymal stem cells, Chemokines/genetics [MeSH], Mustard Gas, Cytokines, Humans, Chemical Warfare Agents, Chemokines, Biomarkers, Cells, Cultured, Cellular Senescence, Cell Proliferation, Skin
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00204-020-02946-5.pdf
https://pubmed.ncbi.nlm.nih.gov/33491125
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7870771
https://kops.uni-konstanz.de/handle/123456789/52592
https://europepmc.org/article/MED/33491125
https://link.springer.com/article/10.1007/s00204-020-02946-5?error=cookies_not_supported&error=cookies_not_supported&error=cookies_not_supported&error=cookies_not_supported&code=4acdcd1c-07dc-4688-98dc-7a7d15fa8a7f&code=df09e271-8065-4d4b-b05c-34949581065b&code=47eae568-eea7-4b42-a913-584eb4f9b0ad&code=9344f33e-3871-43aa-b40f-642603f8f690
https://www.scilit.net/article/516e8cd09414d7681f2a6136e2e99cc2
https://link.springer.com/content/pdf/10.1007/s00204-020-02946-5.pdf
https://repository.publisso.de/resource/frl:6451719 -
17
Autoren: et al.
Quelle: J Cancer Res Clin Oncol
Schlagwörter: Indoles, Organoplatinum Compounds, Carcinoma, Ovarian Epithelial, Maleimides, 03 medical and health sciences, 0302 clinical medicine, Antigens, CD, Cell Movement, Cell Line, Tumor, Antineoplastic Combined Chemotherapy Protocols, Humans, Wnt Signaling Pathway, Neoplasm Staging, Ovarian Neoplasms, ddc:610, Middle Aged, Phenanthrenes, Cadherins, 3. Good health, Drug Resistance, Neoplasm, Epoxy Compounds, Cell Line, Tumor [MeSH], Carcinoma, Ovarian Epithelial/drug therapy [MeSH], Ovarian Neoplasms/drug therapy [MeSH], Cell Movement/drug effects [MeSH], Snail/ slug, Antigens, CD/biosynthesis [MeSH], Ovarian Neoplasms/metabolism [MeSH], Neoplasm Staging [MeSH], Heterocyclic Compounds, 3-Ring/pharmacology [MeSH], Indoles/pharmacology [MeSH], β-catenin, Phenanthrenes/pharmacology [MeSH], Cadherins/metabolism [MeSH], Platinum-resistance, Drug Resistance, Neoplasm [MeSH], Ovarian Neoplasms/pathology [MeSH], Female [MeSH], beta Catenin/metabolism [MeSH], E-cadherin, Original Article – Cancer Research, Maleimides/pharmacology [MeSH], Ovarian cancer, Humans [MeSH], Diterpenes/pharmacology [MeSH], Epoxy Compounds/pharmacology [MeSH], Middle Aged [MeSH], Organoplatinum Compounds/administration, Carcinoma, Ovarian Epithelial/pathology [MeSH], beta Catenin/biosynthesis [MeSH], Carcinoma, Ovarian Epithelial/metabolism [MeSH], Antineoplastic Combined Chemotherapy Protocols/pharmacology [MeSH], Cadherins/biosynthesis [MeSH], Wnt Signaling Pathway/drug effects [MeSH], Organoplatinum Compounds/pharmacology [MeSH], Wnt, Antigens, CD/metabolism [MeSH], Female, Diterpenes, Heterocyclic Compounds, 3-Ring
Dateibeschreibung: application/pdf
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00432-020-03317-4.pdf
https://pubmed.ncbi.nlm.nih.gov/32681294
https://pubmed.ncbi.nlm.nih.gov/32681294/
https://www.ncbi.nlm.nih.gov/pubmed/32681294
https://link.springer.com/content/pdf/10.1007/s00432-020-03317-4.pdf
https://epub.ub.uni-muenchen.de/73190/
http://europepmc.org/article/MED/32681294
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7467966
https://opus.bibliothek.uni-augsburg.de/opus4/files/80467/Kaltofen2020_Article_PotentialOfPlatinum-resensitiz.pdf
https://opus.bibliothek.uni-augsburg.de/opus4/frontdoor/index/index/docId/80467
https://nbn-resolving.org/urn:nbn:de:bvb:384-opus4-804675
https://doi.org/10.1007/s00432-020-03317-4
https://repository.publisso.de/resource/frl:6469356
https://epub.ub.uni-muenchen.de/73190/ -
18
Autoren: et al.
Quelle: J Cancer Res Clin Oncol
Schlagwörter: ddc:610, Computational Biology, Uterine Cervical Neoplasms, Female [MeSH], Signal Transduction/genetics [MeSH], Cell Line, Tumor [MeSH], Cell Movement/genetics [MeSH], Original Article – Cancer Research, Humans [MeSH], Plasminogen activator inhibitor type 1 (PAI-1), Receptors, Prostaglandin E, EP3 Subtype/genetics [MeSH], Survival Analysis [MeSH], Uterine Cervical Neoplasms/drug therapy [MeSH], Cell Movement/drug effects [MeSH], Dinoprostone/analogs, Uterine Cervical Neoplasms/pathology [MeSH], Urokinase-type plasminogen activator receptor (uPAR), Prostaglandin E, Uterine Cervical Neoplasms/genetics [MeSH], Prognosis [MeSH], Dinoprostone/pharmacology [MeSH], Signal Transduction/drug effects [MeSH], Cervical cancer, Dinoprostone/genetics [MeSH], Receptors, Urokinase Plasminogen Activator/genetics [MeSH], HeLa Cells [MeSH], Computational Biology/methods [MeSH], Prognosis, Survival Analysis, Dinoprostone, Receptors, Urokinase Plasminogen Activator, 3. Good health, 03 medical and health sciences, 0302 clinical medicine, Cell Movement, Cell Line, Tumor, Receptors, Prostaglandin E, EP3 Subtype, Humans, Female, HeLa Cells, Signal Transduction
Dateibeschreibung: application/pdf
Zugangs-URL: https://link.springer.com/content/pdf/10.1007/s00432-020-03272-0.pdf
https://pubmed.ncbi.nlm.nih.gov/32488496
https://epub.ub.uni-muenchen.de/73161/
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7382663
https://link.springer.com/content/pdf/10.1007/s00432-020-03272-0.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7382663
https://www.ncbi.nlm.nih.gov/pubmed/32488496
https://link.springer.com/article/10.1007/s00432-020-03272-0
https://opus.bibliothek.uni-augsburg.de/opus4/frontdoor/index/index/docId/77300
https://nbn-resolving.org/urn:nbn:de:bvb:384-opus4-773005
https://opus.bibliothek.uni-augsburg.de/opus4/files/77300/Ye2020_Article_ProstaglandinE2Receptor3EP3Sig.pdf
https://doi.org/10.1007/s00432-020-03272-0
https://repository.publisso.de/resource/frl:6469362
https://epub.ub.uni-muenchen.de/73161/ -
19
Autoren: et al.
Quelle: Proc Natl Acad Sci U S A
Kitchen, G B, Cunningham, P S, Poolman, T M, Iqbal, M, Maidstone, R, Baxter, M, Bagnall, J, Begley, N, Saer, B, Hussell, T, Matthews, L C, Dockrell, D H, Durrington, H J, Gibbs, J E, Blaikley, J F, Loudon, A S & Ray, D W 2020, ' The clock gene Bmal1 inhibits macrophage motility, phagocytosis, and impairs defense against pneumonia ', Proceedings of the National Academy of Sciences, vol. 117, no. 3, pp. 1543-1551 . https://doi.org/10.1073/pnas.1915932117
Kitchen, G B, Cunningham, P S, Poolman, T M, Iqbal, M, Maidstone, R, Baxter, M, Bagnall, J, Begley, N, Saer, B, Hussell, T, Matthews, L C, Dockrell, D H, Durrington, H J, Gibbs, J E, Blaikley, J F, Loudon, A S & Ray, D W 2020, 'The clock gene Bmal1 inhibits macrophage motility, phagocytosis, and impairs defense against pneumonia', Proceedings of the National Academy of Sciences, vol. 117, no. 3, pp. 1543-1551. https://doi.org/10.1073/pnas.1915932117Schlagwörter: 0301 basic medicine, actin cytoskeleton, Disease Resistance/genetics, Circadian Clocks/genetics, Lydia Becker Institute, Knockout, Phagocytosis/drug effects, Pneumococcal/metabolism, Cell Movement/drug effects, Inbred C57BL, sactin cytoskeleton, Mice, 03 medical and health sciences, Macrophages/drug effects, Phagocytosis, Cell Movement, Circadian Clocks, rhoA GTP-Binding Protein/metabolism, Animals, Cytoskeleton, ARNTL Transcription Factors/antagonists & inhibitors, Disease Resistance, Mice, Knockout, 0303 health sciences, Animal, Macrophages, Actin cytoskeleton, Circadian, phagocytosis, ARNTL Transcription Factors, RhoA, Pneumonia, Biological Sciences, Pneumonia, Pneumococcal, Pneumonia, Pneumococcal/metabolism, Streptococcus pneumoniae/pathogenicity, Actins, 3. Good health, Mice, Inbred C57BL, Disease Models, Animal, circadian, Streptococcus pneumoniae, Actins/metabolism, Disease Models, Female, rhoA GTP-Binding Protein, ResearchInstitutes_Networks_Beacons/lydia_becker_institute_of_immunology_and_inflammation, name=Lydia Becker Institute
Dateibeschreibung: application/pdf
Zugangs-URL: https://www.pnas.org/content/pnas/117/3/1543.full.pdf
https://pubmed.ncbi.nlm.nih.gov/31900362
https://eprints.whiterose.ac.uk/155198/7/1543.full.pdf
https://research.manchester.ac.uk/en/publications/3844729e-b110-4fa5-ac2f-72feafdc7875
https://doi.org/10.1073/pnas.1915932117
https://www.pnas.org/content/pnas/117/3/1543.full.pdf
https://www.pnas.org/content/early/2020/01/02/1915932117.abstract
https://www.research.manchester.ac.uk/portal/en/publications/the-clock-gene-bmal1-inhibits-macrophage-motility-phagocytosis-and-impairs-defense-against-pneumonia(3844729e-b110-4fa5-ac2f-72feafdc7875).html
https://pubmed.ncbi.nlm.nih.gov/31900362/
https://europepmc.org/article/MED/31900362
https://www.rdm.ox.ac.uk/publications/1081313
https://ora.ox.ac.uk/objects/uuid:3f25675b-293f-48ce-842b-65f9d38f4b9c
https://doi.org/10.1073/pnas.1915932117
https://www.pure.ed.ac.uk/ws/files/137618653/1543.full.pdf
https://hdl.handle.net/20.500.11820/cf82e31a-b1cd-4eae-82f0-a09ca038791f
https://discovery-pp.ucl.ac.uk/id/eprint/10103180/ -
20
Autoren: et al.
Schlagwörter: Antineoplastic Agents: chemical synthesis, chemistry, pharmacology, Apoptosis: drug effects, Cell Line, Tumor, Cell Movement: drug effects, Cell Proliferation: drug effects, Chemistry Techniques, Synthetic, Dose-Response Relationship, Drug, Drug Design, Drug Screening Assays, Antitumor, Drug Synergism, G1 Phase Cell Cycle Checkpoints: drug effects, Humans, Isoflavones: chemical synthesis, chemistry, pharmacology, MAP Kinase Signaling System: drug effects, Structure-Activity Relationship, Thiocarbamates: chemistry, Wnt Signaling Pathway: drug effects
Dateibeschreibung: application/pdf
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